All Relations between hypertrophic and matrix compartment

Publication Sentence Publish Date Extraction Date Species
G J Breur, M D Lapierre, K Kazmierczak, K M Stechuchak, G P McCab. The domain of hypertrophic chondrocytes in growth plates growing at different rates. Calcified tissue international. vol 61. issue 5. 1998-01-21. PMID:9351885. the data suggest that the domain volume of juxtametaphyseal hypertrophic chondrocytes, as well as the matrix volume produced per cell, may be tightly regulated. 1998-01-21 2023-08-12 rat
N Fujitsuka, A Kurogi, T Hattori, S Shind. [Effects of onpi-to (TJ-8117) and (-)epicatechin-3-O-gallate on the proliferating changes in glomeruli of 5/6 nephrectomized rats]. Nihon Jinzo Gakkai shi. vol 39. issue 7. 1998-01-20. PMID:9396236. in the present study, the effects of tj-8117 and (-)epicatechin-3-o-gallate (ecg), which is a component of rhei rhizoma, on glomerular cell proliferation, extracellular matrix accumulation and glomerular hypertrophy were investigated in 5/6 nephrectomized rats. 1998-01-20 2023-08-12 rat
W S Colucc. Molecular and cellular mechanisms of myocardial failure. The American journal of cardiology. vol 80. issue 11A. 1998-01-20. PMID:9412539. these alterations include hypertrophy and cellular apoptosis of myocytes, changes in the molecular phenotype of the myocardium with reinduction of a fetal gene program, and alterations in the quantity and composition of the extracellular matrix. 1998-01-20 2023-08-12 Not clear
H Yu, A M Gallagher, P M Garfin, M P Print. Prostacyclin release by rat cardiac fibroblasts: inhibition of collagen expression. Hypertension (Dallas, Tex. : 1979). vol 30. issue 5. 1997-12-10. PMID:9369254. cardiac fibroblasts, as the source of extracellular matrix for the left ventricle, subserve important functions to cardiac remodeling and fibrotic development following myocardial infarction or with pressure-overload cardiac hypertrophy. 1997-12-10 2023-08-12 rat
C Shukunami, K Ishizeki, T Atsumi, Y Ohta, F Suzuki, Y Hirak. Cellular hypertrophy and calcification of embryonal carcinoma-derived chondrogenic cell line ATDC5 in vitro. Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research. vol 12. issue 8. 1997-11-20. PMID:9258747. during the process of endochondral bone formation, proliferating chondrocytes give rise to hypertrophic cells, which then deposit a mineralized matrix to form calcified cartilage prior to replacement by bone. 1997-11-20 2023-08-12 Not clear
H Ono, Y On. Nephrosclerosis and hypertension. The Medical clinics of North America. vol 81. issue 6. 1997-11-19. PMID:9356598. in the authors' experience, the kidney of an elderly subject, although with normotensive pressure and normal level of cholesterol, shows an increased mesangial matrix and hypertrophic vascular medial smooth muscle cells. 1997-11-19 2023-08-12 human
G Wolf, F N Ziyade. Renal tubular hypertrophy induced by angiotensin II. Seminars in nephrology. vol 17. issue 5. 1997-11-18. PMID:9316213. angiotensin ii (ang ii) is a prime agent that has been linked to the progression of renal disease by a host of mechanisms, including the induction of tubular epithelial hypertrophy and stimulation of extracellular matrix biosynthesis. 1997-11-18 2023-08-12 Not clear
N Makino, M Sugano, S Otsuka, T Hat. Molecular mechanism of angiotensin II type I and type II receptors in cardiac hypertrophy of spontaneously hypertensive rats. Hypertension (Dallas, Tex. : 1979). vol 30. issue 4. 1997-11-13. PMID:9336375. these results indicate that at1 receptor and ace, but not at2 receptor, play a crucial role in the remodeling of matrix tissue but a smaller role in the development of the hypertrophy of lv myocyte in shr and that the lv/body weight changes do not fully account for the complete suppression of hypertension. 1997-11-13 2023-08-12 rat
K Wong, K R Boheler, M Petrou, M H Yacou. Pharmacological modulation of pressure-overload cardiac hypertrophy: changes in ventricular function, extracellular matrix, and gene expression. Circulation. vol 96. issue 7. 1997-10-31. PMID:9337196. pharmacological modulation of pressure-overload cardiac hypertrophy: changes in ventricular function, extracellular matrix, and gene expression. 1997-10-31 2023-08-12 rat
F H Wezeman, M R Bollno. Immunohistochemical localization of fibroblast growth factor-2 in normal and brachymorphic mouse tibial growth plate and articular cartilage. The Histochemical journal. vol 29. issue 6. 1997-10-08. PMID:9248858. immunostaining was present only in the territorial extracellular matrix immediately adjacent to the chondrocytes of the proliferating and upper hypertrophic zones. 1997-10-08 2023-08-12 mouse
F H Wezeman, M R Bollno. Immunohistochemical localization of fibroblast growth factor-2 in normal and brachymorphic mouse tibial growth plate and articular cartilage. The Histochemical journal. vol 29. issue 6. 1997-10-08. PMID:9248858. in contrast, the reduced size of bm/bm growth plates was accompanied by significantly reduced staining intensity in proliferating and upper hypertrophic chondrocytes, and staining was absent from the territorial extracellular matrix of all zones of the bm/bm growth plate. 1997-10-08 2023-08-12 mouse
J C Woodard, G A Donovan, L W Fishe. Pathogenesis of vitamin (A and D)-induced premature growth-plate closure in calves. Bone. vol 21. issue 2. 1997-10-07. PMID:9267693. columnar-zone cartilage cells appeared immature where the matrix became mineralized and lacked the morphology of hypertrophic chondrocytes. 1997-10-07 2023-08-12 Not clear
J C Woodard, G A Donovan, L W Fishe. Pathogenesis of vitamin (A and D)-induced premature growth-plate closure in calves. Bone. vol 21. issue 2. 1997-10-07. PMID:9267693. biglycan, type i collagen propeptide, type i collagen, osteopontin, osteocalcin, osteonectin, bone sialoprotein, and alkaline phosphatase were localized within the cytoplasm or surrounding matrix of hypertrophic chondrocytes. 1997-10-07 2023-08-12 Not clear
J C Woodard, G A Donovan, L W Fishe. Pathogenesis of vitamin (A and D)-induced premature growth-plate closure in calves. Bone. vol 21. issue 2. 1997-10-07. PMID:9267693. in vitamin-treated calves, these same proteins were found in regions undergoing premature matrix mineralization even though the chondrocytes did not have a hypertrophic morphology. 1997-10-07 2023-08-12 Not clear
J C Woodard, G A Donovan, L W Fishe. Pathogenesis of vitamin (A and D)-induced premature growth-plate closure in calves. Bone. vol 21. issue 2. 1997-10-07. PMID:9267693. therefore, vitamin treatment did not cause just a selective expression, but it caused expression of a large number of matrix proteins normally associated with the hypertrophic chondrocyte phenotype. 1997-10-07 2023-08-12 Not clear
S Bord, A Horner, R M Hembry, J J Reynolds, J E Compsto. Distribution of matrix metalloproteinases and their inhibitor, TIMP-1, in developing human osteophytic bone. Journal of anatomy. vol 191 ( Pt 1). 1997-09-18. PMID:9279657. chondrocytes demonstrated variable levels of collagenase and stromelysin expression throughout the proliferative and hypertrophic regions, stromelysin showing both cell-associated and strong matrix staining. 1997-09-18 2023-08-12 human
S Shibata, S Suzuki, Y Yamashit. An ultrastructural study of cartilage resorption at the site of initial endochondral bone formation in the fetal mouse mandibular condyle. Journal of anatomy. vol 191 ( Pt 1). 1997-09-18. PMID:9279660. after resorbing the bone collar, the osteoclasts extended their cell processes into the cartilage matrix and made contact with hypertrophic chondrocytes. 1997-09-18 2023-08-12 mouse
A G Kleber, V Fas. Molecular and cellular aspects of re-entrant arrhythmias. Basic research in cardiology. vol 92 Suppl 1. 1997-08-27. PMID:9202851. in recent years it has become evident that myocardial tissue undergoes remodeling in diseased states such as myocardial infarction and hypertrophy which affects membrane channels, cell-to-cell coupling as well as the connective tissue matrix. 1997-08-27 2023-08-12 Not clear
P Ren, G N Rowland, J Halpe. Expression of growth factors in chicken growth plate with special reference to tibial dyschondroplasia. Journal of comparative pathology. vol 116. issue 3. 1997-08-14. PMID:9147248. immunolabelling for tgf beta then gradually shifted into cartilaginous matrix and was not observed in cytoplasm of hypertrophic chondrocytes until the late embryonic and post-hatch stages. 1997-08-14 2023-08-12 chicken
M Sutmuller, J A Bruijn, E de Hee. Collagen types VIII and X, two non-fibrillar, short-chain collagens. Structure homologies, functions and involvement in pathology. Histology and histopathology. vol 12. issue 2. 1997-07-17. PMID:9151143. since collagen type x is produced by hypertrophic chondrocytes, this collagen type can only be found in matrix of the hypertrophic zone of the epiphyseal growth plate cartilage. 1997-07-17 2023-08-12 Not clear