| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| G J Breur, M D Lapierre, K Kazmierczak, K M Stechuchak, G P McCab. The domain of hypertrophic chondrocytes in growth plates growing at different rates. Calcified tissue international. vol 61. issue 5. 1998-01-21. PMID:9351885. |
the data suggest that the domain volume of juxtametaphyseal hypertrophic chondrocytes, as well as the matrix volume produced per cell, may be tightly regulated. |
1998-01-21 |
2023-08-12 |
rat |
| N Fujitsuka, A Kurogi, T Hattori, S Shind. [Effects of onpi-to (TJ-8117) and (-)epicatechin-3-O-gallate on the proliferating changes in glomeruli of 5/6 nephrectomized rats]. Nihon Jinzo Gakkai shi. vol 39. issue 7. 1998-01-20. PMID:9396236. |
in the present study, the effects of tj-8117 and (-)epicatechin-3-o-gallate (ecg), which is a component of rhei rhizoma, on glomerular cell proliferation, extracellular matrix accumulation and glomerular hypertrophy were investigated in 5/6 nephrectomized rats. |
1998-01-20 |
2023-08-12 |
rat |
| W S Colucc. Molecular and cellular mechanisms of myocardial failure. The American journal of cardiology. vol 80. issue 11A. 1998-01-20. PMID:9412539. |
these alterations include hypertrophy and cellular apoptosis of myocytes, changes in the molecular phenotype of the myocardium with reinduction of a fetal gene program, and alterations in the quantity and composition of the extracellular matrix. |
1998-01-20 |
2023-08-12 |
Not clear |
| H Yu, A M Gallagher, P M Garfin, M P Print. Prostacyclin release by rat cardiac fibroblasts: inhibition of collagen expression. Hypertension (Dallas, Tex. : 1979). vol 30. issue 5. 1997-12-10. PMID:9369254. |
cardiac fibroblasts, as the source of extracellular matrix for the left ventricle, subserve important functions to cardiac remodeling and fibrotic development following myocardial infarction or with pressure-overload cardiac hypertrophy. |
1997-12-10 |
2023-08-12 |
rat |
| C Shukunami, K Ishizeki, T Atsumi, Y Ohta, F Suzuki, Y Hirak. Cellular hypertrophy and calcification of embryonal carcinoma-derived chondrogenic cell line ATDC5 in vitro. Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research. vol 12. issue 8. 1997-11-20. PMID:9258747. |
during the process of endochondral bone formation, proliferating chondrocytes give rise to hypertrophic cells, which then deposit a mineralized matrix to form calcified cartilage prior to replacement by bone. |
1997-11-20 |
2023-08-12 |
Not clear |
| H Ono, Y On. Nephrosclerosis and hypertension. The Medical clinics of North America. vol 81. issue 6. 1997-11-19. PMID:9356598. |
in the authors' experience, the kidney of an elderly subject, although with normotensive pressure and normal level of cholesterol, shows an increased mesangial matrix and hypertrophic vascular medial smooth muscle cells. |
1997-11-19 |
2023-08-12 |
human |
| G Wolf, F N Ziyade. Renal tubular hypertrophy induced by angiotensin II. Seminars in nephrology. vol 17. issue 5. 1997-11-18. PMID:9316213. |
angiotensin ii (ang ii) is a prime agent that has been linked to the progression of renal disease by a host of mechanisms, including the induction of tubular epithelial hypertrophy and stimulation of extracellular matrix biosynthesis. |
1997-11-18 |
2023-08-12 |
Not clear |
| N Makino, M Sugano, S Otsuka, T Hat. Molecular mechanism of angiotensin II type I and type II receptors in cardiac hypertrophy of spontaneously hypertensive rats. Hypertension (Dallas, Tex. : 1979). vol 30. issue 4. 1997-11-13. PMID:9336375. |
these results indicate that at1 receptor and ace, but not at2 receptor, play a crucial role in the remodeling of matrix tissue but a smaller role in the development of the hypertrophy of lv myocyte in shr and that the lv/body weight changes do not fully account for the complete suppression of hypertension. |
1997-11-13 |
2023-08-12 |
rat |
| K Wong, K R Boheler, M Petrou, M H Yacou. Pharmacological modulation of pressure-overload cardiac hypertrophy: changes in ventricular function, extracellular matrix, and gene expression. Circulation. vol 96. issue 7. 1997-10-31. PMID:9337196. |
pharmacological modulation of pressure-overload cardiac hypertrophy: changes in ventricular function, extracellular matrix, and gene expression. |
1997-10-31 |
2023-08-12 |
rat |
| F H Wezeman, M R Bollno. Immunohistochemical localization of fibroblast growth factor-2 in normal and brachymorphic mouse tibial growth plate and articular cartilage. The Histochemical journal. vol 29. issue 6. 1997-10-08. PMID:9248858. |
immunostaining was present only in the territorial extracellular matrix immediately adjacent to the chondrocytes of the proliferating and upper hypertrophic zones. |
1997-10-08 |
2023-08-12 |
mouse |
| F H Wezeman, M R Bollno. Immunohistochemical localization of fibroblast growth factor-2 in normal and brachymorphic mouse tibial growth plate and articular cartilage. The Histochemical journal. vol 29. issue 6. 1997-10-08. PMID:9248858. |
in contrast, the reduced size of bm/bm growth plates was accompanied by significantly reduced staining intensity in proliferating and upper hypertrophic chondrocytes, and staining was absent from the territorial extracellular matrix of all zones of the bm/bm growth plate. |
1997-10-08 |
2023-08-12 |
mouse |
| J C Woodard, G A Donovan, L W Fishe. Pathogenesis of vitamin (A and D)-induced premature growth-plate closure in calves. Bone. vol 21. issue 2. 1997-10-07. PMID:9267693. |
columnar-zone cartilage cells appeared immature where the matrix became mineralized and lacked the morphology of hypertrophic chondrocytes. |
1997-10-07 |
2023-08-12 |
Not clear |
| J C Woodard, G A Donovan, L W Fishe. Pathogenesis of vitamin (A and D)-induced premature growth-plate closure in calves. Bone. vol 21. issue 2. 1997-10-07. PMID:9267693. |
biglycan, type i collagen propeptide, type i collagen, osteopontin, osteocalcin, osteonectin, bone sialoprotein, and alkaline phosphatase were localized within the cytoplasm or surrounding matrix of hypertrophic chondrocytes. |
1997-10-07 |
2023-08-12 |
Not clear |
| J C Woodard, G A Donovan, L W Fishe. Pathogenesis of vitamin (A and D)-induced premature growth-plate closure in calves. Bone. vol 21. issue 2. 1997-10-07. PMID:9267693. |
in vitamin-treated calves, these same proteins were found in regions undergoing premature matrix mineralization even though the chondrocytes did not have a hypertrophic morphology. |
1997-10-07 |
2023-08-12 |
Not clear |
| J C Woodard, G A Donovan, L W Fishe. Pathogenesis of vitamin (A and D)-induced premature growth-plate closure in calves. Bone. vol 21. issue 2. 1997-10-07. PMID:9267693. |
therefore, vitamin treatment did not cause just a selective expression, but it caused expression of a large number of matrix proteins normally associated with the hypertrophic chondrocyte phenotype. |
1997-10-07 |
2023-08-12 |
Not clear |
| S Bord, A Horner, R M Hembry, J J Reynolds, J E Compsto. Distribution of matrix metalloproteinases and their inhibitor, TIMP-1, in developing human osteophytic bone. Journal of anatomy. vol 191 ( Pt 1). 1997-09-18. PMID:9279657. |
chondrocytes demonstrated variable levels of collagenase and stromelysin expression throughout the proliferative and hypertrophic regions, stromelysin showing both cell-associated and strong matrix staining. |
1997-09-18 |
2023-08-12 |
human |
| S Shibata, S Suzuki, Y Yamashit. An ultrastructural study of cartilage resorption at the site of initial endochondral bone formation in the fetal mouse mandibular condyle. Journal of anatomy. vol 191 ( Pt 1). 1997-09-18. PMID:9279660. |
after resorbing the bone collar, the osteoclasts extended their cell processes into the cartilage matrix and made contact with hypertrophic chondrocytes. |
1997-09-18 |
2023-08-12 |
mouse |
| A G Kleber, V Fas. Molecular and cellular aspects of re-entrant arrhythmias. Basic research in cardiology. vol 92 Suppl 1. 1997-08-27. PMID:9202851. |
in recent years it has become evident that myocardial tissue undergoes remodeling in diseased states such as myocardial infarction and hypertrophy which affects membrane channels, cell-to-cell coupling as well as the connective tissue matrix. |
1997-08-27 |
2023-08-12 |
Not clear |
| P Ren, G N Rowland, J Halpe. Expression of growth factors in chicken growth plate with special reference to tibial dyschondroplasia. Journal of comparative pathology. vol 116. issue 3. 1997-08-14. PMID:9147248. |
immunolabelling for tgf beta then gradually shifted into cartilaginous matrix and was not observed in cytoplasm of hypertrophic chondrocytes until the late embryonic and post-hatch stages. |
1997-08-14 |
2023-08-12 |
chicken |
| M Sutmuller, J A Bruijn, E de Hee. Collagen types VIII and X, two non-fibrillar, short-chain collagens. Structure homologies, functions and involvement in pathology. Histology and histopathology. vol 12. issue 2. 1997-07-17. PMID:9151143. |
since collagen type x is produced by hypertrophic chondrocytes, this collagen type can only be found in matrix of the hypertrophic zone of the epiphyseal growth plate cartilage. |
1997-07-17 |
2023-08-12 |
Not clear |