All Relations between dopaminergic and striatum

Publication Sentence Publish Date Extraction Date Species
P E Vos, G J Bluemink, G Wolterink, J M Van Re. The ACTH-(4-9) analogue ORG 2766 facilitates denervation supersensitivity after a unilateral 6-OHDA lesion of the corpus striatum in rats. Neuropeptides. vol 19. issue 4. 1991-11-19. PMID:1656311. after a unilateral 6-ohda lesion of the right corpus striatum, contralateral rotation was observed upon administration of a high dose of apomorphine 2, 3 and 4 weeks after the lesion, indicating supersensitivity of postsynaptic dopaminergic receptor systems. 1991-11-19 2023-08-11 rat
S B Blunt, P Jenner, C D Marsde. The effect of L-dopa and carbidopa treatment on the survival of rat fetal dopamine grafts assessed by tyrosine hydroxylase immunohistochemistry and [3H]mazindol autoradiography. Neuroscience. vol 43. issue 1. 1991-11-15. PMID:1681462. the effect of treatment with l-3,4-dihydroxyphenylalanine (l-dopa) and carbidopa for five weeks on the survival of rat fetal dopaminergic ventral mesencephalon cells implanted into the denervated striatum of rats with unilateral 6-hydroxydopamine nigrostriatal lesions was assessed. 1991-11-15 2023-08-11 rat
F Filloux, J K Wamsle. Dopaminergic modulation of excitotoxicity in rat striatum: evidence from nigrostriatal lesions. Synapse (New York, N.Y.). vol 8. issue 4. 1991-11-14. PMID:1718049. dopaminergic modulation of excitotoxicity in rat striatum: evidence from nigrostriatal lesions. 1991-11-14 2023-08-11 rat
F Filloux, J K Wamsle. Dopaminergic modulation of excitotoxicity in rat striatum: evidence from nigrostriatal lesions. Synapse (New York, N.Y.). vol 8. issue 4. 1991-11-14. PMID:1718049. prior interruption of the nigrostriatal pathway (resulting in dopaminergic denervation of the ipsilateral striatum) partially protected this latter structure from subsequent injection of ibotenic acid (the extent of the lesion was reduced by 28%, p less than .05). 1991-11-14 2023-08-11 rat
J H Kordower, E Cochran, R D Penn, C G Goet. Putative chromaffin cell survival and enhanced host-derived TH-fiber innervation following a functional adrenal medulla autograft for Parkinson's disease. Annals of neurology. vol 29. issue 4. 1991-11-07. PMID:1681779. this response may represent sprouting by residual host dopaminergic neurons mediated by the vigorous response to injury displayed by the host striatum. 1991-11-07 2023-08-11 human
J F Chen, Z H Qin, F Szele, G Bai, B Weis. Neuronal localization and modulation of the D2 dopamine receptor mRNA in brain of normal mice and mice lesioned with 6-hydroxydopamine. Neuropharmacology. vol 30. issue 9. 1991-11-06. PMID:1833662. these results suggest that the d2 receptor mrna in the substantia nigra pars compacta was localized largely to dopaminergic cell bodies, the terminal projections of which lie in the striatum and codes for d2 autoreceptors and that the d2 receptor mrna of the striatum is in non-dopaminergic cell bodies that are intrinsic to the striatum and probably codes for post-synaptic d2 receptors. 1991-11-06 2023-08-11 mouse
J F Chen, Z H Qin, F Szele, G Bai, B Weis. Neuronal localization and modulation of the D2 dopamine receptor mRNA in brain of normal mice and mice lesioned with 6-hydroxydopamine. Neuropharmacology. vol 30. issue 9. 1991-11-06. PMID:1833662. further, the evidence that lesions of striatum and substantia nigra induced with 6-hydroxydopamine greatly reduced the d2 receptor mrna signal in the substantia nigra, without concomitantly increasing the d2 receptor mrna in the striatum, suggests that the increase in dopamine receptor binding in the striatum that is ipsilateral to the lesion with 6-hydroxydopamine and the enhanced behavioral sensitivity to dopaminergic agonists, cannot be accounted for solely by an increase in d2 receptor mrna. 1991-11-06 2023-08-11 mouse
F C Liu, S B Dunnett, H A Robertson, A M Graybie. Intrastriatal grafts derived from fetal striatal primordia. III. Induction of modular patterns of fos-like immunoreactivity by cocaine. Experimental brain research. vol 85. issue 3. 1991-11-05. PMID:1680735. previous studies have shown that presynaptic and postsynaptic cellular markers of the dopaminergic system in the striatum, including immunostaining for th and dopamine- and adenosine 3':5'-monophosphate-regulated phosphoprotein (darpp-32), and binding for high affinity dopamine uptake sites and for dopamine d1 and d2 receptor sites, are all concentrated in the ache-rich patch regions (p regions) of such embryonic striatal grafts. 1991-11-05 2023-08-11 rat
C Beyer, C Pilgrim, I Reisert, U Misgel. Cells from embryonic rat striatum cocultured with mesencephalic glia express dopaminergic phenotypes. Neuroscience letters. vol 128. issue 1. 1991-11-05. PMID:1681474. cells from embryonic rat striatum cocultured with mesencephalic glia express dopaminergic phenotypes. 1991-11-05 2023-08-11 rat
J Segovia, R Castro, V Notario, K Gal. Transplants of fetal substantia nigra regulate glutamic acid decarboxylase gene expression in host striatal neurons. Brain research. Molecular brain research. vol 10. issue 4. 1991-11-04. PMID:1656162. lesions of the dopaminergic innervation to the striatum result in increased activity of glutamic acid decarboxylase (gad) and increased gad mrna in striatal gabaergic neurons. 1991-11-04 2023-08-11 rat
T Hattori, M Takada, T Moriizumi, D Van der Koo. Single dopaminergic nigrostriatal neurons form two chemically distinct synaptic types: possible transmitter segregation within neurons. The Journal of comparative neurology. vol 309. issue 3. 1991-11-04. PMID:1717519. previous studies have shown that anterograde transport of tritiated labeled proteins from the substantia nigra to the striatum over short survival times primarily labels asymmetric synapses (and that these asymmetric synapses are preferentially vulnerable to selective dopaminergic neurotoxins such as 6-hydroxydopamine). 1991-11-04 2023-08-11 rat
T Hattori, M Takada, T Moriizumi, D Van der Koo. Single dopaminergic nigrostriatal neurons form two chemically distinct synaptic types: possible transmitter segregation within neurons. The Journal of comparative neurology. vol 309. issue 3. 1991-11-04. PMID:1717519. we conclude that both the asymmetric terminal and symmetric en passant synapses take origin from a single nigrostriatal dopaminergic neuronal population and that dopaminergic transmitter markers occur only in one of these synaptic types in the rat striatum. 1991-11-04 2023-08-11 rat
T Moriizumi, M Takada, T Hattor. Morphological changes of striatal dopaminergic presynaptic boutons following intrastriatal kainate injection. Neuropharmacology. vol 30. issue 7. 1991-11-04. PMID:1922691. two to 42 days after kainic acid (2.2-5.0 nmol) injection into the rat striatum, nigrostriatal dopaminergic boutons were examined at the injection site using tyrosine hydroxylase immunohistochemistry. 1991-11-04 2023-08-11 rat
N Mataga, K Imamura, Y Watanab. 6R-tetrahydrobiopterin perfusion enhances dopamine, serotonin, and glutamate outputs in dialysate from rat striatum and frontal cortex. Brain research. vol 551. issue 1-2. 1991-11-01. PMID:1680529. dopaminergic terminals in the striatum were destroyed unilaterally by continuous infusion of 6-hydroxydopamine (6-ohda) using an osmotic minipump system. 1991-11-01 2023-08-11 rat
M Garcia-Munoz, S J Young, P M Grove. Terminal excitability of the corticostriatal pathway. I. Regulation by dopamine receptor stimulation. Brain research. vol 551. issue 1-2. 1991-11-01. PMID:1913151. the influence of dopaminergic transmission on the cortical terminal fields in the striatum was assessed electrophysiologically using the terminal excitability method. 1991-11-01 2023-08-11 Not clear
N T Sándor, J Kiss, A Sándor, B Lendvai, E S Viz. Naloxone enhances the release of acetylcholine from cholinergic interneurons of the striatum if the dopaminergic input is impaired. Brain research. vol 552. issue 2. 1991-10-31. PMID:1913196. naloxone enhances the release of acetylcholine from cholinergic interneurons of the striatum if the dopaminergic input is impaired. 1991-10-31 2023-08-11 rat
D C Mash, J Pablo, B E Buck, J Sanchez-Ramos, W J Weine. Distribution and number of transferrin receptors in Parkinson's disease and in MPTP-treated mice. Experimental neurology. vol 114. issue 1. 1991-10-30. PMID:1915737. to address the possibility that transferrin receptors are located on dopaminergic terminals, we have examined the distribution and number of transferrin receptors in the striatum of mptp-treated mice using in vitro autoradiographic methods. 1991-10-30 2023-08-11 mouse
D C Mash, J Pablo, B E Buck, J Sanchez-Ramos, W J Weine. Distribution and number of transferrin receptors in Parkinson's disease and in MPTP-treated mice. Experimental neurology. vol 114. issue 1. 1991-10-30. PMID:1915737. in these experiments, the loss of dopaminergic terminals in the striatum was visualized by differential [3h]mazindol uptake site autoradiography. 1991-10-30 2023-08-11 mouse
N Kolocouris, G B Foscolos, G Fytas, A Sakellariou, C Raftopoulos, Z Papadopoulou-Daïfoti, A Vamvakidè. [Dopamine amides with essential or fundamental fatty acids. Synthesis and pharmacological study]. Annales pharmaceutiques francaises. vol 49. issue 2. 1991-10-28. PMID:1681774. the experimental results which are obtained from these derivatives (antagonized or non antagonized hypomotility by neuroleptics in low doses on mice, potentiation of the haloperidol catalepsy or not, on rats) could be compatible with a selective action of these amides on the autoreceptors of the cerebral dopaminergic neurons and with a preferential activity of some derivatives on certain cerebral areas (mesencephalon or striatum). 1991-10-28 2023-08-11 mouse
M Manier, D N Abrous, C Feuerstein, M Le Moal, J P Herma. Increase of striatal methionin enkephalin content following lesion of the nigrostriatal dopaminergic pathway in adult rats and reversal following the implantation of embryonic dopaminergic neurons: a quantitative immunohistochemical analysis. Neuroscience. vol 42. issue 2. 1991-10-21. PMID:1896133. the aim of the present study was to test whether intrastriatal implants of embryonic dopaminergic neurons are able to normalize the lesion-induced dysfunction of striatal enkephalinergic neurons, one of the major output systems of the striatum. 1991-10-21 2023-08-11 rat