| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Julie Péron, Didier Grandjean, Sophie Drapier, Marc Véri. Effect of dopamine therapy on nonverbal affect burst recognition in Parkinson's disease. PloS one. vol 9. issue 3. 2014-12-22. PMID:24651759. |
parkinson's disease (pd) provides a model for investigating the involvement of the basal ganglia and mesolimbic dopaminergic system in the recognition of emotions from voices (i.e., emotional prosody). |
2014-12-22 |
2023-08-12 |
Not clear |
| G Herrera-Meza, L Aguirre-Manzo, G A Coria-Avila, M L Lopez-Meraz, R Toledo-Cárdenas, J Manzo, L I Garcia, M Mique. Beyond the basal ganglia: cFOS expression in the cerebellum in response to acute and chronic dopaminergic alterations. Neuroscience. vol 267. 2014-12-09. PMID:24631673. |
beyond the basal ganglia: cfos expression in the cerebellum in response to acute and chronic dopaminergic alterations. |
2014-12-09 |
2023-08-12 |
rat |
| Alexis N Cohen-Oram, Jonathan T Stewart, Kim Bero, Michael W Hoffman. Treatment of gait ignition failure with ropinirole. Journal of movement disorders. vol 7. issue 2. 2014-10-31. PMID:25360234. |
mri was unrevealing, but a positron emission tomography scan showed hypometabolism in the deep frontal aca/mca watershed area; this may have disconnected the basal ganglia from the motor cortex and/or interrupted dopaminergic mesocortical transmission. |
2014-10-31 |
2023-08-13 |
Not clear |
| Marielle Zoetmulder, Heidi B Biernat, Miki Nikolic, Lise Korbo, Lars Friberg, Poul J Jennu. Prepulse inhibition is associated with attention, processing speed, and 123I-FP-CIT SPECT in Parkinson's disease. Journal of Parkinson's disease. vol 4. issue 1. 2014-10-14. PMID:24366928. |
prepulse inhibition is dramatically altered in basal ganglia disorders associated with dysfunction in the midbrain dopaminergic system, and corresponding cognitive information processing deficits such as slowed processing speed. |
2014-10-14 |
2023-08-12 |
Not clear |
| Georg Dirnberger, Marjan Jahanshah. Executive dysfunction in Parkinson's disease: a review. Journal of neuropsychology. vol 7. issue 2. 2014-10-09. PMID:24007368. |
we then discuss how executive deficits relate to pathology in specific territories of the basal ganglia, consider the impact of dopaminergic treatment on executive function (ef) in this context, and review the changes in efs with disease progression. |
2014-10-09 |
2023-08-12 |
Not clear |
| Kara M Smith, Nabila Dahodwal. Sex differences in Parkinson's disease and other movement disorders. Experimental neurology. vol 259. 2014-09-29. PMID:24681088. |
as a result of hormone, chromosome and other unknown effects, there are sexual dimorphisms in the basal ganglia, and at the molecular levels in dopaminergic neurons that may lead to distinct mechanisms of pathogenesis in males and females. |
2014-09-29 |
2023-08-12 |
Not clear |
| Kara M Smith, Nabila Dahodwal. Sex differences in Parkinson's disease and other movement disorders. Experimental neurology. vol 259. 2014-09-29. PMID:24681088. |
we also briefly review how sex differences in basal ganglia function and dopaminergic pathways may impact hd, chorea, and tics/tourette's syndrome. |
2014-09-29 |
2023-08-12 |
Not clear |
| A Delval, C Tard, L Defebvr. Why we should study gait initiation in Parkinson's disease. Neurophysiologie clinique = Clinical neurophysiology. vol 44. issue 1. 2014-09-24. PMID:24502907. |
the fact that emotion, attention, external triggers and dopaminergic drugs can all modify this motor program suggests the existence of a complex pathophysiological mechanism that involves not only locomotor networks but also cortical areas and the basal ganglia system. |
2014-09-24 |
2023-08-12 |
Not clear |
| Henry H Yi. Action, time and the basal ganglia. Philosophical transactions of the Royal Society of London. Series B, Biological sciences. vol 369. issue 1637. 2014-09-22. PMID:24446506. |
the ability to control the speed of movement is compromised in neurological disorders involving the basal ganglia, a set of subcortical cerebral nuclei that receive prominent dopaminergic projections from the midbrain. |
2014-09-22 |
2023-08-12 |
Not clear |
| Ryuji Sakakibar. [Cognitive adverse effects of anticholinergic medication for overactive bladder in PD/DLB]. Rinsho shinkeigaku = Clinical neurology. vol 53. issue 11. 2014-09-19. PMID:24292000. |
overactive bladder (oab) is a common autonomic disorder due mostly to lesions in the micturition-inhibiting area (d1 dopaminergic pathway in the basal ganglia) in parkinson's disease (pd) and dementia with lewy bodies (dlb). |
2014-09-19 |
2023-08-12 |
Not clear |
| Domingo Afonso-Oramas, Ignacio Cruz-Muros, Javier Castro-Hernández, Josmar Salas-Hernández, Pedro Barroso-Chinea, Sonia García-Hernández, José L Lanciego, Tomás González-Hernánde. Striatal vessels receive phosphorylated tyrosine hydroxylase-rich innervation from midbrain dopaminergic neurons. Frontiers in neuroanatomy. vol 8. 2014-09-10. PMID:25206324. |
bearing in mind the evidence of changes in the blood flow of basal ganglia in parkinson's disease (pd), and the pivotal role of the dopaminergic mesostriatal pathway in the pathophysiology of this disease, here we studied whether striatal vessels receive inputs from midbrain dopaminergic neurons. |
2014-09-10 |
2023-08-13 |
rat |
| Asha Kishore, Traian Pop. Cerebellum in levodopa-induced dyskinesias: the unusual suspect in the motor network. Frontiers in neurology. vol 5. 2014-09-03. PMID:25183959. |
artificial cerebellar stimulation might help re-establish the cerebellar and basal ganglia control over the non-salient inputs to the motor areas during synaptic dopaminergic surges. |
2014-09-03 |
2023-08-13 |
Not clear |
| Francisco Espejo-Porras, Javier Fernández-Ruiz, Roger G Pertwee, Raphael Mechoulam, Concepción Garcí. Motor effects of the non-psychotropic phytocannabinoid cannabidiol that are mediated by 5-HT1A receptors. Neuropharmacology. vol 75. 2014-08-19. PMID:23924692. |
the broad presence of cb1 receptors in the basal ganglia, mainly in gaba- or glutamate-containing neurons, as well as the presence of trpv1 receptors in dopaminergic neurons and the identification of cb2 receptors in some neuronal subpopulations within the basal ganglia, explain the powerful motor effects exerted by those cannabinoids that can activate/block these receptors. |
2014-08-19 |
2023-08-12 |
rat |
| Gabriela P Chaves-Kirsten, Caio H Y Mazucanti, Caroline C Real, Bruna M Souza, Luiz R G Britto, Andréa S Torrã. Temporal changes of CB1 cannabinoid receptor in the basal ganglia as a possible structure-specific plasticity process in 6-OHDA lesioned rats. PloS one. vol 8. issue 10. 2014-08-02. PMID:24116178. |
the present study evaluated the cb1 expression in four basal ganglia structures, namely striatum, external globus pallidus (egp), internal globus pallidus (igp) and substantia nigra pars reticulata (snpr) of rats 1, 5, 10, 20, and 60 days after unilateral intrastriatal 6-hydroxydopamine injections, that causes retrograde dopaminergic degeneration. |
2014-08-02 |
2023-08-12 |
rat |
| Anna Czarnecka, Tomasz Lenda, Helena Domin, Jolanta Konieczny, Maria Smiałowska, Elżbieta Lorenc-Koc. Alterations in the expression of nNOS in the substantia nigra and subthalamic nucleus of 6-OHDA-lesioned rats: the effects of chronic treatment with l-DOPA and the nitric oxide donor, molsidomine. Brain research. vol 1541. 2014-07-28. PMID:24129225. |
degeneration of dopaminergic neurons in the substantia nigra (sn) in parkinson's disease leads to disturbances in the nitrergic transmission in the basal ganglia. |
2014-07-28 |
2023-08-12 |
rat |
| Antonella Conte, Nashaba Khan, Giovanni Defazio, John C Rothwell, Alfredo Berardell. Pathophysiology of somatosensory abnormalities in Parkinson disease. Nature reviews. Neurology. vol 9. issue 12. 2014-07-14. PMID:24217516. |
sensory deficits are likely to be a consequence of the dopaminergic denervation of the basal ganglia that is the hallmark of pd. |
2014-07-14 |
2023-08-12 |
Not clear |
| Nathaniel S Miller, Youngbin Kwak, Nicolaas I Bohnen, Martijn L T M Müller, Praveen Dayalu, Rachael D Seidle. The pattern of striatal dopaminergic denervation explains sensorimotor synchronization accuracy in Parkinson's disease. Behavioural brain research. vol 257. 2014-06-25. PMID:24076152. |
these findings provide further evidence for the role of the basal ganglia and dopamine in duration production and suggest that the degree of striatal dopaminergic denervation may explain the heterogeneity of performance between pd patients on the sensorimotor synchronization task. |
2014-06-25 |
2023-08-12 |
Not clear |
| Kaviraja Udupa, Robert Che. Motor cortical plasticity in Parkinson's disease. Frontiers in neurology. vol 4. 2014-06-24. PMID:24027555. |
in parkinson's disease (pd), there are alterations of the basal ganglia (bg) thalamocortical networks, primarily due to degeneration of nigrostriatal dopaminergic neurons. |
2014-06-24 |
2023-08-12 |
human |
| Panagiotis Bargiotas, Spyridon Konitsioti. Levodopa-induced dyskinesias in Parkinson's disease: emerging treatments. Neuropsychiatric disease and treatment. vol 9. 2014-06-24. PMID:24174877. |
lids are the result of profound modifications in the functional organization of the basal ganglia circuitry, possibly related to the chronic and pulsatile stimulation of striatal dopaminergic receptors by levodopa. |
2014-06-24 |
2023-08-12 |
Not clear |
| Ashvin Shah, Kevin N Gurne. Emergent structured transition from variation to repetition in a biologically-plausible model of learning in basal ganglia. Frontiers in psychology. vol 5. 2014-06-24. PMID:24575067. |
while the structure of the pattern can be explained by sophisticated cognitive mechanisms, simpler mechanisms based on dopaminergic modulation of basal ganglia (bg) activity are thought to underlie action discovery (redgrave and gurney, 2006). |
2014-06-24 |
2023-08-12 |
human |