| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| J A Severson, J Marcusson, B Winblad, C E Finc. Age-correlated loss of dopaminergic binding sites in human basal ganglia. Journal of neurochemistry. vol 39. issue 6. 1983-01-07. PMID:7142992. |
age-correlated loss of dopaminergic binding sites in human basal ganglia. |
1983-01-07 |
2023-08-12 |
human |
| K Fuxe, K Andersson, O G Nilsen, R Toftgård, P Eneroth, J A Gustafsso. Toluene and telencephalic dopamine: selective reduction of amine turnover in discrete DA nerve terminal systems of the anterior caudate nucleus by low concentrations of toluene. Toxicology letters. vol 12. issue 2-3. 1982-10-12. PMID:7112606. |
thus, toluene at low concentrations may produce disturbances in dopaminergic mechanisms of the basal ganglia probably leading to functional changes in sensory-motor integration. |
1982-10-12 |
2023-08-12 |
rat |
| R A Smego, D T Durac. The neuroleptic malignant syndrome. Archives of internal medicine. vol 142. issue 6. 1982-08-07. PMID:6124221. |
the nms is attributed to a disturbance of dopaminergic systems within the basal ganglia and hypothalamus. |
1982-08-07 |
2023-08-12 |
Not clear |
| I K Ho, K Fujimori, T P Huang, H Chang-Tus. Neurochemical evaluation of chlordecone toxicity in the mouse. Journal of toxicology and environmental health. vol 8. issue 5-6. 1982-06-24. PMID:6175763. |
on the basis of the data obtained so far, it is suggested that the dopaminergic pathway and its interaction with other neurotransmitter systems in the basal ganglia may be primarily involved in chlordecone-induced neurotoxicity. |
1982-06-24 |
2023-08-12 |
mouse |
| W G Rector, H F Herlong, H Mose. Nonketotic hyperglycemia appearing as choreoathetosis or ballism. Archives of internal medicine. vol 142. issue 1. 1982-02-25. PMID:7053718. |
potential pathogenetic mechanisms include relative dopaminergic hypersensitivity, impaired synthesis of acetylcholine or gamma-aminobutyric acid, or an undefined effect of hyperosmolarity, perhaps unmasking a previously subclinical lesion of the basal ganglia. |
1982-02-25 |
2023-08-12 |
Not clear |
| T W Vickroy, K M Johnso. Stimulation of synaptosomal tyrosine hydroxylation by phencyclidine in vitro. European journal of pharmacology. vol 71. issue 4. 1981-09-15. PMID:7250199. |
phencyclidine (pcp), a potent psychoactive drug, produces some animal behaviors that are believed to be mediated by dopaminergic and/or cholinergic neurons in the basal ganglia. |
1981-09-15 |
2023-08-12 |
rat |
| J F Marshall, J W Critchfield, M R Kozlowsk. Altered succinate dehydrogenase activity of basal ganglia following to mesotelencephalic dopaminergic projection. Brain research. vol 212. issue 2. 1981-07-20. PMID:7225874. |
altered succinate dehydrogenase activity of basal ganglia following to mesotelencephalic dopaminergic projection. |
1981-07-20 |
2023-08-12 |
rat |
| S Lal, P Etienne, J Thavundayil, N P Nair, B Collier, R Rastogi, H Guyda, G Schwart. Effect of choline on central dopaminergic function in normal subjects. Journal of neural transmission. vol 50. issue 1. 1981-05-26. PMID:7205247. |
these data suggest that cholinergic mechanisms may enhance hypothalamic-pituitary dopaminergic function in man in contrast to their inhibitory effect on dopaminergic function in the basal ganglia. |
1981-05-26 |
2023-08-12 |
human |
| O Hornykiewic. [Brain dopamine and brain disorders - and what we have learned from these studies (author's transl)]. Wiener klinische Wochenschrift. vol 92. issue 20. 1981-04-13. PMID:7467350. |
this extensive dopaminergic denervation of the basal ganglia results in compensatory pre- and postsynaptic changes, i.e. |
1981-04-13 |
2023-08-12 |
Not clear |
| Y Kond. [Action sites of morphine--effects on dopaminergic and GABA-containing neurons in the basal ganglia]. [Osaka Daigaku shigaku zasshi] The journal of Osaka University Dental Society. vol 25. issue 1. 1981-03-27. PMID:6256512. |
[action sites of morphine--effects on dopaminergic and gaba-containing neurons in the basal ganglia]. |
1981-03-27 |
2023-08-12 |
Not clear |
| B S Bunney, A A Grace, D W Homme. Changing concepts of nigral dopamine system function within the basal ganglia: relevance to extrapyramidal disorders. Journal of neural transmission. Supplementum. issue 16. 1981-01-16. PMID:6107330. |
one such change has involved our understanding of the role of the dopaminergic system within the basal ganglia. |
1981-01-16 |
2023-08-12 |
Not clear |
| B S Bunney, A A Grace, D W Homme. Changing concepts of nigral dopamine system function within the basal ganglia: relevance to extrapyramidal disorders. Journal of neural transmission. Supplementum. issue 16. 1981-01-16. PMID:6107330. |
these new roles for the nigral dopaminergic system raise important new questions about its function in extrapyramidal motor systems as well as provide possible answers to questions concerning the mechanisms underlying some of the symptoms seen in disorders involving the basal ganglia. |
1981-01-16 |
2023-08-12 |
Not clear |
| R Schwarcz, K Fuxe, T Hökfelt, L Terenius, M Goldstei. Effects of chronic striatal kainate lesions on some dopaminergic parameters and enkephalin immunoreactive neurons in the basal ganglia. Journal of neurochemistry. vol 34. issue 4. 1980-05-23. PMID:6102111. |
effects of chronic striatal kainate lesions on some dopaminergic parameters and enkephalin immunoreactive neurons in the basal ganglia. |
1980-05-23 |
2023-08-12 |
Not clear |
| A I Balakleevsky, N P Bobrova, N V Shevtchenk. Regulation of gamma-aminobutyric acid metabolism in different brain structures by cholinergic and adrenergic drugs. Annali dell'Istituto superiore di sanita. vol 14. issue 1. 1979-12-29. PMID:756168. |
most dopaminergic drugs (apomorphine, l-dopa, cb-154, fusaric acid, 6-hydroxydopamine, haloperidol) fail to influence gaba metabolism in whole brain and in basal ganglia. |
1979-12-29 |
2023-08-11 |
mouse |
| A R Crossman, M A Sambrook, S W Gergies, P Slate. The neurological basis of motor asymmetry following unilateral 6-hydroxydopamine brain lesions in the rat: the effect of motor decortication. Journal of the neurological sciences. vol 34. issue 3. 1978-02-18. PMID:591996. |
it is postulated that subcortical basal ganglia efferent pathways may be involved in the mediation of motor asymmetry after unilateral 6-ohda lesions of the nigrostriatal dopaminergic projection. |
1978-02-18 |
2023-08-11 |
rat |
| E S Sear. Therapeutics of disordered movement. American family physician. vol 16. issue 3. 1977-10-20. PMID:143204. |
human motor behavior is organized around two major neurotransmitter systems in the basal ganglia--dopaminergic and cholinergic. |
1977-10-20 |
2023-08-11 |
human |
| K Lühdorf, M Lun. Phenytoin-induced hyperkinesia. Epilepsia. vol 18. issue 3. 1977-10-14. PMID:891495. |
the hypothesis is presented that phenytoin induces hyperkinesia by increasing dopaminergic and serotonergic activity in the basal ganglia, and that patients with preexisting basal ganglia damage are the most susceptible. |
1977-10-14 |
2023-08-11 |
Not clear |
| M M Cohen, R T Scheif. Pharmacotherapy of Parkinson's disease. American journal of hospital pharmacy. vol 34. issue 5. 1977-07-23. PMID:326045. |
the pathophysiology, anticholinergic therapy and dopaminergic therapy of parkinson's disease are reviewed; an emphasis is placed on the structure and function of the basal ganglia because of their importance in understanding the pharmacotherapy of parkinsonism. |
1977-07-23 |
2023-08-11 |
Not clear |
| J Gerlac. Relationship between tardive dyskinesia, L-Dopa-induced hyperkinesia and parkinsonism. Psychopharmacology. vol 51. issue 3. 1977-05-20. PMID:403541. |
it is concluded that the irreversible neurotoxic effect of neuroleptic drugs may be associated with age-related changes in the oral somatotopic region of the basal ganglia (to be given consideration in any future search for the pathogenetic process underlying irreversible tardive dyskinesia), and that the pathophysiology of involuntary hyperkinesia in neuroleptic-treated psychiatric patients and in l-dopatreated parkinson patients may consist of a primary dopamine deficiency (pharmacological or structural), and a secondary relative hyperactivity in the dopaminergic system ("dopaminergic hypersensitivity") possibly corresponding to hypoactivity in the cholinergic system. |
1977-05-20 |
2023-08-11 |
Not clear |
| B S Bunney, G K Aghajania. Dopaminergic infllence in the basal ganglia: evidence for striatonigral feedback regulation. Research publications - Association for Research in Nervous and Mental Disease. vol 55. 1977-02-24. PMID:826996. |
dopaminergic infllence in the basal ganglia: evidence for striatonigral feedback regulation. |
1977-02-24 |
2023-08-11 |
Not clear |