All Relations between mog and oligodendrocyte

Publication Sentence Publish Date Extraction Date Species
H P Eugster, K Frei, F Winkler, U Koedel, W Pfister, H Lassmann, A Fontan. Superantigen overcomes resistance of IL-6-deficient mice towards MOG-induced EAE by a TNFR1 controlled pathway. European journal of immunology. vol 31. issue 8. 2001-09-13. PMID:11477542. experimental autoimmune encephalomyelitis (eae) induced by myelin oligodendrocyte glycoprotein peptide 35-55 (mog) leads to a chronic form of disease characterized by demyelination, inflammation and gliosis in the central nervous system (cns). 2001-09-13 2023-08-12 mouse
R Weissert, K L de Graaf, M K Storch, S Barth, C Linington, H Lassmann, T Olsso. MHC class II-regulated central nervous system autoaggression and T cell responses in peripheral lymphoid tissues are dissociated in myelin oligodendrocyte glycoprotein-induced experimental autoimmune encephalomyelitis. Journal of immunology (Baltimore, Md. : 1950). vol 166. issue 12. 2001-08-23. PMID:11390515. we dissected the requirements for disease induction of myelin oligodendrocyte glycoprotein (mog)-induced experimental autoimmune encephalomyelitis in mhc (rt1 in rat) congenic rats with overlapping mog peptides. 2001-08-23 2023-08-12 rat
H T Yang, K Bergsteinsdottir, P Wernhoff, C Linington, U Pettersson, R Holmdah. Genetic linkage analysis of the antibody responses to myelin basic protein and myelin oligodendrocyte glycoprotein in rats immunized with rat spinal cord homogenate. Journal of neuroimmunology. vol 117. issue 1-2. 2001-08-23. PMID:11431001. the genetic control of the antibody response to myelin basic protein (mbp) and myelin oligodendrocyte glycoprotein (mog) was analysed in f1 and f2 crosses of da and e3 rats, immunized with rat spinal chord homogenate. 2001-08-23 2023-08-12 rat
Y Sekiguchi, M Ichikawa, A Inoue, M Itoh, C S Ko. Brain-derived gangliosides suppress the chronic relapsing-remitting experimental autoimmune encephalomyelitis in NOD mice induced with myelin oligodendrocyte glycoprotein peptide. Journal of neuroimmunology. vol 116. issue 2. 2001-08-16. PMID:11438174. chronic relapsing-remitting experimental autoimmune encephalomyelitis (creae) induced with myelin oligodendrocyte glycoprotein peptides 35-55 (mog(35-55)) in nod mice was successfully treated with brain-derived gangliosides (ga). 2001-08-16 2023-08-12 mouse
D Offen, J F Kaye, O Bernard, D Merims, C I Coire, H Panet, E Melamed, A Ben-Nu. Mice overexpressing Bcl-2 in their neurons are resistant to myelin oligodendrocyte glycoprotein (MOG)-induced experimental autoimmune encephalomyelitis (EAE). Journal of molecular neuroscience : MN. vol 15. issue 3. 2001-08-09. PMID:11303781. mice overexpressing bcl-2 in their neurons are resistant to myelin oligodendrocyte glycoprotein (mog)-induced experimental autoimmune encephalomyelitis (eae). 2001-08-09 2023-08-12 mouse
D Offen, J F Kaye, O Bernard, D Merims, C I Coire, H Panet, E Melamed, A Ben-Nu. Mice overexpressing Bcl-2 in their neurons are resistant to myelin oligodendrocyte glycoprotein (MOG)-induced experimental autoimmune encephalomyelitis (EAE). Journal of molecular neuroscience : MN. vol 15. issue 3. 2001-08-09. PMID:11303781. to study the role of axonal damage in the pathogenesis of ms-like disease induced by myelin oligodendrocyte glycoprotein (mog), we compared experimental autoimmune encephalomyelitis (eae) in wild-type (wt) and transgenic mice expressing the human bcl-2 gene exclusively in neurons under the control of the neuron-specific enolase (nse) promoter. 2001-08-09 2023-08-12 mouse
A J Slavin, R Maron, H L Weine. Mucosal administration of IL-10 enhances oral tolerance in autoimmune encephalomyelitis and diabetes. International immunology. vol 13. issue 6. 2001-08-09. PMID:11369711. in addition, oral il-10 combined with oral myelin oligodendrocyte glycoprotein (mog) 35-55 reduced relapses in mog-induced eae in the nod mouse, as well as enhanced the protective effect of oral insulin in the nod model of diabetes. 2001-08-09 2023-08-12 mouse
M Onuki, M M Ayers, C C Bernard, J M Oria. Axonal degeneration is an early pathological feature in autoimmune-mediated demyelination in mice. Microscopy research and technique. vol 52. issue 6. 2001-08-02. PMID:11276125. we have described a murine model of relapsing-remitting ms (rr-ms), the most common form of the disease, following immunisation with the myelin component, myelin oligodendrocyte glycoprotein (mog). 2001-08-02 2023-08-12 mouse
A Carotenuto, A M D'Ursi, E Nardi, A M Papini, P Rover. Conformational analysis of a glycosylated human myelin oligodendrocyte glycoprotein peptide epitope able to detect antibody response in multiple sclerosis. Journal of medicinal chemistry. vol 44. issue 14. 2001-07-19. PMID:11428934. myelin oligodendrocyte glycoprotein (mog), a minor myelin component, is an important central nervous system specific target autoantigen for primary demyelination in autoimmune diseases such as multiple sclerosis (ms). 2001-07-19 2023-08-12 human
D A Kahn, D C Archer, D P Gold, C J Kell. Adjuvant immunotherapy is dependent on inducible nitric oxide synthase. The Journal of experimental medicine. vol 193. issue 11. 2001-06-28. PMID:11390433. groups of nontreated and cfa-preimmunized male c57bl/6j or c57bl/6nos2(-/)- mice were immunized with myelin oligodendrocyte glycoprotein (mog) peptide 35-55 in cfa to induce experimental allergic encephalomyelitis (eae). 2001-06-28 2023-08-12 mouse
B Becher, B G Durell, A V Miga, W F Hickey, R J Noell. The clinical course of experimental autoimmune encephalomyelitis and inflammation is controlled by the expression of CD40 within the central nervous system. The Journal of experimental medicine. vol 193. issue 8. 2001-05-21. PMID:11304557. using bone marrow (bm) chimeric mice, the data suggest that the lack of expression of cd40 by cns-resident cells diminishes the intensity and duration of myelin oligodendrocyte glycoprotein (mog)-induced eae and also reduces the degree of inflammatory cell infiltrates into the cns. 2001-05-21 2023-08-12 mouse
M T Heneka, A Sharp, P Murphy, J A Lyons, L Dumitrescu, D L Feinstei. The heat shock response reduces myelin oligodendrocyte glycoprotein-induced experimental autoimmune encephalomyelitis in mice. Journal of neurochemistry. vol 77. issue 2. 2001-05-17. PMID:11299319. eae was actively induced in c57bl/6 mice using an encephalitogenic myelin oligodendrocyte glycoprotein (mog(35-55)) peptide. 2001-05-17 2023-08-12 mouse
b' M Niino, K Iwabuchi, S Kikuchi, M Ato, T Morohashi, A Ogata, K Tashiro, K Ono\\xc3\\xa. Amelioration of experimental autoimmune encephalomyelitis in C57BL/6 mice by an agonist of peroxisome proliferator-activated receptor-gamma. Journal of neuroimmunology. vol 116. issue 1. 2001-05-10. PMID:11311328.' in the present study, we examined whether troglitazone, a selective ppar-gamma agonists, ameliorated experimental autoimmune encephalomyelitis (eae) induced by administration of myelin oligodendrocyte glycoprotein (mog) peptide 35-55 in c57bl/6 mice. 2001-05-10 2023-08-12 mouse
N Hellings, M Barée, C Verhoeven, M B D'hooghe, R Medaer, C C Bernard, J Raus, P Stinisse. T-cell reactivity to multiple myelin antigens in multiple sclerosis patients and healthy controls. Journal of neuroscience research. vol 63. issue 3. 2001-04-26. PMID:11170179. myelin proteins, including myelin basic protein (mbp), proteolipid protein (plp) and myelin oligodendrocyte glycoprotein (mog) are candidate autoantigens in ms. 2001-04-26 2023-08-12 human
T Berger, M Reind. Immunopathogenic and clinical relevance of antibodies against myelin oligodendrocyte glycoprotein (MOG) in Multiple Sclerosis. Journal of neural transmission. Supplementum. issue 60. 2001-04-12. PMID:11205153. immunopathogenic and clinical relevance of antibodies against myelin oligodendrocyte glycoprotein (mog) in multiple sclerosis. 2001-04-12 2023-08-12 Not clear
T Berger, M Reind. Immunopathogenic and clinical relevance of antibodies against myelin oligodendrocyte glycoprotein (MOG) in Multiple Sclerosis. Journal of neural transmission. Supplementum. issue 60. 2001-04-12. PMID:11205153. this present article will discuss recently published and some preliminary data on the immunopathogenic role of antibodies against myelin oligodendrocyte glycoprotein (mog) and other myelin/nonmyelin targets in multiple sclerosis, as well as possible clinical implications for prognosis and therapy in the future. 2001-04-12 2023-08-12 Not clear
C G Haase, J Guggenmos, U Brehm, M Andersson, T Olsson, M Reindl, J M Schneidewind, U K Zettl, F Heidenreich, T Berger, H Wekerle, R Hohlfeld, C Liningto. The fine specificity of the myelin oligodendrocyte glycoprotein autoantibody response in patients with multiple sclerosis and normal healthy controls. Journal of neuroimmunology. vol 114. issue 1-2. 2001-04-12. PMID:11240035. antibodies directed against the extracellular immunoglobulin (ig)-like domain of the myelin oligodendrocyte glycoprotein (mog(igd)) mediate demyelination in experimental autoimmune encephalomyelitis (eae) and are implicated in the immunopathogenesis of multiple sclerosis (ms). 2001-04-12 2023-08-12 human
H Hackstein, A Bitsch, A Bohnert, H Hofmann, F Weber, A Ohly, C Linington, M Mäurer, S Poser, P Rieckmann, G Bei. Analysis of interleukin-4 receptor alpha chain variants in multiple sclerosis. Journal of neuroimmunology. vol 113. issue 2. 2001-03-22. PMID:11164908. analysis of the first 100 ms patients for six il4r variants showed an increased frequency of the r551 variant in ms patients versus healthy controls and carriage of the same il4r variant was weakly associated with myelin oligodendrocyte glycoprotein (mog) autoantibody production. 2001-03-22 2023-08-12 Not clear
J R Kanwar, R K Kanwar, D Wang, G W Krissanse. Prevention of a chronic progressive form of experimental autoimmune encephalomyelitis by an antibody against mucosal addressin cell adhesion molecule-1, given early in the course of disease progression. Immunology and cell biology. vol 78. issue 6. 2001-03-08. PMID:11114975. in the present paper, it is shown that an antibody directed against madcam-1, the preferential ligand for alpha4beta7, effectively prevented the development of a progressive, non-remitting, form of eae, actively induced by injection of myelin oligodendrocyte glycoprotein peptide (mog(35-55)) autoantigen. 2001-03-08 2023-08-12 Not clear
B N Ditte. Evidence that Fas and FasL contribute to the pathogenesis of experimental autoimmune encephalomyelitis. Archivum immunologiae et therapiae experimentalis. vol 48. issue 5. 2001-03-08. PMID:11140465. active immunization of mice with several different protein components of myelin, including myelin basic protein (mbp), proteolipid protein (plp) and myelin oligodendrocyte glycoprotein (mog), are capable of eliciting an immune response resulting in the quintessential symptoms of eae: ascending paralysis involving the tail and then the limbs. 2001-03-08 2023-08-12 mouse