| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Y Oshida, Y Tachi, Y Morishita, K Kitakoshi, N Fuku, Y Q Han, I Ohsawa, Y Sat. Nitric oxide decreases insulin resistance induced by high-fructose feeding. Hormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme. vol 32. issue 9. 2001-01-26. PMID:11014380. |
therefore it could be concluded that no can improve insulin resistance induced by high-fructose feeding. |
2001-01-26 |
2023-08-12 |
rat |
| C A Williams, M F Shih, P V Taberne. Effect of acute and sub-chronic administration of the imidazoline compound S 22068 on in vivo glucose and insulin responses in normal lean CBA/Ca mice. General pharmacology. vol 34. issue 3. 2001-01-25. PMID:11120380. |
sub-chronic treatment with s 22068 did not alter body weight (bw) or food intake, and resulted in tolerance to its effects on glucose metabolism and insulin sensitivity. |
2001-01-25 |
2023-08-12 |
mouse |
| X J Cai, M L Evans, C A Lister, R A Leslie, J R Arch, S Wilson, G William. Hypoglycemia activates orexin neurons and selectively increases hypothalamic orexin-B levels: responses inhibited by feeding and possibly mediated by the nucleus of the solitary tract. Diabetes. vol 50. issue 1. 2001-01-25. PMID:11147774. |
insulin (50 u/kg) lowered plasma glucose by >50% after 5 h and stimulated feeding sixfold compared with saline-injected controls. |
2001-01-25 |
2023-08-12 |
rat |
| M C Père, M Etienne, J Y Dourma. Adaptations of glucose metabolism in multiparous sows: effects of pregnancy and feeding level. Journal of animal science. vol 78. issue 11. 2001-01-02. PMID:11063319. |
the higher feeding level increased plasma insulin and lowered plasma glucose levels. |
2001-01-02 |
2023-08-12 |
Not clear |
| A T Cheung, B Dayanandan, J T Lewis, G S Korbutt, R V Rajotte, M Bryer-Ash, M O Boylan, M M Wolfe, T J Kieffe. Glucose-dependent insulin release from genetically engineered K cells. Science (New York, N.Y.). vol 290. issue 5498. 2000-12-22. PMID:11110661. |
genetic engineering of non-beta cells to release insulin upon feeding could be a therapeutic modality for patients with diabetes. |
2000-12-22 |
2023-08-12 |
mouse |
| L Baggio, F Adatia, T Bock, P L Brubaker, D J Drucke. Sustained expression of exendin-4 does not perturb glucose homeostasis, beta-cell mass, or food intake in metallothionein-preproexendin transgenic mice. The Journal of biological chemistry. vol 275. issue 44. 2000-12-14. PMID:10952981. |
activation of glucagon-like peptide (glp)-1 receptor signaling promotes glucose lowering via multiple mechanisms, including regulation of food intake, glucose-dependent insulin secretion, and stimulation of beta-cell mass. |
2000-12-14 |
2023-08-12 |
mouse |
| M Díaz-Muñoz, O Vázquez-Martínez, R Aguilar-Roblero, C Escoba. Anticipatory changes in liver metabolism and entrainment of insulin, glucagon, and corticosterone in food-restricted rats. American journal of physiology. Regulatory, integrative and comparative physiology. vol 279. issue 6. 2000-12-14. PMID:11080068. |
the present study characterizes the dynamics of circulating corticosterone, insulin, and glucagon and regulatory parameters of liver metabolism in rats under restricted feeding schedules. |
2000-12-14 |
2023-08-12 |
rat |
| J Y Kim, L A Nolte, P A Hansen, D H Han, K Ferguson, P A Thompson, J O Hollosz. High-fat diet-induced muscle insulin resistance: relationship to visceral fat mass. American journal of physiology. Regulatory, integrative and comparative physiology. vol 279. issue 6. 2000-12-14. PMID:11080069. |
in this study we used a number of interventions: fish oil, leptin, caloric restriction, and shorter duration of fat feeding, to try to disassociate an increase in visceral fat from muscle insulin resistance. |
2000-12-14 |
2023-08-12 |
Not clear |
| M J Reed, K Meszaros, L J Entes, M D Claypool, J G Pinkett, T M Gadbois, G M Reave. A new rat model of type 2 diabetes: the fat-fed, streptozotocin-treated rat. Metabolism: clinical and experimental. vol 49. issue 11. 2000-12-14. PMID:11092499. |
plasma insulin concentrations in response to oral glucose (2 g/kg) were increased 2-fold by fat feeding (p < .01), and adipocyte glucose clearance under maximal insulin stimulation was significantly reduced (p < .001), suggesting that fat feeding induced insulin resistance. |
2000-12-14 |
2023-08-12 |
human |
| A Widjaja, J C Levy, R J Morris, K N Frayn, S M Humphreys, R Horn, A von zur Mühlen, R C Turner, G Braban. Determinants of within-subject variation of fasting serum leptin concentrations in healthy subjects. Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association. vol 108. issue 3. 2000-11-30. PMID:10926318. |
the aim of the present study was 1) to assess the day-to-day within-subject variation (biovariability) of serum leptin concentrations in healthy subjects and 2) to investigate whether this variation is associated with food intake, exercise, anthropometric measurements or various metabolic covariates (insulin, c-peptide and glucagon, glucose, lactate, 3-hydroxybutyrate (3-ohb), triglycerides, non-esterified-fatty acids and glycerol). |
2000-11-30 |
2023-08-12 |
human |
| O Gavrilova, B Marcus-Samuels, M L Reitma. Lack of responses to a beta3-adrenergic agonist in lipoatrophic A-ZIP/F-1 mice. Diabetes. vol 49. issue 11. 2000-11-30. PMID:11078459. |
other acute effects include decreased gastrointestinal motility and food intake and increased insulin secretion. |
2000-11-30 |
2023-08-12 |
mouse |
| H Makimura, T M Mizuno, J Roberts, J Silverstein, J Beasley, C V Mobb. Adrenalectomy reverses obese phenotype and restores hypothalamic melanocortin tone in leptin-deficient ob/ob mice. Diabetes. vol 49. issue 11. 2000-11-30. PMID:11078460. |
in ob/ob mice, adrenalectomy reduced the levels of plasma glucose, serum insulin and corticosterone, and food intake toward or below wild-type levels, and it restored hypothalamic pomc and agrp mrna but not npy mrna to wild-type levels. |
2000-11-30 |
2023-08-12 |
mouse |
| A Chicco, J C Basabe, L Karabatas, N Ferraris, A Fortino, Y B Lombard. Troglitazone (CS-045) normalizes hypertriglyceridemia and restores the altered patterns of glucose-stimulated insulin secretion in dyslipidemic rats. Metabolism: clinical and experimental. vol 49. issue 10. 2000-11-28. PMID:11079827. |
the aim of the present study is to investigate whether the hypoglycemic agent troglitazone introduced as a pharmacologic intervention could improve and/or reverse the whole-body insulin insensitivity and related abnormalities present after feeding normal rats with a srd long-term. |
2000-11-28 |
2023-08-12 |
rat |
| T Ikenoue, N Kond. [Pharmacological properties of nateglinide, rapid-onset/short-duration insulinotropic agent, in the treatment of type 2 diabetes]. Nihon yakurigaku zasshi. Folia pharmacologica Japonica. vol 116. issue 3. 2000-11-24. PMID:11031747. |
an early defect in type 2 diabetes is the loss of acute insulin release after food intake, which causes prolonged elevation of postprandial glucose levels. |
2000-11-24 |
2023-08-12 |
Not clear |
| B Ahré. Diurnal variation in circulating leptin is dependent on gender, food intake and circulating insulin in mice. Acta physiologica Scandinavica. vol 169. issue 4. 2000-11-07. PMID:10951124. |
diurnal variation in circulating leptin is dependent on gender, food intake and circulating insulin in mice. |
2000-11-07 |
2023-08-12 |
mouse |
| B Ahré. Diurnal variation in circulating leptin is dependent on gender, food intake and circulating insulin in mice. Acta physiologica Scandinavica. vol 169. issue 4. 2000-11-07. PMID:10951124. |
we examined the influences of gender, feeding state, and plasma insulin and glucose on the diurnal rhythm in normal mice. |
2000-11-07 |
2023-08-12 |
mouse |
| B Ahré. Diurnal variation in circulating leptin is dependent on gender, food intake and circulating insulin in mice. Acta physiologica Scandinavica. vol 169. issue 4. 2000-11-07. PMID:10951124. |
it is suggested that the nocturnal rise in leptin shows gender dependency and is caused by the increase in plasma insulin caused by food intake. |
2000-11-07 |
2023-08-12 |
mouse |
| S Feurté, S Nicolaïdis, K Gerozissi. Is the early increase in leptinemia one of the anorectic signals induced by an essential amino acid-deficient diet in the rat? Endocrinology. vol 141. issue 10. 2000-10-31. PMID:11014250. |
because many hormones regulate feeding behavior, we studied the effect of a meal (46 kj) that was either devoid of threonine or was corrected for the deficiency, on plasma leptin, insulin and glucagon levels using a radio-immunoassay, at 0 to 180 min after the meal. |
2000-10-31 |
2023-08-12 |
rat |
| G Widdup, J M Bryson, D Pawlak, J L Phuyal, G S Denyer, I D Caterso. In vivo and in vitro suppression by leptin of glucose-stimulated insulin hypersecretion in high glucose-fed rats. European journal of endocrinology. vol 143. issue 3. 2000-10-26. PMID:11022188. |
chronic feeding to rats of high glycaemic index (gi) diets results in the hypersecretion of insulin in response to an i.v. |
2000-10-26 |
2023-08-12 |
rat |
| M Senn, S Gross-Lüem, H Leuenberger, W Langhan. Meal patterns and meal-induced metabolic changes in calves fed milk ad lib. Physiology & behavior. vol 70. issue 1-2. 2000-10-23. PMID:10978495. |
the feeding behavior of 11 calves fed milk ad lib was characterized and analyzed at the age of 5 weeks, and the short-term changes in the plasma concentrations of various metabolites (glucose, lactate, free fatty acids, triglycerides, beta-hydroxybutyrate) and insulin in relation to a representative spontaneous milk meal were measured during the following week. |
2000-10-23 |
2023-08-12 |
Not clear |