| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| C Lavigne, F Tremblay, G Asselin, H Jacques, A Marett. Prevention of skeletal muscle insulin resistance by dietary cod protein in high fat-fed rats. American journal of physiology. Endocrinology and metabolism. vol 281. issue 1. 2001-07-12. PMID:11404223. |
however, feeding cod protein fully prevented the development of insulin resistance in high fat-fed rats. |
2001-07-12 |
2023-08-12 |
rat |
| C Lavigne, F Tremblay, G Asselin, H Jacques, A Marett. Prevention of skeletal muscle insulin resistance by dietary cod protein in high fat-fed rats. American journal of physiology. Endocrinology and metabolism. vol 281. issue 1. 2001-07-12. PMID:11404223. |
these data demonstrate that feeding cod protein prevents obesity-induced muscle insulin resistance in high fat-fed obese rats at least in part through a direct action of amino acids on insulin-stimulated glucose uptake in skeletal muscle cells. |
2001-07-12 |
2023-08-12 |
rat |
| S Bi, E E Ladenheim, G J Schwartz, T H Mora. A role for NPY overexpression in the dorsomedial hypothalamus in hyperphagia and obesity of OLETF rats. American journal of physiology. Regulatory, integrative and comparative physiology. vol 281. issue 1. 2001-07-12. PMID:11404301. |
pair feeding oletf rats prevented their increased body weight and elevated levels of plasma insulin and leptin and normalized their elevated pomc and decreased npy mrna expression in the arcuate nucleus. |
2001-07-12 |
2023-08-12 |
rat |
| M Orosco, K Gerozissi. Macronutrient-induced cascade of events leading to parallel changes in hypothalamic serotonin and insulin. Neuroscience and biobehavioral reviews. vol 25. issue 2. 2001-07-05. PMID:11323081. |
this central event may be an important step in a cascade of events triggered by macronutrient ingestion leading to common hypothalamic insulin and 5-ht changes involved in feeding regulation. |
2001-07-05 |
2023-08-12 |
Not clear |
| B O Eijnde, E A Richter, J C Henquin, B Kiens, P Hespe. Effect of creatine supplementation on creatine and glycogen content in rat skeletal muscle. Acta physiologica Scandinavica. vol 171. issue 2. 2001-07-05. PMID:11350277. |
furthermore, plasma insulin concentration was measured in normal rats during creatine feeding, as well as in anaesthetized rats during intravenous creatine infusion. |
2001-07-05 |
2023-08-12 |
rat |
| B O Eijnde, E A Richter, J C Henquin, B Kiens, P Hespe. Effect of creatine supplementation on creatine and glycogen content in rat skeletal muscle. Acta physiologica Scandinavica. vol 171. issue 2. 2001-07-05. PMID:11350277. |
furthermore, high dose creatine feeding raised (p < 0.05) plasma creatine concentration fivefold but did not alter circulating insulin level. |
2001-07-05 |
2023-08-12 |
rat |
| J Wang, V G Yuen, J H McNeil. Effect of vanadium on insulin sensitivity and appetite. Metabolism: clinical and experimental. vol 50. issue 6. 2001-07-05. PMID:11398143. |
bmov treatment significantly reduced food intake, body fat, body weight, plasma insulin levels, and glucose levels in fatty zucker rats. |
2001-07-05 |
2023-08-12 |
rat |
| K Kawanaka, L A Nolte, D H Han, P A Hansen, J O Hollosz. Mechanisms underlying impaired GLUT-4 translocation in glycogen-supercompensated muscles of exercised rats. American journal of physiology. Endocrinology and metabolism. vol 279. issue 6. 2001-06-28. PMID:11093919. |
we previously found that feeding rats a high-carbohydrate diet after exercise, with muscle glycogen supercompensation, results in a decrease in insulin responsiveness so severe that it masks the effect of a training-induced twofold increase in glut-4 on insulin-stimulated muscle glucose transport. |
2001-06-28 |
2023-08-12 |
rat |
| K Ebihara, Y Ogawa, H Masuzaki, M Shintani, F Miyanaga, M Aizawa-Abe, T Hayashi, K Hosoda, G Inoue, Y Yoshimasa, O Gavrilova, M L Reitman, K Naka. Transgenic overexpression of leptin rescues insulin resistance and diabetes in a mouse model of lipoatrophic diabetes. Diabetes. vol 50. issue 6. 2001-06-28. PMID:11375346. |
pair-feeding the a-ziptg/+ mice to the amount of food consumed by leptg/+:a-ziptg/+ mice did not improve their insulin resistance, diabetes, or hepatic steatosis, demonstrating that the beneficial effects of leptin were not due to the decreased food intake. |
2001-06-28 |
2023-08-12 |
mouse |
| A Kalsbeek, E Fliers, J A Romijn, S E La Fleur, J Wortel, O Bakker, E Endert, R M Buij. The suprachiasmatic nucleus generates the diurnal changes in plasma leptin levels. Endocrinology. vol 142. issue 6. 2001-06-21. PMID:11356719. |
at present it is not clear which factors are responsible for the diurnal pattern of plasma leptin levels, although the timing of food intake and circulating hormones such as glucocorticoids and insulin have both been proposed as independent determinants. |
2001-06-21 |
2023-08-12 |
Not clear |
| A Kalsbeek, E Fliers, J A Romijn, S E La Fleur, J Wortel, O Bakker, E Endert, R M Buij. The suprachiasmatic nucleus generates the diurnal changes in plasma leptin levels. Endocrinology. vol 142. issue 6. 2001-06-21. PMID:11356719. |
as neither rhythmic feeding, insulin, or corticosterone signals can completely explain the diurnal plasma leptin rhythm, we conclude that biological clock control of the sympathetic input to the adipocyte is essential for regulation of the daily rhythm in leptin release. |
2001-06-21 |
2023-08-12 |
Not clear |
| F Santilli, S Greene, F Chiarell. Diabetes mellitus in the pre-school child. Minerva pediatrica. vol 52. issue 12. 2001-06-14. PMID:11268929. |
management of diabetes in the pre-school child may result very difficult for both parents and health carers because of the erratic daily pattern of activity, sleep and feeding; however, with a cautious strategy which involves insulin therapy, diet and monitoring it is possible to achieve satisfactorily the following aims: physical well-being of the young child, normal growth, lack of hyperglycaemia or hypoglycaemia, acceptable value of glycosilated haemoglobin. |
2001-06-14 |
2023-08-12 |
Not clear |
| R J Bloomer, G A Sforzo, B A Kelle. Effects of meal form and composition on plasma testosterone, cortisol, and insulin following resistance exercise. International journal of sport nutrition and exercise metabolism. vol 10. issue 4. 2001-06-07. PMID:11099368. |
the purpose of this study was to examine the effects of postexercise feeding on plasma levels of insulin, testosterone, cortisol, and testosterone:cortisol (t:c). |
2001-06-07 |
2023-08-12 |
human |
| R J Bloomer, G A Sforzo, B A Kelle. Effects of meal form and composition on plasma testosterone, cortisol, and insulin following resistance exercise. International journal of sport nutrition and exercise metabolism. vol 10. issue 4. 2001-06-07. PMID:11099368. |
in conclusion, the efficacy of postexercise feeding for optimizing t:c and muscle growth is unclear; however, consumption of sd appears to maximize circulating insulin for several hours following resistance exercise. |
2001-06-07 |
2023-08-12 |
human |
| R Maron, M Guerau-de-Arellano, X Zhang, H L Weine. Oral administration of insulin to neonates suppresses spontaneous and cyclophosphamide induced diabetes in the NOD mouse. Journal of autoimmunity. vol 16. issue 1. 2001-06-07. PMID:11221993. |
our findings that neonatal feeding of human insulin or insulin b-chain peptide is effective in inhibiting diabetes when given to the nod mouse may have applications in preventing diabetes in high risk human populations. |
2001-06-07 |
2023-08-12 |
mouse |
| T H Mora. Cholecystokinin and satiety: current perspectives. Nutrition (Burbank, Los Angeles County, Calif.). vol 16. issue 10. 2001-05-31. PMID:11054590. |
recent work has uncovered roles for the cck satiety pathway in the mediation of the feeding modulatory actions of estradiol, insulin, and leptin. |
2001-05-31 |
2023-08-12 |
rat |
| S C Woods, R J Seele. Adiposity signals and the control of energy homeostasis. Nutrition (Burbank, Los Angeles County, Calif.). vol 16. issue 10. 2001-05-31. PMID:11054594. |
insulin and leptin are the best known of these adiposity signals, and the administration of either into the brain causes a dose-dependent reduction of both food intake and body weight. |
2001-05-31 |
2023-08-12 |
Not clear |
| D B Pawlak, J M Bryson, G S Denyer, J C Brand-Mille. High glycemic index starch promotes hypersecretion of insulin and higher body fat in rats without affecting insulin sensitivity. The Journal of nutrition. vol 131. issue 1. 2001-05-31. PMID:11208944. |
in rats, prolonged feeding of high glycemic index (gi) starch results in basal hyperinsulinemia and an elevated insulin response to an intravenous glucose tolerance test (ivgtt). |
2001-05-31 |
2023-08-12 |
rat |
| D B Pawlak, J M Bryson, G S Denyer, J C Brand-Mille. High glycemic index starch promotes hypersecretion of insulin and higher body fat in rats without affecting insulin sensitivity. The Journal of nutrition. vol 131. issue 1. 2001-05-31. PMID:11208944. |
high gi and high fat feeding both resulted in the higher insulin response during ivgtt, but impaired glucose tolerance was seen only in rats fed high fat. |
2001-05-31 |
2023-08-12 |
rat |
| S G Kim, E J Kim, Y G Kim, M G Le. Expression of cytochrome P-450s and glutathione S-transferases in the rat liver during water deprivation: effects of glucose supplementation. Journal of applied toxicology : JAT. vol 21. issue 2. 2001-05-31. PMID:11288135. |
glucose feeding instead of foods during dehydration prevented p-450 2e1 induction in the absence of recovering the plasma insulin level. |
2001-05-31 |
2023-08-12 |
rat |