All Relations between feeding and hypothalamus

Publication Sentence Publish Date Extraction Date Species
Shigetomo Suyama, Eisuke Takano, Yusaku Iwasaki, Masanori Nakata, Toshihiko Yad. [Roles and functional interplay of the gut, brain stem, hypothalamus and limbic system in regulation of feeding]. Nihon rinsho. Japanese journal of clinical medicine. vol 67. issue 2. 2009-04-21. PMID:19202900. we here review how the hippocampus and amygdala as well as gut, brain stem and hypothalamus function and interact with each other to achieve integrative regulation of feeding. 2009-04-21 2023-08-12 Not clear
Stefany D Primeaux, Christine Blackmon, Maria J Barnes, H Douglas Braymer, George A Bra. Central administration of the RFamide peptides, QRFP-26 and QRFP-43, increases high fat food intake in rats. Peptides. vol 29. issue 11. 2009-04-13. PMID:18765262. in rodents, prepro-qrfp mrna is expressed in localized regions of the mediobasal hypothalamus, a region implicated in feeding behavior. 2009-04-13 2023-08-12 mouse
Luca Passamonti, James B Rowe, Christian Schwarzbauer, Michael P Ewbank, Elisabeth von dem Hagen, Andrew J Calde. Personality predicts the brain's response to viewing appetizing foods: the neural basis of a risk factor for overeating. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 29. issue 1. 2009-04-08. PMID:19129383. animal research has identified the ventral striatum, amygdala, hypothalamus, medial prefrontal and premotor cortices as key interacting structures for feeding. 2009-04-08 2023-08-12 human
Tohru Miyata, Yuji Minai, Minoru Hag. Association of the suckling-weaning transition with development of the hypothalamic arcuate nucleus neurons in rat pups. Experimental animals. vol 58. issue 1. 2009-04-07. PMID:19151514. feeding behavior is regulated by feeding-related peptides in the hypothalamus. 2009-04-07 2023-08-12 rat
M D Lane, M Wolfgang, S-H Cha, Y Da. Regulation of food intake and energy expenditure by hypothalamic malonyl-CoA. International journal of obesity (2005). vol 32 Suppl 4. 2009-04-06. PMID:18719599. stereotactic delivery of a viral malonyl-coa decarboxylase (mcd) vector into the ventral hypothalamus lowers malonyl-coa and increases food intake. 2009-04-06 2023-08-12 mouse
D Carling, M J Sanders, A Wood. The regulation of AMP-activated protein kinase by upstream kinases. International journal of obesity (2005). vol 32 Suppl 4. 2009-04-06. PMID:18719600. recent studies have demonstrated that modulation of ampk activity in the hypothalamus plays a role in feeding. 2009-04-06 2023-08-12 Not clear
Hiroshi Higuchi, Takeshi Niki, Tomohiro Shiiy. Feeding behavior and gene expression of appetite-related neuropeptides in mice lacking for neuropeptide Y Y5 receptor subclass. World journal of gastroenterology. vol 14. issue 41. 2009-04-03. PMID:19009645. rt-pcr and ish suggested that the compensation of feeding is due to change in gene expressions of agrp, cart and pomc in hypothalamus. 2009-04-03 2023-08-12 mouse
Derek Leroit. Foreword. Control of feeding behavior and the peripheral metabolism by the hypothalamus. Endocrinology and metabolism clinics of North America. vol 37. issue 4. 2009-04-02. PMID:19026931. control of feeding behavior and the peripheral metabolism by the hypothalamus. 2009-04-02 2023-08-12 Not clear
L A Kuehn, D J Nonneman, J M Klindt, T H Wis. Genetic relationships of body composition, serum leptin, and age at puberty in gilts. Journal of animal science. vol 87. issue 2. 2009-04-01. PMID:18849393. leptin produced by adipocytes acts through leptin receptors in the hypothalamus to control appetite and food intake and thus communicates information about degree of fatness. 2009-04-01 2023-08-12 Not clear
Paula Mera, Assia Bentebibel, Eduardo López-Viñas, Antonio G Cordente, Chandrashekaran Gurunathan, David Sebastián, Irene Vázquez, Laura Herrero, Xavier Ariza, Paulino Gómez-Puertas, Guillermina Asins, Dolors Serra, Jordi García, Fausto G Hegard. C75 is converted to C75-CoA in the hypothalamus, where it inhibits carnitine palmitoyltransferase 1 and decreases food intake and body weight. Biochemical pharmacology. vol 77. issue 6. 2009-04-01. PMID:19094968. c75 is converted to c75-coa in the hypothalamus, where it inhibits carnitine palmitoyltransferase 1 and decreases food intake and body weight. 2009-04-01 2023-08-12 rat
Paula Mera, Assia Bentebibel, Eduardo López-Viñas, Antonio G Cordente, Chandrashekaran Gurunathan, David Sebastián, Irene Vázquez, Laura Herrero, Xavier Ariza, Paulino Gómez-Puertas, Guillermina Asins, Dolors Serra, Jordi García, Fausto G Hegard. C75 is converted to C75-CoA in the hypothalamus, where it inhibits carnitine palmitoyltransferase 1 and decreases food intake and body weight. Biochemical pharmacology. vol 77. issue 6. 2009-04-01. PMID:19094968. our results suggest that inhibition of cpt1, and thus increased availability of fatty acids in the hypothalamus, contribute to the pharmacological mechanism of c75 to decrease food intake. 2009-04-01 2023-08-12 rat
Li Shen, Michael J Keenan, Roy J Martin, Richard T Tulley, Anne M Raggio, Kathleen L McCutcheon, Jun Zho. Dietary resistant starch increases hypothalamic POMC expression in rats. Obesity (Silver Spring, Md.). vol 17. issue 1. 2009-03-24. PMID:18948970. this study investigates the effect of feeding rs on the neuropeptide messenger rna (mrna) expressions in the arcuate nucleus (arc) of the hypothalamus and whether vagal afferent nerves are involved. 2009-03-24 2023-08-12 rat
Kevin W Williams, Michael M Scott, Joel K Elmquis. From observation to experimentation: leptin action in the mediobasal hypothalamus. The American journal of clinical nutrition. vol 89. issue 3. 2009-03-18. PMID:19176744. the discovery that leptin, an adipose-derived hormone, acts within the mediobasal hypothalamus to control food intake and energy expenditure ushered in a decade of research that went on to describe not only the specific nuclei and cell type, such as proopiomelanocortin neurons of the arcuate nucleus, that respond to leptin but also the signaling cascades that mediated its effects. 2009-03-18 2023-08-12 Not clear
Christy L White, Amy Whittington, Maria J Barnes, Zhong Wang, George A Bray, Christopher D Morriso. HF diets increase hypothalamic PTP1B and induce leptin resistance through both leptin-dependent and -independent mechanisms. American journal of physiology. Endocrinology and metabolism. vol 296. issue 2. 2009-02-27. PMID:19017730. here we report a significant increase in ptp1b protein levels in the mediobasal hypothalamus (p = 0.003) and a concomitant reduction in leptin sensitivity following 28 days of high-fat (hf) feeding in rats. 2009-02-27 2023-08-12 mouse
J E de Leeuw van Weenen, L Hu, K Jansen-Van Zelm, M G de Vries, J T Tamsma, J A Romijn, H Pij. Four weeks high fat feeding induces insulin resistance without affecting dopamine release or gene expression patterns in the hypothalamus of C57Bl6 mice. Brain research. vol 1250. 2009-02-27. PMID:19028458. four weeks high fat feeding induces insulin resistance without affecting dopamine release or gene expression patterns in the hypothalamus of c57bl6 mice. 2009-02-27 2023-08-12 mouse
J E de Leeuw van Weenen, L Hu, K Jansen-Van Zelm, M G de Vries, J T Tamsma, J A Romijn, H Pij. Four weeks high fat feeding induces insulin resistance without affecting dopamine release or gene expression patterns in the hypothalamus of C57Bl6 mice. Brain research. vol 1250. 2009-02-27. PMID:19028458. we show here that short term high fat feeding does not affect dopaminergic neurotransmission in the hypothalamus, whereas it does impair insulin action. 2009-02-27 2023-08-12 mouse
Suresh Ambati, Jiuhua Duan, Yang-Ho Choi, Diane L Hartzell, Mary Anne Della-Fera, Clifton A Bail. ICV vs. VMH injection of leptin: comparative effects on hypothalamic gene expression. Behavioural brain research. vol 196. issue 2. 2009-02-24. PMID:18926857. leptin regulates feeding behavior and body weight by binding to its receptors localized in specific areas of the hypothalamus. 2009-02-24 2023-08-12 rat
Eva Tavares, Francisco J Miñan. Procalcitonin N-terminal peptide causes catabolic effects via the hypothalamus and prostaglandin-dependent pathways. Neuroendocrinology. vol 88. issue 4. 2009-02-19. PMID:18781060. we also show that intracerebroventricular administration of n-pct induced a marked neuronal activation in key thermoregulatory and feeding areas of the hypothalamus. 2009-02-19 2023-08-12 rat
Eva Tavares, Francisco J Miñan. Procalcitonin N-terminal peptide causes catabolic effects via the hypothalamus and prostaglandin-dependent pathways. Neuroendocrinology. vol 88. issue 4. 2009-02-19. PMID:18781060. results support the role of n-pct to the central control of feeding behavior and suggest that n-pct, acting probably through the eicosanoid cyclooxygenase pathway, may act as a signaling molecule in the hypothalamus by regulating the activity of anorexigenic neurons in the hypothalamus. 2009-02-19 2023-08-12 rat
Jinghong Shi, Jianqun Yan, Qi Lei, Junjie Zhao, Ke Chen, Dejun Yang, Xiaolin Zhao, Yuan Zhan. Intragastric administration of evodiamine suppresses NPY and AgRP gene expression in the hypothalamus and decreases food intake in rats. Brain research. vol 1247. 2009-02-19. PMID:18955035. intragastric administration of evodiamine suppresses npy and agrp gene expression in the hypothalamus and decreases food intake in rats. 2009-02-19 2023-08-12 rat