| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| T Hayashi, A Catanzaro, S P Ra. Apoptosis of human monocytes and macrophages by Mycobacterium avium sonicate. Infection and immunity. vol 65. issue 12. 1998-01-02. PMID:9393825. |
in both cases, the reduction in viability was due to apoptosis characterized by chromatin condensation, dna fragmentation by agarose gel electrophoresis, or terminal deoxynucleotidyl transferase-mediated d-utp nick end labeling (tunel) and release of nuclear matrix protein (nmp) into the culture medium. |
1998-01-02 |
2023-08-12 |
human |
| C J Narvaez, J Wels. Differential effects of 1,25-dihydroxyvitamin D3 and tetradecanoylphorbol acetate on cell cycle and apoptosis of MCF-7 cells and a vitamin D3-resistant variant. Endocrinology. vol 138. issue 11. 1997-11-24. PMID:9348195. |
in parental mcf-7 cells, 1,25-(oh)2d3 induced morphological and biochemical markers of apoptosis (chromatin and nuclear matrix condensation and dna fragmentation), whereas tpa induced growth arrest without apoptosis. |
1997-11-24 |
2023-08-12 |
Not clear |
| H Ono, Y On. Nephrosclerosis and hypertension. The Medical clinics of North America. vol 81. issue 6. 1997-11-19. PMID:9356598. |
experimental evidence supports the notion that the pathogenesis of glomerulosclerosis with nephrosclerosis has been demonstrated as important factors: (1) the elevation of pg (glomerular hypertension); (2) mesangial dysfunction, such as mesangiolysis and increased mesangial matrix; and (3) genetic abnormalities (apoptosis) in mesangial cells with glomerular hypertension. |
1997-11-19 |
2023-08-12 |
human |
| L P Wen, J A Fahrni, S Troie, J L Guan, K Orth, G D Rose. Cleavage of focal adhesion kinase by caspases during apoptosis. The Journal of biological chemistry. vol 272. issue 41. 1997-11-13. PMID:9325343. |
the interactions of integrins with the extracellular matrix activates focal adhesion kinase (fak) and suppresses apoptosis in diverse cell types. |
1997-11-13 |
2023-08-12 |
Not clear |
| A M Martelli, R Bareggi, R Bortul, V Grill, P Narducci, M Zweye. The nuclear matrix and apoptosis. Histochemistry and cell biology. vol 108. issue 1. 1997-11-07. PMID:9377219. |
the nuclear matrix and apoptosis. |
1997-11-07 |
2023-08-12 |
Not clear |
| A M Martelli, R Bareggi, R Bortul, V Grill, P Narducci, M Zweye. The nuclear matrix and apoptosis. Histochemistry and cell biology. vol 108. issue 1. 1997-11-07. PMID:9377219. |
recently, attention has been devoted to identifying the modifications that occur during apoptosis in the nuclear matrix, a mainly proteinaceous framework structure which is thought to play a fundamental role in organizing nuclear structure and function. |
1997-11-07 |
2023-08-12 |
Not clear |
| F A Van den Brûle, P L Fernandez, C Buicu, F T Liu, P Jackers, R Lambotte, V Castronov. Differential expression of galectin-1 and galectin-3 during first trimester human embryogenesis. Developmental dynamics : an official publication of the American Association of Anatomists. vol 209. issue 4. 1997-10-29. PMID:9264263. |
galectins are mammalian lectins able to interact with various extracellular matrix glycoconjugates and have been implicated in several biological events including cell attachment, differentiation, apoptosis, embryogenesis, and cancer invasion and metastasis. |
1997-10-29 |
2023-08-12 |
human |
| S Estus, H M Tucker, C van Rooyen, S Wright, E F Brigham, M Wogulis, R E Ryde. Aggregated amyloid-beta protein induces cortical neuronal apoptosis and concomitant "apoptotic" pattern of gene induction. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 17. issue 20. 1997-10-23. PMID:9315895. |
specifically, we (1) report the time-dependent and robust induction of c-jun, junb, c-fos, and fosb, as well as transin, which is induced by c-jun/c-fos heterodimers and encodes an extracellular matrix protease; these gene inductions appear to be selective because other jun and fos family members, i.e., jund and fra-1, are induced only marginally; (2) show that the c-jun induction is widespread, whereas c-fos expression is restricted to a subset of neurons, typically those with condensed chromatin, which is a hallmark of apoptosis; (3) correlate gene induction and neuronal death by showing that each has a similar dose-response to abeta; and (4) demonstrate that both cell death and immediate early gene induction are dependent on abeta aggregation state. |
1997-10-23 |
2023-08-12 |
rat |
| K Antoku, Z Liu, D E Johnso. Inhibition of caspase proteases by CrmA enhances the resistance of human leukemic cells to multiple chemotherapeutic agents. Leukemia. vol 11. issue 10. 1997-10-23. PMID:9324287. |
proteases that are members of the caspase (or interleukin-1beta converting enzyme (ice)) protease family have been shown to be important mediators of apoptosis induced by fas activation, neurotrophic factor withdrawal, and detachment from extracellular matrix. |
1997-10-23 |
2023-08-12 |
human |
| C Yang, S W Li, H J Helminen, J S Khillan, Y Bao, D J Procko. Apoptosis of chondrocytes in transgenic mice lacking collagen II. Experimental cell research. vol 235. issue 2. 1997-10-16. PMID:9299161. |
in the present work, we used the same mice to show that chondrocytes undergo apoptosis in the absence of collagen ii, the major component of the extracellular matrix of cartilage. |
1997-10-16 |
2023-08-12 |
mouse |
| S Leuenroth, E Isaacson, C Lee, H Keeping, H H Simm. Integrin regulation of polymorphonuclear leukocyte apoxis during hypoxia is primarily dependent on very late activation antigens 3 and 5. Surgery. vol 122. issue 2. 1997-10-02. PMID:9288118. |
the regulation of polymorphonuclear leukocyte (pmn) apoptosis during hypoxia has not been previously characterized, and we hypothesized that integrin signaling by matrix proteins (laminin) would regulate pmn apoptosis. |
1997-10-02 |
2023-08-12 |
Not clear |
| N Zamzami, T Hirsch, B Dallaporta, P X Petit, G Kroeme. Mitochondrial implication in accidental and programmed cell death: apoptosis and necrosis. Journal of bioenergetics and biomembranes. vol 29. issue 2. 1997-09-25. PMID:9239543. |
at a second level, the consequences of mitochondrial dysfunction (collapse of the mitochondrial transmembrane potential, uncoupling of the respiratory chain, hyperproduction of superoxide anions, disruption of mitochondrial biogenesis, outflow of matrix calcium and glutathione, and release of soluble intermembrane proteins) can entail a biogenetic catastrophe culminating in the disruption of plasma membrane integrity (necrosis) and/or the activation and action of apoptogenic proteases with secondary endonuclease activation and consequent oligonucleosomal dna fragmentation (apoptosis). |
1997-09-25 |
2023-08-12 |
Not clear |
| G McGill, A Shimamura, R C Bates, R E Savage, D E Fishe. Loss of matrix adhesion triggers rapid transformation-selective apoptosis in fibroblasts. The Journal of cell biology. vol 138. issue 4. 1997-09-15. PMID:9265655. |
loss of matrix adhesion triggers rapid transformation-selective apoptosis in fibroblasts. |
1997-09-15 |
2023-08-12 |
Not clear |
| T Hattori, N Fujitsuka, A Kurogi, S Shind. [Protective effect of onpi-to (TJ-8117) on the expression of apoptosis in 5/6 nephrectomized rats]. Nihon Jinzo Gakkai shi. vol 39. issue 4. 1997-09-08. PMID:9198359. |
these results suggest that tj-8117 may inhibit apoptosis in the late stage of this model by suppressing matrix accumulation and progression of glomerulosclerosis. |
1997-09-08 |
2023-08-12 |
rat |
| M H Cardone, G S Salvesen, C Widmann, G Johnson, S M Frisc. The regulation of anoikis: MEKK-1 activation requires cleavage by caspases. Cell. vol 90. issue 2. 1997-08-28. PMID:9244305. |
certain cell types undergo apoptosis when they lose integrin-mediated contacts with the extracellular matrix ("anoikis"). |
1997-08-28 |
2023-08-12 |
Not clear |
| S Farrier, M Morga. bcl-2 expression in pilomatricoma. The American Journal of dermatopathology. vol 19. issue 3. 1997-08-25. PMID:9185911. |
the normal hair matrix undergoes defined cycles of growth (anagen), regression (catagen), and resting (telogen) that are regulated by programmed cell death (apoptosis). |
1997-08-25 |
2023-08-12 |
Not clear |
| R Gniadecki, M Hansen, H C Wul. Two pathways for induction of apoptosis by ultraviolet radiation in cultured human keratinocytes. The Journal of investigative dermatology. vol 109. issue 2. 1997-08-20. PMID:9242502. |
this phenomenon depended on differences in integrin-mediated adhesion, because blocking of integrin beta1 with a monoclonal antibody increased sensitivity of keratinocyte monolayers to uv and an increase in beta1 integrin receptor occupancy by plating on fibronectin, type iv collagen, or keratinocyte-derived extracellular matrix diminished the uv-dependent apoptosis. |
1997-08-20 |
2023-08-12 |
human |
| M Stanulla, J Wang, D S Chervinsky, S Thandla, P D Apla. DNA cleavage within the MLL breakpoint cluster region is a specific event which occurs as part of higher-order chromatin fragmentation during the initial stages of apoptosis. Molecular and cellular biology. vol 17. issue 7. 1997-07-24. PMID:9199342. |
we also show that site-specific cleavage within the mll bcr can be linked to the higher-order chromatin fragmentation that occurs during the initial stages of apoptosis, possibly through cleavage of dna loops at their anchorage sites to the nuclear matrix. |
1997-07-24 |
2023-08-12 |
Not clear |
| H I Roac. New aspects of endochondral ossification in the chick: chondrocyte apoptosis, bone formation by former chondrocytes, and acid phosphatase activity in the endochondral bone matrix. Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research. vol 12. issue 5. 1997-07-16. PMID:9144346. |
new aspects of endochondral ossification in the chick: chondrocyte apoptosis, bone formation by former chondrocytes, and acid phosphatase activity in the endochondral bone matrix. |
1997-07-16 |
2023-08-12 |
chicken |
| R E Vilar, D Ghael, M Li, D D Bhagat, L M Arrigo, M K Cowman, H S Dweck, L Rosenfel. Nitric oxide degradation of heparin and heparan sulphate. The Biochemical journal. vol 324 ( Pt 2). 1997-07-08. PMID:9182706. |
these observations may be relevant to various pathophysiological processes in which extracellular matrix is degraded, such as bone development, apoptosis, tissue damage from inflammatory responses and possible release of growth factors and cytokines. |
1997-07-08 |
2023-08-12 |
Not clear |