| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Amedeo Anselmi, Marzia Lotrionte, Giuseppe G L Biondi-Zoccai, Leonarda Galiuto, Antonio Abbat. Left ventricular hypertrophy, apoptosis, and progression to heart failure in severe aortic stenosis. European heart journal. vol 26. issue 24. 2006-07-11. PMID:16246825. |
left ventricular hypertrophy, apoptosis, and progression to heart failure in severe aortic stenosis. |
2006-07-11 |
2023-08-12 |
Not clear |
| Tao Hang, Zhaoqi Huang, Shisen Jiang, Jianbin Gong, Chunhui Wang, Dujiang Xie, Hailing Re. Apoptosis in pressure overload-induced cardiac hypertrophy is mediated, in part, by adenine nucleotide translocator-1. Annals of clinical and laboratory science. vol 36. issue 1. 2006-07-06. PMID:16501242. |
apoptosis in pressure overload-induced cardiac hypertrophy is mediated, in part, by adenine nucleotide translocator-1. |
2006-07-06 |
2023-08-12 |
rat |
| Tao Hang, Zhaoqi Huang, Shisen Jiang, Jianbin Gong, Chunhui Wang, Dujiang Xie, Hailing Re. Apoptosis in pressure overload-induced cardiac hypertrophy is mediated, in part, by adenine nucleotide translocator-1. Annals of clinical and laboratory science. vol 36. issue 1. 2006-07-06. PMID:16501242. |
this study explored the role of adenine nucleotide translocator-1 (ant1) in cardiomyocyte apoptosis during left ventricular hypertrophy (lvh) that developed in response to pressure overload. |
2006-07-06 |
2023-08-12 |
rat |
| Nobuo Nagano, Sonoe Miyata, Megumi Abe, Sachiko Wakita, Nami Kobayashi, Michihito Wad. Sevelamer hydrochloride reverses parathyroid gland enlargement via regression of cell hypertrophy but not apoptosis in rats with chronic renal insufficiency. Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association. vol 21. issue 3. 2006-06-29. PMID:16169864. |
sevelamer hydrochloride reverses parathyroid gland enlargement via regression of cell hypertrophy but not apoptosis in rats with chronic renal insufficiency. |
2006-06-29 |
2023-08-12 |
rat |
| Takeshi Hori. [Pathophysiological role of cytokines in heart failure]. Nihon rinsho. Japanese journal of clinical medicine. vol 64. issue 5. 2006-06-15. PMID:16689363. |
in addition, tnf and interleukin induce myocardial apoptosis and promote cardiac hypertrophy and fibrosis, suggesting that these cytokines are involved in the progression of cardiac remodeling. |
2006-06-15 |
2023-08-12 |
Not clear |
| Shintaro Kinugawa, Hiroyuki Tsutsu. [Oxidative stress and heart failure]. Nihon rinsho. Japanese journal of clinical medicine. vol 64. issue 5. 2006-06-15. PMID:16689364. |
the excessive reactive oxygen species (ros), produced by the electron leak in mitochondria of failing myocardium, play an important role in development and progression of heart failure (hf) and cardiac remodeling, which is associated with induction of myocyte hypertrophy and apoptosis and activation of matrix metalloproteinases. |
2006-06-15 |
2023-08-12 |
Not clear |
| Lisa M Mielniczuk, Kenneth L Baughma. Immune modulation therapy in heart failure. Congestive heart failure (Greenwich, Conn.). vol 12. issue 2. 2006-06-13. PMID:16596043. |
several lines of evidence suggest that inflammation plays a pathogenic role in the development and progression of congestive heart failure, influencing heart contractility and hypertrophy, promoting apoptosis, and contributing to the myocardial remodeling process. |
2006-06-13 |
2023-08-12 |
Not clear |
| M P-S Lee, D Orlov, G Sweene. Leptin induces rat glomerular mesangial cell hypertrophy, but does not regulate hyperplasia or apoptosis. International journal of obesity (2005). vol 29. issue 12. 2006-06-09. PMID:15997243. |
leptin induces rat glomerular mesangial cell hypertrophy, but does not regulate hyperplasia or apoptosis. |
2006-06-09 |
2023-08-12 |
rat |
| M P-S Lee, D Orlov, G Sweene. Leptin induces rat glomerular mesangial cell hypertrophy, but does not regulate hyperplasia or apoptosis. International journal of obesity (2005). vol 29. issue 12. 2006-06-09. PMID:15997243. |
one of the major structural changes observed in nephropathy is the increase in kidney size, which may occur due to hypertrophy or changes in the rate of hyperplasia or apoptosis. |
2006-06-09 |
2023-08-12 |
rat |
| Shin-Da Lee, Sheng-Huang Chang, Wu-Hsien Kuo, Tsung-Ho Ying, Wei-Wen Kuo, Ping-Chun Li, Hsi-Hsien Hsu, Min-Chi Lu, Hua Ting, Chih-Yang Huan. Role of mitogen-activated protein kinase kinase in Porphyromonas gingivalis-induced myocardial cell hypertrophy and apoptosis. European journal of oral sciences. vol 114. issue 2. 2006-06-06. PMID:16630308. |
role of mitogen-activated protein kinase kinase in porphyromonas gingivalis-induced myocardial cell hypertrophy and apoptosis. |
2006-06-06 |
2023-08-12 |
Not clear |
| Shin-Da Lee, Sheng-Huang Chang, Wu-Hsien Kuo, Tsung-Ho Ying, Wei-Wen Kuo, Ping-Chun Li, Hsi-Hsien Hsu, Min-Chi Lu, Hua Ting, Chih-Yang Huan. Role of mitogen-activated protein kinase kinase in Porphyromonas gingivalis-induced myocardial cell hypertrophy and apoptosis. European journal of oral sciences. vol 114. issue 2. 2006-06-06. PMID:16630308. |
secreted factors present in the medium following growth of the periodontal pathogen porphyromonas gingivalis cause increased cardiomyocyte hypertrophy and apoptosis, whereas secreted factors from actinobacillus actinomycetemcomitans and prevotella intermedia have no such effects. |
2006-06-06 |
2023-08-12 |
Not clear |
| Shin-Da Lee, Sheng-Huang Chang, Wu-Hsien Kuo, Tsung-Ho Ying, Wei-Wen Kuo, Ping-Chun Li, Hsi-Hsien Hsu, Min-Chi Lu, Hua Ting, Chih-Yang Huan. Role of mitogen-activated protein kinase kinase in Porphyromonas gingivalis-induced myocardial cell hypertrophy and apoptosis. European journal of oral sciences. vol 114. issue 2. 2006-06-06. PMID:16630308. |
the purpose of this study was to clarify the role of mitogen-activated protein kinase (mapk)/extracellular-regulated protein kinase (erk) pathways in p. gingivalis medium-induced h9c2 myocardial cell hypertrophy and apoptosis. |
2006-06-06 |
2023-08-12 |
Not clear |
| Shin-Da Lee, Sheng-Huang Chang, Wu-Hsien Kuo, Tsung-Ho Ying, Wei-Wen Kuo, Ping-Chun Li, Hsi-Hsien Hsu, Min-Chi Lu, Hua Ting, Chih-Yang Huan. Role of mitogen-activated protein kinase kinase in Porphyromonas gingivalis-induced myocardial cell hypertrophy and apoptosis. European journal of oral sciences. vol 114. issue 2. 2006-06-06. PMID:16630308. |
the results suggest that p. gingivalis-secreted factors may initiate mek/erk signal pathways and lead to myocardial cell hypertrophy and apoptosis. |
2006-06-06 |
2023-08-12 |
Not clear |
| Yangxin Li, Yao-Hua Song, Jessica Mohler, Patrick Delafontain. ANG II induces apoptosis of human vascular smooth muscle via extrinsic pathway involving inhibition of Akt phosphorylation and increased FasL expression. American journal of physiology. Heart and circulatory physiology. vol 290. issue 5. 2006-06-01. PMID:16339840. |
ang ii induced apoptosis only in nonquiescent cells but stimulated hypertrophy in quiescent cells. |
2006-06-01 |
2023-08-12 |
human |
| Svitlana Demyanets, Christoph Kaun, Stefan Pfaffenberger, Philipp J Hohensinner, Gersina Rega, Johannes Pammer, Gerald Maurer, Kurt Huber, Johann Wojt. Hydroxymethylglutaryl-coenzyme A reductase inhibitors induce apoptosis in human cardiac myocytes in vitro. Biochemical pharmacology. vol 71. issue 9. 2006-05-22. PMID:16540096. |
we speculate that, similar to findings in animal models, statins might be involved in the attenuation of cardiac hypertrophy and remodeling in humans by modulating the balance between cell survival and apoptosis. |
2006-05-22 |
2023-08-12 |
human |
| Timothy D O'Connell, Philip M Swigart, M C Rodrigo, Shinji Ishizaka, Shuji Joho, Lynne Turnbull, Laurence H Tecott, Anthony J Baker, Elyse Foster, William Grossman, Paul C Simpso. Alpha1-adrenergic receptors prevent a maladaptive cardiac response to pressure overload. The Journal of clinical investigation. vol 116. issue 4. 2006-05-16. PMID:16585965. |
however, ko hearts after tac had increased interstitial fibrosis, increased apoptosis, and failed induction of the fetal hypertrophic genes. |
2006-05-16 |
2023-08-12 |
mouse |
| Sek Won Kong, Natalya Bodyak, Patrick Yue, Zhilin Liu, Jeffrey Brown, Seigo Izumo, Peter M Kan. Genetic expression profiles during physiological and pathological cardiac hypertrophy and heart failure in rats. Physiological genomics. vol 21. issue 1. 2006-05-11. PMID:15623566. |
it revealed significant alterations in genes involved in the apoptosis pathway during pathological hypertrophy, suggesting that the apoptosis pathway may play a role during the transition to heart failure. |
2006-05-11 |
2023-08-12 |
rat |
| Hsiu-Wen Chan, Nicola J Smith, Ross D Hannan, Walter G Thoma. Tackling the EGFR in pathological tissue remodelling. Pulmonary pharmacology & therapeutics. vol 19. issue 1. 2006-05-11. PMID:15979363. |
tissue remodelling is an adaptive physiological event initiated by physical and/or hormonal stimuli and characterised by extracellular matrix modifications, inflammation, cellular hypertrophy, proliferation and/or apoptosis. |
2006-05-11 |
2023-08-12 |
Not clear |
| Narasimman Gurusamy, Kenichi Watanabe, Meilei Ma, Paras Prakash, Kenichi Hirabayashi, Shaosong Zhang, Anthony J Muslin, Makoto Kodama, Yoshifusa Aizaw. Glycogen synthase kinase 3beta together with 14-3-3 protein regulates diabetic cardiomyopathy: effect of losartan and tempol. FEBS letters. vol 580. issue 8. 2006-05-04. PMID:16530186. |
glycogen synthase kinase (gsk) 3beta is a multifunctional protein that positively regulates myocardial apoptosis and negatively regulates hypertrophy. |
2006-05-04 |
2023-08-12 |
mouse |
| Gerald W Dor. Physiologic growth and pathologic genes in cardiac development and cardiomyopathy. Trends in cardiovascular medicine. vol 15. issue 5. 2006-05-03. PMID:16165015. |
here, studies of galphaq and nix, powerful signaling factors for pathologic hypertrophy and cardiomyocyte apoptosis, respectively, are reviewed in terms of their comparative effects on cardiac development and adult cardiac pathology. |
2006-05-03 |
2023-08-12 |
mouse |