| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| C Naujokat, O Sezer, K Possinge. Tumor necrosis factor-alpha and interferon-gamma induce expression of functional Fas ligand on HT29 and MCF7 adenocarcinoma cells. Biochemical and biophysical research communications. vol 264. issue 3. 1999-12-22. PMID:10544014. |
by contrast, mutational deletion of fas cell surface expression as well as inhibition of caspase family proteases involved in fas signaling and monoclonal antibodies neutralizing fasl protect jurkat t cells from apoptosis caused by the fasl-expressing ht29 or mcf7 cells. |
1999-12-22 |
2023-08-12 |
human |
| Sooryanarayana, N Prasad, E Bonnin, A Pashov, K Ben Jilani, J C Ameisen, M D Kazatchkine, S V Kaver. Phosphorylation of Bcl-2 and mitochondrial changes are associated with apoptosis of lymphoblastoid cells induced by normal immunoglobulin G. Biochemical and biophysical research communications. vol 264. issue 3. 1999-12-22. PMID:10544027. |
phosphorylation of bcl-2 and mitochondrial changes are associated with apoptosis of lymphoblastoid cells induced by normal immunoglobulin g. normal human immunoglobulin g induces apoptosis in human lymphoblastoid cells which involves antibody-mediated fas ligation and the activation of caspases. |
1999-12-22 |
2023-08-12 |
human |
| C E Jackson, J M Puc. Autoimmune lymphoproliferative syndrome, a disorder of apoptosis. Current opinion in pediatrics. vol 11. issue 6. 1999-12-22. PMID:10590910. |
this gene, previously known as apoptosis antigen 1 (apt1), encodes the cell surface receptor for the major apoptosis pathway in mature lymphocytes; this receptor has also had many names, including fas (to be used here), cd95, and apo-1. |
1999-12-22 |
2023-08-12 |
Not clear |
| M T Fernández-Figueras, P Armengol, L Puig, J L Molinero, M Esquius, G Sirera, A Ariz. Absence of Fas (CD95) and FasL (CD95L) immunohistochemical expression suggests Fas/FasL-mediated apoptotic signal is not relevant in cutaneous Kaposi's sarcoma lesions. Journal of cutaneous pathology. vol 26. issue 9. 1999-12-17. PMID:10563496. |
in order to evaluate the possible role of fas-mediated apoptosis in kaposi's sarcoma (ks), we have studied the immunocytochemical expression of fas and fasl in biopsy specimens showing different histopathological stages of classic ks (c-ks) and aids-associated ks (aids-ks), as well as in cultured cells derived from c-ks lesions. |
1999-12-17 |
2023-08-12 |
Not clear |
| J F Amara, N L Courage, M Gilma. Cell surface tagging and a suicide mechanism in a single chimeric human protein. Human gene therapy. vol 10. issue 16. 1999-12-16. PMID:10566892. |
treatment of engineered cells with a cell-permeable chemical dimerizing agent that simultaneously binds to two fkbp domains cross-links the chimeric fas protein and induces apoptosis. |
1999-12-16 |
2023-08-12 |
human |
| M Børset, H Hjorth-Hansen, A C Johnsen, C Seidel, A Waage, T Espevik, A Sunda. Apoptosis, proliferation and NF-kappaB activation induced by agonistic Fas antibodies in the human myeloma cell line OH-2: amplification of Fas-mediated apoptosis by tumor necrosis factor. European journal of haematology. vol 63. issue 5. 1999-12-15. PMID:10580567. |
apoptosis, proliferation and nf-kappab activation induced by agonistic fas antibodies in the human myeloma cell line oh-2: amplification of fas-mediated apoptosis by tumor necrosis factor. |
1999-12-15 |
2023-08-12 |
human |
| M Børset, H Hjorth-Hansen, A C Johnsen, C Seidel, A Waage, T Espevik, A Sunda. Apoptosis, proliferation and NF-kappaB activation induced by agonistic Fas antibodies in the human myeloma cell line OH-2: amplification of Fas-mediated apoptosis by tumor necrosis factor. European journal of haematology. vol 63. issue 5. 1999-12-15. PMID:10580567. |
however, in the presence of the agonistic fas antibody ch 11, tnf enhanced the level of apoptosis in cultures of the human myeloma cell line oh-2. |
1999-12-15 |
2023-08-12 |
human |
| H Kimura, H Minakami, I Sekiguchi, K Otsuki, A Shoj. Phorbol ester facilitates apoptosis in murine fibroblasts pretreated by mild ultraviolet radiation. Journal of biochemistry. vol 126. issue 2. 1999-12-13. PMID:10423527. |
exposure to pma alone did not induce fas, fas-l, or apoptosis. |
1999-12-13 |
2023-08-12 |
Not clear |
| H Kimura, H Minakami, I Sekiguchi, K Otsuki, A Shoj. Phorbol ester facilitates apoptosis in murine fibroblasts pretreated by mild ultraviolet radiation. Journal of biochemistry. vol 126. issue 2. 1999-12-13. PMID:10423527. |
cells exposed to mild uv-b irradiation (80 j/m(2)) alone exhibited a slight expression of fas and fas-l 36 to 48 h after the exposure, and exhibited apoptosis as evidenced by dna fragmentation 72 h after exposure. |
1999-12-13 |
2023-08-12 |
Not clear |
| S Irie, T Hachiya, S Rabizadeh, W Maruyama, J Mukai, Y Li, J C Reed, D E Bredesen, T A Sat. Functional interaction of Fas-associated phosphatase-1 (FAP-1) with p75(NTR) and their effect on NF-kappaB activation. FEBS letters. vol 460. issue 2. 1999-12-10. PMID:10544233. |
fap-1 (fas-associated phosphatase-1), which binds to the cytoplasmic tail of fas, was originally identified as a negative regulator of fas-mediated apoptosis. |
1999-12-10 |
2023-08-12 |
Not clear |
| J H Richburg, A Nañez, H Ga. Participation of the Fas-signaling system in the initiation of germ cell apoptosis in young rat testes after exposure to mono-(2-ethylhexyl) phthalate. Toxicology and applied pharmacology. vol 160. issue 3. 1999-12-09. PMID:10544061. |
the fas-signaling system is composed of the interacting proteins fas (cd95/apo-1) and fas ligand (fasl, cd95l, apo-1l) and is proposed to act in the testis as a paracrine signaling mechanism by which fasl-expressing sertoli cells initiate apoptosis of fas-bearing germ cells. |
1999-12-09 |
2023-08-12 |
rat |
| J D Mountz, H G Zhang, H C Hsu, M Fleck, J Wu, M H al-Maini, T Zho. Apoptosis and cell death in the endocrine system. Recent progress in hormone research. vol 54. 1999-12-08. PMID:10548879. |
more recently, these and other autoimmune diseases have been associated with defects in fas apoptosis. |
1999-12-08 |
2023-08-12 |
mouse |
| J D Mountz, H G Zhang, H C Hsu, M Fleck, J Wu, M H al-Maini, T Zho. Apoptosis and cell death in the endocrine system. Recent progress in hormone research. vol 54. 1999-12-08. PMID:10548879. |
however, dysfunction of the fas apoptosis signaling pathway or production of soluble factors, including sfas and sfasl, may be more prevalent. |
1999-12-08 |
2023-08-12 |
mouse |
| J D Mountz, H G Zhang, H C Hsu, M Fleck, J Wu, M H al-Maini, T Zho. Apoptosis and cell death in the endocrine system. Recent progress in hormone research. vol 54. 1999-12-08. PMID:10548879. |
therefore, there are many potential mechanisms by which endocrine glands or hormones can affect the fas apoptosis pathway, resulting in either cell death or a chronic inflammatory disease in the endocrine system, leading to hypothyroidism and diabetes. |
1999-12-08 |
2023-08-12 |
mouse |
| Y Gazit. TRAIL is a potent inducer of apoptosis in myeloma cells derived from multiple myeloma patients and is not cytotoxic to hematopoietic stem cells. Leukemia. vol 13. issue 11. 1999-12-07. PMID:10557057. |
furthermore, we provide first time evidence that similar to fas, trail-induced apoptosis is not blocked by bcl-2 in mm cell lines. |
1999-12-07 |
2023-08-12 |
Not clear |
| T A Bremner, D Chatterjee, Z Han, M F Tsan, J H Wych. THP-1 monocytic leukemia cells express Fas ligand constitutively and kill Fas-positive Jurkat cells. Leukemia research. vol 23. issue 10. 1999-12-07. PMID:10573130. |
normal human monocytes express fas and are susceptible to fas ligand (fasl)-induced apoptosis. |
1999-12-07 |
2023-08-12 |
human |
| J A Houghto. Apoptosis and drug response. Current opinion in oncology. vol 11. issue 6. 1999-12-03. PMID:10550011. |
death receptors for fasl (fas) and for trail (dr4, dr5) are emerging as important regulators of drug-induced apoptosis in human cancers, mediated by caspase activation. |
1999-12-03 |
2023-08-12 |
human |
| K M Murphy, U N Streips, R B Loc. Bax membrane insertion during Fas(CD95)-induced apoptosis precedes cytochrome c release and is inhibited by Bcl-2. Oncogene. vol 18. issue 44. 1999-12-03. PMID:10557088. |
bax membrane insertion during fas(cd95)-induced apoptosis precedes cytochrome c release and is inhibited by bcl-2. |
1999-12-03 |
2023-08-12 |
Not clear |
| K M Murphy, U N Streips, R B Loc. Bax membrane insertion during Fas(CD95)-induced apoptosis precedes cytochrome c release and is inhibited by Bcl-2. Oncogene. vol 18. issue 44. 1999-12-03. PMID:10557088. |
ligation of the fas cell surface receptor leads to activation of caspases and subsequent apoptosis. |
1999-12-03 |
2023-08-12 |
Not clear |
| K M Murphy, U N Streips, R B Loc. Bax membrane insertion during Fas(CD95)-induced apoptosis precedes cytochrome c release and is inhibited by Bcl-2. Oncogene. vol 18. issue 44. 1999-12-03. PMID:10557088. |
these data establish a role for bax mitochondrial insertion during fas-mediated apoptosis, and support a model in which bax insertion amplifies the fas apoptotic cascade through cytochrome c release and complete processing of caspases-3 and -7. |
1999-12-03 |
2023-08-12 |
Not clear |