All Relations between apoptosis and fas

Publication Sentence Publish Date Extraction Date Species
S Inoue, A E Salah-Eldin, K Omoteyam. Apoptosis and anticancer drug resistance. Human cell. vol 14. issue 3. 2002-02-08. PMID:11774740. a bax induces apoptosis mitochondria-dependently, whereas fas can induce apoptosis mitochondria-independently. 2002-02-08 2023-08-12 Not clear
Hannes A Rüdiger, Pierre-Alain Clavie. Tumor necrosis factor alpha, but not Fas, mediates hepatocellular apoptosis in the murine ischemic liver. Gastroenterology. vol 122. issue 1. 2002-02-08. PMID:11781294. tumor necrosis factor alpha, but not fas, mediates hepatocellular apoptosis in the murine ischemic liver. 2002-02-08 2023-08-12 Not clear
T Imanishi, T Hano, I Nishio, D K Han, S M Schwartz, A Karsa. Apoptosis of vascular smooth muscle cells is induced by Fas ligand derived from endothelial cells. Japanese circulation journal. vol 65. issue 6. 2002-02-07. PMID:11407740. apoptosis of vascular smooth muscle cells is induced by fas ligand derived from endothelial cells. 2002-02-07 2023-08-12 human
B N Kang, K S Jeong, S J Park, S J Kim, T H Kim, H J Kim, S Y Ry. Regulation of apoptosis by somatostatin and substance P in peritoneal macrophages. Regulatory peptides. vol 101. issue 1-3. 2002-02-07. PMID:11495678. to define the mechanism of the cytokine production enhancements and inhibitions by som and sp, we examined the expression of apoptosis modulator, p53, bcl-2, bax, inducible nitric oxide synthase (inos), fas, caspase-8 and nitric oxide (no) in thioglycolate-elicited peritoneal macrophages. 2002-02-07 2023-08-12 Not clear
B N Kang, K S Jeong, S J Park, S J Kim, T H Kim, H J Kim, S Y Ry. Regulation of apoptosis by somatostatin and substance P in peritoneal macrophages. Regulatory peptides. vol 101. issue 1-3. 2002-02-07. PMID:11495678. these data suggest that apoptosis by som may occur by a bax- and no-independent p53 accumulation, and through fas and caspase-8 activation pathways, and that the inducible expression of bcl-2 and no production by sp may contribute to prevent the signals of apoptosis by bax, and via fas and caspase-8 activation. 2002-02-07 2023-08-12 Not clear
M Yasuda, Y Tanaka, K Fujii, K Yasumot. CD44 stimulation down-regulates Fas expression and Fas-mediated apoptosis of lung cancer cells. International immunology. vol 13. issue 10. 2002-02-07. PMID:11581176. cd44 stimulation down-regulates fas expression and fas-mediated apoptosis of lung cancer cells. 2002-02-07 2023-08-12 Not clear
M Yasuda, Y Tanaka, K Fujii, K Yasumot. CD44 stimulation down-regulates Fas expression and Fas-mediated apoptosis of lung cancer cells. International immunology. vol 13. issue 10. 2002-02-07. PMID:11581176. we here document a novel function for cd44--using lung cancer cells, we showed that stimulation of cd44 reduced fas expression and fas-mediated apoptosis: (i) lung cancer cells expressed high levels of cd44; (ii) engagement of cd44 on the cells by a specific antibody or fragmented hyaluronan reduced fas expression; (iii) cd44 cross-linking reduced fas-mediated apoptosis; (iv) stimulation of cd44 on lung cancer cells decreased ifn-gamma production by autologous ctl; and (v) cd44 stimulation prevented killing of lung cancer cells by autologous ctl. 2002-02-07 2023-08-12 Not clear
M Yasuda, Y Tanaka, K Fujii, K Yasumot. CD44 stimulation down-regulates Fas expression and Fas-mediated apoptosis of lung cancer cells. International immunology. vol 13. issue 10. 2002-02-07. PMID:11581176. based on these findings, we postulate a new concept--that interaction of cd44 on lung cancer cells with fragments of extracellular hyaluronan present in the surrounding extracellular matrix reduces fas expression as well as fas-mediated apoptosis of cancer cells. 2002-02-07 2023-08-12 Not clear
M J Lebedev, J S Ptitsina, S A Vilkov, S B Korablev, V V Noviko. Membrane and soluble forms of Fas (CD95) in peripheral blood lymphocytes and in serum from burns patients. Burns : journal of the International Society for Burn Injuries. vol 27. issue 7. 2002-02-07. PMID:11600245. apoptosis of immunocytes is associated with the expression of fas antigen. 2002-02-07 2023-08-12 human
M A Hayden, P A Lange, D K Nakayam. Nitric oxide and cyclic guanosine monophosphate stimulate apoptosis via activation of the Fas-FasL pathway. The Journal of surgical research. vol 101. issue 2. 2002-02-07. PMID:11735274. the mechanism by which no causes apoptosis is unknown; however, fas (cd95) and fas ligand (fasl) (cd95l) have been implicated. 2002-02-07 2023-08-12 Not clear
H R de Jonge, B Hogema, B C Till. Protein N-myristoylation: critical role in apoptosis and salt tolerance. Science's STKE : signal transduction knowledge environment. vol 2000. issue 63. 2002-02-07. PMID:11752628. de jonge, hogema, and tilly discuss how n-myristoylation may be involved in triggering fas ligand-induced apoptosis in mammals, and in adapting to conditions of high salt in plants. 2002-02-07 2023-08-12 Not clear
K Goto, J Fukuda, T Hanej. Okadaic acid stimulates apoptosis through expression of Fas receptor and Fas ligand in human oral squamous carcinoma cells. Oral oncology. vol 38. issue 1. 2002-02-07. PMID:11755816. okadaic acid stimulates apoptosis through expression of fas receptor and fas ligand in human oral squamous carcinoma cells. 2002-02-07 2023-08-12 human
K Goto, J Fukuda, T Hanej. Okadaic acid stimulates apoptosis through expression of Fas receptor and Fas ligand in human oral squamous carcinoma cells. Oral oncology. vol 38. issue 1. 2002-02-07. PMID:11755816. the present results indicate that the expression of fas receptor and fas ligand is negatively regulated by a protein phosphatase(s) sensitive to okadaic acid and is involved in okadaic acid-induced apoptosis in scc-25 cells. 2002-02-07 2023-08-12 human
K Goto, J Fukuda, T Hanej. Okadaic acid stimulates apoptosis through expression of Fas receptor and Fas ligand in human oral squamous carcinoma cells. Oral oncology. vol 38. issue 1. 2002-02-07. PMID:11755816. our results also suggest that fas receptor and fas ligand system might regulate apoptosis in scc-25 cells in an autocrine fashion. 2002-02-07 2023-08-12 human
T Tsutamoto, A Wada, K Maeda, N Mabuchi, M Hayashi, T Tsutsui, M Ohnishi, M Fujii, T Matsumoto, T Yamamoto, T Takayama, M Kinoshit. Relationship between plasma levels of cardiac natriuretic peptides and soluble Fas: plasma soluble Fas as a prognostic predictor in patients with congestive heart failure. Journal of cardiac failure. vol 7. issue 4. 2002-02-07. PMID:11782855. cardiac natriuretic peptides may induce apoptosis in myocytes; however, the relationship between plasma levels of cardiac natriuretic peptides and those of soluble fas (sfas) and tumor necrosis factor (tnf)-alpha remains unknown in patients with congestive heart failure (chf). 2002-02-07 2023-08-12 Not clear
E Pluygers, A Sadowska, L Chyczewski, J Nikliński, W Niklińska, E Chyczewsk. The impact of immune responses on lung cancer and the development of new treatment modalities. Lung cancer (Amsterdam, Netherlands). vol 34 Suppl 2. 2002-02-05. PMID:11720745. apoptosis in t-cells is triggered by the interaction of the membrane receptor fas with its normal ligand fas l, or an activating antibody. 2002-02-05 2023-08-12 human
S Faouzi, B E Burckhardt, J C Hanson, C B Campe, L W Schrum, R A Rippe, J J Mahe. Anti-Fas induces hepatic chemokines and promotes inflammation by an NF-kappa B-independent, caspase-3-dependent pathway. The Journal of biological chemistry. vol 276. issue 52. 2002-01-31. PMID:11602613. agonistic antibodies against the fas receptor, when administered to mice in vivo, cause significant apoptosis in the liver. 2002-01-31 2023-08-12 mouse
E A Dearstyne, N I Kerkvlie. Mechanism of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)-induced decrease in anti-CD3-activated CD4(+) T cells: the roles of apoptosis, Fas, and TNF. Toxicology. vol 170. issue 1-2. 2002-01-31. PMID:11750091. mechanism of 2,3,7,8-tetrachlorodibenzo-p-dioxin (tcdd)-induced decrease in anti-cd3-activated cd4(+) t cells: the roles of apoptosis, fas, and tnf. 2002-01-31 2023-08-12 mouse
Carla A Martin, Asit Panj. Cytokine regulation of human intestinal primary epithelial cell susceptibility to Fas-mediated apoptosis. American journal of physiology. Gastrointestinal and liver physiology. vol 282. issue 1. 2002-01-31. PMID:11751162. despite ample expression of fas, hipec were relatively insensitive to fas-mediated apoptosis in that agonist anti-fas antibody (ch11) induced a <25% increase in hipec apoptosis. 2002-01-31 2023-08-12 human
Carla A Martin, Asit Panj. Cytokine regulation of human intestinal primary epithelial cell susceptibility to Fas-mediated apoptosis. American journal of physiology. Gastrointestinal and liver physiology. vol 282. issue 1. 2002-01-31. PMID:11751162. pretreatment of hipec with interferon (ifn)-gamma, but not tumor necrosis factor-alpha or granulocyte-macrophage colony-stimulating factor, significantly increased ch11-induced apoptosis of these cells without increasing fas expression. 2002-01-31 2023-08-12 human