All Relations between Multiple Sclerosis and oligodendrocyte

Publication Sentence Publish Date Extraction Date Species
Victoria Tran, Nicholas Carpo, Sophia Shaka, Joile Zamudio, Sungshin Choi, Carlos Cepeda, Araceli Espinosa-Jeffre. Delayed Maturation of Oligodendrocyte Progenitors by Microgravity: Implications for Multiple Sclerosis and Space Flight. Life (Basel, Switzerland). vol 12. issue 6. 2022-06-24. PMID:35743828. delayed maturation of oligodendrocyte progenitors by microgravity: implications for multiple sclerosis and space flight. 2022-06-24 2023-08-14 human
Samir Alkabie, Adrian Budhra. Testing for Antibodies Against Aquaporin-4 and Myelin Oligodendrocyte Glycoprotein in the Diagnosis of Patients With Suspected Autoimmune Myelopathy. Frontiers in neurology. vol 13. 2022-06-07. PMID:35669883. discoveries of antibodies targeting aquaporin-4 (aqp4-igg) and myelin oligodendrocyte glycoprotein (mog-igg) have facilitated the diagnosis of autoimmune myelopathies that were previously considered to be atypical presentations of multiple sclerosis (ms) or idiopathic, and represent major advancements in the field of autoimmune neurology. 2022-06-07 2023-08-14 Not clear
Er-Chuang Li, Yang Zheng, Meng-Ting Cai, Qi-Lun Lai, Gao-Li Fang, Bing-Qing Du, Chun-Hong Shen, Yin-Xi Zhang, Long-Jun Wu, Mei-Ping Din. Seizures and epilepsy in multiple sclerosis, aquaporin 4 antibody-positive neuromyelitis optica spectrum disorder and myelin oligodendrocyte glycoprotein antibody-associated disease. Epilepsia. 2022-06-02. PMID:35652436. seizure is one of the manifestations of central nervous system (cns) inflammatory demyelinating diseases, which mainly include multiple sclerosis (ms), aquaporin 4 antibody-positive neuromyelitis optica spectrum disorder (aqp4-nmosd) and myelin oligodendrocyte glycoprotein antibody-associated disease (mogad). 2022-06-02 2023-08-14 Not clear
Giulia Fadda, Patrick Waters, Mark Woodhall, Robert A Brown, Julia O'Mahony, Denise A Castro, Giulia Longoni, E Ann Yeh, Ruth Ann Marrie, Douglas L Arnold, Brenda Banwell, Amit Bar-O. Serum MOG-IgG in children meeting multiple sclerosis diagnostic criteria. Multiple sclerosis (Houndmills, Basingstoke, England). 2022-05-18. PMID:35581944. myelin oligodendrocyte glycoprotein antibody-associated disease (mogad) is now recognized as distinct from multiple sclerosis (ms). 2022-05-18 2023-08-13 Not clear
Megan E Rouillard, Jingwen Hu, Pearl A Sutter, Hee Won Kim, Jeffrey K Huang, Stephen J Crocke. The Cellular Senescence Factor Extracellular HMGB1 Directly Inhibits Oligodendrocyte Progenitor Cell Differentiation and Impairs CNS Remyelination. Frontiers in cellular neuroscience. vol 16. 2022-05-16. PMID:35573828. we have recently determined that progenitor cells from multiple sclerosis patients exhibit a cellular senescent phenotype and release extracellular hmgb1 which directly impaired the maturation of oligodendrocyte progenitor cells (opcs) to myelinating oligodendrocytes (ols). 2022-05-16 2023-08-13 Not clear
Xiayin Yang, Xuefen Li, Mengying Lai, Jincui Wang, Shaoying Tan, Henry Ho-Lung Cha. Pain Symptoms in Optic Neuritis. Frontiers in pain research (Lausanne, Switzerland). vol 3. 2022-05-02. PMID:35498555. this article reviews the presence of pain related to on with respect to underlying disorders, including multiple sclerosis (ms), neuromyelitis optica spectrum disorder (nmosd), and myelin oligodendrocyte glycoprotein associated disease (mogad). 2022-05-02 2023-08-13 Not clear
Rui Ma, Yaojuan Chu, Mengmeng Dou, Yilei Jing, Xiaoyu Zhao, Xinyu Li, Silu Li, Yuan Zhang, Limei Wang, Lin Zh. Matrine inhibits the Wnt3a/β-catenin/TCF7L2 signaling pathway in experimental autoimmune encephalomyelitis. Journal of neuroimmunology. vol 367. 2022-04-30. PMID:35489221. oligodendrocyte (ol) death and remyelination failure lead to progressive neurological deficits in multiple sclerosis (ms) and its animal model, experimental autoimmune encephalomyelitis (eae). 2022-04-30 2023-08-13 mouse
Abdulhafeez M Khair, Rahul Nikam, Sumair Husain, Melanie Ortiz, Gurcharanjeet Kau. Para and Post-COVID-19 CNS Acute Demyelinating Disorders in Children: A Case Series on Expanding the Spectrum of Clinical and Radiological Characteristics. Cureus. vol 14. issue 3. 2022-04-27. PMID:35475081. a 16-year-old female with anti-myelin oligodendrocyte glycoprotein (mog) disorder, an eight-year-old male with acute disseminated encephalomyelitis (adem), a 13-year-old female with neuromyelitis optica spectrum disorder (nmosd), and two 14 and 13-year-old females with new-onset multiple sclerosis (ms) are reported, all of whom presented acutely following covid-19 infection. we propose that para and post-infectious cns demyelinating disorders can potentially follow acute covid-19 infection in children. 2022-04-27 2023-08-13 Not clear
Richard A Seidman, Heba Khattab, Jessie J Polanco, Jacqueline E Broome, Fraser J Si. Oscillatory calcium release and sustained store-operated oscillatory calcium signaling prevents differentiation of human oligodendrocyte progenitor cells. Scientific reports. vol 12. issue 1. 2022-04-14. PMID:35418597. endogenous remyelination in demyelinating diseases such as multiple sclerosis is contingent upon the successful differentiation of oligodendrocyte progenitor cells (opcs). 2022-04-14 2023-08-13 human
Aigli G Vakrakou, Maria-Evgenia Brinia, Ioanna Svolaki, Theodore Argyrakos, Leonidas Stefanis, Constantinos Kilidirea. Immunopathology of Tumefactive Demyelinating Lesions-From Idiopathic to Drug-Related Cases. Frontiers in neurology. vol 13. 2022-04-14. PMID:35418930. tdl can emerge not only as part of the spectrum of classic multiple sclerosis (ms) but also can represent an idiopathic monophasic disease, a relapsing disease with recurrent tdl, or could be part of the myelin oligodendrocyte glycoprotein (mog)- and aquaporin-4 (aqp4)-associated disease. 2022-04-14 2023-08-13 Not clear
Sonya Bells, Giulia Longoni, Tara Berenbaum, Cynthia B de Medeiros, Sridar Narayanan, Brenda L Banwell, Douglas L Arnold, Donald J Mabbott, E Ann Ye. Patterns of white and gray structural abnormality associated with paediatric demyelinating disorders. NeuroImage. Clinical. vol 34. 2022-04-05. PMID:35381508. the impact of multiple sclerosis (ms) and myelin oligodendrocyte glycoprotein (mog) - associated disorders (mogad) on brain structure in youth remains poorly understood. 2022-04-05 2023-08-13 Not clear
Rianne P Gorter, Naomi S Dijksman, Wia Baron, Holly Colognat. Investigating demyelination, efficient remyelination and remyelination failure in organotypic cerebellar slice cultures: Workflow and practical tips. Methods in cell biology. vol 168. 2022-04-03. PMID:35366978. thus, regeneration of the myelin sheath by encouraging oligodendrocyte lineage cells to remyelinate the denuded axons is a promising therapeutic target for demyelinating diseases such as multiple sclerosis. 2022-04-03 2023-08-13 Not clear
Sinali O Seneviratne, Mark Marriott, Sudarshini Ramanathan, Wei Yeh, Fabienne Brilot-Turville, Helmut Butzkueven, Mastura Moni. Failure of alemtuzumab therapy in three patients with MOG antibody associated disease. BMC neurology. vol 22. issue 1. 2022-03-10. PMID:35264149. myelin oligodendrocyte glycoprotein antibody-associated disease (mogad) is most classically associated in both children and adults with phenotypes including bilateral and recurrent optic neuritis (on) and transverse myelitis (tm), with the absence of brain lesions characteristic of multiple sclerosis (ms). 2022-03-10 2023-08-13 Not clear
Omri Zveik, Nina Fainstein, Ariel Rechtman, Nitzan Haham, Tal Ganz, Iris Lavon, Livnat Brill, Adi Vaknin-Dembinsk. Cerebrospinal fluid of progressive multiple sclerosis patients reduces differentiation and immune functions of oligodendrocyte progenitor cells. Glia. 2022-03-10. PMID:35266197. cerebrospinal fluid of progressive multiple sclerosis patients reduces differentiation and immune functions of oligodendrocyte progenitor cells. 2022-03-10 2023-08-13 mouse
Vyanka Redenbaugh, Eoin P Flanaga. Monoclonal Antibody Therapies Beyond Complement for NMOSD and MOGAD. Neurotherapeutics : the journal of the American Society for Experimental NeuroTherapeutics. 2022-03-10. PMID:35267170. aquaporin-4 (aqp4)-igg seropositive neuromyelitis optica spectrum disorders (aqp4-igg seropositive nmosd) and myelin oligodendrocyte glycoprotein (mog)-igg-associated disease (mogad) are inflammatory demyelinating disorders distinct from each other and from multiple sclerosis (ms).while anti-cd20 treatments can be used to treat ms and aqp4-igg seropositive nmosd, some ms medications are ineffective or could exacerbate aqp4-igg seropositive nmosd including beta-interferons, natalizumab, and fingolimod. 2022-03-10 2023-08-13 Not clear
Marie L Mather, Marisa A Jeffries, Teresa L Woo. The mechanistic target of rapamycin as a regulator of metabolic function in oligodendroglia during remyelination. Current opinion in pharmacology. vol 63. 2022-03-04. PMID:35245799. despite evidence for prominent metabolic dysfunction within multiple sclerosis (ms) lesions, the mechanisms controlling metabolic shifts in oligodendroglia are poorly understood. 2022-03-04 2023-08-13 Not clear
Yasmine Labrak, Béatrice Heurtault, Benoît Frisch, Patrick Saulnier, Elise Lepeltier, Veronique E Miron, Giulio G Muccioli, Anne des Rieu. Impact of anti-PDGFRα antibody surface functionalization on LNC uptake by oligodendrocyte progenitor cells. International journal of pharmaceutics. 2022-03-01. PMID:35231547. impairment of oligodendrocyte progenitor cell (opc) differentiation into oligodendrocytes and chronic inflammation are key determinants of poor remyelination observed in diseases such as multiple sclerosis. 2022-03-01 2023-08-13 rat
Karolina Salwierak-Głośna, Paweł Piątek, Małgorzata Domowicz, Mariola Świderek-Matysia. Effect of Multiple Sclerosis Cerebrospinal Fluid and Oligodendroglia Cell Line Environment on Human Wharton's Jelly Mesenchymal Stem Cells Secretome. International journal of molecular sciences. vol 23. issue 4. 2022-02-26. PMID:35216294. effect of multiple sclerosis cerebrospinal fluid and oligodendroglia cell line environment on human wharton's jelly mesenchymal stem cells secretome. 2022-02-26 2023-08-13 human
Masoud Etemadifar, Mehri Salari, Mohammad Reza Etemadifar, Fatemeh Sabeti, Sepand Tehrani Fateh, Zahra Aminzad. Centrally-located transverse myelitis would facilitate the differentiation of NMOSD and MOG-AD from MS. Multiple sclerosis and related disorders. vol 60. 2022-02-26. PMID:35219242. centrally-located transverse myelitis would facilitate the differentiation of nmosd and mog-ad from ms. differentiating neuromyelitis optica spectrum disorder (nmosd) and myelin oligodendrocyte glycoprotein antibody disease (mog-ad) from multiple sclerosis (ms) is important since ms therapies might result in progression and relapse of the former diseases. 2022-02-26 2023-08-13 Not clear
Florian Pernin, Julia Luo, Qiao-Ling Cui, Manon Blain, Milton G F Fernandes, Moein Yaqubi, Myriam Srour, Jeff Hall, Roy Dudley, Hélène Jamann, Catherine Larochelle, Stephanie E J Zandee, Alexandre Prat, Jo Anne Stratton, Timothy E Kennedy, Jack P Ante. Diverse injury responses of human oligodendrocyte to mediators implicated in multiple sclerosis. Brain : a journal of neurology. 2022-02-24. PMID:35202462. diverse injury responses of human oligodendrocyte to mediators implicated in multiple sclerosis. 2022-02-24 2023-08-13 human