| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| N Benjelloun, A Ménard, C Charriaut-Marlangue, K Mokhtari, H Perron, J J Hauw, F Riege. Case report: DNA fragmentation in glial cells in a cerebral biopsy from a multiple sclerosis patient. Cellular and molecular biology (Noisy-le-Grand, France). vol 44. issue 4. 1998-10-20. PMID:9678892. |
multiple sclerosis is characterized by myelin destruction and oligodendrocyte loss. |
1998-10-20 |
2023-08-12 |
Not clear |
| C Mertens, D Brassat, J Reboul, S Eichenbaum-Voline, C Vuillemin-Azais, I Cournu, M C Babron, G Semana, G Edan, M Clanet, F Clerget-Darpoux, A Baron-Van Evercooren, O Lyon-Caen, R Liblau, B Fontain. A systematic study of oligodendrocyte growth factors as candidates for genetic susceptibility to MS. French Multiple Sclerosis Genetics Group. Neurology. vol 51. issue 3. 1998-10-20. PMID:9748021. |
a systematic study of oligodendrocyte growth factors as candidates for genetic susceptibility to ms. french multiple sclerosis genetics group. |
1998-10-20 |
2023-08-12 |
Not clear |
| K Akassoglou, J Bauer, G Kassiotis, M Pasparakis, H Lassmann, G Kollias, L Prober. Oligodendrocyte apoptosis and primary demyelination induced by local TNF/p55TNF receptor signaling in the central nervous system of transgenic mice: models for multiple sclerosis with primary oligodendrogliopathy. The American journal of pathology. vol 153. issue 3. 1998-09-24. PMID:9736029. |
oligodendrocyte apoptosis and primary demyelination induced by local tnf/p55tnf receptor signaling in the central nervous system of transgenic mice: models for multiple sclerosis with primary oligodendrogliopathy. |
1998-09-24 |
2023-08-12 |
mouse |
| R Zeine, R Pon, U Ladiwala, J P Antel, L G Filion, M S Freedma. Mechanism of gammadelta T cell-induced human oligodendrocyte cytotoxicity: relevance to multiple sclerosis. Journal of neuroimmunology. vol 87. issue 1-2. 1998-08-13. PMID:9670845. |
mechanism of gammadelta t cell-induced human oligodendrocyte cytotoxicity: relevance to multiple sclerosis. |
1998-08-13 |
2023-08-12 |
human |
| L Zhou, R H Mille. Antibody-mediated oligodendrocyte cell death requires an astrocyte-derived cosignal. Journal of neuroscience research. vol 52. issue 2. 1998-07-28. PMID:9579404. |
such antibody-induced oligodendrocyte cell death may explain in part the loss of myelin-forming cells in demyelination conditions, such as multiple sclerosis. |
1998-07-28 |
2023-08-12 |
rat |
| I Mews, M Bergmann, S Bunkowski, F Gullotta, W Brüc. Oligodendrocyte and axon pathology in clinically silent multiple sclerosis lesions. Multiple sclerosis (Houndmills, Basingstoke, England). vol 4. issue 2. 1998-07-10. PMID:9599334. |
oligodendrocyte and axon pathology in clinically silent multiple sclerosis lesions. |
1998-07-10 |
2023-08-12 |
Not clear |
| I Mews, M Bergmann, S Bunkowski, F Gullotta, W Brüc. Oligodendrocyte and axon pathology in clinically silent multiple sclerosis lesions. Multiple sclerosis (Houndmills, Basingstoke, England). vol 4. issue 2. 1998-07-10. PMID:9599334. |
oligodendrocyte and axon pathology was studied in 11 autopsy cases of clinically silent multiple sclerosis. |
1998-07-10 |
2023-08-12 |
Not clear |
| E A Leadbetter, C R Bourque, B Devaux, C D Olson, G H Sunshine, S Hirani, B P Wallner, D E Smilek, M P Hap. Experimental autoimmune encephalomyelitis induced with a combination of myelin basic protein and myelin oligodendrocyte glycoprotein is ameliorated by administration of a single myelin basic protein peptide. Journal of immunology (Baltimore, Md. : 1950). vol 161. issue 1. 1998-07-09. PMID:9647262. |
multiple sclerosis is an autoimmune disease of the central nervous system in which t cell reactivity to several myelin proteins, including myelin basic protein (mbp), proteolipid protein, and myelin oligodendrocyte glycoprotein (mog), has been implicated in the perpetuation of the disease state. |
1998-07-09 |
2023-08-12 |
mouse |
| J Bettadapura, K K Menon, S Moritz, J Liu, C C Bernar. Expression, purification, and encephalitogenicity of recombinant human myelin oligodendrocyte glycoprotein. Journal of neurochemistry. vol 70. issue 4. 1998-04-23. PMID:9523576. |
myelin oligodendrocyte glycoprotein (mog), a putative autoantigen in multiple sclerosis (ms), is a quantitatively minor component of the cns. |
1998-04-23 |
2023-08-12 |
mouse |
| H S Keirstead, J M Levine, W F Blakemor. Response of the oligodendrocyte progenitor cell population (defined by NG2 labelling) to demyelination of the adult spinal cord. Glia. vol 22. issue 2. 1998-04-09. PMID:9537836. |
elucidation of the response of oligodendrocyte progenitor cell populations to demyelination in the adult central nervous system (cns) is critical to understanding why remyelination fails in multiple sclerosis. |
1998-04-09 |
2023-08-12 |
rat |
| D Rodriguez, B Della Gaspera, B Zalc, J J Hauw, B Fontaine, G Edan, M Clanet, A Dautigny, D Pham-Din. Identification of a Val 145 Ile substitution in the human myelin oligodendrocyte glycoprotein: lack of association with multiple sclerosis. The Réseau de Recherche Clinique INSERM sur la Susceptibilité Génétique à la Sclérose en Plaques. Multiple sclerosis (Houndmills, Basingstoke, England). vol 3. issue 6. 1998-04-02. PMID:9493637. |
identification of a val 145 ile substitution in the human myelin oligodendrocyte glycoprotein: lack of association with multiple sclerosis. |
1998-04-02 |
2023-08-12 |
human |
| J J Archelos, J Trotter, S Previtali, B Weissbrich, K V Toyka, H P Hartun. Isolation and characterization of an oligodendrocyte precursor-derived B-cell epitope in multiple sclerosis. Annals of neurology. vol 43. issue 1. 1998-02-17. PMID:9450764. |
isolation and characterization of an oligodendrocyte precursor-derived b-cell epitope in multiple sclerosis. |
1998-02-17 |
2023-08-12 |
Not clear |
| G Wolswij. Chronic stage multiple sclerosis lesions contain a relatively quiescent population of oligodendrocyte precursor cells. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 18. issue 2. 1998-02-03. PMID:9425002. |
chronic stage multiple sclerosis lesions contain a relatively quiescent population of oligodendrocyte precursor cells. |
1998-02-03 |
2023-08-12 |
human |
| G Wolswij. Chronic stage multiple sclerosis lesions contain a relatively quiescent population of oligodendrocyte precursor cells. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 18. issue 2. 1998-02-03. PMID:9425002. |
much less is known about human oligodendrocyte lineage cells and about the reasons for the failure of the regeneration of the oligodendrocyte population during chronic stages of multiple sclerosis (ms). |
1998-02-03 |
2023-08-12 |
human |
| J Brown, L Hinks, R J Thompso. Lack of association of a Taq 1 polymorphism of the human myelin oligodendrocyte glycoprotein gene with multiple sclerosis in a population of patients from the Southampton area. Journal of neurology, neurosurgery, and psychiatry. vol 64. issue 1. 1998-01-30. PMID:9436746. |
lack of association of a taq 1 polymorphism of the human myelin oligodendrocyte glycoprotein gene with multiple sclerosis in a population of patients from the southampton area. |
1998-01-30 |
2023-08-12 |
human |
| N Kerlero de Rosbo, M Hoffman, I Mendel, I Yust, J Kaye, R Bakimer, S Flechter, O Abramsky, R Milo, A Karni, A Ben-Nu. Predominance of the autoimmune response to myelin oligodendrocyte glycoprotein (MOG) in multiple sclerosis: reactivity to the extracellular domain of MOG is directed against three main regions. European journal of immunology. vol 27. issue 11. 1998-01-02. PMID:9394837. |
predominance of the autoimmune response to myelin oligodendrocyte glycoprotein (mog) in multiple sclerosis: reactivity to the extracellular domain of mog is directed against three main regions. |
1998-01-02 |
2023-08-12 |
human |
| N Kerlero de Rosbo, M Hoffman, I Mendel, I Yust, J Kaye, R Bakimer, S Flechter, O Abramsky, R Milo, A Karni, A Ben-Nu. Predominance of the autoimmune response to myelin oligodendrocyte glycoprotein (MOG) in multiple sclerosis: reactivity to the extracellular domain of MOG is directed against three main regions. European journal of immunology. vol 27. issue 11. 1998-01-02. PMID:9394837. |
our previous analysis of the t cell reactivity to myelin antigens in a group of 24 patients with multiple sclerosis (ms) and 16 control individuals revealed that the autoimmune response to myelin oligodendrocyte glycoprotein (mog) predominates in ms over that to myelin basic protein (mbp), proteolipid protein or myelin-associated glycoprotein, suggesting a prevalent role for the autoimmune response to mog in the pathogenesis of ms. |
1998-01-02 |
2023-08-12 |
human |
| R J Franklin, W F Blakemor. To what extent is oligodendrocyte progenitor migration a limiting factor in the remyelination of multiple sclerosis lesions? Multiple sclerosis (Houndmills, Basingstoke, England). vol 3. issue 2. 1997-10-07. PMID:9291159. |
to what extent is oligodendrocyte progenitor migration a limiting factor in the remyelination of multiple sclerosis lesions? |
1997-10-07 |
2023-08-12 |
Not clear |
| S Jefferson, T Jacques, B W Kiernan, S Scott-Drew, R Milner, C ffrench-Constan. Inhibition of oligodendrocyte precursor motility by oligodendrocyte processes: implications for transplantation-based approaches to multiple sclerosis. Multiple sclerosis (Houndmills, Basingstoke, England). vol 3. issue 2. 1997-10-07. PMID:9291174. |
inhibition of oligodendrocyte precursor motility by oligodendrocyte processes: implications for transplantation-based approaches to multiple sclerosis. |
1997-10-07 |
2023-08-12 |
Not clear |
| S Jefferson, T Jacques, B W Kiernan, S Scott-Drew, R Milner, C ffrench-Constan. Inhibition of oligodendrocyte precursor motility by oligodendrocyte processes: implications for transplantation-based approaches to multiple sclerosis. Multiple sclerosis (Houndmills, Basingstoke, England). vol 3. issue 2. 1997-10-07. PMID:9291174. |
transplantation of oligodendrocyte precursor cells represents a promising approach to the treatment of the chronic demyelinated lesions of multiple sclerosis. |
1997-10-07 |
2023-08-12 |
Not clear |