All Relations between Multiple Sclerosis and oligodendrocyte

Publication Sentence Publish Date Extraction Date Species
S D D'Souza, J P Antel, M S Freedma. Cytokine induction of heat shock protein expression in human oligodendrocytes: an interleukin-1-mediated mechanism. Journal of neuroimmunology. vol 50. issue 1. 1994-03-08. PMID:8300853. in this study, we examined the role of cytokines, known to be in elevated levels in multiple sclerosis (ms) plaques, in regulating oligodendrocyte (odc) expression of heat shock protein (hsp) in human brain-derived glial cell cultures. 1994-03-08 2023-08-12 human
N Kerlero de Rosbo, R Milo, M B Lees, D Burger, C C Bernard, A Ben-Nu. Reactivity to myelin antigens in multiple sclerosis. Peripheral blood lymphocytes respond predominantly to myelin oligodendrocyte glycoprotein. The Journal of clinical investigation. vol 92. issue 6. 1994-01-12. PMID:7504688. although t cell responses to the quantitatively major myelin proteins, myelin basic protein (mbp) and proteolipid protein (plp), are likely to be of importance in the course of multiple sclerosis (ms), cell-mediated autoimmune responses to other myelin antigens, in particular quantitatively minor myelin antigens, such as myelin-associated glycoprotein (mag) and the central nervous system-specific myelin oligodendrocyte glycoprotein (mog), could also play a prevalent role in disease initiation or progression. 1994-01-12 2023-08-12 Not clear
M Rodriguez, B W Scheithauer, G Forbes, P J Kell. Oligodendrocyte injury is an early event in lesions of multiple sclerosis. Mayo Clinic proceedings. vol 68. issue 7. 1993-09-14. PMID:8350635. oligodendrocyte injury is an early event in lesions of multiple sclerosis. 1993-09-14 2023-08-12 Not clear
J E Merrill, L J Ignarro, M P Sherman, J Melinek, T E Lan. Microglial cell cytotoxicity of oligodendrocytes is mediated through nitric oxide. Journal of immunology (Baltimore, Md. : 1950). vol 151. issue 4. 1993-09-08. PMID:8102159. there is a correlation between ameboid microglial cell killing of oligodendrocytes and no2- production suggesting no may be a mechanism of death of the oligodendrocyte and possibly play a role in lesion formation in multiple sclerosis. 1993-09-08 2023-08-12 rat
J B Su. Autoreactive T and B cells in nervous system diseases. Acta neurologica Scandinavica. Supplementum. vol 142. 1993-03-30. PMID:8382894. abnormal autoimmunity could play a role in the pathogenesis of multiple sclerosis (ms), in analogy with its model experimental allergic encephalomyelitis (eae) which can be transferred by t cell lines directed to myelin basic protein (mbp) and myelin proteolipid protein (plp), and worsened with antibody to myelin oligodendrocyte glycoprotein (mog). 1993-03-30 2023-08-12 Not clear
V Jewtoukoff, S Amzazi, R Lebar, M A Bach, P N March. T-cell receptor identification of an oligodendrocyte-specific autoreactive cytotoxic T-cell clone without self restriction. Scandinavian journal of immunology. vol 36. issue 6. 1993-01-08. PMID:1462126. in addition to myelin basic protein (mbp) and proteolipid protein (plp), oligodendrocyte (od) membrane autoantigens, such as the glycoprotein m2/mog, could participate in the pathogenesis of autoimmune demyelinating diseases of the central nervous system (cns), such as experimental allergic encephalomyelitis (eae) or multiple sclerosis (ms). 1993-01-08 2023-08-11 Not clear
J E Merrill, R P Zimmerma. Natural and induced cytotoxicity of oligodendrocytes by microglia is inhibitable by TGF beta. Glia. vol 4. issue 3. 1991-10-18. PMID:1832660. blood macrophages and brain macrophages (microglia) have been implicated in demyelination and destruction of the oligodendrocyte in multiple sclerosis (ms), a disease affecting primarily white matter of the central nervous system (cns). 1991-10-18 2023-08-11 rat
S Fredrikson, J Sun, B G Xiao, H Lin. Cord blood contains cells secreting antibodies to nervous system components. Clinical and experimental immunology. vol 84. issue 2. 1991-06-11. PMID:1709072. umbilical cord blood of newborns and peripheral blood of healthy adults were investigated by an immunospot assay for cells secreting igg, iga and igm antibodies against myelin basic protein (mbp), proteolipid protein (plp), myelin-associated glycoprotein (mag) and myelin oligodendrocyte glycoprotein (mog) which represent putative antigens for an autoimmune attack in multiple sclerosis (ms) and against acetylcholine receptor (achr) which is considered an important autoantigen in myasthenia gravis. 1991-06-11 2023-08-11 Not clear
J W Prineas, E E Kwon, P Z Goldenberg, E S Cho, L R Share. Interaction of astrocytes and newly formed oligodendrocytes in resolving multiple sclerosis lesions. Laboratory investigation; a journal of technical methods and pathology. vol 63. issue 5. 1990-12-19. PMID:1700195. it bears some resemblance, however, to a pattern of slow oligodendrocyte destruction seen previously in organotypic perinatal central nervous tissue cultures exposed to multiple sclerosis serum. 1990-12-19 2023-08-11 Not clear
J W Prineas, E E Kwon, P Z Goldenberg, A A Ilyas, R H Quarles, J A Benjamins, T J Sprinkl. Multiple sclerosis. Oligodendrocyte proliferation and differentiation in fresh lesions. Laboratory investigation; a journal of technical methods and pathology. vol 61. issue 5. 1989-12-21. PMID:2811298. fresh lesions in the brain and spinal cord of patients with multiple sclerosis who died shortly after the onset of symptoms were examined immunocytochemically for myelin and oligodendrocyte antigens that are known to be sequentially expressed during normal development. 1989-12-21 2023-08-11 Not clear
C S Raine, G R Moore, R Hintzen, U Traugot. Induction of oligodendrocyte proliferation and remyelination after chronic demyelination. Relevance to multiple sclerosis. Laboratory investigation; a journal of technical methods and pathology. vol 59. issue 4. 1988-11-21. PMID:2459499. observations from multiple sclerosis lesions showed that oligodendrocyte proliferation and cns remyelination occur in human subcortical white matter, but to a lesser degree than that seen in the cns of mbp/gc/treated guinea pigs. 1988-11-21 2023-08-11 human
S B Elia. Oligodendrocyte development and the natural history of multiple sclerosis. A new hypothesis for the pathogenesis of the disease. Archives of neurology. vol 44. issue 12. 1987-12-17. PMID:3675264. oligodendrocyte development and the natural history of multiple sclerosis. 1987-12-17 2023-08-11 Not clear
A Suzumura, R P Lisak, D H Silberber. Serum cytotoxicity to oligodendrocytes in multiple sclerosis and controls: assessment by 51Cr release assay. Journal of neuroimmunology. vol 11. issue 2. 1986-04-16. PMID:3949965. in order to quantitate oligodendrocyte cytotoxicity induced by sera from patients with multiple sclerosis (ms) and control sera, we established a 51cr release assay system using cultured oligodendrocytes as target cells. 1986-04-16 2023-08-11 Not clear
M Hirayama, R P Lisak, D H Silberber. Serum-mediated oligodendrocyte cytotoxicity in multiple sclerosis patients and controls. Neurology. vol 36. issue 2. 1986-03-05. PMID:3945400. serum-mediated oligodendrocyte cytotoxicity in multiple sclerosis patients and controls. 1986-03-05 2023-08-11 rat
C W Adam. Oligodendroglia in multiple sclerosis. Nature. vol 309. issue 5968. 1984-07-17. PMID:6728010. oligodendroglia in multiple sclerosis. 1984-07-17 2023-08-12 Not clear
A Wajgt, M Górn. CSF antibodies to myelin basic protein and to myelin-associated glycoprotein in multiple sclerosis. Evidence of the intrathecal production of antibodies. Acta neurologica Scandinavica. vol 68. issue 5. 1984-02-29. PMID:6198864. cerebrospinal fluid (csf) from 40 multiple sclerosis (ms) patients was tested by solid-phase radioimmunoassay (ria) for ability to bind 2 common structural components of myelin and oligodendroglia, i.e., to bind myelin basic protein (mbp) and myelin-associated glycoprotein (mag). 1984-02-29 2023-08-12 Not clear
P F Bartlett, I R Macka. The oligodendroglial cell: biology and immunology and relationship to multiple sclerosis. Journal of clinical & laboratory immunology. vol 11. issue 1. 1983-09-09. PMID:6348293. multiple sclerosis (ms) is associated with loss of oligodendrocytes within the lesions and the intractability of the disease compared to other demyelinating conditions, both experimental and clinical, suggests, that the oligodendrocyte is the primary target of this disease. 1983-09-09 2023-08-12 Not clear
S E Poduslo, K Miller, S Zolle. Purification and maintenance in culture of oligodendroglia from human multiple sclerosis brain. Journal of the neurological sciences. vol 54. issue 3. 1982-09-24. PMID:7097309. purification and maintenance in culture of oligodendroglia from human multiple sclerosis brain. 1982-09-24 2023-08-12 human
S E Poduslo, K Miller, S Zolle. Purification and maintenance in culture of oligodendroglia from human multiple sclerosis brain. Journal of the neurological sciences. vol 54. issue 3. 1982-09-24. PMID:7097309. oligodendroglia were isolated from human multiple sclerosis (ms) brain obtained at autopsy. 1982-09-24 2023-08-12 human
B R Mullin, F X Decandis, A J Montanaro, J D Rei. Myelin basic protein interacts with the myelin-specific ganglioside GM4. Brain research. vol 222. issue 1. 1982-01-09. PMID:6170387. demyelinated plaques in multiple sclerosis have decreased amounts of both myelin basic protein and sialosylgalactosylceramide (gm4), a ganglioside specifically localized in myelin and oligodendroglia of the central nervous system. 1982-01-09 2023-08-12 Not clear