| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| J J Archelos, J Trotter, S Previtali, B Weissbrich, K V Toyka, H P Hartun. Isolation and characterization of an oligodendrocyte precursor-derived B-cell epitope in multiple sclerosis. Annals of neurology. vol 43. issue 1. 1998-02-17. PMID:9450764. |
isolation and characterization of an oligodendrocyte precursor-derived b-cell epitope in multiple sclerosis. |
1998-02-17 |
2023-08-12 |
Not clear |
| G Wolswij. Chronic stage multiple sclerosis lesions contain a relatively quiescent population of oligodendrocyte precursor cells. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 18. issue 2. 1998-02-03. PMID:9425002. |
chronic stage multiple sclerosis lesions contain a relatively quiescent population of oligodendrocyte precursor cells. |
1998-02-03 |
2023-08-12 |
human |
| G Wolswij. Chronic stage multiple sclerosis lesions contain a relatively quiescent population of oligodendrocyte precursor cells. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 18. issue 2. 1998-02-03. PMID:9425002. |
much less is known about human oligodendrocyte lineage cells and about the reasons for the failure of the regeneration of the oligodendrocyte population during chronic stages of multiple sclerosis (ms). |
1998-02-03 |
2023-08-12 |
human |
| J Brown, L Hinks, R J Thompso. Lack of association of a Taq 1 polymorphism of the human myelin oligodendrocyte glycoprotein gene with multiple sclerosis in a population of patients from the Southampton area. Journal of neurology, neurosurgery, and psychiatry. vol 64. issue 1. 1998-01-30. PMID:9436746. |
lack of association of a taq 1 polymorphism of the human myelin oligodendrocyte glycoprotein gene with multiple sclerosis in a population of patients from the southampton area. |
1998-01-30 |
2023-08-12 |
human |
| N Kerlero de Rosbo, M Hoffman, I Mendel, I Yust, J Kaye, R Bakimer, S Flechter, O Abramsky, R Milo, A Karni, A Ben-Nu. Predominance of the autoimmune response to myelin oligodendrocyte glycoprotein (MOG) in multiple sclerosis: reactivity to the extracellular domain of MOG is directed against three main regions. European journal of immunology. vol 27. issue 11. 1998-01-02. PMID:9394837. |
predominance of the autoimmune response to myelin oligodendrocyte glycoprotein (mog) in multiple sclerosis: reactivity to the extracellular domain of mog is directed against three main regions. |
1998-01-02 |
2023-08-12 |
human |
| N Kerlero de Rosbo, M Hoffman, I Mendel, I Yust, J Kaye, R Bakimer, S Flechter, O Abramsky, R Milo, A Karni, A Ben-Nu. Predominance of the autoimmune response to myelin oligodendrocyte glycoprotein (MOG) in multiple sclerosis: reactivity to the extracellular domain of MOG is directed against three main regions. European journal of immunology. vol 27. issue 11. 1998-01-02. PMID:9394837. |
our previous analysis of the t cell reactivity to myelin antigens in a group of 24 patients with multiple sclerosis (ms) and 16 control individuals revealed that the autoimmune response to myelin oligodendrocyte glycoprotein (mog) predominates in ms over that to myelin basic protein (mbp), proteolipid protein or myelin-associated glycoprotein, suggesting a prevalent role for the autoimmune response to mog in the pathogenesis of ms. |
1998-01-02 |
2023-08-12 |
human |
| R J Franklin, W F Blakemor. To what extent is oligodendrocyte progenitor migration a limiting factor in the remyelination of multiple sclerosis lesions? Multiple sclerosis (Houndmills, Basingstoke, England). vol 3. issue 2. 1997-10-07. PMID:9291159. |
to what extent is oligodendrocyte progenitor migration a limiting factor in the remyelination of multiple sclerosis lesions? |
1997-10-07 |
2023-08-12 |
Not clear |
| S Jefferson, T Jacques, B W Kiernan, S Scott-Drew, R Milner, C ffrench-Constan. Inhibition of oligodendrocyte precursor motility by oligodendrocyte processes: implications for transplantation-based approaches to multiple sclerosis. Multiple sclerosis (Houndmills, Basingstoke, England). vol 3. issue 2. 1997-10-07. PMID:9291174. |
inhibition of oligodendrocyte precursor motility by oligodendrocyte processes: implications for transplantation-based approaches to multiple sclerosis. |
1997-10-07 |
2023-08-12 |
Not clear |
| S Jefferson, T Jacques, B W Kiernan, S Scott-Drew, R Milner, C ffrench-Constan. Inhibition of oligodendrocyte precursor motility by oligodendrocyte processes: implications for transplantation-based approaches to multiple sclerosis. Multiple sclerosis (Houndmills, Basingstoke, England). vol 3. issue 2. 1997-10-07. PMID:9291174. |
transplantation of oligodendrocyte precursor cells represents a promising approach to the treatment of the chronic demyelinated lesions of multiple sclerosis. |
1997-10-07 |
2023-08-12 |
Not clear |
| G Wolswij. Oligodendrocyte precursor cells in chronic multiple sclerosis lesions. Multiple sclerosis (Houndmills, Basingstoke, England). vol 3. issue 2. 1997-10-07. PMID:9291175. |
oligodendrocyte precursor cells in chronic multiple sclerosis lesions. |
1997-10-07 |
2023-08-12 |
Not clear |
| C S Rain. The Norton Lecture: a review of the oligodendrocyte in the multiple sclerosis lesion. Journal of neuroimmunology. vol 77. issue 2. 1997-08-26. PMID:9258244. |
the norton lecture: a review of the oligodendrocyte in the multiple sclerosis lesion. |
1997-08-26 |
2023-08-12 |
Not clear |
| S Albouz-Abo, J C Wilson, C C Bernard, M von Itzstei. A conformational study of the human and rat encephalitogenic myelin oligodendrocyte glycoprotein peptides 35-55. European journal of biochemistry. vol 246. issue 1. 1997-08-04. PMID:9210466. |
myelin oligodendrocyte glycoprotein (mog), is considered an important central-nervous system-specific target autoantigen for primary demyelination in autoimmune diseases like multiple sclerosis. |
1997-08-04 |
2023-08-12 |
mouse |
| P Dowling, W Husar, J Menonna, H Donnenfeld, S Cook, M Sidh. Cell death and birth in multiple sclerosis brain. Journal of the neurological sciences. vol 149. issue 1. 1997-07-10. PMID:9168159. |
the hallmark of the brain pathology in multiple sclerosis is the white matter plaque, characterized by myelin destruction and oligodendrocyte loss. |
1997-07-10 |
2023-08-12 |
Not clear |
| K Genç, D L Dona, A T Rede. Increased CD80(+) B cells in active multiple sclerosis and reversal by interferon beta-1b therapy. The Journal of clinical investigation. vol 99. issue 11. 1997-07-01. PMID:9169496. |
during multiple sclerosis (ms) exacerbations, circulating immune cells are activated, th1 cytokine levels in the blood are elevated, and blood-derived immune cells destroy brain oligodendroglia. |
1997-07-01 |
2023-08-12 |
Not clear |
| G Giovannoni, S J Heales, N C Silver, J O'Riordan, R F Miller, J M Land, J B Clark, E J Thompso. Raised serum nitrate and nitrite levels in patients with multiple sclerosis. Journal of the neurological sciences. vol 145. issue 1. 1997-06-09. PMID:9073032. |
nitric oxide and its highly reactive derivative peroxynitrite have been implicated as non-specific inflammatory mediators of neuronal and oligodendrocyte damage and death in multiple sclerosis. |
1997-06-09 |
2023-08-12 |
human |
| C C Bernard, T G Johns, A Slavin, M Ichikawa, C Ewing, J Liu, J Bettadapur. Myelin oligodendrocyte glycoprotein: a novel candidate autoantigen in multiple sclerosis. Journal of molecular medicine (Berlin, Germany). vol 75. issue 2. 1997-05-28. PMID:9083925. |
myelin oligodendrocyte glycoprotein: a novel candidate autoantigen in multiple sclerosis. |
1997-05-28 |
2023-08-12 |
mouse |
| J A Kamhol. Regulation of myelin development. Multiple sclerosis (Houndmills, Basingstoke, England). vol 2. issue 5. 1997-03-25. PMID:9050362. |
these three transcription factors are thus important for the normal process of oligodendrocyte differentiation and myelination, and may be involved in the molecular pathogenesis of both demyelination and remyelination in multiple sclerosis. |
1997-03-25 |
2023-08-12 |
Not clear |
| M Ichikawa, T G Johns, M Adelmann, C C Bernar. Antibody response in Lewis rats injected with myelin oligodendrocyte glycoprotein derived peptides. International immunology. vol 8. issue 11. 1997-03-03. PMID:8943561. |
previous studies from our laboratory have demonstrated a predominant response to myelin oligodendrocyte glycoprotein (mog) in patients with multiple sclerosis (ms) and showed that this molecule is able to induce in lewis rats a chronic relapsing ms-like disease with extensive demyelination. |
1997-03-03 |
2023-08-12 |
rat |
| B G Xiao, G X Zhang, C G Ma, H Lin. The cerebrospinal fluid from patients with multiple sclerosis promotes neuronal and oligodendrocyte damage by delayed production of nitric oxide in vitro. Journal of the neurological sciences. vol 142. issue 1-2. 1997-02-07. PMID:8902730. |
the cerebrospinal fluid from patients with multiple sclerosis promotes neuronal and oligodendrocyte damage by delayed production of nitric oxide in vitro. |
1997-02-07 |
2023-08-12 |
Not clear |
| B G Xiao, G X Zhang, C G Ma, H Lin. The cerebrospinal fluid from patients with multiple sclerosis promotes neuronal and oligodendrocyte damage by delayed production of nitric oxide in vitro. Journal of the neurological sciences. vol 142. issue 1-2. 1997-02-07. PMID:8902730. |
nitric oxide (no) may be involved in myelin and oligodendrocyte injury associated with multiple sclerosis (ms), a demyelinating disease of unknown etiology. |
1997-02-07 |
2023-08-12 |
Not clear |