| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| N J Scolding, P J Rayner, J Sussman, C Shaw, D A Compsto. A proliferative adult human oligodendrocyte progenitor. Neuroreport. vol 6. issue 3. 1995-07-06. PMID:7766839. |
the identification of an adult human oligodendrocyte progenitor is the first step towards developing interventional strategies for promoting repair of demyelinated lesions in patients with multiple sclerosis. |
1995-07-06 |
2023-08-12 |
human |
| T G Johns, N Kerlero de Rosbo, K K Menon, S Abo, M F Gonzales, C C Bernar. Myelin oligodendrocyte glycoprotein induces a demyelinating encephalomyelitis resembling multiple sclerosis. Journal of immunology (Baltimore, Md. : 1950). vol 154. issue 10. 1995-05-31. PMID:7537310. |
myelin oligodendrocyte glycoprotein induces a demyelinating encephalomyelitis resembling multiple sclerosis. |
1995-05-31 |
2023-08-12 |
rat |
| T G Johns, N Kerlero de Rosbo, K K Menon, S Abo, M F Gonzales, C C Bernar. Myelin oligodendrocyte glycoprotein induces a demyelinating encephalomyelitis resembling multiple sclerosis. Journal of immunology (Baltimore, Md. : 1950). vol 154. issue 10. 1995-05-31. PMID:7537310. |
the demonstrated involvement of myelin oligodendrocyte glycoprotein as a new demyelinating neural ag may provide a deeper insight into the pathogenesis of multiple sclerosis and its treatment. |
1995-05-31 |
2023-08-12 |
rat |
| K Ozawa, G Suchanek, H Breitschopf, W Brück, H Budka, K Jellinger, H Lassman. Patterns of oligodendroglia pathology in multiple sclerosis. Brain : a journal of neurology. vol 117 ( Pt 6). 1995-02-16. PMID:7820568. |
patterns of oligodendroglia pathology in multiple sclerosis. |
1995-02-16 |
2023-08-12 |
Not clear |
| K Ozawa, G Suchanek, H Breitschopf, W Brück, H Budka, K Jellinger, H Lassman. Patterns of oligodendroglia pathology in multiple sclerosis. Brain : a journal of neurology. vol 117 ( Pt 6). 1995-02-16. PMID:7820568. |
patterns of inflammation, demyelination and oligodendrocyte pathology were studied in acute multiple sclerosis and during early and late exacerbations of chronic multiple sclerosis. |
1995-02-16 |
2023-08-12 |
Not clear |
| J Roddy, I Clark, B L Hazleman, D A Compston, N J Scoldin. Cerebrospinal fluid concentrations of the complement MAC inhibitor CD59 in multiple sclerosis and patients with other neurological disorders. Journal of neurology. vol 241. issue 9. 1995-01-24. PMID:7528270. |
no significant differences were found between patients and controls, or between patients with active and those with clinically inactive demyelinating disease, providing no direct support for oligodendrocyte shedding of cd59 in multiple sclerosis. |
1995-01-24 |
2023-08-12 |
human |
| C Ffrench-Constant, B W Kiernan, R Milner, S Scott-Dre. Developmental studies of oligodendrocyte precursor cell migration and their implications for transplantation as therapy for multiple sclerosis. Eye (London, England). vol 8 ( Pt 2). 1994-12-19. PMID:7958026. |
developmental studies of oligodendrocyte precursor cell migration and their implications for transplantation as therapy for multiple sclerosis. |
1994-12-19 |
2023-08-12 |
Not clear |
| S D D'Souza, J P Antel, M S Freedma. Cytokine induction of heat shock protein expression in human oligodendrocytes: an interleukin-1-mediated mechanism. Journal of neuroimmunology. vol 50. issue 1. 1994-03-08. PMID:8300853. |
in this study, we examined the role of cytokines, known to be in elevated levels in multiple sclerosis (ms) plaques, in regulating oligodendrocyte (odc) expression of heat shock protein (hsp) in human brain-derived glial cell cultures. |
1994-03-08 |
2023-08-12 |
human |
| N Kerlero de Rosbo, R Milo, M B Lees, D Burger, C C Bernard, A Ben-Nu. Reactivity to myelin antigens in multiple sclerosis. Peripheral blood lymphocytes respond predominantly to myelin oligodendrocyte glycoprotein. The Journal of clinical investigation. vol 92. issue 6. 1994-01-12. PMID:7504688. |
although t cell responses to the quantitatively major myelin proteins, myelin basic protein (mbp) and proteolipid protein (plp), are likely to be of importance in the course of multiple sclerosis (ms), cell-mediated autoimmune responses to other myelin antigens, in particular quantitatively minor myelin antigens, such as myelin-associated glycoprotein (mag) and the central nervous system-specific myelin oligodendrocyte glycoprotein (mog), could also play a prevalent role in disease initiation or progression. |
1994-01-12 |
2023-08-12 |
Not clear |
| M Rodriguez, B W Scheithauer, G Forbes, P J Kell. Oligodendrocyte injury is an early event in lesions of multiple sclerosis. Mayo Clinic proceedings. vol 68. issue 7. 1993-09-14. PMID:8350635. |
oligodendrocyte injury is an early event in lesions of multiple sclerosis. |
1993-09-14 |
2023-08-12 |
Not clear |
| J E Merrill, L J Ignarro, M P Sherman, J Melinek, T E Lan. Microglial cell cytotoxicity of oligodendrocytes is mediated through nitric oxide. Journal of immunology (Baltimore, Md. : 1950). vol 151. issue 4. 1993-09-08. PMID:8102159. |
there is a correlation between ameboid microglial cell killing of oligodendrocytes and no2- production suggesting no may be a mechanism of death of the oligodendrocyte and possibly play a role in lesion formation in multiple sclerosis. |
1993-09-08 |
2023-08-12 |
rat |
| J B Su. Autoreactive T and B cells in nervous system diseases. Acta neurologica Scandinavica. Supplementum. vol 142. 1993-03-30. PMID:8382894. |
abnormal autoimmunity could play a role in the pathogenesis of multiple sclerosis (ms), in analogy with its model experimental allergic encephalomyelitis (eae) which can be transferred by t cell lines directed to myelin basic protein (mbp) and myelin proteolipid protein (plp), and worsened with antibody to myelin oligodendrocyte glycoprotein (mog). |
1993-03-30 |
2023-08-12 |
Not clear |
| V Jewtoukoff, S Amzazi, R Lebar, M A Bach, P N March. T-cell receptor identification of an oligodendrocyte-specific autoreactive cytotoxic T-cell clone without self restriction. Scandinavian journal of immunology. vol 36. issue 6. 1993-01-08. PMID:1462126. |
in addition to myelin basic protein (mbp) and proteolipid protein (plp), oligodendrocyte (od) membrane autoantigens, such as the glycoprotein m2/mog, could participate in the pathogenesis of autoimmune demyelinating diseases of the central nervous system (cns), such as experimental allergic encephalomyelitis (eae) or multiple sclerosis (ms). |
1993-01-08 |
2023-08-11 |
Not clear |
| J E Merrill, R P Zimmerma. Natural and induced cytotoxicity of oligodendrocytes by microglia is inhibitable by TGF beta. Glia. vol 4. issue 3. 1991-10-18. PMID:1832660. |
blood macrophages and brain macrophages (microglia) have been implicated in demyelination and destruction of the oligodendrocyte in multiple sclerosis (ms), a disease affecting primarily white matter of the central nervous system (cns). |
1991-10-18 |
2023-08-11 |
rat |
| S Fredrikson, J Sun, B G Xiao, H Lin. Cord blood contains cells secreting antibodies to nervous system components. Clinical and experimental immunology. vol 84. issue 2. 1991-06-11. PMID:1709072. |
umbilical cord blood of newborns and peripheral blood of healthy adults were investigated by an immunospot assay for cells secreting igg, iga and igm antibodies against myelin basic protein (mbp), proteolipid protein (plp), myelin-associated glycoprotein (mag) and myelin oligodendrocyte glycoprotein (mog) which represent putative antigens for an autoimmune attack in multiple sclerosis (ms) and against acetylcholine receptor (achr) which is considered an important autoantigen in myasthenia gravis. |
1991-06-11 |
2023-08-11 |
Not clear |
| J W Prineas, E E Kwon, P Z Goldenberg, E S Cho, L R Share. Interaction of astrocytes and newly formed oligodendrocytes in resolving multiple sclerosis lesions. Laboratory investigation; a journal of technical methods and pathology. vol 63. issue 5. 1990-12-19. PMID:1700195. |
it bears some resemblance, however, to a pattern of slow oligodendrocyte destruction seen previously in organotypic perinatal central nervous tissue cultures exposed to multiple sclerosis serum. |
1990-12-19 |
2023-08-11 |
Not clear |
| J W Prineas, E E Kwon, P Z Goldenberg, A A Ilyas, R H Quarles, J A Benjamins, T J Sprinkl. Multiple sclerosis. Oligodendrocyte proliferation and differentiation in fresh lesions. Laboratory investigation; a journal of technical methods and pathology. vol 61. issue 5. 1989-12-21. PMID:2811298. |
fresh lesions in the brain and spinal cord of patients with multiple sclerosis who died shortly after the onset of symptoms were examined immunocytochemically for myelin and oligodendrocyte antigens that are known to be sequentially expressed during normal development. |
1989-12-21 |
2023-08-11 |
Not clear |
| C S Raine, G R Moore, R Hintzen, U Traugot. Induction of oligodendrocyte proliferation and remyelination after chronic demyelination. Relevance to multiple sclerosis. Laboratory investigation; a journal of technical methods and pathology. vol 59. issue 4. 1988-11-21. PMID:2459499. |
observations from multiple sclerosis lesions showed that oligodendrocyte proliferation and cns remyelination occur in human subcortical white matter, but to a lesser degree than that seen in the cns of mbp/gc/treated guinea pigs. |
1988-11-21 |
2023-08-11 |
human |
| S B Elia. Oligodendrocyte development and the natural history of multiple sclerosis. A new hypothesis for the pathogenesis of the disease. Archives of neurology. vol 44. issue 12. 1987-12-17. PMID:3675264. |
oligodendrocyte development and the natural history of multiple sclerosis. |
1987-12-17 |
2023-08-11 |
Not clear |
| A Suzumura, R P Lisak, D H Silberber. Serum cytotoxicity to oligodendrocytes in multiple sclerosis and controls: assessment by 51Cr release assay. Journal of neuroimmunology. vol 11. issue 2. 1986-04-16. PMID:3949965. |
in order to quantitate oligodendrocyte cytotoxicity induced by sera from patients with multiple sclerosis (ms) and control sera, we established a 51cr release assay system using cultured oligodendrocytes as target cells. |
1986-04-16 |
2023-08-11 |
Not clear |