| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| H Nakayama, A Tamura, I Kanazawa, K San. Time-sequential change of amino acid neurotransmitters--GABA, aspartate and glutamate--in the rat basal ganglia following middle cerebral artery occlusion. Neurological research. vol 12. issue 4. 1991-05-10. PMID:1982166. |
time-sequential change of amino acid neurotransmitters--gaba, aspartate and glutamate--in the rat basal ganglia following middle cerebral artery occlusion. |
1991-05-10 |
2023-08-11 |
rat |
| H Nakayama, A Tamura, I Kanazawa, K San. Time-sequential change of amino acid neurotransmitters--GABA, aspartate and glutamate--in the rat basal ganglia following middle cerebral artery occlusion. Neurological research. vol 12. issue 4. 1991-05-10. PMID:1982166. |
to clarify the involvement of neurotransmitters in the remote metabolic effect following focal ischaemia, amino acid neurotransmitter candidates--gaba, aspartate and glutamate--were measured time-sequentially in the basal ganglia following middle cerebral artery occlusion in the rat, which is known to produce a change in glucose metabolism and blood flow in the remote basal ganglia. |
1991-05-10 |
2023-08-11 |
rat |
| b' V S Kosti\\xc4\\x87, L Mojsilovi\\xc4\\x87, M Stojanovi\\xc4\\x8. Degenerative neurological disorders associated with deficiency of glutamate dehydrogenase. Journal of neurology. vol 236. issue 2. 1989-05-31. PMID:2709051.' |
the activity of glutamate dehydrogenase, the enzyme of glutamate degradation, was measured in platelets of 27 healthy controls and 85 patients with different degenerative cerebellar and/or basal ganglia disorders. |
1989-05-31 |
2023-08-11 |
Not clear |
| U Andersson, J E Häggström, E D Levin, U Bondesson, M Valverius, L M Gunn. Reduced glutamate decarboxylase activity in the subthalamic nucleus in patients with tardive dyskinesia. Movement disorders : official journal of the Movement Disorder Society. vol 4. issue 1. 1989-05-03. PMID:2927401. |
glutamate decarboxylase (gad) activity was measured in the nuclei of the basal ganglia in patients with neuroleptic-induced tardive dyskinesia (td) and controls matched for age and premortem state. |
1989-05-03 |
2023-08-11 |
Not clear |
| T Greenamyre, J B Penney, A B Young, C Hudson, F S Silverstein, M V Johnsto. Evidence for transient perinatal glutamatergic innervation of globus pallidus. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 7. issue 4. 1987-05-29. PMID:2883265. |
because glutamate is an excitotoxin, this pathway may account, in part, for the basal ganglia damage seen in some forms of cerebral palsy after perinatal hypoxia/ischemia. |
1987-05-29 |
2023-08-11 |
human |
| D W Ellison, M F Beal, M F Mazurek, E D Bird, J B Marti. A postmortem study of amino acid neurotransmitters in Alzheimer's disease. Annals of neurology. vol 20. issue 5. 1986-12-24. PMID:2878639. |
glutamate, aspartate, taurine, gamma-aminobutyric acid (gaba), and alanine levels were examined in 9 cortical regions, hippocampus, thalamus, and basal ganglia using high-performance liquid chromatography with electrochemical detection. |
1986-12-24 |
2023-08-11 |
Not clear |
| C Köhler, L W Swanson, L Haglund, J Y W. The cytoarchitecture, histochemistry and projections of the tuberomammillary nucleus in the rat. Neuroscience. vol 16. issue 1. 1986-07-15. PMID:2423918. |
immunohistochemical studies indicate that a majority of the large neurons in all three parts of the nucleus stain with antisera against glutamate decarboxylase and [met]enkephalyl-arg6-phe7 heptapeptide and that a smaller subset of these neurons (about 10%) also stain with an antiserum against substance p. single injections of retrogradely transported fluorescent tracers were made into 18 different sites in 86 animals and the results indicate that all three parts of the tuberomammillary nucleus on one side of the brain send fibers to or through various parts of the neocortex, hippocampal formation, amygdala, basal ganglia, thalamus, superior colliculus and cerebellum on both sides of the brain and that the projection neurons are not organized in a highly topographic way. |
1986-07-15 |
2023-08-11 |
rat |
| b' I V Maslova, N V Shevchenko, A I Balakleevski\\xc4\\xa. [Metabolism of acetylcholine and gamma-aminobutyric acid in the basal ganglia and limbic cortex of rats at different stages in the development of alcohol dependence]. Voprosy meditsinskoi khimii. vol 32. issue 1. 1986-04-02. PMID:3953003.' |
these phase alterations in the activity of the ac structures in basal ganglia were accompanied by the similar alterations in content of glutamate: an increase of glutamate at the i step of alcoholism and a decrease--at the iii step. |
1986-04-02 |
2023-08-11 |
rat |
| C J Carte. Reduced GABA transaminase activity in the Huntington's disease putamen. Neuroscience letters. vol 48. issue 3. 1984-11-09. PMID:6237280. |
in both cases, the reduction in gaba transaminase activity is comparable with the reduction in glutamate decarboxylase activity, supporting the suspected localization of this enzyme to gaba neurones within the basal ganglia. |
1984-11-09 |
2023-08-12 |
rat |
| W H Oertel, E Mugnain. Immunocytochemical studies of GABAergic neurons in rat basal ganglia and their relations to other neuronal systems. Neuroscience letters. vol 47. issue 3. 1984-10-15. PMID:6147799. |
gabaergic neurons were localized in the rat basal ganglia by glutamate decarboxylase (gad) immunohistochemistry. |
1984-10-15 |
2023-08-12 |
rat |
| E G McGeer, W A Staines, P L McGee. Neurotransmitters in the basal ganglia. The Canadian journal of neurological sciences. Le journal canadien des sciences neurologiques. vol 11. issue 1 Suppl. 1984-05-29. PMID:6143612. |
emphasis is placed upon the diverse roles played by gaba and glutamate as transmitters in motor pathways and upon the probability that the substantia nigra pars reticulata plays a pivotal role in the output of the basal ganglia. |
1984-05-29 |
2023-08-12 |
Not clear |
| A Plaitakis, S Ber. Oral glutamate loading in disorders with spinocerebellar and extrapyramidal involvement: effect on plasma glutamate, aspartate and taurine. Journal of neural transmission. Supplementum. vol 19. 1984-04-04. PMID:6142089. |
to further investigate the specificity of these results, oral glutamate loading tests were performed in healthy controls, patients with gdh deficient opca as well as patients with non-gdh deficient degenerative disorders affecting primarily the function of the cerebellum and/or the basal ganglia. |
1984-04-04 |
2023-08-12 |
Not clear |
| A Plaitakis, S Berl, M D Yah. Neurological disorders associated with deficiency of glutamate dehydrogenase. Annals of neurology. vol 15. issue 2. 1984-03-30. PMID:6703655. |
glutamate dehydrogenase (gdh) activity was measured in leukocytes from 88 patients with various types of degenerative neurological disorders affecting primarily the cerebellum and/or the basal ganglia, and 26 healthy control subjects. |
1984-03-30 |
2023-08-12 |
human |
| C J Pycock, C J Carter, R W Kerwi. Effect of 6-hydroxydopamine lesions of the medial prefrontal cortex on neurotransmitter systems in subcortical sites in the rat. Journal of neurochemistry. vol 34. issue 1. 1981-03-17. PMID:6161214. |
decreased activity of the neurotransmitter-synthesizing enzyme glutamate decarboxylase and choline acetyltransferase was also recorded in certain regions of the basal ganglia. |
1981-03-17 |
2023-08-12 |
rat |