All Relations between serotonergic and brainstem

Publication Sentence Publish Date Extraction Date Species
Emily A Karanges, Linnet Ramos, Bruno Dampney, Anastasia S Suraev, Kong M Li, Iain S McGregor, Glenn E Hun. Contrasting regional Fos expression in adolescent and young adult rats following acute administration of the antidepressant paroxetine. Brain research bulletin. vol 121. 2016-12-13. PMID:26876759. these results indicate a different c-fos signature of acute paroxetine in adolescent rats, with greater activation in key mesolimbic and serotonergic regions, but a more subdued cortical, brainstem and hypothalamic response. 2016-12-13 2023-08-13 rat
Jong-Hoon Kim, Young-Don Son, Jeong-Hee Kim, Eun-Jung Choi, Sang-Yoon Lee, Yo-Han Joo, Young-Bo Kim, Zang-Hee Ch. Self-transcendence trait and its relationship with in vivo serotonin transporter availability in brainstem raphe nuclei: An ultra-high resolution PET-MRI study. Brain research. vol 1629. 2016-09-22. PMID:26459992. we examined the relationship between self-transcendence and serotonin transporter (sert) availability in brainstem raphe nuclei, which are collections of five different serotonergic nuclei with rostro-caudal extension, using ultra-high resolution magnetic resonance imaging (mri) and positron emission tomography (pet) with (11)c-3-amino-4-(2-dimethylaminomethylphenylthio)benzonitrile ([(11)c]dasb) to elucidate potential roles of serotonergic neuronal activities in this personality trait. 2016-09-22 2023-08-13 human
Melissa J Maguire, Cerian F Jackson, Anthony G Marson, Sarah J Nola. Treatments for the prevention of Sudden Unexpected Death in Epilepsy (SUDEP). The Cochrane database of systematic reviews. vol 7. 2016-09-21. PMID:27434597. preventative strategies for sudep include reducing the occurrence of gtcs by timely referral for presurgical evaluation in people with lesional epilepsy and advice on lifestyle measures; detecting cardiorespiratory distress through clinical observation and seizure, respiratory, and heart rate monitoring devices; preventing airway obstruction through nocturnal supervision and safety pillows; reducing central hypoventilation through physical stimulation and enhancing serotonergic mechanisms of respiratory regulation using selective serotonin reuptake inhibitors (ssris); reducing adenosine and endogenous opioid-induced brain and brainstem depression. 2016-09-21 2023-08-13 Not clear
Qiong Zhan, Gordon F Buchanan, Joshua E Motelow, John Andrews, Petr Vitkovskiy, William C Chen, Florian Serout, Abhijeet Gummadavelli, Adam Kundishora, Moran Furman, Wei Li, Xiao Bo, George B Richerson, Hal Blumenfel. Impaired Serotonergic Brainstem Function during and after Seizures. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 36. issue 9. 2016-08-16. PMID:26937010. impaired serotonergic brainstem function during and after seizures. 2016-08-16 2023-08-13 rat
Qiong Zhan, Gordon F Buchanan, Joshua E Motelow, John Andrews, Petr Vitkovskiy, William C Chen, Florian Serout, Abhijeet Gummadavelli, Adam Kundishora, Moran Furman, Wei Li, Xiao Bo, George B Richerson, Hal Blumenfel. Impaired Serotonergic Brainstem Function during and after Seizures. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 36. issue 9. 2016-08-16. PMID:26937010. lower brainstem serotonergic systems are postulated to play an important role in cardiorespiratory changes during and after seizures, whereas upper brainstem serotonergic and other systems regulate arousal. 2016-08-16 2023-08-13 rat
Qiong Zhan, Gordon F Buchanan, Joshua E Motelow, John Andrews, Petr Vitkovskiy, William C Chen, Florian Serout, Abhijeet Gummadavelli, Adam Kundishora, Moran Furman, Wei Li, Xiao Bo, George B Richerson, Hal Blumenfel. Impaired Serotonergic Brainstem Function during and after Seizures. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 36. issue 9. 2016-08-16. PMID:26937010. however, direct demonstration of seizure-associated neuronal activity changes in brainstem serotonergic regions has been lacking. 2016-08-16 2023-08-13 rat
Karlene T Barrett, Ryan T Dosumu-Johnson, J Andrew Daubenspeck, Rachael D Brust, Vasileios Kreouzis, Jun Chul Kim, Aihua Li, Susan M Dymecki, Eugene E Natti. Partial Raphe Dysfunction in Neurotransmission Is Sufficient to Increase Mortality after Anoxic Exposures in Mice at a Critical Period in Postnatal Development. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 36. issue 14. 2016-08-15. PMID:27053202. sudden infant death syndrome (sids) cases often have abnormalities of the brainstem raphe serotonergic (5-ht) system. 2016-08-15 2023-08-13 mouse
B J Dougherty, E J Gonzalez-Rothi, K Z Lee, H H Ross, P J Reier, D D Fulle. Respiratory outcomes after mid-cervical transplantation of embryonic medullary cells in rats with cervical spinal cord injury. Experimental neurology. vol 278. 2016-07-14. PMID:26808660. we hypothesized that intraspinal transplantation of embryonic midline brainstem (mb) cells rich in serotonergic raphé neurons would improve respiratory outcomes after csci. 2016-07-14 2023-08-13 rat
Petr Šilhán, Monika Jelínková, Uwe Walter, Ján Pavlov Praško, Roman Herzig, Kateřina Langová, David Školoudí. Transcranial sonography of brainstem structures in panic disorder. Psychiatry research. vol 234. issue 1. 2016-06-27. PMID:26371456. patients with panic disorder exhibit changes of brainstem raphe on tcs suggesting an alteration of the central serotonergic system. 2016-06-27 2023-08-13 human
Maria Cristina Greco, Pierluigi Navarra, Giuseppe Tringal. The analgesic agent tapentadol inhibits calcitonin gene-related peptide release from isolated rat brainstem via a serotonergic mechanism. Life sciences. vol 145. 2016-06-13. PMID:26706288. the analgesic agent tapentadol inhibits calcitonin gene-related peptide release from isolated rat brainstem via a serotonergic mechanism. 2016-06-13 2023-08-13 rat
Takashi Matsui, Shingo Soya, Kentaro Kawanaka, Hideaki Soy. Brain Glycogen Decreases During Intense Exercise Without Hypoglycemia: The Possible Involvement of Serotonin. Neurochemical research. vol 40. issue 7. 2016-04-11. PMID:26037553. activated serotonergic regulation is a possible underlying mechanism for intense exercise-induced glycogenolysis at least in the hippocampus and brainstem. 2016-04-11 2023-08-13 rat
Eunkyung Park, Yu Mi Hwang, Min Kyung Chu, Ki-Young Jun. Increased Brainstem Serotonergic Transporter Availability in Adult Migraineurs: an [(18)F]FP-CIT PET Imaging Pilot Study. Nuclear medicine and molecular imaging. vol 50. issue 1. 2016-03-04. PMID:26941862. increased brainstem serotonergic transporter availability in adult migraineurs: an [(18)f]fp-cit pet imaging pilot study. 2016-03-04 2023-08-13 Not clear
Janina Deusser, Stefanie Schmidt, Benjamin Ettle, Sonja Plötz, Sabine Huber, Christian P Müller, Eliezer Masliah, Jürgen Winkler, Zacharias Koh. Serotonergic dysfunction in the A53T alpha-synuclein mouse model of Parkinson's disease. Journal of neurochemistry. vol 135. issue 3. 2016-02-10. PMID:26201615. we detected human α-synuclein in the perikarya of brainstem median and dorsal raphe neurons as well as in serotonergic fibers in the hippocampus. 2016-02-10 2023-08-13 mouse
Dario Arnaldi, Francesco Famà, Fabrizio De Carli, Silvia Morbelli, Michela Ferrara, Agnese Picco, Jennifer Accardo, Alberto Primavera, Gianmario Sambuceti, Flavio Nobil. The Role of the Serotonergic System in REM Sleep Behavior Disorder. Sleep. vol 38. issue 9. 2016-01-14. PMID:25845692. we aimed to study brain stem serotonergic system integrity, by means of (123)i-fp-cit-spect, in a group of irbd patients as compared to normal subjects. 2016-01-14 2023-08-13 human
Hector Mateo-Carrasco, Eva María Muñoz-Aguilera, Juan Manuel García-Torrecillas, Hiba Abu Al-Rob. Serotonin syndrome probably triggered by a morphine-phenelzine interaction. Pharmacotherapy. vol 35. issue 6. 2016-01-04. PMID:25903219. serotonin syndrome is a potentially life-threatening condition caused by excessive central and peripheral stimulation of serotonin brainstem receptors, usually triggered by inadvertent interactions between agents with serotonergic activity. 2016-01-04 2023-08-13 Not clear
Zahi Qamhawi, David Towey, Bina Shah, Gennaro Pagano, John Seibyl, Kenneth Marek, Per Borghammer, David James Brooks, Nicola Paves. Clinical correlates of raphe serotonergic dysfunction in early Parkinson's disease. Brain : a journal of neurology. vol 138. issue Pt 10. 2015-12-28. PMID:26209314. post-mortem and neuroimaging studies suggest that the serotonergic system, which originates from the brainstem raphe nuclei, is disrupted in parkinson's disease. 2015-12-28 2023-08-13 human
Patrice G Guyenet, Douglas A Baylis. Neural Control of Breathing and CO2 Homeostasis. Neuron. vol 87. issue 5. 2015-11-23. PMID:26335642. specific brainstem neurons (e.g., retrotrapezoid nucleus, rtn; serotonergic) are activated by pco2 and stimulate breathing. 2015-11-23 2023-08-13 Not clear
Blair A Johnston, Serenella Tolomeo, Victoria Gradin, David Christmas, Keith Matthews, J Douglas Steel. Failure of hippocampal deactivation during loss events in treatment-resistant depression. Brain : a journal of neurology. vol 138. issue Pt 9. 2015-11-16. PMID:26133661. we conclude that the neuroimaging findings were largely consistent with deaken and graeff's predictions, abnormally increased hippocampal activity during loss events was an especially consistent abnormality, and brainstem serotonergic nuclei merit further study in depressive illness. 2015-11-16 2023-08-13 human
H E Reinebrant, J A Wixey, K M Bulle. Neonatal hypoxia-ischaemia disrupts descending neural inputs to dorsal raphé nuclei. Neuroscience. vol 248. 2015-11-03. PMID:23806712. nonetheless the mechanisms contributing to losses of serotonergic neurons in the brainstem remain to be elucidated. 2015-11-03 2023-08-12 rat
H E Reinebrant, J A Wixey, K M Bulle. Neonatal hypoxia-ischaemia disrupts descending neural inputs to dorsal raphé nuclei. Neuroscience. vol 248. 2015-11-03. PMID:23806712. since losses of descending inputs are associated with decreases in serotonergic neurons in the brainstem raphé nuclei, we propose that disruption of certain descending neural inputs from the forebrain to the dr dorsal and the dr ventral nuclei may contribute to losses of serotonergic neurons after p3 hi. 2015-11-03 2023-08-12 rat