All Relations between noradrenergic and dopamine

Publication Sentence Publish Date Extraction Date Species
S Jiang, R Xin, S Lin, Y Qian, G Tang, D Wang, X W. Linkage studies between attention-deficit hyperactivity disorder and the monoamine oxidase genes. American journal of medical genetics. vol 105. issue 8. 2002-03-05. PMID:11803531. molecular genetic and pharmacological studies suggest the involvement of dopaminergic and noradrenergic neurotransmitter systems in adhd, e.g., several reports have found association between adhd and the dopamine receptor gene drd-4, the dopamine transporter gene dat1, and the catecholamine clearance enzyme catechol-o-methyltransferase. 2002-03-05 2023-08-12 Not clear
Kengo Funakoshi, Masato Nakano, Yoshitoshi Atobe, Tetsuo Kadota, Richard C Goris, Reiji Kishid. Catecholaminergic innervation of the sympathetic preganglionic cell column of the filefish Stephanolepis cirrhifer. The Journal of comparative neurology. vol 442. issue 3. 2002-02-25. PMID:11774336. fluorescent double labeling distinguished noradrenergic nerve fibers immunoreactive for both tyrosine hydroxylase and dopamine beta hydroxylase, and dopaminergic fibers immunoreactive only for tyrosine hydroxylase. 2002-02-25 2023-08-12 Not clear
Kengo Funakoshi, Masato Nakano, Yoshitoshi Atobe, Tetsuo Kadota, Richard C Goris, Reiji Kishid. Catecholaminergic innervation of the sympathetic preganglionic cell column of the filefish Stephanolepis cirrhifer. The Journal of comparative neurology. vol 442. issue 3. 2002-02-25. PMID:11774336. double labeling of tyrosine hydroxylase and dopamine beta hydroxylase showed that the neurons in the locus coeruleus were all noradrenergic, and those in the caudal dorsomedial medulla were mostly noradrenergic, whereas the area postrema contained both noradrenergic and dopaminergic neurons. 2002-02-25 2023-08-12 Not clear
M D Jones, M E Williams, E J Hes. Expression of catecholaminergic mRNAs in the hyperactive mouse mutant coloboma. Brain research. Molecular brain research. vol 96. issue 1-2. 2002-02-14. PMID:11731016. alpha(2)-adrenergic receptors are important modulators of central noradrenergic function and dopamine release. 2002-02-14 2023-08-12 mouse
T Y Chen, S L Duh, C C Huang, T B Lin, D Y Ku. Evidence for the involvement of dopamine D(1) and D(2) receptors in mediating the decrease of food intake during repeated treatment with amphetamine. Journal of biomedical science. vol 8. issue 6. 2002-01-30. PMID:11702009. it is unknown whether these feeding changes are related to dopamine (da) and/or noradrenergic neurotransmission. 2002-01-30 2023-08-12 Not clear
Candice Drouin, Gérard Blanc, Anne-Sophie Villégier, Jacques Glowinski, Jean-Pol Tassi. Critical role of alpha1-adrenergic receptors in acute and sensitized locomotor effects of D-amphetamine, cocaine, and GBR 12783: influence of preexposure conditions and pharmacological characteristics. Synapse (New York, N.Y.). vol 43. issue 1. 2002-01-24. PMID:11746733. this possibility was tested on locomotor responses induced either by d-amphetamine and cocaine, which both interfere with noradrenergic and dopaminergic transmissions, or by gbr 12783, a specific dopamine reuptake inhibitor. 2002-01-24 2023-08-12 rat
P Devoto, G Flore, L Pani, G L Gess. Evidence for co-release of noradrenaline and dopamine from noradrenergic neurons in the cerebral cortex. Molecular psychiatry. vol 6. issue 6. 2002-01-23. PMID:11673793. evidence for co-release of noradrenaline and dopamine from noradrenergic neurons in the cerebral cortex. 2002-01-23 2023-08-12 Not clear
P Devoto, G Flore, L Pani, G L Gess. Evidence for co-release of noradrenaline and dopamine from noradrenergic neurons in the cerebral cortex. Molecular psychiatry. vol 6. issue 6. 2002-01-23. PMID:11673793. the aim of this study was to determine whether extracellular dopamine (da) in the prefrontal cortex (pfc) might originate other than from da neurons, also from noradrenergic (na) ones. 2002-01-23 2023-08-12 Not clear
G Eisenhofe. The role of neuronal and extraneuronal plasma membrane transporters in the inactivation of peripheral catecholamines. Pharmacology & therapeutics. vol 91. issue 1. 2001-12-19. PMID:11707293. in mammals, neuronal uptake of catecholamines involves the dopamine transporter (dat) at dopaminergic neurons and the norepinephrine transporter (net) at noradrenergic neurons. 2001-12-19 2023-08-12 Not clear
C Gottesmann, S Jonca. Letter to the editor: hypothesis for the neurophysiology of dreaming. Sleep research online : SRO. vol 3. issue 1. 2001-12-04. PMID:11382893. we hypothesize that, in addition to this unusual state, the persistent release of dopamine associated with the specific silence of noradrenergic neurons could explain the characteristics of dream mental activity which are somewhat similar to psychotic symptoms. 2001-12-04 2023-08-12 Not clear
K Yui, K Goto, S Ikemoto, K Nishijima, T Yoshino, T Ishigur. Susceptibility to subsequent episodes of spontaneous recurrence of methamphetamine psychosis. Drug and alcohol dependence. vol 64. issue 2. 2001-12-04. PMID:11543983. robust noradrenergic hyperactivity with slightly increased dopamine release may therefore predict subsequent flashbacks. 2001-12-04 2023-08-12 Not clear
D Y Hwang, W A Carlezon, O Isacson, K S Ki. A high-efficiency synthetic promoter that drives transgene expression selectively in noradrenergic neurons. Human gene therapy. vol 12. issue 14. 2001-12-04. PMID:11560767. to generate an efficient promoter system specific for noradrenergic (na) neurons, we multimerized an na-specific cis-regulatory element (prs2) identified in the human dopamine beta-hydroxylase (hdbh) promoter, and combined it with a minimal promoter (containing a tata box and transcription start site). 2001-12-04 2023-08-12 human
S Nishino, J Mao, R Sampathkumaran, J Shelto. Increased dopaminergic transmission mediates the wake-promoting effects of CNS stimulants. Sleep research online : SRO. vol 1. issue 1. 2001-10-18. PMID:11382857. in order to evaluate the relative importance of dopaminergic and noradrenergic transmission in the mediation of these effects, five dopamine (da) uptake inhibitors (mazindol, gbr-12909, bupropion, nomifensine and amineptine), two norepinephrine (ne) uptake inhibitors (nisoxetine and desipramine), d-amphetamine, and modafinil, a non-amphetamine stimulant, were tested in control and narcoleptic canines. 2001-10-18 2023-08-12 Not clear
P Szot, D Weinshenker, J M Rho, T W Storey, P A Schwartzkroi. Norepinephrine is required for the anticonvulsant effect of the ketogenic diet. Brain research. Developmental brain research. vol 129. issue 2. 2001-09-27. PMID:11506865. to determine if the noradrenergic system has a role in mediating the anticonvulsant action of the kd, dopamine beta-hydroxylase knockout (dbh -/-) mice that lack norepinephrine (ne) and dbh +/- littermates that have normal ne content were fed either a standard rodent chow or the kd. 2001-09-27 2023-08-12 mouse
Y Nod. [Possible mechanisms in latent learning formation investigated by using mutant mice]. Nihon yakurigaku zasshi. Folia pharmacologica Japonica. vol 117. issue 3. 2001-07-19. PMID:11288486. mice that received dopamine agonists, a noradrenergic neurotoxin or a traumatic brain injury showed impairment of latent learning. 2001-07-19 2023-08-12 mouse
W P De Potter, C P Chanh, F De Smet, A F De Schaepdryve. The presence of dopamine beta-hydroxylase in the cerebrospinal fluid of rabbits and its increased concentration after stimulation of peripheral nerves and cold stress. Neuroscience. vol 1. issue 6. 2001-07-12. PMID:11370247. it is suggested that the dopamine beta-hydroxylase activity in cerebrospinal fluid may be derived from noradrenergic neurons within the brain and that the enzyme is released together with noradrenaline. 2001-07-12 2023-08-12 rabbit
S F Bartlett, H Lagercrantz, A D Smit. Gel electrophoresis of soluble and insoluble proteins of noradrenergic vesicles from ox splenic nerve: a comparison with proteins of adrenal chromaffin granules. Neuroscience. vol 1. issue 4. 2001-07-12. PMID:11370518. the relative staining intensities of the latter two proteins were different: in noradrenergic vesicles, there was more dopamine beta-hydroxylase than chromogranin a; whereas in chromaffin granules, chromogranin a was the major protein. 2001-07-12 2023-08-12 Not clear
S F Bartlett, H Lagercrantz, A D Smit. Gel electrophoresis of soluble and insoluble proteins of noradrenergic vesicles from ox splenic nerve: a comparison with proteins of adrenal chromaffin granules. Neuroscience. vol 1. issue 4. 2001-07-12. PMID:11370518. these findings are consistent with earlier studies in which dopamine beta-hydroxylase, chromogranin a and chromomembrin b have been identified as constituents of noradrenergic vesicles by enzymatic or immunochemical assay methods. 2001-07-12 2023-08-12 Not clear
S F Bartlett, H Lagercrantz, A D Smit. Gel electrophoresis of soluble and insoluble proteins of noradrenergic vesicles from ox splenic nerve: a comparison with proteins of adrenal chromaffin granules. Neuroscience. vol 1. issue 4. 2001-07-12. PMID:11370518. however, gel electrophoresis also confirms that quantitative differences exist between the relative proportions of the soluble proteins and shows that dopamine beta-hydroxylase is the major soluble protein of the noradrenergic vesicles isolated from splenic nerve trunks. 2001-07-12 2023-08-12 Not clear
D S Melchitzky, D A Lewi. Dopamine transporter-immunoreactive axons in the mediodorsal thalamic nucleus of the macaque monkey. Neuroscience. vol 103. issue 4. 2001-06-28. PMID:11301210. in contrast, noradrenergic axons, as revealed by dopamine beta-hydroxylase immunoreactivity, were present in higher density and were more evenly distributed throughout the mediodorsal thalamic nucleus. 2001-06-28 2023-08-12 monkey