All Relations between noradrenergic and dopamine

Publication Sentence Publish Date Extraction Date Species
Paola Devoto, Giovanna Flore, Luigi Pira, Giorgio Longu, Gian Luigi Gess. Mirtazapine-induced corelease of dopamine and noradrenaline from noradrenergic neurons in the medial prefrontal and occipital cortex. European journal of pharmacology. vol 487. issue 1-3. 2004-11-24. PMID:15033381. our previous studies indicate that extracellular dopamine in the cerebral cortex originates largely from noradrenergic terminals, such release being controlled by alpha(2)-adrenoceptors. 2004-11-24 2023-08-12 Not clear
Paola Devoto, Giovanna Flore, Luigi Pira, Giorgio Longu, Gian Luigi Gess. Mirtazapine-induced corelease of dopamine and noradrenaline from noradrenergic neurons in the medial prefrontal and occipital cortex. European journal of pharmacology. vol 487. issue 1-3. 2004-11-24. PMID:15033381. by means of microdialysis, the effect of mirtazapine on extracellular dopamine, 3,4-dihydroxyphenylacetic acid (dopac) and noradrenaline in the medial prefrontal cortex, densely innervated by dopaminergic and noradrenergic neurons, and in the occipital cortex, receiving equal noradrenergic but scarce dopaminergic projections, was compared. 2004-11-24 2023-08-12 Not clear
Paola Devoto, Giovanna Flore, Luigi Pira, Giorgio Longu, Gian Luigi Gess. Mirtazapine-induced corelease of dopamine and noradrenaline from noradrenergic neurons in the medial prefrontal and occipital cortex. European journal of pharmacology. vol 487. issue 1-3. 2004-11-24. PMID:15033381. basal extracellular concentration of noradrenaline was similar in both cortices, while dopamine in the occipital cortex was only about 50% lower than in the medial prefrontal cortex, reflecting noradrenergic rather than dopaminergic projections. 2004-11-24 2023-08-12 Not clear
Paola Devoto, Giovanna Flore, Luigi Pira, Giorgio Longu, Gian Luigi Gess. Mirtazapine-induced corelease of dopamine and noradrenaline from noradrenergic neurons in the medial prefrontal and occipital cortex. European journal of pharmacology. vol 487. issue 1-3. 2004-11-24. PMID:15033381. the results suggest that mirtazapine by inhibiting alpha(2)-adrenoceptors produces a corelease of noradrenaline and dopamine from noradrenergic terminals in the cerebral cortex. 2004-11-24 2023-08-12 Not clear
Anna Zacharko, Marcin Bartłomiej Arciszewski, Krzysztof Wasowic. Origin of the primary efferent neurons projecting to the prostate of the dog. Annals of anatomy = Anatomischer Anzeiger : official organ of the Anatomische Gesellschaft. vol 186. issue 4. 2004-11-22. PMID:15481842. immunohistochemistry revealed that in all tyrosine hydroxylase immunoreactive fast blue-positive neurons immunoreactivity for dopamine beta-hydroxylase was also found (noradrenergic neurons) while all tyrosine hydroxylase-negative fast blue-positive neurons did not express dopamine beta-hydroxylase (non-noradrenergic neurons). 2004-11-22 2023-08-12 dog
Johannes Tauscher, Tabasum Hussain, Ofer Agid, N Paul L G Verhoeff, Alan A Wilson, Sylvain Houle, Gary Remington, Robert B Zipursky, Shitij Kapu. Equivalent occupancy of dopamine D1 and D2 receptors with clozapine: differentiation from other atypical antipsychotics. The American journal of psychiatry. vol 161. issue 9. 2004-10-07. PMID:15337652. its affinity for dopamine d(4) receptors, serotonin 5-ht(2a) receptor antagonism, effects on the noradrenergic system, and its relatively moderate occupancy of d(2) receptors are unlikely to be the critical mechanism underlying its efficacy. 2004-10-07 2023-08-12 Not clear
René Hummerich, Gerald Reischl, Walter Ehrlichmann, Hans-Jürgen Machulla, Andreas Heinz, Patrick Schlos. DASB -in vitro binding characteristics on human recombinant monoamine transporters with regard to its potential as positron emission tomography (PET) tracer. Journal of neurochemistry. vol 90. issue 5. 2004-09-30. PMID:15312176. previous positron emission tomography (pet) and single photon emission computed tomography (spect) studies were often carried out using radioligands that bind with high affinity to serts in the brainstem but also exhibit high affinity for dopamine and norepinephrine transporters and therefore did not allow quantification of serotonergic innervations in brain regions also containing dopaminergic or noradrenergic terminals. 2004-09-30 2023-08-12 human
Claude Gottesman. Brain inhibitory mechanisms involved in basic and higher integrated sleep processes. Brain research. Brain research reviews. vol 45. issue 3. 2004-09-07. PMID:15210306. for sleep mentation, the noradrenergic and serotonergic silence is thought, today, to be directly, or indirectly, responsible for dopamine predominance and glutamate decrease in the nucleus accumbens, which could be the background of the well-known psychotic-like mental activity of dreaming. 2004-09-07 2023-08-12 Not clear
Carlos A Paladini, John T William. Noradrenergic inhibition of midbrain dopamine neurons. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 24. issue 19. 2004-08-13. PMID:15140928. noradrenergic inhibition of midbrain dopamine neurons. 2004-08-13 2023-08-12 Not clear
Carlos A Paladini, John T William. Noradrenergic inhibition of midbrain dopamine neurons. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 24. issue 19. 2004-08-13. PMID:15140928. the results suggest that noradrenergic innervation of dopamine cells can directly inhibit the activity of dopamine cells. 2004-08-13 2023-08-12 Not clear
James R Shoblock, Isabelle M Maisonneuve, Stanley D Glic. Differential interactions of desipramine with amphetamine and methamphetamine: evidence that amphetamine releases dopamine from noradrenergic neurons in the medial prefrontal cortex. Neurochemical research. vol 29. issue 7. 2004-08-10. PMID:15202777. differential interactions of desipramine with amphetamine and methamphetamine: evidence that amphetamine releases dopamine from noradrenergic neurons in the medial prefrontal cortex. 2004-08-10 2023-08-12 rat
Wynn H T Pan, Sze-Yuan Yang, Shi-Kwang Li. Neurochemical interaction between dopaminergic and noradrenergic neurons in the medial prefrontal cortex. Synapse (New York, N.Y.). vol 53. issue 1. 2004-07-16. PMID:15150740. growing evidence indicates that there is an interaction between the transmission of dopamine (da) and norepinephrine (ne) in the noradrenergic and dopaminergic projections that converge in the medial prefrontal cortex (mpfc). 2004-07-16 2023-08-12 rat
Carlos A Paladini, Jennifer M Mitchell, John T Williams, Gregory P Mar. Cocaine self-administration selectively decreases noradrenergic regulation of metabotropic glutamate receptor-mediated inhibition in dopamine neurons. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 24. issue 22. 2004-07-09. PMID:15175391. cocaine self-administration selectively decreases noradrenergic regulation of metabotropic glutamate receptor-mediated inhibition in dopamine neurons. 2004-07-09 2023-08-12 rat
Carlos A Paladini, Jennifer M Mitchell, John T Williams, Gregory P Mar. Cocaine self-administration selectively decreases noradrenergic regulation of metabotropic glutamate receptor-mediated inhibition in dopamine neurons. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 24. issue 22. 2004-07-09. PMID:15175391. stimulant drugs of abuse have several effects on neural activity, including altering the excitability of dopamine neurons via the noradrenergic and glutamatergic systems. 2004-07-09 2023-08-12 rat
Carlos A Paladini, Jennifer M Mitchell, John T Williams, Gregory P Mar. Cocaine self-administration selectively decreases noradrenergic regulation of metabotropic glutamate receptor-mediated inhibition in dopamine neurons. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 24. issue 22. 2004-07-09. PMID:15175391. the noradrenergic system could therefore serve to alter the reward value of stimuli that have significant effects on dopamine neuron firing pattern through mglurs. 2004-07-09 2023-08-12 rat
Suzan Dziennis, Beth A Habecke. Ciliary neurotrophic factor suppresses Phox2a in sympathetic neurons. Neuroreport. vol 15. issue 1. 2004-05-27. PMID:15106827. in the rat, this includes suppression of the noradrenergic enzymes tyrosine hydroxylase and dopamine beta-hydroxylase. 2004-05-27 2023-08-12 mouse
David S Goldstei. Imaging of the autonomic nervous system: focus on cardiac sympathetic innervation. Seminars in neurology. vol 23. issue 4. 2004-05-19. PMID:15088263. because parkinson's disease entails a postganglionic sympathetic noradrenergic lesion, the disease appears to be not only a movement disorder, with dopamine loss in the nigrostriatal system of the brain, but also a dysautonomia, with noradrenaline loss in the sympathetic nervous system of the heart. 2004-05-19 2023-08-12 Not clear
Erik G Jönsson, Rami Abou Jamra, Johannes Schumacher, Lena Flyckt, Gunnar Edman, Kaj Forslund, Marja Mattila-Evenden, Gunnar Rylander, Marie Asberg, Lars Bjerkenstedt, Frits-Axel Wiesel, Peter Propping, Sven Cichon, Markus M Nöthen, Göran C Sedval. No association between a putative functional promoter variant in the dopamine beta-hydroxylase gene and schizophrenia. Psychiatric genetics. vol 13. issue 3. 2004-05-05. PMID:12960750. dopamine beta-hydroxylase catalyses the conversion of dopamine to norepinephrine in noradrenergic cells. 2004-05-05 2023-08-12 Not clear
J Srinivasan, W J Schmid. Functional recovery of locus coeruleus noradrenergic neurons after DSP-4 lesion: effects on dopamine levels and neuroleptic induced-parkinsonian symptoms in rats. Journal of neural transmission (Vienna, Austria : 1996). vol 111. issue 1. 2004-04-30. PMID:14714212. functional recovery of locus coeruleus noradrenergic neurons after dsp-4 lesion: effects on dopamine levels and neuroleptic induced-parkinsonian symptoms in rats. 2004-04-30 2023-08-12 rat
J Srinivasan, W J Schmid. Functional recovery of locus coeruleus noradrenergic neurons after DSP-4 lesion: effects on dopamine levels and neuroleptic induced-parkinsonian symptoms in rats. Journal of neural transmission (Vienna, Austria : 1996). vol 111. issue 1. 2004-04-30. PMID:14714212. noradrenergic lesion with n-(2-chloroethyl)-n-ethyl-2-bromobenzylamine (dsp-4) decreases dopamine release in the striatum and enhances catalepsy in experimental models of parkinson's disease. 2004-04-30 2023-08-12 rat