All Relations between hypertrophic and matrix compartment

Publication Sentence Publish Date Extraction Date Species
Jung-Tak Kim, Sang-Heum Baek, Sang-Han Lee, Eui Kyun Park, Eun-Cheol Kim, In-Sook Kwun, Hong-In Shi. Zinc-deficient diet decreases fetal long bone growth through decreased bone matrix formation in mice. Journal of medicinal food. vol 12. issue 1. 2009-05-29. PMID:19298204. the fetal developing tibia of the zinc-deficient group showed marked shortening of diaphysis and a mild narrowing of the hypertrophic chondrocyte zone width with increased osteoclast number, but there was no influence on the mineralization of bone matrix. 2009-05-29 2023-08-12 mouse
E J Mackie, Y A Ahmed, L Tatarczuch, K-S Chen, M Miram. Endochondral ossification: how cartilage is converted into bone in the developing skeleton. The international journal of biochemistry & cell biology. vol 40. issue 1. 2009-05-01. PMID:17659995. during endochondral ossification, chondrocytes proliferate, undergo hypertrophy and die; the cartilage extracellular matrix they construct is then invaded by blood vessels, osteoclasts, bone marrow cells and osteoblasts, the last of which deposit bone on remnants of cartilage matrix. 2009-05-01 2023-08-12 Not clear
Denise L Cecil, C Thomas G Appleton, Monika D Polewski, John S Mort, Ann Marie Schmidt, Alison Bendele, Frank Beier, Robert Terkeltau. The pattern recognition receptor CD36 is a chondrocyte hypertrophy marker associated with suppression of catabolic responses and promotion of repair responses to inflammatory stimuli. Journal of immunology (Baltimore, Md. : 1950). vol 182. issue 8. 2009-04-23. PMID:19342682. multiple inflammatory mediators in osteoarthritis (oa) cartilage, including s100/calgranulin ligands of receptor for advanced glycation end products (rage), promote chondrocyte hypertrophy, a differentiation state associated with matrix catabolism. 2009-04-23 2023-08-12 mouse
Denise L Cecil, C Thomas G Appleton, Monika D Polewski, John S Mort, Ann Marie Schmidt, Alison Bendele, Frank Beier, Robert Terkeltau. The pattern recognition receptor CD36 is a chondrocyte hypertrophy marker associated with suppression of catabolic responses and promotion of repair responses to inflammatory stimuli. Journal of immunology (Baltimore, Md. : 1950). vol 182. issue 8. 2009-04-23. PMID:19342682. however, cd36 transfection in normal human knee-immortalized chondrocytes (ch-8 cells) was associated with decreased capacity of s100a11 and tnf-alpha to induce chondrocyte hypertrophy and adamts-4 and matrix metalloproteinase 13 expression. 2009-04-23 2023-08-12 mouse
Serafim M Oliveira, Isabel F Amaral, Mário A Barbosa, Cristina C Teixeir. Engineering endochondral bone: in vitro studies. Tissue engineering. Part A. vol 15. issue 3. 2009-04-16. PMID:18759672. ra treatment caused chondrocyte hypertrophy, characterized by the presence of type x collagen in the extracellular matrix and increased alkaline phosphatase activity. 2009-04-16 2023-08-12 chicken
Luisa Lassová, Zeling Niu, Eleanor B Golden, Arthur J Cohen, Sherrill L Adam. Thyroid hormone treatment of cultured chondrocytes mimics in vivo stimulation of collagen X mRNA by increasing BMP 4 expression. Journal of cellular physiology. vol 219. issue 3. 2009-04-16. PMID:19170125. during endochondral bone formation, chondrocytes undergo terminal differentiation, during which the rate of proliferation decreases, cells become hypertrophic, and the extracellular matrix is altered by production of collagen x, as well as proteins required for matrix mineralization. 2009-04-16 2023-08-12 Not clear
Konstantinos Tziomalos, Joshua M Har. Role of xanthine oxidoreductase in cardiac nitroso-redox imbalance. Frontiers in bioscience (Landmark edition). vol 14. 2009-04-07. PMID:19273066. within the heart, xor activity stimulates cardiomyocyte hypertrophy, apoptosis, and impairs matrix structure. 2009-04-07 2023-08-12 Not clear
Narayana Anilkumar, Alexander Sirker, Ajay M Sha. Redox sensitive signaling pathways in cardiac remodeling, hypertrophy and failure. Frontiers in bioscience (Landmark edition). vol 14. 2009-04-07. PMID:19273265. in particular, the tightly regulated generation of ros by nadph oxidases appears especially important in key signaling events that drive the development of cardiomyocyte hypertrophy, fibrosis, extracellular matrix remodelling and cell apoptosis. 2009-04-07 2023-08-12 Not clear
Kristin Schram, Gary Sweene. Implications of myocardial matrix remodeling by adipokines in obesity-related heart failure. Trends in cardiovascular medicine. vol 18. issue 6. 2009-04-02. PMID:19185809. the events that these adipokines can regulate include alterations in myocardial metabolism, cardiomyocyte hypertrophy, cell death, and structure and composition of the extracellular matrix. 2009-04-02 2023-08-12 Not clear
George Osol, Maurizio Mandal. Maternal uterine vascular remodeling during pregnancy. Physiology (Bethesda, Md.). vol 24. 2009-04-01. PMID:19196652. the process of remodeling involves a number of cellular processes, including hyperplasia and hypertrophy, rearrangement of existing elements, and changes in extracellular matrix. 2009-04-01 2023-08-12 Not clear
S Tanriverdi-Akhisaroglu, A Menderes, G Okta. Matrix metalloproteinase-2 and -9 activities in human keloids, hypertrophic and atrophic scars: a pilot study. Cell biochemistry and function. vol 27. issue 2. 2009-03-30. PMID:19165813. matrix metalloproteinase-2 and -9 activities in human keloids, hypertrophic and atrophic scars: a pilot study. 2009-03-30 2023-08-12 human
Hiroyuki Tsutsui, Shintaro Kinugawa, Shouji Matsushim. Mitochondrial oxidative stress and dysfunction in myocardial remodelling. Cardiovascular research. vol 81. issue 3. 2009-03-26. PMID:18854381. reactive oxygen species induce myocyte hypertrophy, apoptosis, and interstitial fibrosis by activating matrix metalloproteinases. 2009-03-26 2023-08-12 Not clear
Kevin V Lemle. Diabetes and chronic kidney disease: lessons from the Pima Indians. Pediatric nephrology (Berlin, Germany). vol 23. issue 11. 2009-03-23. PMID:18320236. this population well illustrates the often inexorable progression from glomerular hyperfiltration to microalbuminuria to overt proteinuria and loss of glomerular filtration rate (gfr), paralleled by the accumulation of mesangial matrix and basement membrane, glomerular hypertrophy, loss of podocytes and eventual glomerular sclerosis and interstitial fibrosis. 2009-03-23 2023-08-12 Not clear
Mohamed S Elkelini, Karen Aitken, Darius J Bagli, Magdy M Hassoun. Effects of doxycycline on voiding behaviour of rats with bladder outlet obstruction. BJU international. vol 103. issue 4. 2009-03-23. PMID:18990141. to examine the voiding behaviour changes in rats with bladder outlet obstruction (boo) while inhibiting matrix metalloproteinase (mmp) activity with doxycycline, as increased mmp activity may be involved in obstruction-induced bladder hypertrophy. 2009-03-23 2023-08-12 rat
C Tselepis, J A Hoyland, R E Barber, B H Thorp, A P Kwa. Expression and distribution of cartilage matrix macromolecules in avian tibial dyschondroplasia. Avian pathology : journal of the W.V.P.A. vol 25. issue 2. 2009-03-10. PMID:18645860. normal hypertrophic cartilage showed a strong matrix labelling for aggrecan and biglycan. 2009-03-10 2023-08-12 Not clear
C Tselepis, J A Hoyland, R E Barber, B H Thorp, A P Kwa. Expression and distribution of cartilage matrix macromolecules in avian tibial dyschondroplasia. Avian pathology : journal of the W.V.P.A. vol 25. issue 2. 2009-03-10. PMID:18645860. type x collagen in the normal hypertrophic cartilage showed strong pericellular and matrix distribution, whereas in td cartilage labelling for aggrecan, biglycan and collagen x was located intracellularly with a very low level of signal in the matrix. 2009-03-10 2023-08-12 Not clear
Abdi Ghaffari, Ruhangiz T Kilani, Aziz Ghahar. Keratinocyte-conditioned media regulate collagen expression in dermal fibroblasts. The Journal of investigative dermatology. vol 129. issue 2. 2009-02-27. PMID:18787532. excessive extracellular matrix (ecm) production during dermal wound healing often leads to fibrotic conditions such as keloids and hypertrophic scarring (hsc). 2009-02-27 2023-08-12 human
Muneyoshi Okada, Ryuta Kikuzuki, Toshiyuki Harada, Yasutomo Hori, Hideyuki Yamawaki, Yukio Har. Captopril attenuates matrix metalloproteinase-2 and -9 in monocrotaline-induced right ventricular hypertrophy in rats. Journal of pharmacological sciences. vol 108. issue 4. 2009-02-19. PMID:19057128. captopril attenuates matrix metalloproteinase-2 and -9 in monocrotaline-induced right ventricular hypertrophy in rats. 2009-02-19 2023-08-12 rat
Fuad N Ziyadeh, Gunter Wol. Pathogenesis of the podocytopathy and proteinuria in diabetic glomerulopathy. Current diabetes reviews. vol 4. issue 1. 2009-02-13. PMID:18220694. transforming growth factor-beta 1 (tgf-beta1) has emerged as an important downstream mediator for the development of renal hypertrophy and the accumulation of mesangial extracellular matrix components, but there is limited evidence to support its role in the development of albuminuria. 2009-02-13 2023-08-12 Not clear
Cyril Thouverey, Agnieszka Strzelecka-Kiliszek, Marcin Balcerzak, René Buchet, Slawomir Pikul. Matrix vesicles originate from apical membrane microvilli of mineralizing osteoblast-like Saos-2 cells. Journal of cellular biochemistry. vol 106. issue 1. 2009-02-13. PMID:19009559. in bone, mineralization is tightly regulated by osteoblasts and hypertrophic chondrocytes which release matrix vesicles (mvs) and control extracellular ionic conditions and matrix composition. 2009-02-13 2023-08-12 Not clear