| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| E J Karle, K D Anderson, L Medina, A Reine. Light and electron microscopic immunohistochemical study of dopaminergic terminals in the striatal portion of the pigeon basal ganglia using antisera against tyrosine hydroxylase and dopamine. The Journal of comparative neurology. vol 369. issue 1. 1996-11-07. PMID:8723706. |
light and electron microscopic immunohistochemical study of dopaminergic terminals in the striatal portion of the pigeon basal ganglia using antisera against tyrosine hydroxylase and dopamine. |
1996-11-07 |
2023-08-12 |
pigeon |
| E J Karle, K D Anderson, L Medina, A Reine. Light and electron microscopic immunohistochemical study of dopaminergic terminals in the striatal portion of the pigeon basal ganglia using antisera against tyrosine hydroxylase and dopamine. The Journal of comparative neurology. vol 369. issue 1. 1996-11-07. PMID:8723706. |
in the present study, we used immunohistochemical labeling with antibodies against tyrosine hydroxylase (th) or dopamine (da) to study the dopaminergic input to the striatal portion of the basal ganglia in pigeons (i.e., lobus parolfactorius and paleostriatum augmentatum). |
1996-11-07 |
2023-08-12 |
pigeon |
| A Pinna, G di Chiara, J Wardas, M Morell. Blockade of A2a adenosine receptors positively modulates turning behaviour and c-Fos expression induced by D1 agonists in dopamine-denervated rats. The European journal of neuroscience. vol 8. issue 6. 1996-11-06. PMID:8752587. |
in rats with unilateral 6-hydroxydopamine lesions of the dopaminergic nigrostriatal pathway, administration of the a2a adenosine antagonist sch 58261 alone did not induce any motor asymmetry but strongly potentiated the contralateral turning behaviour induced by the dopamine d1 agonist skf 38393. |
1996-11-06 |
2023-08-12 |
rat |
| B A Bennett, D E Rusyniak, C K Hollingswort. HIV-1 gp120-induced neurotoxicity to midbrain dopamine cultures. Brain research. vol 705. issue 1-2. 1996-11-06. PMID:8821747. |
exposure of rat midbrain dopamine cultures to gp120 for 3 days reduced the ability of dopaminergic cells to transport this amine and also resulted in a reduction in dopamine neuron process length while it did not alter either dopamine cell number or the total number of neuronal cells. |
1996-11-06 |
2023-08-12 |
rat |
| O Dkhissi, E Chanut, C Versaux-Botteri, F Minvielle, J H Trouvin, J Nguyen-Legro. Changes in retinal dopaminergic cells and dopamine rhythmic metabolism during the development of a glaucoma-like disorder in quails. Investigative ophthalmology & visual science. vol 37. issue 11. 1996-11-06. PMID:8843918. |
changes in retinal dopaminergic cells and dopamine rhythmic metabolism during the development of a glaucoma-like disorder in quails. |
1996-11-06 |
2023-08-12 |
Not clear |
| A B Mulder, I Manshanden, P E Vos, G Wolterink, J M van Ree, F H Lopes da Silv. Modifications in glutamatergic transmission after dopamine depletion of the nucleus accumbens. A combined in vivo/in vitro electrophysiological study in the rat. Neuroscience. vol 72. issue 4. 1996-11-01. PMID:8735226. |
the interaction between the glutamatergic and dopaminergic input in the nucleus accumbens was examined by studying the effects of dopamine depletion of the nucleus accumbens on the local field potentials, and the l-glutamate elicited responses of the nucleus accumbens in anaesthetized rats in vivo. |
1996-11-01 |
2023-08-12 |
rat |
| W H Frishman, H Hotchkis. Selective and nonselective dopamine receptor agonists: an innovative approach to cardiovascular disease treatment. American heart journal. vol 132. issue 4. 1996-11-01. PMID:8831378. |
dopamine and a new group of selective and nonselective peripheral dopaminergic receptor effectors are being evaluated for the treatment of various cardiovascular disorders, including shock, chf, and systemic hypertension. |
1996-11-01 |
2023-08-12 |
Not clear |
| M Laruelle, A Abi-Dargham, C H van Dyck, R Gil, C D D'Souza, J Erdos, E McCance, W Rosenblatt, C Fingado, S S Zoghbi, R M Baldwin, J P Seibyl, J H Krystal, D S Charney, R B Inni. Single photon emission computerized tomography imaging of amphetamine-induced dopamine release in drug-free schizophrenic subjects. Proceedings of the National Academy of Sciences of the United States of America. vol 93. issue 17. 1996-10-31. PMID:8799184. |
the dopamine hypothesis of schizophrenia proposes that hyperactivity of dopaminergic transmission is associated with this illness, but direct observation of abnormalities of dopamine function in schizophrenia has remained elusive. |
1996-10-31 |
2023-08-12 |
human |
| L M Shulman, D A Fo. Dopamine inhibits mammalian photoreceptor Na+,K+-ATPase activity via a selective effect on the alpha3 isozyme. Proceedings of the National Academy of Sciences of the United States of America. vol 93. issue 15. 1996-10-29. PMID:8755598. |
the rat retina contains dopaminergic interplexiform cells that send processes to the outer plexiform layer where dopamine is released in a light-dependent manner. |
1996-10-29 |
2023-08-12 |
rat |
| M Contin, R Riva, F Albani, A Baruzz. Pharmacokinetic optimisation in the treatment of Parkinson's disease. Clinical pharmacokinetics. vol 30. issue 6. 1996-10-29. PMID:8792058. |
the current symptomatic treatment of parkinson's disease mainly relies on agents which are able to restore dopaminergic transmission in the nigrostriatal pathway, such as the dopamine precursor levodopa or direct agonists of dopamine receptors. |
1996-10-29 |
2023-08-12 |
Not clear |
| J L Cornish, M van den Buus. Stimulation of the rat mesolimbic dopaminergic system produces a pressor response which is mediated by dopamine D-1 and D-2 receptor activation and the release of vasopressin. Brain research. vol 701. issue 1-2. 1996-10-25. PMID:8925292. |
stimulation of the rat mesolimbic dopaminergic system produces a pressor response which is mediated by dopamine d-1 and d-2 receptor activation and the release of vasopressin. |
1996-10-25 |
2023-08-12 |
rat |
| J L Cornish, M van den Buus. Stimulation of the rat mesolimbic dopaminergic system produces a pressor response which is mediated by dopamine D-1 and D-2 receptor activation and the release of vasopressin. Brain research. vol 701. issue 1-2. 1996-10-25. PMID:8925292. |
we have investigated the effect on blood pressure and heart rate of stimulating the release of endogenous dopamine in the brain from the mesolimbic/mesocortical (a10) dopaminergic system of conscious sprague-dawley rats. |
1996-10-25 |
2023-08-12 |
rat |
| J L Cornish, M van den Buus. Stimulation of the rat mesolimbic dopaminergic system produces a pressor response which is mediated by dopamine D-1 and D-2 receptor activation and the release of vasopressin. Brain research. vol 701. issue 1-2. 1996-10-25. PMID:8925292. |
these results suggest an involvement of the mesolimbic a10 dopaminergic system in the regulation of blood pressure and heart rate through the activation of dopamine d-1 and d-2 receptors and vasopressin release. |
1996-10-25 |
2023-08-12 |
rat |
| N K Popova, E M Nikulina, N N Voĭtenko, D F Avgustinovich, O V Trapezo. [Effect of a single-locus mutation of the silver-blue color allele in mink on the dopaminergic system of the brain]. Genetika. vol 32. issue 3. 1996-10-24. PMID:8723640. |
a pp mutation was found to affect the dopamine turnover in corpus striatum, which is one of the major dopaminergic brain structures. |
1996-10-24 |
2023-08-12 |
Not clear |
| J A Gray, M H Joseph, D R Hemsley, A M Young, E C Warburton, P Boulenguez, G A Grigoryan, S L Peters, J N Rawlins, C T Tai. The role of mesolimbic dopaminergic and retrohippocampal afferents to the nucleus accumbens in latent inhibition: implications for schizophrenia. Behavioural brain research. vol 71. issue 1-2. 1996-10-24. PMID:8747172. |
we review here evidence in support of the model, including experiments tending to show that: (1) disruption of li is characteristic of acute, positively-symptomatic schizophrenia; (2) li depends upon dopaminergic activity; (3) li depends specifically upon dopamine release in n. accumbens; (4) li depends upon the integrity of the hippocampal formation and the retrohippocampal region reciprocally connected to the hippocampal formation; (5) the roles of n. accumbens and the hippocampal system in li are interconnected. |
1996-10-24 |
2023-08-12 |
Not clear |
| F Fujiyama, S Masuk. Association of dopaminergic terminals and neurons releasing nitric oxide in the rat striatum: an electron microscopic study using NADPH-diaphorase histochemistry and tyrosine hydroxylase immunohistochemistry. Brain research bulletin. vol 40. issue 2. 1996-10-23. PMID:8724430. |
these results suggest that activity of the striatal neurons that release nitric oxide may be regulated by direct synaptic input from dopaminergic neurons and also suggest that the th-immunoreactive terminals associated with the dendrites of nitric oxide synthase-expressing neurons provide the sites where nitric oxide influences dopamine release from neighboring terminals. |
1996-10-23 |
2023-08-12 |
rat |
| Y Kadota, T Kondo, K Sat. Akinetic mutism and involuntary movements following radical resection of hypothalamic glioma--case report. Neurologia medico-chirurgica. vol 36. issue 7. 1996-10-23. PMID:8741374. |
marked neurological improvement was obtained by treatment with dopamine agonists, suggesting that the disruption of the dopaminergic pathway via the hypothalamus was the cause of these neurological symptoms. |
1996-10-23 |
2023-08-12 |
Not clear |
| E Melame. Does treatment with dopamine agonists affect utilization of exogenous levodopa in the parkinsonian striatum? Journal of neural transmission. Supplementum. vol 45. 1996-10-23. PMID:8748609. |
this could theoretically suppress both discharge of surviving dopaminergic neurons and their ability to convert exogenous levodopa to dopamine. |
1996-10-23 |
2023-08-12 |
rat |
| S Fenu, A Carta, M Morell. Intranigral injections of glutamate antagonists modulate dopamine D1-mediated turning behavior and striatal c-fos expression. Journal of neural transmission. Supplementum. vol 45. 1996-10-23. PMID:8748612. |
the contribution of the substantia nigra (sn) in the positive interaction between dopamine d1 receptor agonists and glutamate antagonists was studied in rats with a unilateral 6-hydroxydopamine (6-ohda) lesion of dopaminergic nigro-striatal pathway. |
1996-10-23 |
2023-08-12 |
rat |
| G M Gonzalez-Alvear, L L Werlin. sigma1 Receptors in rat striatum regulate NMDA-stimulated [3H]dopamine release via a presynaptic mechanism. European journal of pharmacology. vol 294. issue 2-3. 1996-10-23. PMID:8750737. |
furthermore, bd737 and (+)-pentazocine inhibited stimulated release in the presence of tetrodotoxin, suggesting that sigma1 receptors regulating dopamine release are located on dopaminergic nerve terminals. |
1996-10-23 |
2023-08-12 |
rat |