| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| G K Wenning, R Granata, P M Laboyrie, N P Quinn, P Jenner, C D Marsde. Reversal of behavioural abnormalities by fetal allografts in a novel rat model of striatonigral degeneration. Movement disorders : official journal of the Movement Disorder Society. vol 11. issue 5. 1997-01-10. PMID:8866493. |
dopaminergic reinnervation of denervated and lesioned adult striatum itself was absent, suggesting that rotational recovery was due to diffuse dopamine release. |
1997-01-10 |
2023-08-12 |
rat |
| H Kamei, T Kameyama, T Nabeshim. (+)-SKF-10,047 and dextromethorphan ameliorate conditioned fear stress via dopaminergic systems linked to phenytoin-regulated sigma 1 sites. European journal of pharmacology. vol 309. issue 2. 1997-01-08. PMID:8874133. |
sch 23390 (r-(+)-7-chloro-8-hydroxy-3-methyl-1-phenyl-2,3,4,5-tetrahydro-1h-3- benzazepine), and the dopamine d2 receptor antagonist, (-)-sulpiride, and they were also attenuated by 6-hydroxydopamine-induced lesions of dopaminergic neurons. |
1997-01-08 |
2023-08-12 |
mouse |
| D B Miller, J P O'Callagha. Neurotoxicity of d-amphetamine in the C57BL/6J and CD-1 mouse. Interactions with stress and the adrenal system. Annals of the New York Academy of Sciences. vol 801. 1997-01-08. PMID:8959030. |
our previous work demonstrated that a variety of amps, including d-meth, d-mda, d-mdma but not d-fen are able to damage dopaminergic elements of the striatum as shown by decreases in dopamine and tyrosine hydroxylase. |
1997-01-08 |
2023-08-12 |
mouse |
| W Timmerman, E D Abercrombi. Amphetamine-induced release of dendritic dopamine in substantia nigra pars reticulata: D1-mediated behavioral and electrophysiological effects. Synapse (New York, N.Y.). vol 23. issue 4. 1997-01-07. PMID:8855513. |
dopamine (da) released from dendrites of substantia nigra dopaminergic neurons potentially is in a position to modulate basal ganglia outputs from the substantia nigra pars reticulata (snr) via stimulation of d1 receptors on the terminals of striatonigral afferents. |
1997-01-07 |
2023-08-12 |
rat |
| C D Dunigan, A E Shamo. Cation requirements of basal and ATP-regulated dopamine transport in rat pheochromocytoma cells. Neuroscience. vol 74. issue 1. 1997-01-06. PMID:8843092. |
the transport of dopamine into presynaptic nerve terminals is the primary mechanism for the termination of dopaminergic neurotransmission. |
1997-01-06 |
2023-08-12 |
rat |
| N A Otmakhova, J E Lisma. D1/D5 dopamine receptor activation increases the magnitude of early long-term potentiation at CA1 hippocampal synapses. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 16. issue 23. 1997-01-06. PMID:8922403. |
the role of the mesolimbic dopaminergic system in the reinforcement of learning suggests that dopamine should be able to modulate activity-dependent synaptic plasticity. |
1997-01-06 |
2023-08-12 |
Not clear |
| T Momiyama, J A Si. Modulation of inhibitory transmission by dopamine in rat basal forebrain nuclei: activation of presynaptic D1-like dopaminergic receptors. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 16. issue 23. 1997-01-06. PMID:8922406. |
modulation of inhibitory transmission by dopamine in rat basal forebrain nuclei: activation of presynaptic d1-like dopaminergic receptors. |
1997-01-06 |
2023-08-12 |
rat |
| S E Bachus, J E Kleinma. The neuropathology of schizophrenia. The Journal of clinical psychiatry. vol 57 Suppl 11. 1997-01-02. PMID:8941174. |
yet, despite concerted efforts to identify the source of putative dopaminergic hyperactivity in the brain in schizophrenia, definitive evidence for the "dopamine hypothesis of schizophrenia" remains elusive. |
1997-01-02 |
2023-08-12 |
Not clear |
| S Narayanan, L Wallace, N Uretsk. Spontaneous and drug-stimulated locomotor activity after the administration of pertussis toxin into the ventral tegmental area. Journal of psychiatry & neuroscience : JPN. vol 21. issue 3. 1996-12-27. PMID:8935329. |
in the present study, we have evaluated the role of dopamine receptors on spontaneous locomotor activity and the enhanced locomotor response to dopaminergic agonists after the administration of ptx into the vta. |
1996-12-27 |
2023-08-12 |
rat |
| J Romero, R de Miguel, E García-Palomero, J J Fernández-Ruiz, J A Ramo. Time-course of the effects of anandamide, the putative endogenous cannabinoid receptor ligand, on extrapyramidal function. Brain research. vol 694. issue 1-2. 1996-12-27. PMID:8974649. |
this was followed by an increase in th activity and dopamine and l-3,4-dihydroxyphenylacetic acid contents at 60 min after treatment, which would likely reflect a compensatory stimulation of these neurons, whereas restoration of control values was found at 180 min after aea administration, suggesting that the motor deficit observed at this time was not dependent on dopaminergic influence. |
1996-12-27 |
2023-08-12 |
rat |
| M Contin, R Riva, P Martinelli, E J Triggs, F Albani, A Baruzz. Rate of motor response to oral levodopa and the clinical progression of Parkinson's disease. Neurology. vol 46. issue 4. 1996-12-23. PMID:8780090. |
the rate of motor response to oral levodopa may reflect the rate of dopamine interaction with the postsynaptic receptors, providing an indirect objective index of presynaptic dopaminergic homeostasis. |
1996-12-23 |
2023-08-12 |
Not clear |
| J T Hutton, W C Koller, J E Ahlskog, R Pahwa, H I Hurtig, M B Stern, B C Hiner, A Lieberman, R F Pfeiffer, R L Rodnitzky, C H Waters, M D Muenter, C H Adler, J L Morri. Multicenter, placebo-controlled trial of cabergoline taken once daily in the treatment of Parkinson's disease. Neurology. vol 46. issue 4. 1996-12-23. PMID:8780092. |
cabergoline is a dopaminergic agonist relatively specific for the d2 receptor and much longer-acting than other dopamine agonists. |
1996-12-23 |
2023-08-12 |
Not clear |
| D W Howells, G T Liberatore, J Y Wong, G A Donna. Dopaminergic responses to striatal damage. Journal of the neurological sciences. vol 139. issue 1. 1996-12-23. PMID:8836983. |
we provide evidence in a mouse model of striatal damage that surgical cavitation induces a concerted response from the dopaminergic system with proliferation of striatal presynaptic dopamine uptake sites, increased tyrosine hydroxylase activity, increased concentrations of dopamine, dihydroxyphenylacetic acid and homovanillic acid. |
1996-12-23 |
2023-08-12 |
mouse |
| L Pinilla, D Gonzalez, M Tena-Sempere, R Aguilar, E Aguila. Mechanisms of inhibitory action of kainic acid on prolactin secretion in male rats. The Journal of endocrinology. vol 151. issue 1. 1996-12-23. PMID:8943780. |
the decrease in serum prl levels after ka administration was accompanied by an increase in pituitary concentrations of dopamine, and the ka effect on prl disappeared in males pretreated with domperidone, an antagonist of dopaminergic receptors. |
1996-12-23 |
2023-08-12 |
rat |
| A B Jung, J P Bennet. Development of striatal dopaminergic function. I. Pre- and postnatal development of mRNAs and binding sites for striatal D1 (D1a) and D2 (D2a) receptors. Brain research. Developmental brain research. vol 94. issue 2. 1996-12-20. PMID:8836569. |
quantitative receptor autoradiography with iodinated ligands, quantitative in situ hybridization histochemistry and reverse transcriptase-polymerase chain reaction (rt-pcr) were used to describe the prenatal and early postnatal ontogeny (embryonic day 14 to postnatal day 7, or e14 to p7) of striatal d1 and d2 dopamine receptor binding sites and mrna levels, respectively, in relation to the development of dopaminergic nigrostriatal innervation d1 dopamine receptor, measured by [125i]sch23982 binding, and dopamine transporter binding sites, measured by [125i]rti-55 binding, were present in low amounts beginning on e14 (2-3% and 0.3-0.6% of adult values, respectively) and increased slowly during the prenatal period. |
1996-12-20 |
2023-08-12 |
Not clear |
| T Ishikawa, V Dhawan, K Kazumata, T Chaly, F Mandel, J Neumeyer, C Margouleff, B Babchyck, I Zanzi, D Eidelber. Comparative nigrostriatal dopaminergic imaging with iodine-123-beta CIT-FP/SPECT and fluorine-18-FDOPA/PET. Journal of nuclear medicine : official publication, Society of Nuclear Medicine. vol 37. issue 11. 1996-12-20. PMID:8917170. |
spect imaging of the dopamine transporter is now an alternative to pet in the quantification of nigrostriatal dopaminergic function. |
1996-12-20 |
2023-08-12 |
Not clear |
| M Hasegawa, K Yamada, T Hasegawa, T Nabeshim. Role of dopaminergic neuronal system in dizocilpine-induced acetylcholine release in the rat brain. Journal of neural transmission (Vienna, Austria : 1996). vol 103. issue 6. 1996-12-17. PMID:8836927. |
these results suggest that the dopaminergic system is involved in the dizocilpine-induced increase in the extracellular ach level in the parietal cortex in two ways, through both dopamine d1 and d2 receptors. |
1996-12-17 |
2023-08-12 |
rat |
| C Trenkwalder, A S Walters, W Henin. Periodic limb movements and restless legs syndrome. Neurologic clinics. vol 14. issue 3. 1996-12-17. PMID:8871980. |
treatment of first choice consists of dopaminergic drugs or dopamine agonist followed by opioids or benzodiazepines. |
1996-12-17 |
2023-08-12 |
Not clear |
| A Mao, K A Freeman, R J Tallarid. Transient loss of dopamine autoreceptor control in the presence of highly potent dopamine agonists. Life sciences. vol 59. issue 21. 1996-12-17. PMID:8937509. |
the dopaminergic system is an excellent example of a control system in which a negative feedback signal is associated with receptor occupancy of a d2-like dopamine autoreceptor. |
1996-12-17 |
2023-08-12 |
rat |
| J L Labandeira-Garcia, G Rozas, E Lopez-Martin, I Liste, M J Guerr. Time course of striatal changes induced by 6-hydroxydopamine lesion of the nigrostriatal pathway, as studied by combined evaluation of rotational behaviour and striatal Fos expression. Experimental brain research. vol 108. issue 1. 1996-12-16. PMID:8721156. |
these results indicate that the severe and progressive loss of dopaminergic terminals is counteracted by an early and rapidly progressing dopamine supersensitivity, together with a higher susceptibility to drug-induced dopamine release. |
1996-12-16 |
2023-08-12 |
rat |