| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| E Acquas, H C Fibige. Dopaminergic regulation of striatal acetylcholine release: the critical role of acetylcholinesterase inhibition. Journal of neurochemistry. vol 70. issue 3. 1998-03-12. PMID:9489729. |
these results demonstrate that the concentration of an ache inhibitor in the perfusion solution can quantitatively and even qualitatively influence the manner in which dopaminergic agents regulate ach overflow in the striatum. |
1998-03-12 |
2023-08-12 |
Not clear |
| T Deacon, J Dinsmore, L C Costantini, J Ratliff, O Isacso. Blastula-stage stem cells can differentiate into dopaminergic and serotonergic neurons after transplantation. Experimental neurology. vol 149. issue 1. 1998-03-06. PMID:9454612. |
these findings demonstrate that transplantation to the brain or kidney capsule can induce a significant fraction of totipotent embryonic stem cells to become putative dopaminergic or serotonergic neurons and that when transplanted to the brain these neurons are capable of innervating the adult host striatum. |
1998-03-06 |
2023-08-12 |
mouse |
| J Sautter, J L Tseng, D Braguglia, P Aebischer, C Spenger, R W Seiler, H R Widmer, A D Zur. Implants of polymer-encapsulated genetically modified cells releasing glial cell line-derived neurotrophic factor improve survival, growth, and function of fetal dopaminergic grafts. Experimental neurology. vol 149. issue 1. 1998-03-06. PMID:9454632. |
we demonstrate here that the implantation of polymer-encapsulated cells genetically engineered to continuously secrete glial cell line-derived neurotrophic factor to the adult rat striatum improves dopaminergic graft survival and function. |
1998-03-06 |
2023-08-12 |
rat |
| M J Guerra, I Liste, J L Labandeira-Garci. Effects of lesions of the nigrostriatal pathway and of nigral grafts on striatal serotonergic innervation in adult rats. Neuroreport. vol 8. issue 16. 1998-02-26. PMID:9427312. |
neonatal destruction of the nigrostriatal dopaminergic system leads to serotonergic hyperinnervation of the striatum. |
1998-02-26 |
2023-08-12 |
rat |
| S Chapman, D M Michaelso. Specific neurochemical derangements of brain projecting neurons in apolipoprotein E-deficient mice. Journal of neurochemistry. vol 70. issue 2. 1998-02-24. PMID:9453565. |
in contrast, the nerve terminal density of the dopaminergic neurons that project from the substantia nigra to the striatum was unaffected and was similar to that of the controls. |
1998-02-24 |
2023-08-12 |
mouse |
| L M Farkas, C Suter-Crazzolara, K Unsicke. GDNF induces the calretinin phenotype in cultures of embryonic striatal neurons. Journal of neuroscience research. vol 50. issue 3. 1998-02-19. PMID:9364321. |
we conclude that striatal gdnf, in addition to its retrograde trophic role for nigrostriatal dopaminergic neurons, may also act locally within the striatum (e.g., by inducing the car phenotype and protecting these cells against toxic insult). |
1998-02-19 |
2023-08-12 |
rat |
| S Saporta, C Borlongan, J Moore, E Mejia-Millan, S L Jones, P Bonness, T S Randall, R C Allen, T B Freeman, P R Sanber. Microcarrier enhanced survival of human and rat fetal ventral mesencephalon cells implanted in the rat striatum. Cell transplantation. vol 6. issue 6. 1998-02-12. PMID:9440867. |
recent evidence has suggested that cytodex microcarriers enhance the survival of dopaminergic rat chromaffin cells transplanted into the rat striatum in the absence of immunosuppression. |
1998-02-12 |
2023-08-12 |
human |
| L R Faro, R Durán, J L do Nascimento, M Alfonso, C W Picanço-Dini. Effects of methyl mercury on the in vivo release of dopamine and its acidic metabolites DOPAC and HVA from striatum of rats. Ecotoxicology and environmental safety. vol 38. issue 2. 1998-02-06. PMID:9417850. |
in the present work, the effects of chronic doses of methyl mercury (mehg) were studied on the dopaminergic system of the rat striatum, using microdialysis coupled to high-performance liquid chromatography in order to quantify the in vivo release of dopamine (da) and its acidic metabolites dopac and hva. |
1998-02-06 |
2023-08-12 |
rat |
| R Dall'olio, R Rimondini, O Gandolf. Effects of competitive and non-competitive NMDA receptor antagonists on behavioral responses induced by 7-OH-DPAT and quinpirole in rats. Pharmacological research. vol 36. issue 3. 1998-02-05. PMID:9367665. |
this suggests that both nmda receptor antagonists could potentiate dopaminergic function in the striatum, a region critically involved in the generation of stereotyped behaviour. |
1998-02-05 |
2023-08-12 |
rat |
| T A Samad, W Krezel, P Chambon, E Borrell. Regulation of dopaminergic pathways by retinoids: activation of the D2 receptor promoter by members of the retinoic acid receptor-retinoid X receptor family. Proceedings of the National Academy of Sciences of the United States of America. vol 94. issue 26. 1998-02-02. PMID:9405615. |
among them, dopamine d2 receptor is highly expressed in the striatum and the pituitary gland as well as by mesencephalic dopaminergic neurons. |
1998-02-02 |
2023-08-12 |
mouse |
| T Blank, I Nijholt, U Teichert, H Kügler, H Behrsing, A Fienberg, P Greengard, J Spies. The phosphoprotein DARPP-32 mediates cAMP-dependent potentiation of striatal N-methyl-D-aspartate responses. Proceedings of the National Academy of Sciences of the United States of America. vol 94. issue 26. 1998-02-02. PMID:9405704. |
the results provide evidence that darpp-32, which is enriched in the striatum, may participate in the signaling between the two major afferent striatal pathways, the glutamatergic and the dopaminergic projections, by the camp-dependent regulation of striatal nmda currents. |
1998-02-02 |
2023-08-12 |
rat |
| R Lalonde, T Botez-Marquar. The neurobiological basis of movement initiation. Reviews in the neurosciences. vol 8. issue 1. 1998-01-30. PMID:9402644. |
this system is modulated by dopaminergic and glutamatergic pathways to the striatum. |
1998-01-30 |
2023-08-12 |
Not clear |
| A D Crocke. The regulation of motor control: an evaluation of the role of dopamine receptors in the substantia nigra. Reviews in the neurosciences. vol 8. issue 1. 1998-01-30. PMID:9402645. |
the importance of the nigrostriatal dopaminergic pathway in motor control is widely accepted and it is generally believed that the motor symptoms of parkinson's disease result solely from reduced release of dopamine from terminals in the striatum. |
1998-01-30 |
2023-08-12 |
Not clear |
| C Watanabe, Y Kasanuma, H Sato. Deficiency of selenium enhances the K+-induced release of dopamine in the striatum of mice. Neuroscience letters. vol 236. issue 1. 1998-01-29. PMID:9404949. |
to determine whether a selenium (se) deficiency in the brain leads to a functional change in dopaminergic transmission in the striatum, in vivo microdialysis was conducted in mice fed a low-se diet. |
1998-01-29 |
2023-08-12 |
mouse |
| J A Obeso, M C Rodríguez, A Gorospe, J Guridi, L Alvarez, R Macia. Surgical treatment of Parkinson's disease. Bailliere's clinical neurology. vol 6. issue 1. 1998-01-28. PMID:9426872. |
grafting aims to repair the nigrostriatal pathway and restore dopaminergic function in the striatum. |
1998-01-28 |
2023-08-12 |
Not clear |
| G M Gonzalez, L L Werlin. Release of [3H]dopamine from guinea pig striatal slices is modulated by sigma1 receptor agonists. Naunyn-Schmiedeberg's archives of pharmacology. vol 356. issue 4. 1998-01-21. PMID:9349631. |
their location in guinea pig striatum, a terminal field of dopaminergic projections arising from the substantia nigra, suggested that this tissue would be a logical choice in which to examine physiological properties of sigma receptor activation. |
1998-01-21 |
2023-08-12 |
rat |
| D J Brook. Motor disturbance and brain functional imaging in Parkinson's disease. European neurology. vol 38 Suppl 2. 1998-01-20. PMID:9387800. |
activation of these cortical areas can be restored by administering dopaminergic medication, implanting the striatum with fetal mesencephalic tissue, and by pallidotomy. |
1998-01-20 |
2023-08-12 |
Not clear |
| F Tamar. Disturbances in higher function in Parkinson's disease. European neurology. vol 38 Suppl 2. 1998-01-20. PMID:9387801. |
as the activity of these circuits can be modulated at the striatum by the nigrostriatal dopaminergic system, cognitive function can be influenced by the pathological change in the substantia nigra in parkinson's disease. |
1998-01-20 |
2023-08-12 |
Not clear |
| D Dluze. Estrogen decreases corpus striatal neurotoxicity in response to 6-hydroxydopamine. Brain research. vol 767. issue 2. 1998-01-15. PMID:9367266. |
various parameters of nigrostriatal dopaminergic function as derived from measurements of dopamine and dihydroxyphenylacetic acid (dopac) concentrations were determined from the 6-ohda lesioned and non-lesioned sides of the corpus striatum in these animals. |
1998-01-15 |
2023-08-12 |
rat |
| D Dluze. Estrogen decreases corpus striatal neurotoxicity in response to 6-hydroxydopamine. Brain research. vol 767. issue 2. 1998-01-15. PMID:9367266. |
such findings suggest that estrogen may function as an important modulatory factor capable of attenuating degeneration within the corpus striatum, and in this way serve as a neuroprotectant of the nigrostriatal dopaminergic system. |
1998-01-15 |
2023-08-12 |
rat |