| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| H H Samson, C W Hodge, H L Erickson, J S Niehus, G A Gerhardt, P W Kalivas, E A Floy. The effects of local application of ethanol in the n. accumbens on dopamine overflow and clearance. Alcohol (Fayetteville, N.Y.). vol 14. issue 5. 1997-10-31. PMID:9305464. |
taken together, the data suggest that the actions of ethanol to increase extracellular dopamine levels in the n. accumbens are most likely not an effect of ethanol at the level of the accumbens but rather an action which increases neural activity within the mesoaccumbens pathway, perhaps via actions at the ventral tegmental area. |
1997-10-31 |
2023-08-12 |
rat |
| M Rubinstein, T J Phillips, J R Bunzow, T L Falzone, G Dziewczapolski, G Zhang, Y Fang, J L Larson, J A McDougall, J A Chester, C Saez, T A Pugsley, O Gershanik, M J Low, D K Grand. Mice lacking dopamine D4 receptors are supersensitive to ethanol, cocaine, and methamphetamine. Cell. vol 90. issue 6. 1997-10-20. PMID:9323127. |
mice lacking dopamine d4 receptors are supersensitive to ethanol, cocaine, and methamphetamine. |
1997-10-20 |
2023-08-12 |
mouse |
| G F Koob, E J Nestle. The neurobiology of drug addiction. The Journal of neuropsychiatry and clinical neurosciences. vol 9. issue 3. 1997-10-16. PMID:9276849. |
ethanol's reinforcing effects may result from multiple neurotransmitter interactions including gamma-aminobutyric acid, glutamate, dopamine, opioid peptides, and serotonin. |
1997-10-16 |
2023-08-12 |
Not clear |
| G Iusco, V Boido, F Sparatore, G Colombo, P L Saba, Z Rossetti, A Vaccar. New benzamide-derived 5-HT3 receptor antagonists which prevent the effects of ethanol on extracellular dopamine, and fail to reduce voluntary alcohol intake in rats. Farmaco (Societa chimica italiana : 1989). vol 52. issue 3. 1997-09-12. PMID:9212448. |
new benzamide-derived 5-ht3 receptor antagonists which prevent the effects of ethanol on extracellular dopamine, and fail to reduce voluntary alcohol intake in rats. |
1997-09-12 |
2023-08-12 |
rat |
| G Iusco, V Boido, F Sparatore, G Colombo, P L Saba, Z Rossetti, A Vaccar. New benzamide-derived 5-HT3 receptor antagonists which prevent the effects of ethanol on extracellular dopamine, and fail to reduce voluntary alcohol intake in rats. Farmaco (Societa chimica italiana : 1989). vol 52. issue 3. 1997-09-12. PMID:9212448. |
thus the 5-ht3-mediated inhibition of alcohol-induced striatal release of dopamine by substituted benzamides is not a requisite for affecting ethanol intake. |
1997-09-12 |
2023-08-12 |
rat |
| E Gili-Martín, A Fernández-Briera, P Calv. Effects of chronic ethanol treatment and ethanol withdrawal on [3H]SCH23390 binding to rat striatal membranes. Neuropharmacology. vol 36. issue 1. 1997-08-18. PMID:9144646. |
the effects of chronic ethanol administration and ethanol withdrawal on the kinetic and pharmacological properties of [3h]sch23390 binding sites and the labelling of central dopamine d-1 receptors were studied in the striatum of the rat. |
1997-08-18 |
2023-08-12 |
rat |
| E Gili-Martín, A Fernández-Briera, P Calv. Effects of chronic ethanol treatment and ethanol withdrawal on [3H]SCH23390 binding to rat striatal membranes. Neuropharmacology. vol 36. issue 1. 1997-08-18. PMID:9144646. |
chronic 40 day ethanol treatment produced a statistically significant decrease (p < 0.05) in maximum binding (bmax) on striatal dopamine d-1 receptors of the rat, kd remaining unaltered. |
1997-08-18 |
2023-08-12 |
rat |
| E Gili-Martín, A Fernández-Briera, P Calv. Effects of chronic ethanol treatment and ethanol withdrawal on [3H]SCH23390 binding to rat striatal membranes. Neuropharmacology. vol 36. issue 1. 1997-08-18. PMID:9144646. |
the results show that rat striatal [3h]sch23390-labelled binding sites are affected by different conditions of ethanol exposure, possibly suggesting the medication of striatal dopamine pathways in the responses to ethanol intoxication. |
1997-08-18 |
2023-08-12 |
rat |
| R A Gillespie, J Eriksen, H L Hao, M J Drus. Effects of maternal ethanol consumption and buspirone treatment on dopamine and norepinephrine reuptake sites and D1 receptors in offspring. Alcoholism, clinical and experimental research. vol 21. issue 3. 1997-08-07. PMID:9161605. |
effects of maternal ethanol consumption and buspirone treatment on dopamine and norepinephrine reuptake sites and d1 receptors in offspring. |
1997-08-07 |
2023-08-12 |
rat |
| K Blum, E R Braverman, S Wu, J G Cull, T J Chen, J Gill, R Wood, A Eisenberg, M Sherman, K R Davis, D Matthews, L Fischer, N Schnautz, W Walsh, A A Pontius, M Zedar, G Kaats, D E Coming. Association of polymorphisms of dopamine D2 receptor (DRD2), and dopamine transporter (DAT1) genes with schizoid/avoidant behaviors (SAB). Molecular psychiatry. vol 2. issue 3. 1997-07-30. PMID:9152988. |
dysfunction of the d2 dopamine receptors leads to aberrant substance-seeking behaviors (ethanol, drugs, tobacco, and food) and other related behaviors (pathological gambling, tourette's disorder, attention-deficit/hyperactivity disorder). |
1997-07-30 |
2023-08-12 |
Not clear |
| D M Wilson, A P Ferko, E J Barbieri, G J DiGregorio, E Bobyock, R McMichae. The interaction of dopamine, cocaine, and cocaethylene with ethanol on central nervous system depression in mice. Pharmacology, biochemistry, and behavior. vol 57. issue 1-2. 1997-07-30. PMID:9164556. |
the interaction of dopamine, cocaine, and cocaethylene with ethanol on central nervous system depression in mice. |
1997-07-30 |
2023-08-12 |
mouse |
| D M Wilson, A P Ferko, E J Barbieri, G J DiGregorio, E Bobyock, R McMichae. The interaction of dopamine, cocaine, and cocaethylene with ethanol on central nervous system depression in mice. Pharmacology, biochemistry, and behavior. vol 57. issue 1-2. 1997-07-30. PMID:9164556. |
the interactions between dopamine, cocaine, cocaethylene, and ethanol were studied in swiss-webster mice. |
1997-07-30 |
2023-08-12 |
mouse |
| D M Wilson, A P Ferko, E J Barbieri, G J DiGregorio, E Bobyock, R McMichae. The interaction of dopamine, cocaine, and cocaethylene with ethanol on central nervous system depression in mice. Pharmacology, biochemistry, and behavior. vol 57. issue 1-2. 1997-07-30. PMID:9164556. |
the dopamine d2-receptor antagonist sulpiride and the d1-receptor antagonist fluphenazine were given acutely icv with dopamine in the presence of systemic ethanol to examine whether these antagonists could block the return to the lorr produced by dopamine. |
1997-07-30 |
2023-08-12 |
mouse |
| D M Wilson, A P Ferko, E J Barbieri, G J DiGregorio, E Bobyock, R McMichae. The interaction of dopamine, cocaine, and cocaethylene with ethanol on central nervous system depression in mice. Pharmacology, biochemistry, and behavior. vol 57. issue 1-2. 1997-07-30. PMID:9164556. |
sulpiride, however, actually enhanced the interaction between ethanol and dopamine in a dose-dependent manner as measured by the lorr; fluphenazine neither blocked nor enhanced the effect of dopamine in the presence of systemic ethanol. |
1997-07-30 |
2023-08-12 |
mouse |
| D M Wilson, A P Ferko, E J Barbieri, G J DiGregorio, E Bobyock, R McMichae. The interaction of dopamine, cocaine, and cocaethylene with ethanol on central nervous system depression in mice. Pharmacology, biochemistry, and behavior. vol 57. issue 1-2. 1997-07-30. PMID:9164556. |
the results of this study showed that the neurotransmitter dopamine and both cocaine and cocaethylene can promote further cns depression in the presence of systemic ethanol, and that dopamine was significantly more potent than cocaine and cocaethylene as measured by the return to the lorr. |
1997-07-30 |
2023-08-12 |
mouse |
| A D Lê, D Tomkins, G Higgins, B Quan, E M Seller. Effects of 5-HT3, D1 and D2 receptor antagonists on ethanol- and cocaine-induced locomotion. Pharmacology, biochemistry, and behavior. vol 57. issue 1-2. 1997-07-30. PMID:9164590. |
in contrast, pretreatment with a 10 micrograms/kg dose of either sch 23390 or spiperone, a d1 and d2 dopamine (da) receptor antagonist respectively, completely antagonized the stimulation of lma induced by ethanol. |
1997-07-30 |
2023-08-12 |
mouse |
| S L Dewey, C S Chaurasia, C E Chen, N D Volkow, F A Clarkson, S P Porter, R M Straughter-Moore, D L Alexoff, D Tedeschi, N B Russo, J S Fowler, J D Brodi. GABAergic attenuation of cocaine-induced dopamine release and locomotor activity. Synapse (New York, N.Y.). vol 25. issue 4. 1997-07-24. PMID:9097399. |
ethanol, lorazepam (ativan), and gamma-vinyl gaba (gvg) significantly and dose-dependently attenuated cocaine-induced dopamine release in the corpus striatum of freely moving animals. |
1997-07-24 |
2023-08-12 |
Not clear |
| S L Dewey, C S Chaurasia, C E Chen, N D Volkow, F A Clarkson, S P Porter, R M Straughter-Moore, D L Alexoff, D Tedeschi, N B Russo, J S Fowler, J D Brodi. GABAergic attenuation of cocaine-induced dopamine release and locomotor activity. Synapse (New York, N.Y.). vol 25. issue 4. 1997-07-24. PMID:9097399. |
unlike ethanol or lorazepam, however, gvg is not a sedative hypnotic in the doses used, and hence the strategy of selectively increasing gabaergic activity by suicide inhibition of the catabolic enzyme, gaba-transaminase, offers the unique advantage of attenuating cocaine-induced dopamine release without the apparent side effects typically associated with sedative hypnotics. |
1997-07-24 |
2023-08-12 |
Not clear |
| S J Manley, H J Littl. Enhancement of amphetamine- and cocaine-induced locomotor activity after chronic ethanol administration. The Journal of pharmacology and experimental therapeutics. vol 281. issue 3. 1997-07-21. PMID:9190869. |
it is possible that alterations in mesolimbic dopamine transmission are responsible for the effects of the ethanol treatment. |
1997-07-21 |
2023-08-12 |
mouse |
| M Z Wrona, J Waskiewicz, Q P Han, J Han, H Li, G Dryhurs. Putative oxidative metabolites of 1-methyl-6-hydroxy-1,2,3,4-tetrahydro-beta-carboline of potential relevance to the addictive and neurodegenerative consequences of ethanol abuse. Alcohol (Fayetteville, N.Y.). vol 14. issue 3. 1997-07-10. PMID:9160798. |
accordingly, it is speculated that ethanol drinking might potentiate formation of 6 that contributes to elevated release of several neurotransmitters including dopamine (da) and endogenous opioids in regions of the brain innervated by serotonergic axon terminals. |
1997-07-10 |
2023-08-12 |
mouse |