All Relations between reward and dopamine

Publication Sentence Publish Date Extraction Date Species
J R Stellar, M Waraczynski, J P Brun. Neonatal dopamine depletions spare lateral hypothalamic stimulation reward in adult rats. Pharmacology, biochemistry, and behavior. vol 30. issue 2. 1988-11-01. PMID:3174768. neonatal dopamine depletions spare lateral hypothalamic stimulation reward in adult rats. 1988-11-01 2023-08-11 rat
G J Schaefe. Opiate antagonists and rewarding brain stimulation. Neuroscience and biobehavioral reviews. vol 12. issue 1. 1988-07-01. PMID:3287237. the results produced by naloxone are consistent with a modulatory effect of endogenous opioid systems on reward, and suggest that the opiate and dopamine systems together exert significant control over icss. 1988-07-01 2023-08-11 Not clear
L Hernandez, B G Hoebe. Food reward and cocaine increase extracellular dopamine in the nucleus accumbens as measured by microdialysis. Life sciences. vol 42. issue 18. 1988-06-03. PMID:3362036. food reward and cocaine increase extracellular dopamine in the nucleus accumbens as measured by microdialysis. 1988-06-03 2023-08-11 rat
L Hernandez, B G Hoebe. Food reward and cocaine increase extracellular dopamine in the nucleus accumbens as measured by microdialysis. Life sciences. vol 42. issue 18. 1988-06-03. PMID:3362036. extracellular dopamine increased 37% when the animals pressed a lever for food reward. 1988-06-03 2023-08-11 rat
K D Carr, T H Ba. Lateral hypothalamic stimulation-produced analgesia: inferred refractory period of directly stimulated neurons and resistance to pimozide antagonism. Physiology & behavior. vol 41. issue 4. 1988-03-15. PMID:3432389. the dopamine antagonist pimozide, at doses known to diminish eslh-induced feeding and reward (0.25 and 0.5 mg/kg), failed to affect analgesia. 1988-03-15 2023-08-11 Not clear
M J Blake, E A Stei. Brain stimulation of the ventral tegmental area attenuates footshock escape: an in vivo autoradiographic analysis of opiate receptors. Brain research. vol 435. issue 1-2. 1988-03-10. PMID:2827852. it appears, further, that ics can inhibit the release of endogenous opioid peptides in areas along the mesotelencephalic dopamine pathways, possibly to regulate the activity of neurons conveying reward information. 1988-03-10 2023-08-11 rat
R A Wis. The role of reward pathways in the development of drug dependence. Pharmacology & therapeutics. vol 35. issue 1-2. 1988-02-20. PMID:3321101. ethanol, nicotine, caffeine and phencyclidine stimulate both locomotor activity and dopamine turnover, but their sites of interaction with reward pathways have not yet been identified. 1988-02-20 2023-08-11 Not clear
R A Wis. The role of reward pathways in the development of drug dependence. Pharmacology & therapeutics. vol 35. issue 1-2. 1988-02-20. PMID:3321101. benzodiazepines and barbiturates stimulate locomotor activity without stimulating dopamine turnover; they may interact with reward pathways at a synapse efferent to the dopaminergic link in the pathways. 1988-02-20 2023-08-11 Not clear
G F Koob, H T Le, I Crees. The D1 dopamine receptor antagonist SCH 23390 increases cocaine self-administration in the rat. Neuroscience letters. vol 79. issue 3. 1987-11-20. PMID:2958724. these results suggest that the d1 antagonist may be more effective at blocking mesolimbic dopamine activity, and that selective d1 receptor activation may be an important component of psychostimulant reward. 1987-11-20 2023-08-11 rat
R J Beninger, M Cheng, B L Hahn, D C Hoffman, E J Mazurski, M A Morency, P Ramm, R J Stewar. Effects of extinction, pimozide, SCH 23390, and metoclopramide on food-rewarded operant responding of rats. Psychopharmacology. vol 92. issue 3. 1987-10-16. PMID:2957718. the similarity in the pattern of responding produced by extinction and dopamine (da) receptor blockers has led to the suggestion that da neurons may participate in the usual effects of reward on behaviour. 1987-10-16 2023-08-11 rat
L M Colle, R A Wis. Opposite effects of unilateral forebrain ablations on ipsilateral and contralateral hypothalamic self-stimulation. Brain research. vol 407. issue 2. 1987-06-17. PMID:3494485. the small effects of large ablations on ipsilateral self-stimulation confirm similar observations of huston and stellar and their co-workers and raise questions for current theories regarding the role of dopamine in brain stimulation reward. 1987-06-17 2023-08-11 Not clear
J M Van Ree, N Ramse. The dopamine hypothesis of opiate reward challenged. European journal of pharmacology. vol 134. issue 2. 1987-05-29. PMID:3032655. the dopamine hypothesis of opiate reward challenged. 1987-05-29 2023-08-11 rat
J M Van Ree, N Ramse. The dopamine hypothesis of opiate reward challenged. European journal of pharmacology. vol 134. issue 2. 1987-05-29. PMID:3032655. the dopamine hypothesis of opiate reward was directly tested by blocking dopamine receptor systems with haloperidol. 1987-05-29 2023-08-11 rat
J M Van Ree, N Ramse. The dopamine hypothesis of opiate reward challenged. European journal of pharmacology. vol 134. issue 2. 1987-05-29. PMID:3032655. it is concluded that dopamine is not critically involved in opiate reward and that multiple endogenous reward systems are present in the brain. 1987-05-29 2023-08-11 rat
A G Phillips, A Jakubovic, H C Fibige. Increased in vivo tyrosine hydroxylase activity in rat telencephalon produced by self-stimulation of the ventral tegmental area. Brain research. vol 402. issue 1. 1987-05-14. PMID:2881597. these results are consistent with a role for dopamine in brain-stimulation reward obtained from electrical stimulation of the ventral tegmental area but do not provide evidence for dopaminergic mediation of the rewarding properties of food. 1987-05-14 2023-08-11 human
R J Care. An examination of parkinsonian versus anhedonia contributions to self-stimulation impairments induced by dopamine dysfunction. Behavioural brain research. vol 22. issue 2. 1987-02-10. PMID:3024663. interference with brain dopamine neurotransmission can severely impair brain stimulation reward behavior. 1987-02-10 2023-08-11 Not clear
R J Care. An examination of parkinsonian versus anhedonia contributions to self-stimulation impairments induced by dopamine dysfunction. Behavioural brain research. vol 22. issue 2. 1987-02-10. PMID:3024663. that is, it has been difficult to determine whether the dopamine dysfunction has attenuated the reward effect of the stimulation or has merely rendered the animal less able to generate the behavior required to obtain reinforcement. 1987-02-10 2023-08-11 Not clear
R J Care. An examination of parkinsonian versus anhedonia contributions to self-stimulation impairments induced by dopamine dysfunction. Behavioural brain research. vol 22. issue 2. 1987-02-10. PMID:3024663. to experimentally re-assess this issue, the present studies examine the effect of unilateral 6-hydroxydopamine lesions of forebrain dopamine neurons on brain stimulation reward in animals. 1987-02-10 2023-08-11 Not clear
S C Fowler, M M LaCerra, A Ettenber. Effects of haloperidol on the biophysical characteristics of operant responding: implications for motor and reinforcement processes. Pharmacology, biochemistry, and behavior. vol 25. issue 4. 1987-01-13. PMID:3786339. implications for motor and reward interpretations of the actions of dopamine antagonists are also discussed. 1987-01-13 2023-08-11 rat
A Ettenberg, C H Cam. Haloperidol induces a partial reinforcement extinction effect in rats: implications for a dopamine involvement in food reward. Pharmacology, biochemistry, and behavior. vol 25. issue 4. 1987-01-13. PMID:3786340. haloperidol induces a partial reinforcement extinction effect in rats: implications for a dopamine involvement in food reward. 1987-01-13 2023-08-11 rat