All Relations between reward and dopamine

Publication Sentence Publish Date Extraction Date Species
W Schultz, L Tremblay, J R Hollerma. Reward prediction in primate basal ganglia and frontal cortex. Neuropharmacology. vol 37. issue 4-5. 1999-05-26. PMID:9704983. dopamine neurons respond phasically to primary rewards and reward-predicting stimuli depending on reward unpredictability but without discriminating between rewards. 1999-05-26 2023-08-12 Not clear
W Schultz, L Tremblay, J R Hollerma. Reward prediction in primate basal ganglia and frontal cortex. Neuropharmacology. vol 37. issue 4-5. 1999-05-26. PMID:9704983. whereas dopamine neurons emit a reward teaching signal without indicating the specific reward, striatal neurons adapt expectation activity to new reward situations, and orbitofrontal neurons process the specific nature of rewards. 1999-05-26 2023-08-12 Not clear
J R Taylor, B A Horge. Enhanced responding for conditioned reward produced by intra-accumbens amphetamine is potentiated after cocaine sensitization. Psychopharmacology. vol 142. issue 1. 1999-05-26. PMID:10102780. the mesolimbic dopamine (da) system has been implicated in conditioned reward (cr), locomotor sensitization, and the reinforcing properties of psychomotor stimulants. 1999-05-26 2023-08-12 human
C Cohen, G Perrault, D J Sange. Effects of D1 dopamine receptor agonists on oral ethanol self-administration in rats: comparison with their efficacy to produce grooming and hyperactivity. Psychopharmacology. vol 142. issue 1. 1999-05-26. PMID:10102789. the present results underline the involvement of d1 dopamine receptors in reward processes. 1999-05-26 2023-08-12 rat
S Z Sabol, M L Nelson, C Fisher, L Gunzerath, C L Brody, S Hu, L A Sirota, S E Marcus, B D Greenberg, F R Lucas, J Benjamin, D L Murphy, D H Hame. A genetic association for cigarette smoking behavior. Health psychology : official journal of the Division of Health Psychology, American Psychological Association. vol 18. issue 1. 1999-05-20. PMID:9925040. it is hypothesized that individuals carrying the slc6a3-9 polymorphism have altered dopamine transmission, which reduces their need for novelty and reward by external stimuli, including cigarettes. 1999-05-20 2023-08-12 Not clear
Y Hurd, N Lindefor. [Increasing abuse of psychostimulants is alarming. New findings confirm the mechanisms behind cocaine and amphetamine addiction]. Lakartidningen. vol 96. issue 15. 1999-05-19. PMID:10319645. dopamine pathways extending from the brain stem to the basal ganglia, limbic structures, and cerebral cortex are generally considered to constitute the neuroanatomical substrates underlying motivation, reward, and motor function. 1999-05-19 2023-08-12 human
P K Hitchcott, G D Phillip. Effects of intra-amygdala R(+) 7-OH-DPAT on intra-accumbens d-amphetamine-associated learning. I. Pavlovian conditioning. Psychopharmacology. vol 140. issue 3. 1999-04-21. PMID:9877010. we have previously obtained evidence that the mesoamygdaloid dopamine projection modulates the acquisition of a conditioned response (cr) elicited by presentation of a conditioned stimulus (cs) predicting the availability of a natural (sucrose) reward. 1999-04-21 2023-08-12 rat
K D Carr, N Kutchukhidze, T H Par. Differential effects of mu and kappa opioid antagonists on Fos-like immunoreactivity in extended amygdala. Brain research. vol 822. issue 1-2. 1999-04-21. PMID:10082881. considering the involvement of mesoaccumbens dopamine neurons and components of the 'extended amygdala' in motivated behavior and reward, it was hypothesized that the induction of c-fos by naltrexone accounts for the motivational-affective consequences of opioid antagonism. 1999-04-21 2023-08-12 human
J R Hollerman, W Schult. Dopamine neurons report an error in the temporal prediction of reward during learning. Nature neuroscience. vol 1. issue 4. 1999-04-21. PMID:10195164. dopamine neurons report an error in the temporal prediction of reward during learning. 1999-04-21 2023-08-12 monkey
A Nehli. Are we dependent upon coffee and caffeine? A review on human and animal data. Neuroscience and biobehavioral reviews. vol 23. issue 4. 1999-03-26. PMID:10073894. the classical drugs of abuse lead to quite specific increases in cerebral functional activity and dopamine release in the shell of the nucleus accumbens, the key structure for reward, motivation and addiction. 1999-03-26 2023-08-12 human
P A Garris, M Kilpatrick, M A Bunin, D Michael, Q D Walker, R M Wightma. Dissociation of dopamine release in the nucleus accumbens from intracranial self-stimulation. Nature. vol 398. issue 6722. 1999-03-25. PMID:10078530. dopamine may therefore be a neural substrate for novelty or reward expectation rather than reward itself. 1999-03-25 2023-08-12 rat
T M Tzschentk. Measuring reward with the conditioned place preference paradigm: a comprehensive review of drug effects, recent progress and new issues. Progress in neurobiology. vol 56. issue 6. 1999-03-23. PMID:9871940. transmitter systems that have been investigated with respect to their involvement in brain reward mechanisms include dopamine, opioids, acetylcholine, gaba, serotonin, glutamate, substance p, and cholecystokinin, the motivational significance of which has been examined either directly, by using respective agonist or antagonist drugs, or indirectly, by studying the effects of these drugs on the reward induced by other drugs. 1999-03-23 2023-08-12 Not clear
I Sziráki, H Sershen, M Benuck, A Hashim, A Lajth. Receptor systems participating in nicotine-specific effects. Neurochemistry international. vol 33. issue 5. 1999-03-18. PMID:9874095. it is generally accepted that self-administration of drugs is prompted primarily by a reward system driven by an increase in extracellular dopamine in the nucleus accumbens. 1999-03-18 2023-08-12 rat
I Sziráki, H Sershen, M Benuck, A Hashim, A Lajth. Receptor systems participating in nicotine-specific effects. Neurochemistry international. vol 33. issue 5. 1999-03-18. PMID:9874095. recent findings that dopamine increase in the accumbens can be caused by many other factors, among them stress, suggest a more complex mechanism, and possibly differences in the reward system for different compounds. 1999-03-18 2023-08-12 rat
I Sziráki, H Sershen, M Benuck, A Hashim, A Lajth. Receptor systems participating in nicotine-specific effects. Neurochemistry international. vol 33. issue 5. 1999-03-18. PMID:9874095. sch-23390, a dopamine d1 receptor antagonist, blocked both nicotine- and cocaine-induced effects, indicating the possible role of this receptor in these reward effects. 1999-03-18 2023-08-12 rat
B T Lett, V L Grant, L L Gabork. Wheel running simultaneously induces CTA and facilitates feeding in non-deprived rats. Conditioned taste aversion. Appetite. vol 31. issue 3. 1999-03-18. PMID:9920687. a role for the mesolimbic dopamine reward system in these effects was considered. 1999-03-18 2023-08-12 rat
E N Pothos, D Sulzer, B G Hoebe. Plasticity of quantal size in ventral midbrain dopamine neurons: possible implications for the neurochemistry of feeding and reward. Appetite. vol 31. issue 3. 1999-03-18. PMID:9920693. plasticity of quantal size in ventral midbrain dopamine neurons: possible implications for the neurochemistry of feeding and reward. 1999-03-18 2023-08-12 Not clear
K C Berridge, T E Robinso. What is the role of dopamine in reward: hedonic impact, reward learning, or incentive salience? Brain research. Brain research reviews. vol 28. issue 3. 1999-03-03. PMID:9858756. what is the role of dopamine in reward: hedonic impact, reward learning, or incentive salience? 1999-03-03 2023-08-12 rat
K C Berridge, T E Robinso. What is the role of dopamine in reward: hedonic impact, reward learning, or incentive salience? Brain research. Brain research reviews. vol 28. issue 3. 1999-03-03. PMID:9858756. what roles do mesolimbic and neostriatal dopamine systems play in reward? 1999-03-03 2023-08-12 rat
K C Berridge, T E Robinso. What is the role of dopamine in reward: hedonic impact, reward learning, or incentive salience? Brain research. Brain research reviews. vol 28. issue 3. 1999-03-03. PMID:9858756. our review of the literature, together with results of a new study of residual reward capacity after dopamine depletion, indicates the answer to both questions is 'no'. 1999-03-03 2023-08-12 rat