| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Ariel D Quiroga, Lena Li, Martin Trötzmüller, Randy Nelson, Spencer D Proctor, Harald Köfeler, Richard Lehne. Deficiency of carboxylesterase 1/esterase-x results in obesity, hepatic steatosis, and hyperlipidemia. Hepatology (Baltimore, Md.). vol 56. issue 6. 2013-02-28. PMID:22806626. |
consequently, ces1/es-x knockout mice develop obesity, fatty liver, hyperinsulinemia, and insulin insensitivity on chow diet without change in food intake and present with decreased energy expenditure. |
2013-02-28 |
2023-08-12 |
mouse |
| S S Runchey, M N Pollak, L M Valsta, G D Coronado, Y Schwarz, K L Breymeyer, C Wang, C-Y Wang, J W Lampe, M L Neuhouse. Glycemic load effect on fasting and post-prandial serum glucose, insulin, IGF-1 and IGFBP-3 in a randomized, controlled feeding study. European journal of clinical nutrition. vol 66. issue 10. 2013-02-27. PMID:22892437. |
glycemic load effect on fasting and post-prandial serum glucose, insulin, igf-1 and igfbp-3 in a randomized, controlled feeding study. |
2013-02-27 |
2023-08-12 |
Not clear |
| Stephen C Woods, Wolfgang Langhan. Inconsistencies in the assessment of food intake. American journal of physiology. Endocrinology and metabolism. vol 303. issue 12. 2013-02-25. PMID:23074241. |
for example, cholecystokinin (cck), a duodenal peptide secreted during meals that aids in digestion, also reduces ongoing food intake, thereby contributing to satiation; and insulin and leptin, hormones secreted in direct proportion to body fat, act in the brain to help control adiposity by reducing energy intake. |
2013-02-25 |
2023-08-12 |
Not clear |
| Anne-Maj Samuelsson, Phillippa A Matthews, Eugene Jansen, Paul D Taylor, Lucilla Posto. Sucrose feeding in mouse pregnancy leads to hypertension, and sex-linked obesity and insulin resistance in female offspring. Frontiers in physiology. vol 4. 2013-02-21. PMID:23423541. |
sucrose feeding in mouse pregnancy leads to hypertension, and sex-linked obesity and insulin resistance in female offspring. |
2013-02-21 |
2023-08-12 |
mouse |
| Haiyan Zhang, Wen-Jun Shen, Yuan Cortez, Fredric B Kraemer, Salman Azha. Nordihydroguaiaretic acid improves metabolic dysregulation and aberrant hepatic lipid metabolism in mice by both PPARα-dependent and -independent pathways. American journal of physiology. Gastrointestinal and liver physiology. vol 304. issue 1. 2013-02-20. PMID:23104557. |
feeding ob/ob mice a chow diet supplemented with either low (0.83 g/kg diet) or high-dose (2.5 g/kg diet) ndga for 16 wk significantly improved plasma triglyceride (tg), inflammatory chemokine levels, hyperinsulinemia, insulin sensitivity, and glucose intolerance. |
2013-02-20 |
2023-08-12 |
mouse |
| A T Grazul-Bilska, E Borowczyk, J J Bilski, L P Reynolds, D A Redmer, J S Caton, K A Vonnahm. Overfeeding and underfeeding have detrimental effects on oocyte quality measured by in vitro fertilization and early embryonic development in sheep. Domestic animal endocrinology. vol 43. issue 4. 2013-02-19. PMID:22652013. |
these data show that inadequate feeding has a negative effect on oocyte quality which results in lower oocyte cleavage after ivf and morula and blastocyst formation; overfeeding increased serum insulin and underfeeding increased serum e(2) but not t(3) or t(4). |
2013-02-19 |
2023-08-12 |
Not clear |
| Van Anh Nguyen, Tran Le, Ming Tong, Elizabeth Silbermann, Fusun Gundogan, Suzanne M de la Mont. Impaired insulin/IGF signaling in experimental alcohol-related myopathy. Nutrients. vol 4. issue 8. 2013-02-19. PMID:23016132. |
chronic ethanol feeding reduced myofiber size and mrna expression of igf-1 polypeptide, insulin, igf-1, and igf-2 receptors, irs-1, and irs-2. |
2013-02-19 |
2023-08-12 |
rat |
| Magdolna Szántó, Attila Brunyánszki, Borbála Kiss, Lilla Nagy, Pál Gergely, László Virág, Péter Ba. Poly(ADP-ribose) polymerase-2: emerging transcriptional roles of a DNA-repair protein. Cellular and molecular life sciences : CMLS. vol 69. issue 24. 2013-02-18. PMID:22581363. |
altered gene expression gives rise to a complex phenotype characterized primarily by enhanced mitochondrial activity that results both in beneficial (loss of fat, enhanced insulin sensitivity) and in disadvantageous (pancreatic beta cell hypofunction upon high fat feeding) consequences. |
2013-02-18 |
2023-08-12 |
Not clear |
| K E Borer, S R Bailey, N J Menzies-Gow, P A Harris, J Elliot. Effect of feeding glucose, fructose, and inulin on blood glucose and insulin concentrations in normal ponies and those predisposed to laminitis. Journal of animal science. vol 90. issue 9. 2013-02-15. PMID:22966077. |
effect of feeding glucose, fructose, and inulin on blood glucose and insulin concentrations in normal ponies and those predisposed to laminitis. |
2013-02-15 |
2023-08-12 |
Not clear |
| K E Borer, S R Bailey, N J Menzies-Gow, P A Harris, J Elliot. Effect of feeding glucose, fructose, and inulin on blood glucose and insulin concentrations in normal ponies and those predisposed to laminitis. Journal of animal science. vol 90. issue 9. 2013-02-15. PMID:22966077. |
insulin resistance may be diagnosed by feeding supplementary water-soluble carbohydrate (wsc) and measuring blood glucose and insulin concentrations. |
2013-02-15 |
2023-08-12 |
Not clear |
| K E Borer, S R Bailey, N J Menzies-Gow, P A Harris, J Elliot. Effect of feeding glucose, fructose, and inulin on blood glucose and insulin concentrations in normal ponies and those predisposed to laminitis. Journal of animal science. vol 90. issue 9. 2013-02-15. PMID:22966077. |
blood samples were taken for 12 h after feeding on each day, and baseline and peak concentrations and area under the curve (auc) for glucose and insulin were recorded. |
2013-02-15 |
2023-08-12 |
Not clear |
| K E Borer, S R Bailey, N J Menzies-Gow, P A Harris, J Elliot. Effect of feeding glucose, fructose, and inulin on blood glucose and insulin concentrations in normal ponies and those predisposed to laminitis. Journal of animal science. vol 90. issue 9. 2013-02-15. PMID:22966077. |
differences between plp and np groups were most marked after glucose feeding with differences in peak glucose (p = 0.02) and peak insulin (p = 0.016) concentrations. |
2013-02-15 |
2023-08-12 |
Not clear |
| K E Borer, S R Bailey, N J Menzies-Gow, P A Harris, J Elliot. Effect of feeding glucose, fructose, and inulin on blood glucose and insulin concentrations in normal ponies and those predisposed to laminitis. Journal of animal science. vol 90. issue 9. 2013-02-15. PMID:22966077. |
peak concentrations of glucose and insulin occurred 2 to 4 h after wsc feeding. |
2013-02-15 |
2023-08-12 |
Not clear |
| K E Borer, S R Bailey, N J Menzies-Gow, P A Harris, J Elliot. Effect of feeding glucose, fructose, and inulin on blood glucose and insulin concentrations in normal ponies and those predisposed to laminitis. Journal of animal science. vol 90. issue 9. 2013-02-15. PMID:22966077. |
insulin auc was greater in plp than np after feeding both glucose and fructose (p = 0.017), but there were no differences between plp and np in glucose auc. |
2013-02-15 |
2023-08-12 |
Not clear |
| K E Borer, S R Bailey, N J Menzies-Gow, P A Harris, J Elliot. Effect of feeding glucose, fructose, and inulin on blood glucose and insulin concentrations in normal ponies and those predisposed to laminitis. Journal of animal science. vol 90. issue 9. 2013-02-15. PMID:22966077. |
minimal changes in glucose and insulin concentrations occurred after inulin feeding. |
2013-02-15 |
2023-08-12 |
Not clear |
| K E Borer, S R Bailey, N J Menzies-Gow, P A Harris, J Elliot. Effect of feeding glucose, fructose, and inulin on blood glucose and insulin concentrations in normal ponies and those predisposed to laminitis. Journal of animal science. vol 90. issue 9. 2013-02-15. PMID:22966077. |
measurement of peak insulin 2 h after feeding of a single dose of glucose (1 g/kg) may be a simple and practical way to aid identification of laminitis-prone ponies before the onset of clinical disease, particularly when ponies are adapted to eating fall pasture. |
2013-02-15 |
2023-08-12 |
Not clear |
| Krystal L Edwards, Megan Stapleton, Jessica Weis, Brian K Iron. An update in incretin-based therapy: a focus on glucagon-like peptide-1 receptor agonists. Diabetes technology & therapeutics. vol 14. issue 10. 2013-02-12. PMID:22845681. |
their mechanism of action is to increase insulin secretion, decrease glucagon release, reduce food intake, and slow gastric emptying. |
2013-02-12 |
2023-08-12 |
Not clear |
| Melissa L Borg, Simin Fallah Omran, Jacquelyn Weir, Peter J Meikle, Matthew J Wat. Consumption of a high-fat diet, but not regular endurance exercise training, regulates hypothalamic lipid accumulation in mice. The Journal of physiology. vol 590. issue 17. 2013-02-11. PMID:22674717. |
insulin resistance also develops in the central nervous system with high-fat feeding. |
2013-02-11 |
2023-08-12 |
mouse |
| Michael E Doche, Elena G Bochukova, Hsiao-Wen Su, Laura R Pearce, Julia M Keogh, Elana Henning, Joel M Cline, Sadia Saeed, Anne Dale, Tim Cheetham, Inês Barroso, Lawrence S Argetsinger, Stephen O'Rahilly, Liangyou Rui, Christin Carter-Su, I Sadaf Farooq. Human SH2B1 mutations are associated with maladaptive behaviors and obesity. The Journal of clinical investigation. vol 122. issue 12. 2013-02-04. PMID:23160192. |
targeted deletion of sh2b1 in mice results in increased food intake, obesity, and insulin resistance, with an intermediate phenotype seen in heterozygous null mice on a high-fat diet. |
2013-02-04 |
2023-08-12 |
mouse |
| Chisato Sugiyama, Michiyo Yamamoto, Tomomi Kotani, Fumitaka Kikkawa, Yoshiharu Murata, Yoshitaka Hayash. Fertility and pregnancy-associated ß-cell proliferation in mice deficient in proglucagon-derived peptides. PloS one. vol 7. issue 8. 2013-01-29. PMID:22928026. |
however, serum insulin levels in pregnant gcg(gfp/gfp) females were lower than those in control pregnant females under ad libitum feeding, and blood glucose levels in pregnant gcg(gfp/gfp) females were higher after gestational day 12. |
2013-01-29 |
2023-08-12 |
mouse |