All Relations between feeding and insulin

Publication Sentence Publish Date Extraction Date Species
Katsutaka Oishi, Manami Kasamatsu, Norio Ishid. Gene- and tissue-specific alterations of circadian clock gene expression in streptozotocin-induced diabetic mice under restricted feeding. Biochemical and biophysical research communications. vol 317. issue 2. 2004-06-01. PMID:15063761. the expression phase of the circadian output gene, albumin d-site binding protein (dbp), was completely shifted by rf both in normal and in diabetic mice, suggesting that the endogenous insulin is not essential for the entrainment of peripheral clocks to feeding cycles in mice. 2004-06-01 2023-08-12 mouse
Gordon L Carlso. Hunterian Lecture: Insulin resistance in human sepsis: implications for the nutritional and metabolic care of the critically ill surgical patient. Annals of the Royal College of Surgeons of England. vol 86. issue 2. 2004-05-27. PMID:15005922. loss of the anabolic effect of insulin (insulin resistance) is a key component of the adverse metabolic consequences of sepsis and may contribute to the apparent lack of efficacy of feeding regimens in critically ill patients. 2004-05-27 2023-08-12 human
W Yin, D Liao, M Kusunoki, S Xi, K Tsutsumi, Z Wang, X Lian, T Koike, J Fan, Y Yang, C Tan. NO-1886 decreases ectopic lipid deposition and protects pancreatic beta cells in diet-induced diabetic swine. The Journal of endocrinology. vol 180. issue 3. 2004-05-27. PMID:15012594. our results showed that feeding this diet to miniature swine caused insulin resistance, increased lipid deposition in non-adipose tissue, such as in the heart, skeletal muscle, liver and pancreas, and also caused pancreatic beta cell damage. 2004-05-27 2023-08-12 Not clear
Jeppe Sturis, Carsten F Gotfredsen, John Rømer, Bidda Rolin, Ulla Ribel, Christian L Brand, Michael Wilken, Karsten Wassermann, Carolyn F Deacon, Richard D Carr, Lotte Bjerre Knudse. GLP-1 derivative liraglutide in rats with beta-cell deficiencies: influence of metabolic state on beta-cell mass dynamics. British journal of pharmacology. vol 140. issue 1. 2004-05-25. PMID:12967942. blood glucose was approximately 12 mm lower compared to vehicle (p<0.0002), and plasma insulin was 2-3-fold higher during a normal 24-h feeding period (p<0.001). 2004-05-25 2023-08-12 rat
Dongzhe Song, Emi Arikawa, Denise Galipeau, Mary Battell, John H McNeil. Androgens are necessary for the development of fructose-induced hypertension. Hypertension (Dallas, Tex. : 1979). vol 43. issue 3. 2004-05-24. PMID:14757778. blood pressure was measured by a tail-cuff method, and an oral glucose tolerance test was performed to assess insulin sensitivity after 8 weeks of fructose feeding. 2004-05-24 2023-08-12 rat
Amy J Davidoff, Meredith M Mason, Michael B Davidson, Marybeth W Carmody, Kadon K Hintz, Loren E Wold, Deborah A Podolin, Jun Re. Sucrose-induced cardiomyocyte dysfunction is both preventable and reversible with clinically relevant treatments. American journal of physiology. Endocrinology and metabolism. vol 286. issue 5. 2004-05-19. PMID:15102617. su feeding produced hyperinsulinemia and hypertriglyceridemia, with euglycemia, and induced characteristic whole body insulin resistance. 2004-05-19 2023-08-12 rat
Ladislav Macho, Stefan Zorad, Zofia Radikova, Patricia Patterson-Buckedahl, Richard Kvetnansk. Ethanol consumption affects stress response and insulin binding in tissues of rats. Endocrine regulations. vol 37. issue 4. 2004-05-13. PMID:15106816. the aim of present study was to investigate the effect of short-term and long-term ethanol consumption on food intake, plasma levels of corticosterone (cs), glucose (g) and insulin (ins) in intact rats and in animals exposed to immobilization stress (imo) or restrain stress (restr). 2004-05-13 2023-08-12 rat
F Thabet, J Bourgeois, B Guy, G Pute. Continuous insulin infusion in hyperglycaemic very-low-birth-weight infants receiving parenteral nutrition. Clinical nutrition (Edinburgh, Scotland). vol 22. issue 6. 2004-05-12. PMID:14613756. during continuous insulin infusion, enteral feeding was started in all infants at 124.9+/-75.8 h of life. 2004-05-12 2023-08-12 Not clear
Cédric Asensio, Philippe Cettour-Rose, Claudia Theander-Carrillo, Françoise Rohner-Jeanrenaud, Patrick Muzzi. Changes in glycemia by leptin administration or high- fat feeding in rodent models of obesity/type 2 diabetes suggest a link between resistin expression and control of glucose homeostasis. Endocrinology. vol 145. issue 5. 2004-05-11. PMID:14962997. high-fat feeding in c57bl/6j wild-type mice, which is known to induce the development of obesity and insulin resistance, produced an increase in resistin expression. 2004-05-11 2023-08-12 mouse
Simonetta Bellone, Nadia Castellino, Fabio Broglio, Anna Rapa, Daniela Vivenza, Giorgio Radetti, Jaele Bellone, Cristina Gottero, Ezio Ghigo, Gianni Bon. Ghrelin secretion in childhood is refractory to the inhibitory effect of feeding. The Journal of clinical endocrinology and metabolism. vol 89. issue 4. 2004-05-11. PMID:15070928. ghrelin secretion is increased by fasting and energy restriction but decreased by food intake, glucose, insulin, and somatostatin. 2004-05-11 2023-08-12 Not clear
S Davail, N Rideau, G Guy, J M André, R Hoo-Pari. Pancreatic hormonal and metabolic responses in overfed ducks. Hormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme. vol 35. issue 7. 2004-05-05. PMID:12931276. with similar food intake, mule ducks (moderately overfed) showed global blood insulin lower than that of pekin ducks (intensively overfed) despite similar blood sugar levels, suggesting a trend towards reduced pancreas response to glucose in mule ducks. 2004-05-05 2023-08-12 Not clear
Dongmei Jia, Mitsuyoshi Yamamoto, Munenori Otani, Makoto Otsuk. Bezafibrate on lipids and glucose metabolism in obese diabetic Otsuka Long-Evans Tokushima fatty rats. Metabolism: clinical and experimental. vol 53. issue 4. 2004-05-04. PMID:15045684. bezafibrate treatment significantly reduced serum triglyceride (tg) and free fatty acid (ffa) levels, suppressed the increase in islet size, and inhibited the expression of alpha-smooth muscle actin, a marker for activated pancreatic stellate cells that are involved in the fibrosis of the pancreas, in the islets in oletf rats, but had no influences on food intake, body weight gain, abdominal adipose depots, and pancreatic insulin content in both strains of rats. 2004-05-04 2023-08-12 human
Alexandre Loktiono. Common gene polymorphisms and nutrition: emerging links with pathogenesis of multifactorial chronic diseases (review). The Journal of nutritional biochemistry. vol 14. issue 8. 2004-04-29. PMID:12948874. food intake control may be affected by polymorphisms in the genes encoding taste receptors and a number of peripheral signaling peptides such as insulin, leptin, ghrelin, cholecystokinin, and corresponding receptors. 2004-04-29 2023-08-12 human
Farid F Chehab, Jun Qiu, Scott Ogu. The use of animal models to dissect the biology of leptin. Recent progress in hormone research. vol 59. 2004-04-29. PMID:14749505. the vast amount of biology that has been ascribed to leptin encompasses effects on food intake, insulin sensitivity, adiposity, thermogenesis, reproduction, immunity, and bone regulation. 2004-04-29 2023-08-12 mouse
Stephen C Benoit, Deborah J Clegg, Randy J Seeley, Stephen C Wood. Insulin and leptin as adiposity signals. Recent progress in hormone research. vol 59. 2004-04-29. PMID:14749506. there is now considerable consensus that the adipocyte hormone leptin and the pancreatic hormone insulin are important regulators of food intake and energy balance. 2004-04-29 2023-08-12 Not clear
Stephen C Benoit, Deborah J Clegg, Randy J Seeley, Stephen C Wood. Insulin and leptin as adiposity signals. Recent progress in hormone research. vol 59. 2004-04-29. PMID:14749506. the brain expresses both insulin and leptin receptors in areas important in the control of food intake and energy balance. 2004-04-29 2023-08-12 Not clear
Stephen C Benoit, Deborah J Clegg, Randy J Seeley, Stephen C Wood. Insulin and leptin as adiposity signals. Recent progress in hormone research. vol 59. 2004-04-29. PMID:14749506. consistent with their roles as adiposity signals, exogenous leptin and insulin both reduce food intake when administered locally into the brain in a number of species under different experimental paradigms. 2004-04-29 2023-08-12 Not clear
Stephen C Benoit, Deborah J Clegg, Randy J Seeley, Stephen C Wood. Insulin and leptin as adiposity signals. Recent progress in hormone research. vol 59. 2004-04-29. PMID:14749506. additionally, central administration of insulin antibodies increases food intake and body weight. 2004-04-29 2023-08-12 Not clear
Jonathan R Seckl, Nik M Morton, Karen E Chapman, Brian R Walke. Glucocorticoids and 11beta-hydroxysteroid dehydrogenase in adipose tissue. Recent progress in hormone research. vol 59. 2004-04-29. PMID:14749510. conversely, 11beta-hsd1(-/-) mice resist the metabolic consequences of stress and high-fat feeding via insulin sensitisation and other advantageous effects in the liver and adipose tissue. 2004-04-29 2023-08-12 mouse
George A Bray, Samara Joy Nielsen, Barry M Popki. Consumption of high-fructose corn syrup in beverages may play a role in the epidemic of obesity. The American journal of clinical nutrition. vol 79. issue 4. 2004-04-27. PMID:15051594. because insulin and leptin act as key afferent signals in the regulation of food intake and body weight, this suggests that dietary fructose may contribute to increased energy intake and weight gain. 2004-04-27 2023-08-12 Not clear