| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Mariam Zaka, David A Wenge. Psychosine-induced apoptosis in a mouse oligodendrocyte progenitor cell line is mediated by caspase activation. Neuroscience letters. vol 358. issue 3. 2004-07-16. PMID:15039117. |
psychosine-induced apoptosis in a mouse oligodendrocyte progenitor cell line is mediated by caspase activation. |
2004-07-16 |
2023-08-12 |
mouse |
| Jagat R Kanwar, Rupinder K Kanwar, Geoffrey W Krissanse. Simultaneous neuroprotection and blockade of inflammation reverses autoimmune encephalomyelitis. Brain : a journal of neurology. vol 127. issue Pt 6. 2004-07-13. PMID:15130951. |
remarkably, administration at an advanced stage of unremitting eae of either a combination of nbqx and gpe, or preferably all three latter reagents, resulted in amelioration of disease and repair of the cns, as assessed by increased oligodendrocyte survival and remyelination, and corresponding decreased paralysis, inflammation, cns apoptosis and axonal damage. |
2004-07-13 |
2023-08-12 |
mouse |
| Jeffrey L Mason, Arrel Toews, Janell D Hostettler, Pierre Morell, Kinuko Suzuki, James E Goldman, Glenn K Matsushim. Oligodendrocytes and progenitors become progressively depleted within chronically demyelinated lesions. The American journal of pathology. vol 164. issue 5. 2004-06-04. PMID:15111314. |
although there is rapid regeneration of the oligodendrocyte population following an acute lesion, most of these newly regenerated cells undergo apoptosis if mice remain on a cuprizone diet. |
2004-06-04 |
2023-08-12 |
mouse |
| Jennifer K Ness, Russell C Scaduto, Teresa L Woo. IGF-I prevents glutamate-mediated bax translocation and cytochrome C release in O4+ oligodendrocyte progenitors. Glia. vol 46. issue 2. 2004-05-28. PMID:15042585. |
the results of these experiments define the mechanisms by which glutamate kills oligodendrocyte progenitor cells and by which igf-i blocks glutamate-induced apoptosis in these cells. |
2004-05-28 |
2023-08-12 |
Not clear |
| Christos Profyris, Surindar S Cheema, DaWei Zang, Michael F Azari, Kristy Boyle, Steven Petrato. Degenerative and regenerative mechanisms governing spinal cord injury. Neurobiology of disease. vol 15. issue 3. 2004-05-27. PMID:15056450. |
by indulging into the molecular mechanisms that cause oligodendrocyte apoptosis and identifying potential targets for therapeutic intervention, the prevention of this apoptotic wave will be of tremendous value to individuals living with sci. |
2004-05-27 |
2023-08-12 |
Not clear |
| David P Stirling, Kourosh Khodarahmi, Jie Liu, Lowell T McPhail, Christopher B McBride, John D Steeves, Matt S Ramer, Wolfram Tetzlaf. Minocycline treatment reduces delayed oligodendrocyte death, attenuates axonal dieback, and improves functional outcome after spinal cord injury. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 24. issue 9. 2004-05-14. PMID:14999069. |
we examined the ability of minocycline to reduce oligodendrocyte apoptosis, microglial/macrophage activation, corticospinal tract (cst) dieback, and lesion size and to improve functional outcome after sci. |
2004-05-14 |
2023-08-12 |
rat |
| Michael H Barnett, John W Prinea. Relapsing and remitting multiple sclerosis: pathology of the newly forming lesion. Annals of neurology. vol 55. issue 4. 2004-05-07. PMID:15048884. |
pathological changes not previously associated with the formation of new symptomatic lesions were observed in seven cases, namely, extensive oligodendrocyte apoptosis and microglial activation in myelinated tissue containing few or no lymphocytes or myelin phagocytes. |
2004-05-07 |
2023-08-12 |
Not clear |
| Pascale Giraudon, Peggy Vincent, Carine Vuaillat, Olivier Verlaeten, Luis Cartier, Anne Marie-Cardine, Mireille Mutin, Armand Bensussan, Marie-Françoise Belin, Laurence Boumsel. Semaphorin CD100 from activated T lymphocytes induces process extension collapse in oligodendrocytes and death of immature neural cells. Journal of immunology (Baltimore, Md. : 1950). vol 172. issue 2. 2004-04-20. PMID:14707103. |
blockade by specific abs of soluble cd100 (scd100)/semaphorin 4d released by activated t cells, or treatment with rscd100, demonstrated that this immune semaphorin has the ability to collapse oligodendrocyte process extensions and to trigger neural cell apoptosis, most likely through receptors of the plexin family. |
2004-04-20 |
2023-08-12 |
human |
| David H Kim, Alexander R Vaccaro, Fraser C Henderson, Edward C Benze. Molecular biology of cervical myelopathy and spinal cord injury: role of oligodendrocyte apoptosis. The spine journal : official journal of the North American Spine Society. vol 3. issue 6. 2004-03-24. PMID:14609697. |
molecular biology of cervical myelopathy and spinal cord injury: role of oligodendrocyte apoptosis. |
2004-03-24 |
2023-08-12 |
Not clear |
| Mary R L Dawson, Annabella Polito, Joel M Levine, Richard Reynold. NG2-expressing glial progenitor cells: an abundant and widespread population of cycling cells in the adult rat CNS. Molecular and cellular neurosciences. vol 24. issue 2. 2004-01-23. PMID:14572468. |
since very little apoptosis was detected and brdu became increasingly present in oligodendrocytes after a 10-day pulse chase, with a concomitant decrease in ng2(+) brdu incorporating cells, we suggest that the size of the oligodendrocyte population may actually increase during adult life. |
2004-01-23 |
2023-08-12 |
rat |
| H Tomimoto, M Ihara, H Wakita, R Ohtani, J-X Lin, I Akiguchi, M Kinoshita, H Shibasak. Chronic cerebral hypoperfusion induces white matter lesions and loss of oligodendroglia with DNA fragmentation in the rat. Acta neuropathologica. vol 106. issue 6. 2004-01-16. PMID:13680276. |
in humans, cerebrovascular white matter lesions are accompanied by apoptosis of oligodendroglia, and have been thought to be caused by chronic cerebral ischemia. |
2004-01-16 |
2023-08-12 |
rat |
| H Tomimoto, M Ihara, H Wakita, R Ohtani, J-X Lin, I Akiguchi, M Kinoshita, H Shibasak. Chronic cerebral hypoperfusion induces white matter lesions and loss of oligodendroglia with DNA fragmentation in the rat. Acta neuropathologica. vol 106. issue 6. 2004-01-16. PMID:13680276. |
in the present study, we tested whether chronic cerebral hypoperfusion induces white matter lesions and apoptosis of oligodendroglia in the rat. |
2004-01-16 |
2023-08-12 |
rat |
| Yin Liu, Yingyun Cai, Xuming Zhan. Induction of caspase-dependent apoptosis in cultured rat oligodendrocytes by murine coronavirus is mediated during cell entry and does not require virus replication. Journal of virology. vol 77. issue 22. 2003-11-25. PMID:14581532. |
however, whether mhv can directly induce oligodendrocyte apoptosis has not been documented. |
2003-11-25 |
2023-08-12 |
mouse |
| Yin Liu, Yingyun Cai, Xuming Zhan. Induction of caspase-dependent apoptosis in cultured rat oligodendrocytes by murine coronavirus is mediated during cell entry and does not require virus replication. Journal of virology. vol 77. issue 22. 2003-11-25. PMID:14581532. |
this finding provides an explanation for the destruction of oligodendrocytes and the damage of myelin sheath in mhv-infected cns and suggests that oligodendrocyte apoptosis may be one of the underlying mechanisms for the pathogenesis of mhv-induced demyelinating diseases in animals. |
2003-11-25 |
2023-08-12 |
mouse |
| Christopher B McBride, Lowell T McPhail, Jacqueline L Vanderluit, Wolfram Tetzlaff, John D Steeve. Caspase inhibition attenuates transection-induced oligodendrocyte apoptosis in the developing chick spinal cord. Molecular and cellular neurosciences. vol 23. issue 3. 2003-09-11. PMID:12837623. |
caspase inhibition attenuates transection-induced oligodendrocyte apoptosis in the developing chick spinal cord. |
2003-09-11 |
2023-08-12 |
chicken |
| Christopher B McBride, Lowell T McPhail, Jacqueline L Vanderluit, Wolfram Tetzlaff, John D Steeve. Caspase inhibition attenuates transection-induced oligodendrocyte apoptosis in the developing chick spinal cord. Molecular and cellular neurosciences. vol 23. issue 3. 2003-09-11. PMID:12837623. |
a developmental model of spinal cord injury in the embryonic chick was specifically developed to characterize the involvement of caspases in injury-induced oligodendrocyte apoptosis remote from the lesion and the ability of caspase inhibitors to attenuate this process. |
2003-09-11 |
2023-08-12 |
chicken |
| Ju Young Kim, Qin Sun, Michael Oglesbee, Sung Ok Yoo. The role of ErbB2 signaling in the onset of terminal differentiation of oligodendrocytes in vivo. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 23. issue 13. 2003-08-14. PMID:12843257. |
we report that a reduction in oligodendrocyte differentiation is attributed in part to apoptosis of oligodendrocyte progenitors as they exit the cell cycle. |
2003-08-14 |
2023-08-12 |
mouse |
| Amani Khorchid, Gabriela Fragoso, Gordon Shore, Guillermina Almaza. Catecholamine-induced oligodendrocyte cell death in culture is developmentally regulated and involves free radical generation and differential activation of caspase-3. Glia. vol 40. issue 3. 2003-08-01. PMID:12420309. |
a role for oxidative stress and apoptosis on the mechanism of action of catecholamines on oligodendrocyte cell death was next assessed. |
2003-08-01 |
2023-08-12 |
Not clear |
| Yun Cao, Alistair Jan Gunn, Laura Bennet, David Wu, Sherly George, Peter D Gluckman, Xiao-Mei Shao, Jian Gua. Insulin-like growth factor (IGF)-1 suppresses oligodendrocyte caspase-3 activation and increases glial proliferation after ischemia in near-term fetal sheep. Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism. vol 23. issue 6. 2003-07-17. PMID:12796722. |
thus, increased oligodendrocyte numbers after igf-1 treatment is partly due to suppression of apoptosis, and partly to increased proliferation. |
2003-07-17 |
2023-08-12 |
Not clear |
| Hans Lassmann, Markus Reindl, Helmut Rauschka, Johannes Berger, Fahmy Aboul-Enein, Thomas Berger, Andreas Zurbriggen, Andreas Lutterotti, Wolfgang Brück, Jörg R Weber, Robert Ullrich, Manfred Schmidbauer, Kurt Jellinger, Marc Vandeveld. A new paraclinical CSF marker for hypoxia-like tissue damage in multiple sclerosis lesions. Brain : a journal of neurology. vol 126. issue Pt 6. 2003-07-01. PMID:12764056. |
one of these patterns of demyelination, characterized by oligodendrocyte dystrophy and apoptosis, closely mimics myelin destruction in acute white matter ischaemia. |
2003-07-01 |
2023-08-12 |
Not clear |