| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| H Hu. Fas-mediated apoptosis in tumor formation and defense. Biological chemistry. vol 378. issue 12. 1998-03-18. PMID:9461339. |
tumor cells which became resistant to fas-mediated apoptosis should be insensitive to cytotoxic drugs that kill drug-sensitive cells by inducing the fas system. |
1998-03-18 |
2023-08-12 |
Not clear |
| S A Memon, J Hou, M B Moreno, C M Zacharchu. Apoptosis induced by a chimeric Fas/FLICE receptor: lack of requirement for Fas- or FADD-binding proteins. Journal of immunology (Baltimore, Md. : 1950). vol 160. issue 5. 1998-03-18. PMID:9498739. |
current models for fas (cd95)-mediated apoptosis suggest that flice/caspase-8 is recruited and activated, which results in cell death. |
1998-03-18 |
2023-08-12 |
Not clear |
| S A Memon, J Hou, M B Moreno, C M Zacharchu. Apoptosis induced by a chimeric Fas/FLICE receptor: lack of requirement for Fas- or FADD-binding proteins. Journal of immunology (Baltimore, Md. : 1950). vol 160. issue 5. 1998-03-18. PMID:9498739. |
a chimeric fas/flice (f/f) receptor, containing the extracellular/transmembrane portion of fas and the caspase region of flice, mediated anti-fas apoptosis. |
1998-03-18 |
2023-08-12 |
Not clear |
| S A Memon, J Hou, M B Moreno, C M Zacharchu. Apoptosis induced by a chimeric Fas/FLICE receptor: lack of requirement for Fas- or FADD-binding proteins. Journal of immunology (Baltimore, Md. : 1950). vol 160. issue 5. 1998-03-18. PMID:9498739. |
apoptosis triggered through fas and f/f was inhibited by coexpression of crma and p35, but not bcl-xl. |
1998-03-18 |
2023-08-12 |
Not clear |
| S A Memon, J Hou, M B Moreno, C M Zacharchu. Apoptosis induced by a chimeric Fas/FLICE receptor: lack of requirement for Fas- or FADD-binding proteins. Journal of immunology (Baltimore, Md. : 1950). vol 160. issue 5. 1998-03-18. PMID:9498739. |
these results show that: 1) f/f induces cell death, indicating that flice activation is sufficient for apoptosis and does not require additional fas- or fadd-binding proteins; and 2) f/f bypasses proximal defects in fas signaling that prevent flice recruitment or activation. |
1998-03-18 |
2023-08-12 |
Not clear |
| L Van Parijs, A Biuckians, A K Abba. Functional roles of Fas and Bcl-2-regulated apoptosis of T lymphocytes. Journal of immunology (Baltimore, Md. : 1950). vol 160. issue 5. 1998-03-18. PMID:9498742. |
functional roles of fas and bcl-2-regulated apoptosis of t lymphocytes. |
1998-03-18 |
2023-08-12 |
mouse |
| L Van Parijs, A Biuckians, A K Abba. Functional roles of Fas and Bcl-2-regulated apoptosis of T lymphocytes. Journal of immunology (Baltimore, Md. : 1950). vol 160. issue 5. 1998-03-18. PMID:9498742. |
these results demonstrate that fas and bcl-2 regulate different pathways of apoptosis that may serve distinct functions in lymphocyte homeostasis and in the maintenance of t cell tolerance. |
1998-03-18 |
2023-08-12 |
mouse |
| D M Lewinsohn, T T Bement, J Xu, D H Lynch, K H Grabstein, S G Reed, M R Alderso. Human purified protein derivative-specific CD4+ T cells use both CD95-dependent and CD95-independent cytolytic mechanisms. Journal of immunology (Baltimore, Md. : 1950). vol 160. issue 5. 1998-03-18. PMID:9498779. |
data generated using murine t cells have suggested a critical role for cd95 (fas, apo-1) in cd4+ t cell-induced apoptosis of target cells. |
1998-03-18 |
2023-08-12 |
human |
| B Combadière, C Reis e Sousa, R N Germain, M J Lenard. Selective induction of apoptosis in mature T lymphocytes by variant T cell receptor ligands. The Journal of experimental medicine. vol 187. issue 3. 1998-03-13. PMID:9449715. |
here we describe altered ligands for mature mouse cd4(+) t helper 1 cells that lead to t cell apoptosis by the selective expression of fas ligand (fasl) and tumor necrosis factor (tnf) without concomitant il-2, il-3, or interferon gamma production. |
1998-03-13 |
2023-08-12 |
mouse |
| H Hebestreit, B Dibbert, I Balatti, D Braun, A Schapowal, K Blaser, H U Simo. Disruption of fas receptor signaling by nitric oxide in eosinophils. The Journal of experimental medicine. vol 187. issue 3. 1998-03-13. PMID:9449721. |
it has been suggested that fas ligand-fas receptor interactions are involved in the regulation of eosinophil apoptosis and that dysfunctions in this system could contribute to the accumulation of these cells in allergic and asthmatic diseases. |
1998-03-13 |
2023-08-12 |
human |
| H Hebestreit, B Dibbert, I Balatti, D Braun, A Schapowal, K Blaser, H U Simo. Disruption of fas receptor signaling by nitric oxide in eosinophils. The Journal of experimental medicine. vol 187. issue 3. 1998-03-13. PMID:9449721. |
here, we demonstrate that nitric oxide (no) specifically prevents fas receptor-mediated apoptosis in freshly isolated human eosinophils. |
1998-03-13 |
2023-08-12 |
human |
| H Hebestreit, B Dibbert, I Balatti, D Braun, A Schapowal, K Blaser, H U Simo. Disruption of fas receptor signaling by nitric oxide in eosinophils. The Journal of experimental medicine. vol 187. issue 3. 1998-03-13. PMID:9449721. |
in contrast, rapid acceleration of eosinophil apoptosis by activation of the fas receptor occurs in the presence of eosinophil hematopoietins. |
1998-03-13 |
2023-08-12 |
human |
| H Hebestreit, B Dibbert, I Balatti, D Braun, A Schapowal, K Blaser, H U Simo. Disruption of fas receptor signaling by nitric oxide in eosinophils. The Journal of experimental medicine. vol 187. issue 3. 1998-03-13. PMID:9449721. |
therefore, no concentrations within allergic inflammatory sites may be important in determining whether an eosinophil survives or undergoes apoptosis upon fas ligand stimulation. |
1998-03-13 |
2023-08-12 |
human |
| Y Nishimura-Morita, M Nose, T Inoue, S Yonehar. Amelioration of systemic autoimmune disease by the stimulation of apoptosis-promoting receptor Fas with anti-Fas mAb. International immunology. vol 9. issue 12. 1998-03-13. PMID:9466307. |
administration of the hamster anti-mouse fas mab rk-8, which induced apoptosis both in vivo and in vitro, did not kill adult mice, whereas those given another hamster anti-mouse fas mab jo2 rapidly die of fulminant hepatitis with hemorrhage. |
1998-03-13 |
2023-08-12 |
mouse |
| T L Yue, X L Ma, X Wang, A M Romanic, G L Liu, C Louden, J L Gu, S Kumar, G Poste, R R Ruffolo, G Z Feuerstei. Possible involvement of stress-activated protein kinase signaling pathway and Fas receptor expression in prevention of ischemia/reperfusion-induced cardiomyocyte apoptosis by carvedilol. Circulation research. vol 82. issue 2. 1998-03-12. PMID:9468187. |
possible involvement of stress-activated protein kinase signaling pathway and fas receptor expression in prevention of ischemia/reperfusion-induced cardiomyocyte apoptosis by carvedilol. |
1998-03-12 |
2023-08-12 |
rabbit |
| T L Yue, X L Ma, X Wang, A M Romanic, G L Liu, C Louden, J L Gu, S Kumar, G Poste, R R Ruffolo, G Z Feuerstei. Possible involvement of stress-activated protein kinase signaling pathway and Fas receptor expression in prevention of ischemia/reperfusion-induced cardiomyocyte apoptosis by carvedilol. Circulation research. vol 82. issue 2. 1998-03-12. PMID:9468187. |
taken together, these results suggest that carvedilol prevents myocardial ischemia/reperfusion-induced apoptosis in cardiomyocytes possibly by downregulation of the sapk signaling pathway, by inhibition of fas receptor expression, and by beta-adrenergic blockade. |
1998-03-12 |
2023-08-12 |
rabbit |
| A Jelaska, J H Kor. Anti-Fas induces apoptosis and proliferation in human dermal fibroblasts: differences between foreskin and adult fibroblasts. Journal of cellular physiology. vol 175. issue 1. 1998-03-12. PMID:9491777. |
thus, this difference in apoptosis between adult dermal and foreskin fibroblasts appears to be related to the level of fas expression. |
1998-03-12 |
2023-08-12 |
human |
| A Jelaska, J H Kor. Anti-Fas induces apoptosis and proliferation in human dermal fibroblasts: differences between foreskin and adult fibroblasts. Journal of cellular physiology. vol 175. issue 1. 1998-03-12. PMID:9491777. |
when clones of foreskin fibroblasts were examined, there was heterogeneity of anti-fas antibody-induced apoptosis and proliferation in the cloned fibroblast subpopulations, but this was not correlated with differences in fas expression. |
1998-03-12 |
2023-08-12 |
human |
| M Sata, H Perlman, D A Muruve, M Silver, M Ikebe, T A Libermann, P Oettgen, K Wals. Fas ligand gene transfer to the vessel wall inhibits neointima formation and overrides the adenovirus-mediated T cell response. Proceedings of the National Academy of Sciences of the United States of America. vol 95. issue 3. 1998-03-11. PMID:9448311. |
fas ligand (fasl) induces apoptosis in fas-bearing cells, and its expression on activated t cells contributes to the regulation of the immune response and physiological cell turnover. |
1998-03-11 |
2023-08-12 |
rat |
| D R Hinton, S He, P F Lope. Apoptosis in surgically excised choroidal neovascular membranes in age-related macular degeneration. Archives of ophthalmology (Chicago, Ill. : 1960). vol 116. issue 2. 1998-03-09. PMID:9488273. |
the purpose of this study was to determine whether apoptosis was involved in the evolution of these changes, what cell types are involved, and whether there was an association with the fas antigen (fas or cd95) and fas ligand (fasl). |
1998-03-09 |
2023-08-12 |
Not clear |