All Relations between Multiple Sclerosis and oligodendrocyte

Publication Sentence Publish Date Extraction Date Species
Falak Sher, Veerakumar Balasubramaniyan, Erik Boddeke, Sjef Copra. Oligodendrocyte differentiation and implantation: new insights for remyelinating cell therapy. Current opinion in neurology. vol 21. issue 5. 2008-10-20. PMID:18769257. in this review, we highlight the current knowledge on oligodendrocyte differentiation and discuss the novel possibilities of neural stem cell-derived oligodendrocytes for graft-based remyelination therapy, for example, for multiple sclerosis. 2008-10-20 2023-08-12 Not clear
Eva Herrero-Herranz, Luis A Pardo, Ralf Gold, Ralf A Linke. Pattern of axonal injury in murine myelin oligodendrocyte glycoprotein induced experimental autoimmune encephalomyelitis: implications for multiple sclerosis. Neurobiology of disease. vol 30. issue 2. 2008-10-13. PMID:18342527. pattern of axonal injury in murine myelin oligodendrocyte glycoprotein induced experimental autoimmune encephalomyelitis: implications for multiple sclerosis. 2008-10-13 2023-08-12 human
Cecilia B Marta, Rashmi Bansal, Steven E Pfeiffe. Microglial Fc receptors mediate physiological changes resulting from antibody cross-linking of myelin oligodendrocyte glycoprotein. Journal of neuroimmunology. vol 196. issue 1-2. 2008-09-24. PMID:18406472. antibodies to myelin oligodendrocyte glycoprotein (mog) have been implicated in multiple sclerosis demyelination through activation of complement and/or macrophage-effector processes. 2008-09-24 2023-08-12 Not clear
Andre M Mueller, Xiomara Pedré, Thomas Stempfl, Ingo Kleiter, Sebastien Couillard-Despres, Ludwig Aigner, Gerhard Giegerich, Andreas Steinbreche. Novel role for SLPI in MOG-induced EAE revealed by spinal cord expression analysis. Journal of neuroinflammation. vol 5. 2008-08-21. PMID:18501024. experimental autoimmune encephalomyelitis (eae) induced by myelin oligodendrocyte protein (mog) in female dark agouti (da) rats is a chronic demyelinating animal model of multiple sclerosis (ms). 2008-08-21 2023-08-12 rat
Constanze Breithaupt, Beatrix Schäfer, Hannah Pellkofer, Robert Huber, Christopher Linington, Uwe Jaco. Demyelinating myelin oligodendrocyte glycoprotein-specific autoantibody response is focused on one dominant conformational epitope region in rodents. Journal of immunology (Baltimore, Md. : 1950). vol 181. issue 2. 2008-08-12. PMID:18606679. conformational epitopes of myelin oligodendrocyte glycoprotein (mog) provide a major target for demyelinating autoantibodies in experimental autoimmune encephalomyelitis and recent studies indicate that a similar situation may exist in multiple sclerosis. 2008-08-12 2023-08-12 mouse
Vincent Pernet, Sandrine Joly, Franziska Christ, Leda Dimou, Martin E Schwa. Nogo-A and myelin-associated glycoprotein differently regulate oligodendrocyte maturation and myelin formation. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 28. issue 29. 2008-08-12. PMID:18632947. together, our results demonstrate that nogo-a and mag are differently involved in oligodendrocyte maturation in vivo, and suggest that nogo-a may influence also remyelination in pathological conditions such as multiple sclerosis. 2008-08-12 2023-08-12 mouse
Celia Michèle Kassmann, Klaus-Armin Nav. Oligodendroglial impact on axonal function and survival - a hypothesis. Current opinion in neurology. vol 21. issue 3. 2008-07-25. PMID:18451704. although multiple sclerosis is considered the prototype of a primary autoimmune disease in the central nervous system, there is emerging evidence that primary oligodendrocyte dysfunctions can suffice to trigger a secondary immune response in the nervous system. 2008-07-25 2023-08-12 Not clear
Yasunori Uemura, Hiroaki Ohno, Yumiko Ohzeki, Hiromi Takanashi, Hideko Murooka, Kazuo Kubo, Isao Serizaw. The selective M-CSF receptor tyrosine kinase inhibitor Ki20227 suppresses experimental autoimmune encephalomyelitis. Journal of neuroimmunology. vol 195. issue 1-2. 2008-07-10. PMID:18378004. experimental autoimmune encephalomyelitis (eae), an animal model of multiple sclerosis (ms), can be induced by the immunization of mice with myelin antigens in the form of myelin oligodendrocyte glycoprotein (mog). 2008-07-10 2023-08-12 mouse
Olatz Pampliega, María Domercq, Pablo Villoslada, Jorge Sepulcre, Alfredo Rodríguez-Antigüedad, Carlos Matut. Association of an EAAT2 polymorphism with higher glutamate concentration in relapsing multiple sclerosis. Journal of neuroimmunology. vol 195. issue 1-2. 2008-07-10. PMID:18378006. glutamate excitotoxicity contributes to oligodendrocyte and tissue damage in multiple sclerosis (ms). 2008-07-10 2023-08-12 human
Ralf A Linker, Charlotte Weller, Fred Lühder, Alexander Mohr, Jens Schmidt, Michael Knauth, Josbert M Metselaar, Ralf Gol. Liposomal glucocorticosteroids in treatment of chronic autoimmune demyelination: long-term protective effects and enhanced efficacy of methylprednisolone formulations. Experimental neurology. vol 211. issue 2. 2008-07-07. PMID:18394606. here we compare liposomal prednisolone (pl) to liposomal methylprednisolone (mpl) in chronic-relapsing myelin oligodendrocyte glycoprotein-induced experimental autoimmune encephalomyelitis (eae), a model closely reflecting aspects of multiple sclerosis (ms). 2008-07-07 2023-08-12 rat
W F Blakemor. Regeneration and repair in multiple sclerosis: the view of experimental pathology. Journal of the neurological sciences. vol 265. issue 1-2. 2008-04-11. PMID:17459413. in order to devise a strategy to enhance remyelination in multiple sclerosis (ms) it is necessary to understand the cause of remyelination failure in ms. a case is made that areas of chronic demyelination arise because of concurrent loss of oligodendrocyte progenitor cells (opcs) and oligodendrocytes and that because of the slow rate of repopulation that occurs in old individuals the recruited opcs are not exposed to the acute inflammatory environment required to generate remyelinating oligodendrocytes. 2008-04-11 2023-08-12 Not clear
S Amor, G Giovannon. Antibodies to myelin oligodendrocyte glycoprotein as a biomarker in multiple sclerosis--are we there yet? Multiple sclerosis (Houndmills, Basingstoke, England). vol 13. issue 9. 2008-02-21. PMID:17967836. antibodies to myelin oligodendrocyte glycoprotein as a biomarker in multiple sclerosis--are we there yet? 2008-02-21 2023-08-12 Not clear
Huiliang Li, Yan Lu, Hazel K Smith, William D Richardso. Olig1 and Sox10 interact synergistically to drive myelin basic protein transcription in oligodendrocytes. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 27. issue 52. 2008-02-05. PMID:18160645. olig1 is important for differentiation of oligodendrocyte precursors into myelin-forming oligodendrocytes during development and is thought to play a crucial role in remyelination during multiple sclerosis. 2008-02-05 2023-08-12 mouse
Denise T Chung, Thomas Korn, Julie Richard, Melanie Ruzek, Adam P Kohm, Stephen Miller, Sharon Nahill, Mohamed Oukk. Anti-thymocyte globulin (ATG) prevents autoimmune encephalomyelitis by expanding myelin antigen-specific Foxp3+ regulatory T cells. International immunology. vol 19. issue 8. 2008-01-25. PMID:17698561. in order to test whether atg is effective in autoimmune disease, we used foxp3gfp 'knock-in' mice in combination with a myelin oligodendrocyte glycoprotein (mog)(35-55)/ia(b) tetramer to study more closely the effect of atg treatment on antigen-specific t cell responses in vivo during mog-induced experimental autoimmune encephalomyelitis (eae), an animal model for multiple sclerosis. 2008-01-25 2023-08-12 mouse
Amanda L Sheldon, Michael B Robinso. The role of glutamate transporters in neurodegenerative diseases and potential opportunities for intervention. Neurochemistry international. vol 51. issue 6-7. 2007-12-21. PMID:17517448. the latter phenomenon is known as 'excitotoxicity' and has been associated with a wide range of acute and chronic neurodegenerative disorders, as well as disorders that result in the loss of non-neural cells such as oligodendroglia in multiple sclerosis. 2007-12-21 2023-08-12 Not clear
Thomas Korn, Jayagopala Reddy, Wenda Gao, Estelle Bettelli, Amit Awasthi, Troels R Petersen, B Thomas Bäckström, Raymond A Sobel, Kai W Wucherpfennig, Terry B Strom, Mohamed Oukka, Vijay K Kuchro. Myelin-specific regulatory T cells accumulate in the CNS but fail to control autoimmune inflammation. Nature medicine. vol 13. issue 4. 2007-11-21. PMID:17384649. thus, we developed foxp3gfp knock-in (foxp3gfp.ki) mice and myelin oligodendrocyte glycoprotein (mog)(35-55)/ia(b) (mhc class ii) tetramers to track autoantigen-specific effector t cells (t-eff) and t-reg in vivo during experimental autoimmune encephalomyelitis (eae), an animal model for multiple sclerosis. 2007-11-21 2023-08-12 mouse
Anna Williams, Gabrièle Piaton, Catherine Lubetzk. Astrocytes--friends or foes in multiple sclerosis? Glia. vol 55. issue 13. 2007-11-13. PMID:17626262. in multiple sclerosis (ms), the presence of demyelinating plaques has concentrated researchers' minds on the role of the oligodendrocyte in its pathophysiology. 2007-11-13 2023-08-12 Not clear
Thomas Stahnke, Christine Stadelmann, Anne Netzler, Wolfgang Brück, Christiane Richter-Landsber. Differential upregulation of heme oxygenase-1 (HSP32) in glial cells after oxidative stress and in demyelinating disorders. Journal of molecular neuroscience : MN. vol 32. issue 1. 2007-10-26. PMID:17873285. in this study we analyzed autopsy and biopsy brain samples of patients with multiple sclerosis (ms) and adem (acute disseminated leucoencephalomyelits) and spinal cord lesions of mouse eae (experimental autoimmune encephalomyelitis), which was actively induced by immunization with myelin oligodendrocyte glycoprotein (mog35-55) peptide, for the presence of ho-1. 2007-10-26 2023-08-12 mouse
Louise H Boyle, James A Traherne, Gemma Plotnek, Rosemary Ward, John Trowsdal. Splice variation in the cytoplasmic domains of myelin oligodendrocyte glycoprotein affects its cellular localisation and transport. Journal of neurochemistry. vol 102. issue 6. 2007-10-19. PMID:17573820. our findings highlight myelin oligodendrocyte glycoprotein splicing as a factor that could be critical to the phenotypic expression of multiple sclerosis. 2007-10-19 2023-08-12 Not clear
Barbara Cannella, Stefanie Gaupp, Kakuri M Omari, Cedric S Rain. Multiple sclerosis: death receptor expression and oligodendrocyte apoptosis in established lesions. Journal of neuroimmunology. vol 188. issue 1-2. 2007-10-19. PMID:17610960. multiple sclerosis: death receptor expression and oligodendrocyte apoptosis in established lesions. 2007-10-19 2023-08-12 human