| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| M Carbon, M Niethammer, S Peng, D Raymond, V Dhawan, T Chaly, Y Ma, S Bressman, D Eidelber. Abnormal striatal and thalamic dopamine neurotransmission: Genotype-related features of dystonia. Neurology. vol 72. issue 24. 2009-07-31. PMID:19528516. |
abnormal striatal and thalamic dopamine neurotransmission: genotype-related features of dystonia. |
2009-07-31 |
2023-08-12 |
Not clear |
| Ryuji Kaji, Kenta Sato, Wataru Sako, Satoshi Got. [Diagnosis and treatment of dystonia]. Rinsho shinkeigaku = Clinical neurology. vol 48. issue 11. 2009-04-08. PMID:19198096. |
the pathogenesis of dystonia is now being clarified, and includes abnormal neuroplasticity caused by the relative excess of dopamine in the matrix compartment of the striatum, the possible primary lesion being the striosome. |
2009-04-08 |
2023-08-12 |
Not clear |
| Haruo Shintak. [Biopterin and child neurologic disease]. No to hattatsu = Brain and development. vol 41. issue 1. 2009-03-30. PMID:19172809. |
the former is segawa's disease, which is characterized by dopa-responsive dystonia with marked diurnal fluctuation and is caused by a defect of gtpch, and the latter is sr deficiency, which is characterized by progressive psychomotor retardation, dystonia, and severe dopamine and serotonin deficiencies. |
2009-03-30 |
2023-08-12 |
Not clear |
| Jing-Fang Yang, Tao Wu, Jian-Yu Li, Yong-Jie Li, Yan-Li Zhang, Piu Cha. DYT1 mutations in early onset primary torsion dystonia and Parkinson disease patients in Chinese populations. Neuroscience letters. vol 450. issue 2. 2009-03-24. PMID:19038309. |
moreover, dystonia and parkinson disease share the common feature of reduced dopamine neurotransmission in the striatum, so we assumed that mutations in the dyt1 gene might have the same role in cases of early onset primary torsion dystonia (eoptd) and early onset parkinson disease (eopd) that present dystonia. |
2009-03-24 |
2023-08-12 |
human |
| Adam Harper, Mark Bayliss, Romi Saha, Amanda Scutt, Angus Nisbe. Late onset dopa-responsive dystonia with tremor, gait freezing and behavioural disturbance and a normal dopamine transporter scan. Age and ageing. vol 37. issue 6. 2009-02-09. PMID:18922834. |
late onset dopa-responsive dystonia with tremor, gait freezing and behavioural disturbance and a normal dopamine transporter scan. |
2009-02-09 |
2023-08-12 |
Not clear |
| Mary V Seeman, David Clodman, Gary Remingto. Transient tardive dystonia: overview and case presentation. Journal of psychiatric practice. vol 14. issue 4. 2008-12-12. PMID:18664896. |
tardive dystonia is a form of the disorder that starts after longer term use of dopamine antagonists. |
2008-12-12 |
2023-08-12 |
Not clear |
| Kenta Sato, Chiho Sumi-Ichinose, Ryuji Kaji, Kazuhisa Ikemoto, Takahide Nomura, Ikuko Nagatsu, Hiroshi Ichinose, Masayuki Ito, Wataru Sako, Shinji Nagahiro, Ann M Graybiel, Satoshi Got. Differential involvement of striosome and matrix dopamine systems in a transgenic model of dopa-responsive dystonia. Proceedings of the National Academy of Sciences of the United States of America. vol 105. issue 34. 2008-10-24. PMID:18713855. |
differential involvement of striosome and matrix dopamine systems in a transgenic model of dopa-responsive dystonia. |
2008-10-24 |
2023-08-12 |
mouse |
| Kenta Sato, Chiho Sumi-Ichinose, Ryuji Kaji, Kazuhisa Ikemoto, Takahide Nomura, Ikuko Nagatsu, Hiroshi Ichinose, Masayuki Ito, Wataru Sako, Shinji Nagahiro, Ann M Graybiel, Satoshi Got. Differential involvement of striosome and matrix dopamine systems in a transgenic model of dopa-responsive dystonia. Proceedings of the National Academy of Sciences of the United States of America. vol 105. issue 34. 2008-10-24. PMID:18713855. |
the mechanisms underlying the genesis of dystonia in drd present a challenge to models of basal ganglia movement control, given that striatal dopamine deficiency is the hallmark of parkinson's disease. |
2008-10-24 |
2023-08-12 |
mouse |
| Kenta Sato, Chiho Sumi-Ichinose, Ryuji Kaji, Kazuhisa Ikemoto, Takahide Nomura, Ikuko Nagatsu, Hiroshi Ichinose, Masayuki Ito, Wataru Sako, Shinji Nagahiro, Ann M Graybiel, Satoshi Got. Differential involvement of striosome and matrix dopamine systems in a transgenic model of dopa-responsive dystonia. Proceedings of the National Academy of Sciences of the United States of America. vol 105. issue 34. 2008-10-24. PMID:18713855. |
we suggest that the differential striosome-matrix pattern of dopamine loss could be a key to identifying the mechanisms underlying the genesis of dystonia in drd. |
2008-10-24 |
2023-08-12 |
mouse |
| Benjamin N R Cheyette, Sarah N R Cheyette, Kristina Cusmano-Ozog, Gregory M Enn. Dopa-responsive dystonia presenting as delayed and awkward gait. Pediatric neurology. vol 38. issue 4. 2008-07-10. PMID:18358407. |
dopa-responsive dystonia is a hereditary disease characterized by inadequate dopamine production. |
2008-07-10 |
2023-08-12 |
Not clear |
| Yu Zhao, Michael DeCuypere, Mark S LeDou. Abnormal motor function and dopamine neurotransmission in DYT1 DeltaGAG transgenic mice. Experimental neurology. vol 210. issue 2. 2008-06-20. PMID:18299128. |
increased striatal dopamine turnover in hmt1 mice suggests that the nigrostriatal pathway may be a site of functional neuropathology in dyt1 dystonia. |
2008-06-20 |
2023-08-12 |
mouse |
| Masaya Segaw. [Segawa disease]. Brain and nerve = Shinkei kenkyu no shinpo. vol 60. issue 1. 2008-04-11. PMID:18232327. |
however, it was also clarified existence of two types, one, classic type, postural dystonia and the other action dystonia with vigorous dystonic movements besides dystonic posture, which, is postulated to be caused by the dopamine neuron innervating to the subthalamic nucleus with d1 neuron. |
2008-04-11 |
2023-08-12 |
Not clear |
| Aygul Balcioglu, Mee-Ohk Kim, Nutan Sharma, Jang-Ho Cha, Xandra O Breakefield, David G Standaer. Dopamine release is impaired in a mouse model of DYT1 dystonia. Journal of neurochemistry. vol 102. issue 3. 2007-10-15. PMID:17550429. |
dopamine release is impaired in a mouse model of dyt1 dystonia. |
2007-10-15 |
2023-08-12 |
mouse |
| S D Tabbal, J W Mink, J A V Antenor, J L Carl, S M Moerlein, J S Perlmutte. 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine-induced acute transient dystonia in monkeys associated with low striatal dopamine. Neuroscience. vol 141. issue 3. 2006-10-18. PMID:16766129. |
1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine-induced acute transient dystonia in monkeys associated with low striatal dopamine. |
2006-10-18 |
2023-08-12 |
monkey |
| S D Tabbal, J W Mink, J A V Antenor, J L Carl, S M Moerlein, J S Perlmutte. 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine-induced acute transient dystonia in monkeys associated with low striatal dopamine. Neuroscience. vol 141. issue 3. 2006-10-18. PMID:16766129. |
effects were assessed with blinded clinical ratings of dystonia and parkinsonism; [18f]-6-fluoro-dopa (fdopa) positron emission tomography; and postmortem measurements of striatal dopamine content. |
2006-10-18 |
2023-08-12 |
monkey |
| S D Tabbal, J W Mink, J A V Antenor, J L Carl, S M Moerlein, J S Perlmutte. 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine-induced acute transient dystonia in monkeys associated with low striatal dopamine. Neuroscience. vol 141. issue 3. 2006-10-18. PMID:16766129. |
these findings extend the model of mptp-induced transient dystonia followed by chronic hemiparkinsonism to m. nemestrina and m. fascicularis and demonstrate that the early dystonic phase is accompanied by striatal dopamine deficiency. |
2006-10-18 |
2023-08-12 |
monkey |
| Ryuji Kaji, Satoshi Goto, Gen Tamiya, L V Le. [Molecular and anatomical bases of dystonia: X-linked recessive dystonia-parkinsonism (DYT3)]. Rinsho shinkeigaku = Clinical neurology. vol 45. issue 11. 2006-02-15. PMID:16447732. |
because striosomal neurons inhibit nigrostriatal dopaminergic neurons via gabaergic innervation, the striosomal lesion could account for dopamine excess in the striatum, which in turn causes a hyperkinetic state or dystonia. |
2006-02-15 |
2023-08-12 |
Not clear |
| Ryuji Kaji, Satoshi Goto, Gen Tamiya, Satoshi Ando, Satoshi Makino, L V Le. Molecular dissection and anatomical basis of dystonia: X-linked recessive dystonia-parkinsonism (DYT3). The journal of medical investigation : JMI. vol 52 Suppl. 2006-02-03. PMID:16366515. |
because striosomal neurons inhibit nigrostriatal dopaminergic neurons via gabaergic innervation, the striosomal lesion could account for dopamine excess in the striatum, which in turn causes a hyperkinetic state or dystonia. |
2006-02-03 |
2023-08-12 |
Not clear |
| Anjum Misbahuddin, Mark R Placzek, Jan-Willem Taanman, Steve Gschmeissner, Giampietro Schiavo, J Mark Cooper, Thomas T Warne. Mutant torsinA, which causes early-onset primary torsion dystonia, is redistributed to membranous structures enriched in vesicular monoamine transporter in cultured human SH-SY5Y cells. Movement disorders : official journal of the Movement Disorder Society. vol 20. issue 4. 2005-08-25. PMID:15593317. |
abnormal processing, transport, or entrapment of vmat2 within the mutant torsina membranous inclusions, therefore, may affect cellular dopamine release, providing a potential pathogenic mechanism for the dyt1-dependent dystonia. |
2005-08-25 |
2023-08-12 |
human |
| Melanie Hamann, Angelika Richte. Striatal increase of extracellular dopamine levels during dystonic episodes in a genetic model of paroxysmal dyskinesia. Neurobiology of disease. vol 16. issue 1. 2004-08-18. PMID:15207264. |
consistent with previous pharmacologic findings, paroxysmal dystonia in mutant hamsters is associated with temporary increases of extracellular dopamine levels in the striatum. |
2004-08-18 |
2023-08-12 |
Not clear |