| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Sh V Vashadz. [Serotonin and depression]. Klinicheskaia laboratornaia diagnostika. issue 10. 2007-01-10. PMID:17144541. |
[serotonin and depression]. |
2007-01-10 |
2023-08-12 |
Not clear |
| Leslie S Kersun, Anne E Kaza. Prescribing practices of selective serotonin reuptake inhibitors (SSRIs) among pediatric oncologists: a single institution experience. Pediatric blood & cancer. vol 47. issue 3. 2007-01-09. PMID:16007589. |
although children and adolescents with cancer often experience symptoms of anxiety and depression, there are no published data regarding the use of selective serotonin reuptake inhibitors (ssris) for treating depression in the pediatric oncology population. |
2007-01-09 |
2023-08-12 |
Not clear |
| Donald Mattison, Anne Zajice. Gaps in knowledge in treating pregnant women. Gender medicine. vol 3. issue 3. 2007-01-09. PMID:17081950. |
among women with depression who are treated with selective serotonin reuptake inhibitors, the dose needed to maintain efficacy increases across the course of pregnancy. |
2007-01-09 |
2023-08-12 |
human |
| . ACOG Committee Opinion No. 354: Treatment with selective serotonin reuptake inhibitors during pregnancy. Obstetrics and gynecology. vol 108. issue 6. 2007-01-09. PMID:17138801. |
depression is a common condition among women of reproductive age, and selective serotonin reuptake inhibitors (ssris) are frequently used for the treatment of depression. |
2007-01-09 |
2023-08-12 |
Not clear |
| Jitendra N Singh, Rajesh Gupta, Shripad B Deshpand. Ptychodiscus brevis toxin-induced depression of spinal reflexes involves 5-HT via 5-HT3 receptors modulated by NMDA receptor. Neuroscience letters. vol 409. issue 1. 2007-01-04. PMID:17014958. |
ptychodiscus brevis toxin-induced depression of spinal reflexes involves 5-ht via 5-ht3 receptors modulated by nmda receptor. |
2007-01-04 |
2023-08-12 |
rat |
| Jitendra N Singh, Rajesh Gupta, Shripad B Deshpand. Ptychodiscus brevis toxin-induced depression of spinal reflexes involves 5-HT via 5-HT3 receptors modulated by NMDA receptor. Neuroscience letters. vol 409. issue 1. 2007-01-04. PMID:17014958. |
the involvement of 5-hydroxytryptaminergic (5-ht) system for the ptychodiscus brevis toxin (pbtx)-induced depression of spinal reflexes was evaluated. |
2007-01-04 |
2023-08-12 |
rat |
| Jitendra N Singh, Rajesh Gupta, Shripad B Deshpand. Ptychodiscus brevis toxin-induced depression of spinal reflexes involves 5-HT via 5-HT3 receptors modulated by NMDA receptor. Neuroscience letters. vol 409. issue 1. 2007-01-04. PMID:17014958. |
ondansetron (0.1microm), a 5-ht(3) receptor antagonist, blocked the pbtx-induced depression of msr and psr. |
2007-01-04 |
2023-08-12 |
rat |
| Jitendra N Singh, Rajesh Gupta, Shripad B Deshpand. Ptychodiscus brevis toxin-induced depression of spinal reflexes involves 5-HT via 5-HT3 receptors modulated by NMDA receptor. Neuroscience letters. vol 409. issue 1. 2007-01-04. PMID:17014958. |
spiperone (a 5-ht(2a) antagonist) or ketanserin (5-ht(2a/2c) antagonist and also at 5-ht(1b/1d)) failed to block the pbtx-induced depression of the reflexes. |
2007-01-04 |
2023-08-12 |
rat |
| Jitendra N Singh, Rajesh Gupta, Shripad B Deshpand. Ptychodiscus brevis toxin-induced depression of spinal reflexes involves 5-HT via 5-HT3 receptors modulated by NMDA receptor. Neuroscience letters. vol 409. issue 1. 2007-01-04. PMID:17014958. |
superfusion of 5-ht or phenylbiguanide (pbg, a 5-ht(3) receptor agonist) also produced depression of the spinal reflexes in a concentration-dependent manner. |
2007-01-04 |
2023-08-12 |
rat |
| Jitendra N Singh, Rajesh Gupta, Shripad B Deshpand. Ptychodiscus brevis toxin-induced depression of spinal reflexes involves 5-HT via 5-HT3 receptors modulated by NMDA receptor. Neuroscience letters. vol 409. issue 1. 2007-01-04. PMID:17014958. |
the results demonstrate that the pbtx-induced depression of spinal reflexes involves 5-hydroxytryptamine via 5-ht(3) receptors modulated by nmda receptor. |
2007-01-04 |
2023-08-12 |
rat |
| Toru Kobayashi, Kazuo Washiyama, Kazutaka Iked. Inhibition of G protein-activated inwardly rectifying K+ channels by the antidepressant paroxetine. Journal of pharmacological sciences. vol 102. issue 3. 2007-01-04. PMID:17072103. |
paroxetine is commonly used as a selective serotonin reuptake inhibitor for the treatment of depression and other psychiatric disorders. |
2007-01-04 |
2023-08-12 |
xenopus_laevis |
| H Husum, S Aznar, S Høyer-Hansen, M Hald Larsen, J D Mikkelsen, A Møller, A A Mathé, G Wörtwei. Exacerbated loss of cell survival, neuropeptide Y-immunoreactive (IR) cells, and serotonin-IR fiber lengths in the dorsal hippocampus of the aged flinders sensitive line "depressed" rat: Implications for the pathophysiology of depression? Journal of neuroscience research. vol 84. issue 6. 2007-01-04. PMID:17099915. |
the flinders-sensitive line (fsl) rat represents a genetic model of depression with characterized 5-ht and npy abnormalities in the hippocampus. |
2007-01-04 |
2023-08-12 |
rat |
| Su-Jane Wang, Kai-Yun Wang, Wei-Chieh Wang, Talvinder S Sihr. Unexpected inhibitory regulation of glutamate release from rat cerebrocortical nerve terminals by presynaptic 5-hydroxytryptamine-2A receptors. Journal of neuroscience research. vol 84. issue 7. 2007-01-03. PMID:17016851. |
together, these results suggest that presynaptic 5-ht(2a) receptors present on glutamatergic terminals effect an unexpected depression of glutamate release by negatively modulating nerve terminal excitability and downstream vdcc activation through a signaling cascade involving phospholipase c/protein kinase c. these observations invoke presynaptic inhibitory 5-ht(2a) receptor function as a potential target for drugs to mitigate the effects of excessive glutamatergic transmission. |
2007-01-03 |
2023-08-12 |
rat |
| S Totterdel. The anatomy of co-morbid neuropsychiatric disorders based on cortico-limbic synaptic interactions. Neurotoxicity research. vol 10. issue 2. 2007-01-03. PMID:17062369. |
for example, depression may involve a lack of serotonin signalling, particularly in the prefrontal cortex, while in schizophrenia there is evidence for reduced dopamine signalling in the same brain region, co-existing with hyperactivity in the mesolimbic dopamine pathway. |
2007-01-03 |
2023-08-12 |
Not clear |
| S Logsdon, A F M Johnstone, K Viele, R L Coope. Regulation of synaptic vesicles pools within motor nerve terminals during short-term facilitation and neuromodulation. Journal of applied physiology (Bethesda, Md. : 1985). vol 100. issue 2. 2006-12-28. PMID:16210437. |
quantal recordings made directly at nerve terminals revealed smaller quantal sizes with tboa exposure with a marked increase in quantal size as well as a continual appearance of smaller quanta upon 5-ht treatment after tboa-induced depression. |
2006-12-28 |
2023-08-12 |
Not clear |
| David Heal. Manufacturing consensus. Culture, medicine and psychiatry. vol 30. issue 2. 2006-12-28. PMID:16804639. |
this paper explores how pharmaceutical companies manufacture an apparent academic consensus and, in so doing, gives a case study of the recent controversies surrounding the marketing of selective serotonin reuptake inhibitor (ssri) drugs for adolescent depression. |
2006-12-28 |
2023-08-12 |
Not clear |
| V Vazquez, C Gury, X Laqueill. [Methadone: from pharmacokinetic profile to clinical pharmacology]. L'Encephale. vol 32. issue 4 Pt 1. 2006-12-28. PMID:17099560. |
metabolism inhibitors such as selective serotonin reuptake inhibitors (fluvoxamine, fluoxetine, paroxetine, sertraline) or antifungals of the azol groups could enhance plasma concentration and may sometimes lead to respiratory depression or death. |
2006-12-28 |
2023-08-12 |
human |
| A Richard Gree. Neuropharmacology of 5-hydroxytryptamine. British journal of pharmacology. vol 147 Suppl 1. 2006-12-27. PMID:16402098. |
a considerable literature also now exists on the mechanisms by which many of the drugs used to treat psychiatric illness alter the functional activity of 5-ht, particularly the drugs used to treat depression. |
2006-12-27 |
2023-08-12 |
Not clear |
| A L T Uusitalo, E Vanninen, M Valkonen-Korhonen, J T Kuikk. Brain serotonin reuptake did not change during one year in overtrained athletes. International journal of sports medicine. vol 27. issue 9. 2006-12-22. PMID:16586330. |
5-ht reuptake did not correlate with the depression scores either in the whole group or in the oa. |
2006-12-22 |
2023-08-12 |
Not clear |
| A L T Uusitalo, E Vanninen, M Valkonen-Korhonen, J T Kuikk. Brain serotonin reuptake did not change during one year in overtrained athletes. International journal of sports medicine. vol 27. issue 9. 2006-12-22. PMID:16586330. |
furthermore, depression of overtrained athletes may be its own variant having no correlation with 5-ht reuptake in midbrain. |
2006-12-22 |
2023-08-12 |
Not clear |