| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| J E Madl, S M Roye. Glutamate in synaptic terminals is reduced by lack of glucose but not hypoxia in rat hippocampal slices. Neuroscience. vol 94. issue 2. 1999-12-21. PMID:10579205. |
glutamate in synaptic terminals is reduced by lack of glucose but not hypoxia in rat hippocampal slices. |
1999-12-21 |
2023-08-12 |
rat |
| J E Madl, S M Roye. Glutamate in synaptic terminals is reduced by lack of glucose but not hypoxia in rat hippocampal slices. Neuroscience. vol 94. issue 2. 1999-12-21. PMID:10579205. |
we examined the effects of two components of ischemia, hypoxia and hypoglycemia, on glutamate loss from rat hippocampal slices. |
1999-12-21 |
2023-08-12 |
rat |
| M Yuzaki, J A Conno. Characterization of L-homocysteate-induced currents in Purkinje cells from wild-type and NMDA receptor knockout mice. Journal of neurophysiology. vol 82. issue 5. 1999-12-17. PMID:10561449. |
we also found that in hippocampal neurons, the ec(50)s of hca for non-nmda receptors and for nmda receptors were more similar than originally reported; this finding indicates that hca is similar to other mixed agonists, such as glutamate. |
1999-12-17 |
2023-08-12 |
mouse |
| K Ikemune, A Mitani, S Namba, K Kataoka, T Ara. Functional changes of N-methyl-D-aspartic acid and alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionate channels in gerbil hippocampal CA1, in relation to postischemic enhancement of glutamate receptor-mediated responses. Neuroscience letters. vol 275. issue 2. 1999-12-16. PMID:10568515. |
gerbils were subjected to 5-min ischemia to induce the enhancement of glutamate receptor-mediated responses and the hippocampal slices were prepared 3 h after ischemia. |
1999-12-16 |
2023-08-12 |
Not clear |
| S M Lasley, M C Green, M E Gilber. Influence of exposure period on in vivo hippocampal glutamate and GABA release in rats chronically exposed to lead. Neurotoxicology. vol 20. issue 4. 1999-12-10. PMID:10499360. |
influence of exposure period on in vivo hippocampal glutamate and gaba release in rats chronically exposed to lead. |
1999-12-10 |
2023-08-12 |
rat |
| S M Lasley, M C Green, M E Gilber. Influence of exposure period on in vivo hippocampal glutamate and GABA release in rats chronically exposed to lead. Neurotoxicology. vol 20. issue 4. 1999-12-10. PMID:10499360. |
previous work has demonstrated that continual exposure to 0.2% lead (pb) beginning at birth diminishes depolarization-induced hippocampal glutamate (glu) and gaba release in vivo. |
1999-12-10 |
2023-08-12 |
rat |
| M G Packar. Glutamate infused posttraining into the hippocampus or caudate-putamen differentially strengthens place and response learning. Proceedings of the National Academy of Sciences of the United States of America. vol 96. issue 22. 1999-12-10. PMID:10536017. |
glutamate infused posttraining into the hippocampus or caudate-putamen differentially strengthens place and response learning. |
1999-12-10 |
2023-08-12 |
rat |
| M G Packar. Glutamate infused posttraining into the hippocampus or caudate-putamen differentially strengthens place and response learning. Proceedings of the National Academy of Sciences of the United States of America. vol 96. issue 22. 1999-12-10. PMID:10536017. |
rats receiving intrahippocampal injections of glutamate predominantly displayed place learning on days 8 and 16, indicating that manipulation of the hippocampus produced a blockade of the shift to response learning. |
1999-12-10 |
2023-08-12 |
rat |
| M G Packar. Glutamate infused posttraining into the hippocampus or caudate-putamen differentially strengthens place and response learning. Proceedings of the National Academy of Sciences of the United States of America. vol 96. issue 22. 1999-12-10. PMID:10536017. |
the findings suggest that posttraining intracerebral glutamate infusions can (i) modulate the distinct memory processes mediated by the hippocampus and caudate-putamen and (ii) bias the brain toward the use of a specific memory system to control learned behavior and thereby influence the timing of the switch from the use of cognitive memory to habit learning to guide behavior. |
1999-12-10 |
2023-08-12 |
rat |
| C Jang, Y Park, R W Rockhold, I K H. Autoradiographic study of NMDA-displaceable [3H]glutamate and [3H]MK-801 binding during butorphanol withdrawal in the rat brain. Brain research. vol 845. issue 2. 1999-12-10. PMID:10536204. |
nmda-displaceable [3h]glutamate binding was markedly increased in the cortical area, striatum, septum, hippocampus, thalamus, and cerebellum in 7 h withdrawal group and was significantly increased in the striatum, hippocampus, and thalamus in 24 h withdrawal group. |
1999-12-10 |
2023-08-12 |
rat |
| Y Xiao, X L. Polyunsaturated fatty acids modify mouse hippocampal neuronal excitability during excitotoxic or convulsant stimulation. Brain research. vol 846. issue 1. 1999-12-10. PMID:10536218. |
bath application of pentylenetetrazole (ptz) or glutamate reduced the stimulatory threshold and increased the frequency of action potentials of hippocampal neurons. |
1999-12-10 |
2023-08-12 |
mouse |
| J G Liu, R Li, G Q Li. l-S.R-daurisoline protects cultured hippocampal neurons against glutamate neurotoxicity by reducing nitric oxide production. Zhongguo yao li xue bao = Acta pharmacologica Sinica. vol 20. issue 1. 1999-12-09. PMID:10437119. |
l-s.r-daurisoline protects cultured hippocampal neurons against glutamate neurotoxicity by reducing nitric oxide production. |
1999-12-09 |
2023-08-12 |
Not clear |
| J G Liu, R Li, G Q Li. l-S.R-daurisoline protects cultured hippocampal neurons against glutamate neurotoxicity by reducing nitric oxide production. Zhongguo yao li xue bao = Acta pharmacologica Sinica. vol 20. issue 1. 1999-12-09. PMID:10437119. |
to explore mechanisms of l-s.r-daurisoline (ds)-mediated protection of cultured hippocampal neurons from sodium glutamate (glu) cytotoxicity. |
1999-12-09 |
2023-08-12 |
Not clear |
| F Angenstein, G Riedel, K G Reyman, S Staa. Transient translocation of protein kinase Cgamma in hippocampal long-term potentiation depends on activation of metabotropic glutamate receptors. Neuroscience. vol 93. issue 4. 1999-12-09. PMID:10501452. |
transient translocation of protein kinase cgamma in hippocampal long-term potentiation depends on activation of metabotropic glutamate receptors. |
1999-12-09 |
2023-08-12 |
rat |
| Y Zheng, P F Smith, C L Darlingto. Subregional analysis of amino acid levels in the guinea pig hippocampus following unilateral vestibular deafferentation. Journal of vestibular research : equilibrium & orientation. vol 9. issue 5. 1999-12-08. PMID:10544372. |
the aim of the present study was to examine levels of glutamine, glutamate, gamma-amino-butyric acid (gaba), glycine, serine, threonine and taurine in the ca1, ca2, ca3 and dentate gyrus (dg) subregions of the hippocampus at 10 hs following uvd in guinea pig, using high-performance liquid chromatography (hplc) with electrochemical detection (ecd). |
1999-12-08 |
2023-08-12 |
Not clear |
| E M Blanc, M Jallageas, M Recasens, J Guiraman. Potentiation of glutamatergic agonist-induced inositol phosphate formation by basic fibroblast growth factor is related to developmental features in hippocampal cultures: neuronal survival and glial cell proliferation. The European journal of neuroscience. vol 11. issue 10. 1999-12-08. PMID:10564345. |
we investigated the modulation by growth factors of phospholipase c (plc)-linked glutamate receptors during in vitro development of hippocampal cultures. |
1999-12-08 |
2023-08-12 |
Not clear |
| E M Blanc, M Jallageas, M Recasens, J Guiraman. Potentiation of glutamatergic agonist-induced inositol phosphate formation by basic fibroblast growth factor is related to developmental features in hippocampal cultures: neuronal survival and glial cell proliferation. The European journal of neuroscience. vol 11. issue 10. 1999-12-08. PMID:10564345. |
these results suggest that, in hippocampal cultures, bfgf modulates ampa and metabotropic glutamate receptors linked to the ip cascade, possibly in relation to the regulation of neuronal survival and glial cell proliferation, respectively. |
1999-12-08 |
2023-08-12 |
Not clear |
| D M Hodgson, A N Taylor, Z Zhang, A Rosenber. Lysosphingomyelin prevents behavioral aberrations and hippocampal neuron loss induced by the metabotropic glutamate receptor agonist quisqualate. Progress in neuro-psychopharmacology & biological psychiatry. vol 23. issue 5. 1999-12-02. PMID:10509381. |
lysosphingomyelin prevents behavioral aberrations and hippocampal neuron loss induced by the metabotropic glutamate receptor agonist quisqualate. |
1999-12-02 |
2023-08-12 |
rat |
| D M Hodgson, A N Taylor, Z Zhang, A Rosenber. Lysosphingomyelin prevents behavioral aberrations and hippocampal neuron loss induced by the metabotropic glutamate receptor agonist quisqualate. Progress in neuro-psychopharmacology & biological psychiatry. vol 23. issue 5. 1999-12-02. PMID:10509381. |
administration of the group 1 metabotropic glutamate receptor (mglur) agonist, quisqualate, produced seizures, akinesia, destruction of hippocampal pyramidal cell dendritic microtubule-associated protein-2, and major loss of hippocampal ca sector neurons. |
1999-12-02 |
2023-08-12 |
rat |
| Z L Chen, J A Indyk, T H Bugge, K W Kombrinck, J L Degen, S Stricklan. Neuronal death and blood-brain barrier breakdown after excitotoxic injury are independent processes. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 19. issue 22. 1999-12-02. PMID:10559390. |
we have used a glutamate analog injection model and genetically altered mice to investigate the relationship between these two processes in the hippocampus. |
1999-12-02 |
2023-08-12 |
mouse |