All Relations between hypertrophic and matrix compartment

Publication Sentence Publish Date Extraction Date Species
M N Campos, A R Cru. Relative growth of the condylar cartilage of the rat mandibular head during postnatal life. Gegenbaurs morphologisches Jahrbuch. vol 133. issue 4. 1987-12-21. PMID:3678782. the approximate 15% decrease of the cell contingent, the approximate 48% increase in cartilage matrix, the abrupt changes in the descriptive curve around the 15th to 20th d of postnatal life and the analysis of the relative growth pattern of the fibrous, embryogenic, cartilage, and hypertrophic cartilage layers were related to the functional adaptations which regulate the growth of the mandible condyle by a direct or indirect action on the temporal-mandibular articulation system. 1987-12-21 2023-08-11 rat
E Livne, C Oliver, R D Leapman, L C Rosenberg, A R Poole, M Silberman. Age-related changes in the role of matrix vesicles in the mandibular condylar cartilage. Journal of anatomy. vol 150. 1987-11-16. PMID:3308799. in one week old animals the most intense staining was observed in the matrix around the hypertrophic cells in the mineralising zone, to a lesser degree around the cells in the zone of chondroblasts, while no staining was noted in the zone of chondroprogenitor cells and in the matrix around the early hypertrophic cells. 1987-11-16 2023-08-11 mouse
E Livne, C Oliver, R D Leapman, L C Rosenberg, A R Poole, M Silberman. Age-related changes in the role of matrix vesicles in the mandibular condylar cartilage. Journal of anatomy. vol 150. 1987-11-16. PMID:3308799. the matrix between the early hypertrophic cells as shown by transmission electron microscopy revealed the presence of matrix vesicles and demonstrated a gradual accumulation of hydroxyapatite in the mineralising zone. 1987-11-16 2023-08-11 mouse
J Wroblewsk. Elemental changes associated with chondrocyte differentiation in rat rib growth plate. Histochemistry. vol 87. issue 2. 1987-10-22. PMID:3623998. levels of na, p, s, cl, k, and ca were determined in the cells and extracellular matrix in different zones of the cartilage--resting, proliferative, and hypertrophic. 1987-10-22 2023-08-11 rat
E Vignon, J Bejui, P Mathieu, J D Hartmann, G Ville, J C Evreux, J Descote. Histological cartilage changes in a rabbit model of osteoarthritis. The Journal of rheumatology. vol 14 Spec No. 1987-10-05. PMID:3625662. histological changes including cartilage hypertrophy, reduced cell density, matrix alterations, cystic lesions, and enlarged perichondrocytic lacunae were found to precede cartilage fibrillation. 1987-10-05 2023-08-11 rabbit
W T Grant, G J Wang, G Balia. Type X collagen synthesis during endochondral ossification in fracture repair. The Journal of biological chemistry. vol 262. issue 20. 1987-08-13. PMID:3597444. type x synthesis occurred in the areas of cartilaginous callus composed of hypertrophic and degenerative chondrocytes that were associated with increased vascularity and matrix mineralization. 1987-08-13 2023-08-11 chicken
P Hadjiisky, N Peyri, Y Grosgogea. Tunica media changes in the spontaneously hypertensive rat (SHR). Atherosclerosis. vol 65. issue 1-2. 1987-08-10. PMID:3606728. it seems due to three processes: early generalized hypertrophy of smooth muscle cells (smc); connective matrix neogenesis and smc proliferation, more evident in peripheral vasculature. 1987-08-10 2023-08-11 rat
M Jayo, H W Leipold, S M Dennis, F E Eldridg. Brachygnathia superior and degenerative joint disease: a new lethal syndrome in Angus calves. Veterinary pathology. vol 24. issue 2. 1987-05-22. PMID:3576909. growth plates had defective chondrocyte proliferation and hypertrophy with aberrant ossification of calcified cartilaginous matrix. 1987-05-22 2023-08-11 Not clear
E B Hunziker, R K Schenk, L M Cruz-Oriv. Quantitation of chondrocyte performance in growth-plate cartilage during longitudinal bone growth. The Journal of bone and joint surgery. American volume. vol 69. issue 2. 1987-03-23. PMID:3543020. the two most prominent stages are those of cellular proliferation and hypertrophy (including the mineralization of matrix) before the resorption of tissue during vascular invasion. 1987-03-23 2023-08-11 rat
E B Hunziker, R K Schenk, L M Cruz-Oriv. Quantitation of chondrocyte performance in growth-plate cartilage during longitudinal bone growth. The Journal of bone and joint surgery. American volume. vol 69. issue 2. 1987-03-23. PMID:3543020. by applying recently developed stereological procedures and improved methods for the fixation of cartilage, we compared cellular shape modulation, various ultrastructural parameters (surface areas or volumes of endoplasmic reticulum, golgi membranes, and mitochondria), the production of matrix, and cellular turnover for proliferating and hypertrophic chondrocytes within the proximal tibial growth plate of the rat. 1987-03-23 2023-08-11 rat
E B Hunziker, R K Schenk, L M Cruz-Oriv. Quantitation of chondrocyte performance in growth-plate cartilage during longitudinal bone growth. The Journal of bone and joint surgery. American volume. vol 69. issue 2. 1987-03-23. PMID:3543020. by the late hypertrophic stage, fourfold and tenfold increases in the mean cellular height and volume, respectively, and a threefold increase in the mean volume of the matrix per cell were achieved. 1987-03-23 2023-08-11 rat
S D Boden, F S Kaplan, M D Fallon, R Ruddy, J Belik, E Anday, E Zackai, J Elli. Metatropic dwarfism. Uncoupling of endochondral and perichondral growth. The Journal of bone and joint surgery. American volume. vol 69. issue 2. 1987-03-23. PMID:3805078. other observations included prominence of the cartilaginous canals and vascular channels in the reserve zone; clumping of chondrocytes with enhanced staining of the pericellular matrix in the proliferative zone; a decreased ratio of cells to matrix in the hypertrophic zone, with intracellular metachromatic granules and incomplete evolution of chondrocytes; complete absence of an alcian-blue-positive zone of provisional calcification; and, finally, islands of dysplastic chondrocytes in the metaphysis. 1987-03-23 2023-08-11 Not clear
M E Rosenfeld, T Tsukada, A Chait, E L Bierman, A M Gown, R Ros. Fatty streak expansion and maturation in Watanabe Heritable Hyperlipemic and comparably hypercholesterolemic fat-fed rabbits. Arteriosclerosis (Dallas, Tex.). vol 7. issue 1. 1987-03-17. PMID:3813974. in both groups of animals, the fatty streaks expanded due to: 1) the formation of multiple layers of a mixed population of macrophage-derived foam cells and lipid-containing smooth muscle cells, 2) the hypertrophy of the macrophage-derived foam cells, 3) the continued accumulation of extracellular matrix, 4) the insudation of plasma components. 1987-03-17 2023-08-11 Not clear
K T Weber, J S Janicki, R Pick, C Abrahams, S G Shroff, R I Bashey, R M Che. Collagen in the hypertrophied, pressure-overloaded myocardium. Circulation. vol 75. issue 1 Pt 2. 1987-02-05. PMID:2947751. the functional consequences of a remodeling of the collagen matrix that accompanied myocardial hypertrophy remain to be elucidated. 1987-02-05 2023-08-11 Not clear
L A de Freitas, M Chevallier, D Louis, J A Grimau. Human extrahepatic biliary atresia: portal connective tissue activation related to ductular proliferation. Liver. vol 6. issue 5. 1987-01-20. PMID:3537604. the connective matrix pattern observed in periductular areas can be compared to that described in cicatricial and hypertrophic processes where the myofibroblastic cell population is known to play an important role in fibrosis development. 1987-01-20 2023-08-11 human
N Kawabe, M G Ehrlich, H J Manki. In vivo degradation systems of the epiphyseal cartilage. Clinical orthopaedics and related research. issue 211. 1986-12-01. PMID:3769264. accumulated evidence suggests that in the growth cartilage during endochondral ossification, proteoglycans in the extracellular matrix of the lower hypertrophic zone are degraded by proteases and removed before mineralization. 1986-12-01 2023-08-11 human
B de Bernard, P Bianco, E Bonucci, M Costantini, G C Lunazzi, P Martinuzzi, C Modricky, L Moro, E Panfili, P Pollesell. Biochemical and immunohistochemical evidence that in cartilage an alkaline phosphatase is a Ca2+-binding glycoprotein. The Journal of cell biology. vol 103. issue 4. 1986-11-26. PMID:3771650. in the cartilage plate, between the proliferating and the hypertrophic region, a weak immune reactivity is seen in the cytoplasm, whereas in the intercolumnar matrix the collagen fibers appear clearly stained. 1986-11-26 2023-08-11 Not clear
B de Bernard, P Bianco, E Bonucci, M Costantini, G C Lunazzi, P Martinuzzi, C Modricky, L Moro, E Panfili, P Pollesell. Biochemical and immunohistochemical evidence that in cartilage an alkaline phosphatase is a Ca2+-binding glycoprotein. The Journal of cell biology. vol 103. issue 4. 1986-11-26. PMID:3771650. whereas resting cartilage, the intercellular matrix of the resting zone, and calcified matrix do not exhibit any enzyme activity, the zones of maturing and hypertrophic chondrocytes are highly reactive. 1986-11-26 2023-08-11 Not clear
B Wikström, A Hjerpe, F P Reinholt, B Engfeld. Stereological studies on the epiphyseal growth cartilage and characterization of costal cartilage proteoglycans in the achondroplastic (cn/cn) mouse. Collagen and related research. vol 6. issue 3. 1986-11-25. PMID:3769423. the pattern of matrix mineralization seems to be unaffected, even in areas where hypertrophic chondrocytes are completely absent. 1986-11-25 2023-08-11 mouse
R D Campo, J E Roman. Changes in cartilage proteoglycans associated with calcification. Calcified tissue international. vol 39. issue 3. 1986-11-20. PMID:3093029. based on in vivo experiments in which 35 s-sodium sulfate was injected into rabbits, it is shown that proteoglycans from the hypertrophic region becomes part of the calcified cartilage matrix which is to be incorporated into the metaphysis. 1986-11-20 2023-08-11 Not clear