All Relations between dopaminergic and dopamine

Publication Sentence Publish Date Extraction Date Species
N J Smatresk, S Lahir. Aortic body chemoreceptor responses to dopamine, haloperidol, and pargyline. Journal of applied physiology: respiratory, environmental and exercise physiology. vol 53. issue 3. 1982-12-18. PMID:7129979. aortic chemoreceptor activity, from single- or few-fiber afferent nerve preparations, was measured in response to dopamine and a dopaminergic blocker, haloperidol, in 18 anesthetized cats. 1982-12-18 2023-08-12 Not clear
M Toru, T Nishikawa, N Mataga, M Takashim. Dopamine metabolism increases in post-mortem schizophrenic basal ganglia. Journal of neural transmission. vol 54. issue 3-4. 1982-12-16. PMID:6127373. a remarkable low level of dopamine and a high level of homovanillic acid measured indicate this case would have had an increased turnover rate of dopamine in the dopaminergic nerve terminals. 1982-12-16 2023-08-12 Not clear
D R Sibley, S E Leff, I Crees. Interactions of novel dopaminergic ligands with D-1 and D-2 dopamine receptors. Life sciences. vol 31. issue 7. 1982-12-16. PMID:6127585. interactions of novel dopaminergic ligands with d-1 and d-2 dopamine receptors. 1982-12-16 2023-08-12 rat
D R Sibley, S E Leff, I Crees. Interactions of novel dopaminergic ligands with D-1 and D-2 dopamine receptors. Life sciences. vol 31. issue 7. 1982-12-16. PMID:6127585. the interactions of three novel dopaminergic ligands, skf38393, skf82526 and skf83742, with d-1 and d-2 dopamine (da) receptors have been investigated using radioligand binding techniques and computer modeling procedures. 1982-12-16 2023-08-12 rat
M W Hamblin, I Crees. Phenoxybenzamine treatment differentiates dopaminergic 3H-ligand binding sites in bovine caudate membranes. Molecular pharmacology. vol 21. issue 1. 1982-12-16. PMID:6127621. the effect is site-directed, as the dopaminergic agonists dopamine and apomorphine and the antagonist domperidone are able to protect against phenoxybenzamine-mediated attack in proportion to their affinities for d-2 sites. 1982-12-16 2023-08-12 cattle
K Bech, C P Hovendal, D Anderse. Effect of dopamine on pentagastrin-stimulated gastric antral motility in dogs with gastric fistula. Scandinavian journal of gastroenterology. vol 17. issue 1. 1982-12-16. PMID:6127786. dopamine, an endogenous catecholamine, was used alone and in conjunction with selective blockade or adrenergic and dopaminergic receptors. 1982-12-16 2023-08-12 Not clear
K Bech, C P Hovendal, D Anderse. Effect of dopamine on pentagastrin-stimulated gastric antral motility in dogs with gastric fistula. Scandinavian journal of gastroenterology. vol 17. issue 1. 1982-12-16. PMID:6127786. consequently, this study indicates that dopamine acts on gastric antral motility through dopaminergic receptors. 1982-12-16 2023-08-12 Not clear
C P Hovendal, K Bech, F Gottrup, D Anderse. Effect of dopamine on pentagastrin-stimulated gastric acid secretion and mucosal blood flow in dogs with gastric fistula. Scandinavian journal of gastroenterology. vol 17. issue 1. 1982-12-16. PMID:6127791. dopamine was used alone and in conjunction with selective blockade of alpha-, beta-, and dopaminergic receptors. 1982-12-16 2023-08-12 Not clear
C P Hovendal, K Bech, F Gottrup, D Anderse. Effect of dopamine on pentagastrin-stimulated gastric acid secretion and mucosal blood flow in dogs with gastric fistula. Scandinavian journal of gastroenterology. vol 17. issue 1. 1982-12-16. PMID:6127791. the anti-secretory effect dopamine was significantly blocked by non-selective beta-blockade or by selective beta-blockade but not by alpha- or dopaminergic receptor blockade. 1982-12-16 2023-08-12 Not clear
M W Hamblin, I Crees. Heat treatment mimics guanosine-5'-triphosphate effects on dopaminergic 3H-ligand binding to bovine caudate membranes. Molecular pharmacology. vol 21. issue 1. 1982-12-16. PMID:7132962. exposure of bovine caudate homogenates to 53 degrees rapidly (less than 4 min) abolishes subsequent specific binding of the agonist ligand [3h]dopamine to d-3 sites but not that of the butyrophenone dopaminergic antagonist [3h]spiroperidol to d-2 sites in bovine caudate membranes. 1982-12-16 2023-08-12 cattle
A E Pontiroli, G Loda, A Roggia, P Scagliola, L Falsett. Benserazide and nomifensine in the diagnosis of prolactin-secreting pituitary adenomas. Acta endocrinologica. vol 101. issue 2. 1982-12-16. PMID:7136447. benserazide, an inhibitor of dopa-decarboxylase, stimulates prolactin (prl) release in normal women and in puerperae; nomifensine, a dopaminergic drug able to release dopamine and to inhibit its re-uptake at the post-synaptic level, inhibits prl release in the same subjects. 1982-12-16 2023-08-12 human
M Itoh, B L Furman, J E Geric. Dopaminergic suppression of pancreatic somatostatin secretion. Acta endocrinologica. vol 101. issue 1. 1982-12-03. PMID:6126981. to characterize dopaminergic influences on pancreatic islet d cell function and its potential interaction with islet a and b cell function, the effect of dopamine (0.5-100 micro m) on immunoreactive somatostatin (irs), insulin (iri), and glucagon (irg) release from rat islets incubated in vitro was studied. 1982-12-03 2023-08-12 rat
M Itoh, B L Furman, J E Geric. Dopaminergic suppression of pancreatic somatostatin secretion. Acta endocrinologica. vol 101. issue 1. 1982-12-03. PMID:6126981. maximum suppression of irs and iri release was evident at 20 micro m dopamine with half-maximal suppression occurring at 0.5-1 micro m. maximal stimulation of irg release was observed at 100 micro m dopamine with a half-maximal response occurring at 5-10 micro m. suppression of irs secretion by dopamine (20 micro m) was completely reversed by the dopaminergic antagonists haloperidol (5 micro m) and pimozide (5 micro m) but was only partially reversed by the alpha adrenergic antagonist phentolamine (2 micro m), and was further suppressed by the beta adrenergic antagonist phentolamine (2 micro m). 1982-12-03 2023-08-12 rat
M Itoh, B L Furman, J E Geric. Dopaminergic suppression of pancreatic somatostatin secretion. Acta endocrinologica. vol 101. issue 1. 1982-12-03. PMID:6126981. dopamine suppresses irs secretion predominantly through activation of dopaminergic receptors, whereas it suppresses iri release through an alpha adrenergic mechanism and stimulates irg release through a beta adrenergic mechanism. 1982-12-03 2023-08-12 rat
C Ferrari, P Rampini, R Benco, R Caldara, C Scarduelli, P G Crosignan. Functional characterization of hypothalamic hyperprolactinemia. The Journal of clinical endocrinology and metabolism. vol 55. issue 5. 1982-12-03. PMID:6811605. the administration of substances resulting in stimulation of pituitary dopamine receptors, such as dopamine and l-dopa, induced a normal prl suppression in 7 patients with hypothalamic disease so tested, whereas central nervous system-acting dopaminergic drugs, such as carbidopa plus l-dopa and nomifensine, failed to lower prl levels in most cases (even when normoprolactinemic after surgery). 1982-12-03 2023-08-12 human
S J List, P Seema. [3H]dopamine labeling of D3 dopaminergic sites in human, rat, and calf brain. Journal of neurochemistry. vol 39. issue 5. 1982-12-03. PMID:7119802. [3h]dopamine labeling of d3 dopaminergic sites in human, rat, and calf brain. 1982-12-03 2023-08-12 human
S J List, P Seema. [3H]dopamine labeling of D3 dopaminergic sites in human, rat, and calf brain. Journal of neurochemistry. vol 39. issue 5. 1982-12-03. PMID:7119802. dopaminergic catecholamines (dopamine, apomorphine, 6,7-dihydroxy-2-aminotetralin, and n-propylnorapomorphine) inhibited the binding of [3h]dopamine in all three species, at low concentrations, with ic50 values of 1.5 to 6 nm. 1982-12-03 2023-08-12 human
S J List, P Seema. [3H]dopamine labeling of D3 dopaminergic sites in human, rat, and calf brain. Journal of neurochemistry. vol 39. issue 5. 1982-12-03. PMID:7119802. the optimum conditions for selective labeling of the d3 dopaminergic sites, using [3h]dopamine, required the presence of edta and ascorbate. 1982-12-03 2023-08-12 human
J C van Oene, H A Houwing, A S Hor. Evidence that the purported dopaminergic agonist (3,4-dihydroxyphenylimino)-2-imidazolidine (DPI) may reduce rat striatal dopamine turnover by an alpha 2-adrenergic mechanism. European journal of pharmacology. vol 81. issue 1. 1982-12-02. PMID:6126370. evidence that the purported dopaminergic agonist (3,4-dihydroxyphenylimino)-2-imidazolidine (dpi) may reduce rat striatal dopamine turnover by an alpha 2-adrenergic mechanism. 1982-12-02 2023-08-12 rat
J C van Oene, H A Houwing, A S Hor. Evidence that the purported dopaminergic agonist (3,4-dihydroxyphenylimino)-2-imidazolidine (DPI) may reduce rat striatal dopamine turnover by an alpha 2-adrenergic mechanism. European journal of pharmacology. vol 81. issue 1. 1982-12-02. PMID:6126370. the potent alpha-adrenergic agonist dpi, which has also been claimed to be a selective dopaminergic agonist, was shown to reduce rat striatal dopamine (da) synthesis, da utilization and da metabolism following intraperitoneal administration (25 mumol/kg). 1982-12-02 2023-08-12 rat