| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| D M Yurek, W Lu, S Hipkens, S J Wiegan. BDNF enhances the functional reinnervation of the striatum by grafted fetal dopamine neurons. Experimental neurology. vol 137. issue 1. 1996-03-01. PMID:8566202. |
transplantation of fetal dopaminergic neurons to the striatum can ameliorate neurological deficits exhibited by experimental animals and human graft recipients with parkinson's disease. |
1996-03-01 |
2023-08-12 |
human |
| D W Howells, J Y Wong, A J Churchyard, G A Donna. Leukaemia inhibitory factor prevents injury induced proliferation of striatal dopamine uptake sites. Neuroreport. vol 6. issue 14. 1996-02-22. PMID:8547584. |
the injury associated with implantation of an inert gelatin matrix (gel foam) into normal mouse striatum induces a long-lived increase in binding of [3h]mazindol to presynaptic dopamine uptake sites, probably due to proliferation of striatal dopaminergic terminals. |
1996-02-22 |
2023-08-12 |
mouse |
| Q Y Zhou, R D Palmite. Dopamine-deficient mice are severely hypoactive, adipsic, and aphagic. Cell. vol 83. issue 7. 1996-02-21. PMID:8548806. |
midbrain dopaminergic neurons, their projections, and most characteristics of their target neurons in the striatum appeared normal. |
1996-02-21 |
2023-08-12 |
mouse |
| S K Yeghiayan, A E Kelle. Serotonergic stimulation of the ventrolateral striatum induces orofacial stereotypy. Pharmacology, biochemistry, and behavior. vol 52. issue 3. 1996-02-14. PMID:8545465. |
dopaminergic (da) stimulation of the ventrolateral striatum produces a syndrome of intense orofacial stereotypies. |
1996-02-14 |
2023-08-12 |
Not clear |
| S K Yeghiayan, A E Kelle. Serotonergic stimulation of the ventrolateral striatum induces orofacial stereotypy. Pharmacology, biochemistry, and behavior. vol 52. issue 3. 1996-02-14. PMID:8545465. |
in addition to dopaminergic projections from the substantia nigra, the striatum receives serotonergic (5-ht) inputs arising from the raphe nuclei. |
1996-02-14 |
2023-08-12 |
Not clear |
| M Asanuma, N Ogawa, S Nishibayashi, M Kawai, Y Kondo, E Iwat. Protective effects of pergolide on dopamine levels in the 6-hydroxydopamine-lesioned mouse brain. Archives internationales de pharmacodynamie et de therapie. vol 329. issue 2. 1996-02-06. PMID:8540762. |
to determine whether or not pergolide protects against dopaminergic neuronal damage, via its activity on monoamine metabolism, we studied the effects of pergolide pretreatment on changes in monomaines and their metabolites in the mouse striatum after intracerebroventricular injection of 6-hydroxydopamine with pretreatment of desipramine. |
1996-02-06 |
2023-08-12 |
mouse |
| N Hayase, K Tomiyoshi, K Watanabe, S Horikoshi, T Shibasaki, C Ohy. Positron emission tomography with 4-[18F]fluoro-L-m-tyrosine in MPTP-induced hemiparkinsonian monkeys. Annals of nuclear medicine. vol 9. issue 3. 1996-02-05. PMID:8534583. |
fmt-pet studies were able to detect dopaminergic terminals in both normal and hemiparkinsonian monkeys, and clearly showed a reduction in aromatic l-amino acid decarboxylase (aaad) activities in the mptp-lesioned striatum. |
1996-02-05 |
2023-08-12 |
monkey |
| M O Kreb. [Glutamatergic hypothesis of schizophrenia: psychoses induced by phencyclidine and cortical-subcortical imbalance]. L'Encephale. vol 21. issue 5. 1996-01-29. PMID:8529568. |
positive symptoms could also be due to a secondary hyperdopaminergia, since a part of striatum, the striosomes, connected with limbic system, control the activity of dopaminergic neurons. |
1996-01-29 |
2023-08-12 |
human |
| P Remy, H Hosseini, J D Degos, Y Samson, Y Agid, J M Warter, M Vidailhe. Striatal dopaminergic denervation in pallidopyramidal disease demonstrated by positron emission tomography. Annals of neurology. vol 38. issue 6. 1996-01-23. PMID:8526470. |
in the present report, [18f]fluorodopa positron emission tomography in 2 patients showed a marked dopaminergic denervation of the striatum, suggesting that this disease corresponds more to a nigropallidopyramidal dysfunction. |
1996-01-23 |
2023-08-12 |
Not clear |
| P T Wong, H Feng, W L Te. Interaction of the dopaminergic and serotonergic systems in the rat striatum: effects of selective antagonists and uptake inhibitors. Neuroscience research. vol 23. issue 1. 1996-01-18. PMID:7501296. |
interaction of the dopaminergic and serotonergic systems in the rat striatum: effects of selective antagonists and uptake inhibitors. |
1996-01-18 |
2023-08-12 |
rat |
| P T Wong, H Feng, W L Te. Interaction of the dopaminergic and serotonergic systems in the rat striatum: effects of selective antagonists and uptake inhibitors. Neuroscience research. vol 23. issue 1. 1996-01-18. PMID:7501296. |
they also support the idea that, in the rat striatum, presynaptic inhibitory heteroreceptors are a major mechanism that contributes to a reciprocal interaction of the dopaminergic and serotonergic systems. |
1996-01-18 |
2023-08-12 |
rat |
| Y Akaneya, M Takahashi, H Hatanak. Survival of and 1-methyl-4-phenylpyridinium (MPP+) neurotoxicity against dopaminergic neurons in coculture of rat mesencephalon with their target on non-target regions. Neuroscience research. vol 23. issue 1. 1996-01-18. PMID:7501301. |
it is known that dopaminergic neurons in the substantia nigra of the mesencephalon mainly project to the corpus striatum and neocortex, while the hippocampus receives major cholinergic projection from the septum. |
1996-01-18 |
2023-08-12 |
rat |
| Y Akaneya, M Takahashi, H Hatanak. Survival of and 1-methyl-4-phenylpyridinium (MPP+) neurotoxicity against dopaminergic neurons in coculture of rat mesencephalon with their target on non-target regions. Neuroscience research. vol 23. issue 1. 1996-01-18. PMID:7501301. |
in the present study, the ventral mesencephalon was cocultured with target regions of its dopaminergic neurons, the striatum and neocortex, and with non-target regions, the hippocampus, thalamus, colliculus and cerebellum, using embryonic day-17 (e17) rats. |
1996-01-18 |
2023-08-12 |
rat |
| Y Akaneya, M Takahashi, H Hatanak. Survival of and 1-methyl-4-phenylpyridinium (MPP+) neurotoxicity against dopaminergic neurons in coculture of rat mesencephalon with their target on non-target regions. Neuroscience research. vol 23. issue 1. 1996-01-18. PMID:7501301. |
the numbers of viable dopaminergic neurons were enhanced in coculture not only with corpus striatum or neocortex, but also with hippocampus or cerebellum. |
1996-01-18 |
2023-08-12 |
rat |
| R L Dorri. Evidence for an action of araboascorbic acid on dopaminergic pathways of the corpus striatum. Pharmacology, biochemistry, and behavior. vol 52. issue 1. 1996-01-16. PMID:7501673. |
evidence for an action of araboascorbic acid on dopaminergic pathways of the corpus striatum. |
1996-01-16 |
2023-08-12 |
mouse |
| R L Dorri. Evidence for an action of araboascorbic acid on dopaminergic pathways of the corpus striatum. Pharmacology, biochemistry, and behavior. vol 52. issue 1. 1996-01-16. PMID:7501673. |
the relative decrease in the 2 h accumulation of administered [3h]-spiperone (spi)-2 muci/kg, 0.0004 mg/kg, s.c. - in mouse corpus striatum, a brain area with a high dopaminergic input (specific plus nonspecific dopamine receptor ligand binding) and the cerebellum, a brain area with little, if any, dopaminergic input (an index of nonspecific dopamine receptor ligand binding) were used to compare the influence of araboascorbic acid (araa) with ascorbic acid (asa) on the dopamine receptor. |
1996-01-16 |
2023-08-12 |
mouse |
| P M Laduro. Functional consequences of retrograde axonal transport of receptor-bound neurotensin. Trends in pharmacological sciences. vol 16. issue 10. 1996-01-04. PMID:7491711. |
the most compelling evidence that retrograde axonal transport of receptor-bound neuropeptide represents a long-distance signalling system in gene expression, discussed here by pierre laduron, comes from the recent observation that neurotensin injected in the striatum undergoes retrograde transport in dopaminergic neurones to the substantia nigra, where it induces in the cell bodies an increase of tyrosine hydroxylase mrna. |
1996-01-04 |
2023-08-12 |
Not clear |
| J Tiihonen, J Kuikka, K Bergström, P Hakola, J Karhu, O P Ryynänen, J Föh. Altered striatal dopamine re-uptake site densities in habitually violent and non-violent alcoholics. Nature medicine. vol 1. issue 7. 1995-12-28. PMID:7585146. |
animal studies suggest that development of substance dependence is associated with dopaminergic activity in striatum and the limbic system. |
1995-12-28 |
2023-08-12 |
human |
| Z Elazar, N Peleg, M Paz, G Rin. The striatal dopaminergic catalepsy mechanism is not necessary for the expression of pontine catalepsy produced by carbachol injections into the pontine reticular formation. Naunyn-Schmiedeberg's archives of pharmacology. vol 352. issue 2. 1995-12-27. PMID:7477442. |
in the experiments described in the present article we studied the possibility that the pontine carbachol catalepsy is generated via the intermediary of the dopaminergic cataleptogenic mechanism in the striatum. |
1995-12-27 |
2023-08-12 |
rat |
| A Gobert, J M Rivet, V Audinot, L Cistarelli, M Spedding, J Vian, J L Peglion, M J Milla. Functional correlates of dopamine D3 receptor activation in the rat in vivo and their modulation by the selective antagonist, (+)-S 14297: II. Both D2 and "silent" D3 autoreceptors control synthesis and release in mesolimbic, mesocortical and nigrostriatal pathways. The Journal of pharmacology and experimental therapeutics. vol 275. issue 2. 1995-12-19. PMID:7473181. |
in conclusion, these data suggest that d3 (auto)receptors control synthesis and release of da in dopaminergic pathways innervating the limbic system, cortex and striatum. |
1995-12-19 |
2023-08-12 |
rat |