All Relations between dopaminergic and striatum

Publication Sentence Publish Date Extraction Date Species
N Kayadjanian, R P Heavens, M J Besson, D J Sirinathsinghj. Striatal NMDAR2B mRNA expression after bilateral cortical and unilateral nigral deafferentation. Neuroreport. vol 7. issue 3. 1996-11-05. PMID:8733728. we have studies by in situ hybridization histochemistry the regulation of nmdar2b mrna expression by dopaminergic (da) and cortical glutamatergic afferent fibres in the rat striatum. 1996-11-05 2023-08-12 rat
K Kashihara, T Ishihara, K Akiyama, S Kuroda, T Morimasa, T Shomor. Levodopa induces AP-1 and CREB DNA-binding activities in the rat striatum. Psychiatry and clinical neurosciences. vol 49. issue 5-6. 1996-10-29. PMID:8726116. present results suggest that levodopa but not bromocriptine induces these transcription-regulating proteins in the striatum with normal dopaminergic functioning. 1996-10-29 2023-08-12 rat
N K Popova, E M Nikulina, N N Voĭtenko, D F Avgustinovich, O V Trapezo. [Effect of a single-locus mutation of the silver-blue color allele in mink on the dopaminergic system of the brain]. Genetika. vol 32. issue 3. 1996-10-24. PMID:8723640. a pp mutation was found to affect the dopamine turnover in corpus striatum, which is one of the major dopaminergic brain structures. 1996-10-24 2023-08-12 Not clear
F Fujiyama, S Masuk. Association of dopaminergic terminals and neurons releasing nitric oxide in the rat striatum: an electron microscopic study using NADPH-diaphorase histochemistry and tyrosine hydroxylase immunohistochemistry. Brain research bulletin. vol 40. issue 2. 1996-10-23. PMID:8724430. association of dopaminergic terminals and neurons releasing nitric oxide in the rat striatum: an electron microscopic study using nadph-diaphorase histochemistry and tyrosine hydroxylase immunohistochemistry. 1996-10-23 2023-08-12 rat
V Silani, S Bernasconi, A Pizzuti, A Sampietro, A Brioschi, M Buscaglia, G Scarlat. Molecules with neurotrophic effects on the human developing mesencephalic dopaminergic neurons. Journal of neural transmission. Supplementum. vol 45. 1996-10-23. PMID:8748637. parkinson's disease (pd) is characterized by the degeneration of the mesencephalic dopaminergic (mesda) neurons innervating the striatum. 1996-10-23 2023-08-12 human
M Benoit-Guyod, E Nicolle, A Namil, C Coulombeau, G Lecler. Syntheses and molecular structures of 3-N, N-di-n-propylamino-2-chromanones as new analogues of dopamine. Bioorganic & medicinal chemistry. vol 3. issue 12. 1996-10-23. PMID:8770390. pharmacological activities were determined in vitro on rat striatum, by examining their capacity to displace the specific binding of the labeled dopaminergic ligand 3h sulpiride or 3h spiperone and 3h sch 23390 for d2 and d1 sites, respectively. 1996-10-23 2023-08-12 rat
J P Doucet, Y Nakabeppu, P J Bedard, B T Hope, E J Nestler, B J Jasmin, J S Chen, M J Iadarola, M St-Jean, N Wigle, P Blanchet, R Grondin, G S Robertso. Chronic alterations in dopaminergic neurotransmission produce a persistent elevation of deltaFosB-like protein(s) in both the rodent and primate striatum. The European journal of neuroscience. vol 8. issue 2. 1996-10-22. PMID:8714707. chronic alterations in dopaminergic neurotransmission produce a persistent elevation of deltafosb-like protein(s) in both the rodent and primate striatum. 1996-10-22 2023-08-12 rat
J P Doucet, Y Nakabeppu, P J Bedard, B T Hope, E J Nestler, B J Jasmin, J S Chen, M J Iadarola, M St-Jean, N Wigle, P Blanchet, R Grondin, G S Robertso. Chronic alterations in dopaminergic neurotransmission produce a persistent elevation of deltaFosB-like protein(s) in both the rodent and primate striatum. The European journal of neuroscience. vol 8. issue 2. 1996-10-22. PMID:8714707. taken together, these results demonstrate that chronic alterations in dopaminergic neurotransmission produce a persistent elevation of deltafosb-like protein(s) in both the rodent and primate striatum. 1996-10-22 2023-08-12 rat
S Patel, J Roberts, J Moorman, C Reavil. Localization of serotonin-4 receptors in the striatonigral pathway in rat brain. Neuroscience. vol 69. issue 4. 1996-10-22. PMID:8848104. however, serotonin-4 receptors are not located on dopaminergic neurons, either on their cell bodies in the substantia nigra or terminals in the striatum. 1996-10-22 2023-08-12 rat
C Gilber. Optimal physical performance in athletes: key roles of dopamine in a specific neurotransmitter/hormonal mechanism. Mechanisms of ageing and development. vol 84. issue 2. 1996-10-18. PMID:8788237. the present analysis, based on studies of sedentary and exercise trained humans, proposes the following: that (1) the ans profile in exercise consists in enhanced dopaminergic (da) relative to noradrenergic (na) activity and increased vagal tone; (2) da is the principal catecholamine neuromodulator/neurotransmitter of the brain, directly involved in motor control in the striatum and is key to the mechanism underlying increased and maintained efficiency of exercise trained humans; (3) da is a major participant in many aspects of motor function which include the regulation of cardiovascular and renal function (heart rate, blood pressure, and other), muscle tone, visual processing, calcium homeostasis, protein synthesis and conceivably the optimal utilization of food intake; (4) the peripheral actions of da reflect and are functionally interrelated to the observed global activation of brain da systems in exercising animals, and probably man; (5) that a different enzyme profile evolves in exercise training which may potentiate da synthesis and preserve the structural and functional integrity of central da neurons; (6) that a shift to enhanced da vs. na activity occurs in exercise trained whites which resembles the norm for sedentary africans and confers distinct physiological advantages; (7) there is unequivocal evidence for the physiological efficiency of a da dominated ans profile which can be correlated to the low incidence of da related diseases in aging africans; (8) data suggests that the superiority of top-class african athletes in distance running and their endurance capacity are related to an inherent neurophysiological advantage, to efficient da and protein synthesis, a decreased rate of da decline during aging and to improved calcium homeostasis, inter alia. 1996-10-18 2023-08-12 human
M Menegoz, L F Lau, D Hervé, R L Huganir, J A Giraul. Tyrosine phosphorylation of NMDA receptor in rat striatum: effects of 6-OH-dopamine lesions. Neuroreport. vol 7. issue 1. 1996-10-17. PMID:8742433. two weeks after unilateral lesioning of nigrostriatal dopaminergic neurones with 6-hydroxydopamine, tyrosine phosphorylation of nr2b was increased by approximately 20% in the ipsilateral striatum. 1996-10-17 2023-08-12 human
A Mura, D Jackson, M S Manley, S J Young, P M Grove. Aromatic L-amino acid decarboxylase immunoreactive cells in the rat striatum: a possible site for the conversion of exogenous L-DOPA to dopamine. Brain research. vol 704. issue 1. 1996-10-17. PMID:8750961. in the present study, we used immunocytochemical techniques to examine the localization of aadc and da in the striatum of rats with a unilateral 6-hydroxydopamine (6-ohda) lesion of the nigrostriatal dopaminergic projection. 1996-10-17 2023-08-12 rat
M P Viveros, C de Cabo, M I Colado, M I Martí. Naltrexone administration during the preweanling period affects striatal and hypothalamic serotonergic systems, but not midbrain serotonergic or striatal dopaminergic systems in the adult rat. Neuroscience letters. vol 201. issue 3. 1996-09-26. PMID:8786838. we have recently reported that daily administration from birth of the opioid antagonist naltrexone (1 mg/kg, s.c.) affected dopaminergic and serotonergic systems in the striatum, hypothalamus and midbrain in rats of 7, 14 and 22 days of age. 1996-09-26 2023-08-12 rat
S C Cheetham, C J Kettle, K F Martin, D J Hea. D1 receptor binding in rat striatum: modification by various D1 and D2 antagonists, but not by sibutramine hydrochloride, antidepressants or treatments which enhance central dopaminergic function. Journal of neural transmission. General section. vol 102. issue 1. 1996-09-20. PMID:8785022. d1 receptor binding in rat striatum: modification by various d1 and d2 antagonists, but not by sibutramine hydrochloride, antidepressants or treatments which enhance central dopaminergic function. 1996-09-20 2023-08-12 rat
K D Beck, I Irwin, J Valverde, T J Brennan, J W Langston, F Heft. GDNF induces a dystonia-like state in neonatal rats and stimulates dopamine and serotonin synthesis. Neuron. vol 16. issue 3. 1996-09-20. PMID:8785063. to test whether glial cell line-derived neurotrophic factor (gdnf) regulates the development of nigral dopaminergic neurons in vivo, neonatal rats received bilateral injections of gdnf into the striatum. 1996-09-20 2023-08-12 rat
M P Gygi, J W Gibb, G R Hanso. Methcathinone: an initial study of its effects on monoaminergic systems. The Journal of pharmacology and experimental therapeutics. vol 276. issue 3. 1996-09-20. PMID:8786536. multiple doses of 30 mg/kg methcathinone caused dramatic decreases in neurochemical parameters associated with both dopaminergic and serotonergic systems in the striatum; corresponding decreases also occurred in serotonergic parameters in hippocampus and frontal cortex. 1996-09-20 2023-08-12 rat
E Gil-Martín, I Colado, A Fernández-López, A Fernández-Briera, P Calv. Effects of an acute dose of ethanol on dopaminergic and serotonergic systems from rat cerebral cortex and striatum. Comparative biochemistry and physiology. Part C, Pharmacology, toxicology & endocrinology. vol 113. issue 3. 1996-09-05. PMID:8697197. effects of an acute dose of ethanol on dopaminergic and serotonergic systems from rat cerebral cortex and striatum. 1996-09-05 2023-08-12 rat
T G Hastings, D A Lewis, M J Zigmon. Role of oxidation in the neurotoxic effects of intrastriatal dopamine injections. Proceedings of the National Academy of Sciences of the United States of America. vol 93. issue 5. 1996-09-05. PMID:8700866. thus, following its oxidation, dopamine in high concentrations binds to protein in the striatum, an event that is correlated with the specific loss of dopaminergic terminals. 1996-09-05 2023-08-12 rat
J D Elsworth, M S Brittan, J R Taylor, J R Sladek, M S al-Tikriti, Y Zea-Ponce, R B Innis, D E Redmond, R H Rot. Restoration of dopamine transporter density in the striatum of fetal ventral mesencephalon-grafted, but not sham-grafted, MPTP-treated parkinsonian monkeys. Cell transplantation. vol 5. issue 2. 1996-08-27. PMID:8689042. it is possible that the number of fetal dopamine neurons that survive transplantation is insufficient to restore dopaminergic innervation of the large human striatum to a level where striking recovery is obtained. 1996-08-27 2023-08-12 human
W R Galpern, L H Burns, T W Deacon, J Dinsmore, O Isacso. Xenotransplantation of porcine fetal ventral mesencephalon in a rat model of Parkinson's disease: functional recovery and graft morphology. Experimental neurology. vol 140. issue 1. 1996-08-19. PMID:8682173. using the 6-hydroxydopamine rat model of pd, we transplanted dopaminergic neurons derived from embryonic day 27 porcine fetuses into the denervated striatum of cyclosporine-a (cya)-treated or non-cya-treated rats. 1996-08-19 2023-08-12 human