| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| T Hasegawa, M Shimohira, M Itoh, M Hayashi, J Kohyama, S Kumada, Y Iwakaw. [Polygraphical sleep study on typical absence: relationship between the effect of sodium valproate and nigrostriatal function]. No to hattatsu = Brain and development. vol 26. issue 3. 1994-06-23. PMID:8185977. |
since tms are thought to be controlled by the nigrostriatal dopaminergic pathway, the different response of basal ganglia to vpa among cases with absence epilepsy would have some relation to the different effectiveness of vpa in controlling seizures. |
1994-06-23 |
2023-08-12 |
Not clear |
| A Priori, A Berardelli, M Inghilleri, N Accornero, M Manfred. Motor cortical inhibition and the dopaminergic system. Pharmacological changes in the silent period after transcranial brain stimulation in normal subjects, patients with Parkinson's disease and drug-induced parkinsonism. Brain : a journal of neurology. vol 117 ( Pt 2). 1994-06-22. PMID:8186958. |
dopaminergic drugs modulate the duration of the cortical silent periods in patients and in normal subjects, through mechanisms acting mainly at basal ganglia and possibly also directly at cortical level. |
1994-06-22 |
2023-08-12 |
human |
| O Suchowersk. Gilles de la Tourette syndrome. The Canadian journal of neurological sciences. Le journal canadien des sciences neurologiques. vol 21. issue 1. 1994-06-15. PMID:8180905. |
neuropathophysiology of tourette syndrome is unknown but investigations have suggested that the cause may be an abnormality either in the dopaminergic or endorphin receptor system within the basal ganglia. |
1994-06-15 |
2023-08-12 |
Not clear |
| Z H Qin, S P Zhang, B Weis. Dopaminergic and glutamatergic blocking drugs differentially regulate glutamic acid decarboxylase mRNA in mouse brain. Brain research. Molecular brain research. vol 21. issue 3-4. 1994-06-01. PMID:8170353. |
dopaminergic and glutamatergic inputs play an important role in regulating the activity of gabaergic neurons in basal ganglia. |
1994-06-01 |
2023-08-12 |
mouse |
| H S Markus, D C Costa, A J Lee. HMPAO SPECT in Parkinson's disease before and after levodopa: correlation with dopaminergic responsiveness. Journal of neurology, neurosurgery, and psychiatry. vol 57. issue 2. 1994-04-14. PMID:8126501. |
basal ganglia perfusion assessed by this technique is unlikely to be of use in the diagnosis of parkinson's disease that is responsive to dopaminergic treatment. |
1994-04-14 |
2023-08-12 |
human |
| A Takahashi, M Murasak. [Drug-induced movement disorders]. Nihon rinsho. Japanese journal of clinical medicine. vol 51. issue 11. 1994-02-04. PMID:7903986. |
most of these drugs interfere with dopaminergic transmission within the basal ganglia. |
1994-02-04 |
2023-08-12 |
Not clear |
| P Jalil, M C Miranda, C Frenke. [Progressive hereditary dystonia with diurnal fluctuations: report of 2 cases]. Revista medica de Chile. vol 121. issue 5. 1994-02-03. PMID:7903816. |
the pathogenesis of this disease could be a functional alteration of basal ganglia in the dopaminergic striatonigral region. |
1994-02-03 |
2023-08-12 |
Not clear |
| A Benazzouz, C Gross, J Féger, T Boraud, B Bioula. Reversal of rigidity and improvement in motor performance by subthalamic high-frequency stimulation in MPTP-treated monkeys. The European journal of neuroscience. vol 5. issue 4. 1994-01-27. PMID:8261116. |
in parkinson's disease the loss of dopaminergic neurons in the substantia nigra is associated with global disorganization of basal ganglia activity and, in particular, with increased activity of the excitatory glutamatergic neurons of the subthalamic nucleus. |
1994-01-27 |
2023-08-12 |
monkey |
| B K Lipska, D R Weinberge. Delayed effects of neonatal hippocampal damage on haloperidol-induced catalepsy and apomorphine-induced stereotypic behaviors in the rat. Brain research. Developmental brain research. vol 75. issue 2. 1994-01-26. PMID:7903225. |
the developmental effects of neonatal excitotoxic ventral hippocampal (vh) damage on behaviors related to dopaminergic (da) transmission in the basal ganglia were investigated in the rat. |
1994-01-26 |
2023-08-12 |
rat |
| R Hitzemann, Y Qian, B Hitzeman. Dopamine and acetylcholine cell density in the neuroleptic responsive (NR) and neuroleptic nonresponsive (NNR) lines of mice. The Journal of pharmacology and experimental therapeutics. vol 266. issue 1. 1993-08-19. PMID:8101223. |
overall, these data illustrate that selective breeding for neuroleptic response has yielded lines of mice with significant differences in both the cholinergic and dopaminergic systems of the basal ganglia. |
1993-08-19 |
2023-08-12 |
mouse |
| J Kornhuber, M Welle. Amantadine and the glutamate hypothesis of schizophrenia. Experiences in the treatment of neuroleptic malignant syndrome. Journal of neural transmission. General section. vol 92. issue 1. 1993-08-17. PMID:8101093. |
our observations support the glutamate hypothesis of schizophrenia which suggests that reduced glutamatergic transmission causes a relative dopaminergic excess in the basal ganglia and the limbic system. |
1993-08-17 |
2023-08-12 |
Not clear |
| W Timmerma. Dopaminergic receptor agents and the basal ganglia: pharmacological properties and interactions with the GABA-ergic system. Pharmacy world & science : PWS. vol 15. issue 2. 1993-06-11. PMID:8098242. |
dopaminergic receptor agents and the basal ganglia: pharmacological properties and interactions with the gaba-ergic system. |
1993-06-11 |
2023-08-12 |
Not clear |
| M S Aldrich, Z Hollingsworth, J B Penne. Autoradiographic studies of post-mortem human narcoleptic brain. Neurophysiologie clinique = Clinical neurophysiology. vol 23. issue 1. 1993-04-08. PMID:8383284. |
we used quantitative autoradiography to assess binding of dopaminergic, noradrenergic, and cholinergic ligands to basal ganglia and amygdala of five narcoleptic and 17 control human brains. |
1993-04-08 |
2023-08-12 |
human |
| S J Patrick, L K Snelling, L R Men. Infantile chorea following abrupt withdrawal of diazepam and pentobarbital therapy. Journal of toxicology. Clinical toxicology. vol 31. issue 1. 1993-03-17. PMID:8433409. |
we speculate that the mechanism by which chorea may occur as a withdrawal phenomenon to benzodiazepine/barbiturate treatment is by a functional overactivity of the dopaminergic system in the basal ganglia. |
1993-03-17 |
2023-08-12 |
Not clear |
| D J Cohe. [Tourette's syndrome: psychopathology of development in a model of neuropsychiatric dysfunction in children]. La Psychiatrie de l'enfant. vol 35. issue 2. 1993-03-08. PMID:1494599. |
no specific neuroanatomical or neurochemical basis has yet been discovered, but the basal ganglia and dopaminergic systems have been implicated. |
1993-03-08 |
2023-08-11 |
Not clear |
| P A Löschmann, M Kunow, H Wachte. Synergism of NBQX with dopamine agonists in the 6-OHDA rat model of Parkinson's disease. Journal of neural transmission. Supplementum. vol 38. 1993-02-26. PMID:1491248. |
advance in understanding of the anatomy, physiology and pharmacology of basal ganglia organisation over the past decade revealed a functional relation between excitatory glutamatergic and the degenerated dopaminergic nigrostriatal transmitter systems which could serve as targets for pharmacological interventions in parkinson's disease. |
1993-02-26 |
2023-08-11 |
rat |
| A Nieoullon, L Kerkerian-Le Gof. Cellular interactions in the striatum involving neuronal systems using "classical" neurotransmitters: possible functional implications. Movement disorders : official journal of the Movement Disorder Society. vol 7. issue 4. 1993-02-18. PMID:1362449. |
consequently, an impairment of the dopaminergic transmission such as that occurring in parkinsonism may lead to an increase in the corticostriatal glutamatergic transmission, which may further contribute towards reinforcing the "imbalance" between subsets of striatal neuronal systems controlling the output of the basal ganglia. |
1993-02-18 |
2023-08-11 |
Not clear |
| M Weller, J Kornhube. [Pathophysiology and therapy of malignant neuroleptic syndrome]. Der Nervenarzt. vol 63. issue 11. 1993-01-22. PMID:1361662. |
an acute dopaminergic transmission block in the basal ganglia and the hypothalamus is thought to be the pathophysiological mechanism of nms. |
1993-01-22 |
2023-08-11 |
Not clear |
| H Wachtel, M Kunow, P A Löschman. NBQX (6-nitro-sulfamoyl-benzo-quinoxaline-dione) and CPP (3-carboxy-piperazin-propyl phosphonic acid) potentiate dopamine agonist induced rotations in substantia nigra lesioned rats. Neuroscience letters. vol 142. issue 2. 1993-01-06. PMID:1280793. |
degeneration of dopaminergic nigrostriatal neurons in primate models of parkinson's disease (pd) leads to an overactivity of excitatory glutamatergic projections from the subthalamic nucleus (stn) to the output nuclei of the basal ganglia resulting in rigidity and akinesia. |
1993-01-06 |
2023-08-11 |
rat |
| C R Dermon, M Tzagournissakis, H E Savak. Bilateral cerebral metabolic effects of pharmacological manipulation of the substantia nigra in the rat: unilateral intranigral application of the putative excitatory neurotransmitter substance P. Neuroscience. vol 50. issue 4. 1992-12-29. PMID:1280349. |
furthermore, it is suggested that the ipsilateral basal ganglia effects induced by intranigral substance p application are mediated via both the compacta dopaminergic nigrostriatal projection and the reticulata gabaergic nigro-thalamocortico-striatal loop, whereas the contralateral basal ganglia and associated thalamocortical effects are due to the activation of the gabaergic reticulata efferents and are mediated via an interthalamic circuitry involving the motor, reticular and intralaminar thalamic nuclei. |
1992-12-29 |
2023-08-11 |
rat |