| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| D L Bliwise, R L Watts, N Watts, D B Rye, D Irbe, M Hughe. Disruptive nocturnal behavior in Parkinson's disease and Alzheimer's disease. Journal of geriatric psychiatry and neurology. vol 8. issue 2. 1995-08-03. PMID:7794473. |
these findings raise the possibility that sundowning in pd patients may be a manifestation of dopaminergic depletion within the basal ganglia or other abnormalities involving the cholinergic, serotoninergic and/or noradrenergic systems in the brainstem. |
1995-08-03 |
2023-08-12 |
Not clear |
| G B Frisoni, G Pizzolato, O Zanetti, A Bianchetti, F Chierichetti, M Trabucch. Corticobasal degeneration: neuropsychological assessment and dopamine D2 receptor SPECT analysis. European neurology. vol 35. issue 1. 1995-06-02. PMID:7737248. |
ibzm spect, which gives information about postsynaptic dopaminergic d2 receptors, showed severe reduction of tracer uptake in the right basal ganglia. |
1995-06-02 |
2023-08-12 |
Not clear |
| G B Frisoni, G Pizzolato, O Zanetti, A Bianchetti, F Chierichetti, M Trabucch. Corticobasal degeneration: neuropsychological assessment and dopamine D2 receptor SPECT analysis. European neurology. vol 35. issue 1. 1995-06-02. PMID:7737248. |
our findings suggest that a postsynaptic lesion in the basal ganglia might account for the lack of response of extrapyramidal motor symptoms to dopaminergic agonists in corticobasal degeneration. |
1995-06-02 |
2023-08-12 |
Not clear |
| S A Factor, G D Podskalny, K D Barro. Persistent neuroleptic-induced rigidity and dystonia in AIDS dementia complex: a clinico-pathological case report. Journal of the neurological sciences. vol 127. issue 1. 1995-04-28. PMID:7699386. |
it has been hypothesized, based on clinical evidence, that hypersensitivity to these drugs in adc is probably related to direct invasion of the basal ganglia by the hiv virus and a secondary alteration in dopaminergic mechanisms. |
1995-04-28 |
2023-08-12 |
Not clear |
| R M Ridle. The psychology of perserverative and stereotyped behaviour. Progress in neurobiology. vol 44. issue 2. 1995-02-17. PMID:7831478. |
in experimental studies in animals, stereotyped behaviour seems to be related mainly to excess dopaminergic activity in the basal ganglia while perserverative behaviour can be produced by lesions of the frontal lobes. |
1995-02-17 |
2023-08-12 |
Not clear |
| C C Chiueh, R M Wu, K P Mohanakumar, L M Sternberger, G Krishna, T Obata, D L Murph. In vivo generation of hydroxyl radicals and MPTP-induced dopaminergic toxicity in the basal ganglia. Annals of the New York Academy of Sciences. vol 738. 1995-02-17. PMID:7832434. |
in vivo generation of hydroxyl radicals and mptp-induced dopaminergic toxicity in the basal ganglia. |
1995-02-17 |
2023-08-12 |
Not clear |
| C C Chiueh, R M Wu, K P Mohanakumar, L M Sternberger, G Krishna, T Obata, D L Murph. In vivo generation of hydroxyl radicals and MPTP-induced dopaminergic toxicity in the basal ganglia. Annals of the New York Academy of Sciences. vol 738. 1995-02-17. PMID:7832434. |
in conclusion, this in vivo free-radical trapping procedure provides evidence to support a current working hypothesis that a site-specific formation of cytotoxic .oh free radicals in the basal ganglia may be one of the neurotoxic mechanisms underlying nigrostriatal degeneration and parkinsonism caused by the dopaminergic neurotoxin mptp. |
1995-02-17 |
2023-08-12 |
Not clear |
| M J Twery, L A Thompson, J R Walter. Intracellularly recorded response of rat striatal neurons in vitro to fenoldopam and SKF 38393 following lesions of midbrain dopamine cells. Synapse (New York, N.Y.). vol 18. issue 1. 1995-02-16. PMID:7825125. |
similar regulation of striatal neurons in vivo may contribute to dopaminergic regulation of basal ganglia output and the ability of dopaminomimetic agents to ameliorate symptoms of dopaminergic deficiency in parkinson's disease. |
1995-02-16 |
2023-08-12 |
rat |
| J C Hedreen, S E Folstei. Early loss of neostriatal striosome neurons in Huntington's disease. Journal of neuropathology and experimental neurology. vol 54. issue 1. 1995-02-08. PMID:7815073. |
analysis of the functional circuitry of the basal ganglia suggests that early degeneration of striosomal neurons may produce hyperactivity of the nigrostriatal dopaminergic pathway, causing chorea and other early clinical manifestations of hd. |
1995-02-08 |
2023-08-12 |
Not clear |
| K W Lange, P Riederer, M B Youdi. Biochemical actions of l-deprenyl (selegiline). Clinical pharmacology and therapeutics. vol 56. issue 6 Pt 2. 1995-01-18. PMID:7995015. |
because parkinson's disease is characterized by a decrease in dopaminergic neurotransmission in the basal ganglia, the selective inhibition of mao-b should lead to diminished metabolism of dopamine in the nigrostriatal system and a significant increase in the concentration of the neurotransmitter. |
1995-01-18 |
2023-08-12 |
human |
| K X Huang, J R Walter. Electrophysiological effects of SKF 38393 in rats with reserpine treatment and 6-hydroxydopamine-induced nigrostriatal lesions reveal two types of plasticity in D1 dopamine receptor modulation of basal ganglia output. The Journal of pharmacology and experimental therapeutics. vol 271. issue 3. 1995-01-17. PMID:7996456. |
to examine the role of d1 dopamine receptors in modulating basal ganglia output and how this role may be altered by changes in dopaminergic transmission, the effects of a d1 dopamine agonist on firing rates of substantia nigra pars reticulata (snpr) neurons were determined in reserpine-treated rats and compared with effects observed in nigrostriatal-lesioned and normal rats. |
1995-01-17 |
2023-08-12 |
rat |
| M Xu, R Moratalla, L H Gold, N Hiroi, G F Koob, A M Graybiel, S Tonegaw. Dopamine D1 receptor mutant mice are deficient in striatal expression of dynorphin and in dopamine-mediated behavioral responses. Cell. vol 79. issue 4. 1994-12-27. PMID:7954836. |
the brain dopaminergic system is a critical modulator of basal ganglia function and plasticity. |
1994-12-27 |
2023-08-12 |
mouse |
| R H Porter, J G Greene, D S Higgins, J T Greenamyr. Polysynaptic regulation of glutamate receptors and mitochondrial enzyme activities in the basal ganglia of rats with unilateral dopamine depletion. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 14. issue 11 Pt 2. 1994-12-02. PMID:7965108. |
after nigrostriatal dopaminergic denervation, the output nuclei of the basal ganglia, the medial globus pallidus and substantia nigra pars reticulata (snr), become overactive, in part, because of increased activity of excitatory afferents from the subthalamic nucleus (stn). |
1994-12-02 |
2023-08-12 |
rat |
| J M Casto, G F Bal. Characterization and localization of D1 dopamine receptors in the sexually dimorphic vocal control nucleus, area X, and the basal ganglia of European starlings. Journal of neurobiology. vol 25. issue 7. 1994-10-20. PMID:8089655. |
this circuit includes a sexually dimorphic nucleus, area x, that is a subregion of the parolfactory lobe of the basal ganglia and is known from work on zebra finches to receive dopaminergic input from the area ventralis of tsai. |
1994-10-20 |
2023-08-12 |
Not clear |
| D G Cole, M Di Figli. Reserpine increases Fos activity in the rat basal ganglia via a quinpirole-sensitive mechanism. Neuroscience. vol 60. issue 1. 1994-09-08. PMID:7914358. |
c-fos and fos protein have been widely used to investigate basal ganglia responses to changes in dopaminergic neurotransmission. |
1994-09-08 |
2023-08-12 |
rat |
| D G Cole, M Di Figli. Reserpine increases Fos activity in the rat basal ganglia via a quinpirole-sensitive mechanism. Neuroscience. vol 60. issue 1. 1994-09-08. PMID:7914358. |
because dopamine is believed to excite striatal neurons via d1 receptors and inhibit them via d2 receptors, we hypothesized that acute dopamine depletion would increase fos activity in basal ganglia circuits normally inhibited by dopaminergic input to d2 receptors. |
1994-09-08 |
2023-08-12 |
rat |
| N Tsuda, K Negoro, M Morimats. [A case of multiple cerebral infarctions associated with hemichorea induced by amantadine hydrochloride]. Rinsho shinkeigaku = Clinical neurology. vol 34. issue 4. 1994-08-09. PMID:8026137. |
these findings suggest hemichorea was related to hyperfunction of dopaminergic neurons in the left basal ganglia. |
1994-08-09 |
2023-08-12 |
Not clear |
| T Di Paol. Modulation of brain dopamine transmission by sex steroids. Reviews in the neurosciences. vol 5. issue 1. 1994-08-04. PMID:8019704. |
biochemical and behavioral studies in animals have also shown the effect of sex steroids on dopaminergic activity in the basal ganglia; however, both activating and inhibiting effects have been reported. |
1994-08-04 |
2023-08-12 |
rat |
| E Bächli, C Alban. [Akinetic crisis in Parkinson disease]. Schweizerische medizinische Wochenschrift. vol 124. issue 23. 1994-08-04. PMID:8023100. |
while nms is related to dopaminergic receptor blockade or dopaminergic depletion, akinetic crisis can occur despite adequate dopaminergic therapy as a symptom of severe basal ganglia dysfunction related to the advanced stages of parkinson's disease. |
1994-08-04 |
2023-08-12 |
Not clear |
| F Rodríguez de Fonseca, J L Martín Calderón, R Mechoulam, M Navarr. Repeated stimulation of D1 dopamine receptors enhances (-)-11-hydroxy-delta 8-tetrahydrocannabinol-dimethyl-heptyl-induced catalepsy in male rats. Neuroreport. vol 5. issue 7. 1994-07-29. PMID:7912554. |
dopaminergic and cannabinoid receptors are localized in the outflow nuclei of the basal ganglia. |
1994-07-29 |
2023-08-12 |
rat |