| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| K Choong, R She. Prenatal ethanol exposure alters the postnatal development of the spontaneous electrical activity of dopamine neurons in the ventral tegmental area. Neuroscience. vol 126. issue 4. 2004-10-08. PMID:15207341. |
prenatal ethanol exposure causes a persistent reduction in the spontaneous electrical activity of dopamine (da) neurons in the ventral tegmental area (vta) in adult animals. |
2004-10-08 |
2023-08-12 |
Not clear |
| A E Sahr, R J Thielen, L Lumeng, T-K Li, W J McBrid. Long-lasting alterations of the mesolimbic dopamine system after periadolescent ethanol drinking by alcohol-preferring rats. Alcoholism, clinical and experimental research. vol 28. issue 5. 2004-09-09. PMID:15166644. |
this study tested the hypothesis that ethanol consumption by alcohol-preferring (p) rats during the periadolescent period causes persistent alterations in the mesolimbic dopamine (da) system. |
2004-09-09 |
2023-08-12 |
rat |
| L M Oswald, G S Wan. Opioids and alcoholism. Physiology & behavior. vol 81. issue 2. 2004-09-07. PMID:15159175. |
findings from preclinical research indicate that ethanol consumption and ethanol-induced dopamine (da) release are both reduced by opioid antagonists. |
2004-09-07 |
2023-08-12 |
human |
| Qing-Shan Ya. Involvement of non-exocytotic mechanisms in ethanol-induced in vivo dopamine release: comparisons with cocaine. European journal of pharmacology. vol 477. issue 1. 2004-08-24. PMID:14512096. |
effects of ethanol on dopamine release in the nucleus accumbens were compared with those by cocaine, a drug that increases synaptic dopamine by a mechanism, which depends on neuronal activity and involves an exocytotic process. |
2004-08-24 |
2023-08-12 |
rat |
| Qing-Shan Ya. Involvement of non-exocytotic mechanisms in ethanol-induced in vivo dopamine release: comparisons with cocaine. European journal of pharmacology. vol 477. issue 1. 2004-08-24. PMID:14512096. |
administration of ethanol (80 mm) or cocaine (10 microm) via a dialysis probe increased extracellular dopamine concentrations in the nucleus accumbens. |
2004-08-24 |
2023-08-12 |
rat |
| Qing-Shan Ya. Involvement of non-exocytotic mechanisms in ethanol-induced in vivo dopamine release: comparisons with cocaine. European journal of pharmacology. vol 477. issue 1. 2004-08-24. PMID:14512096. |
pretreatments with tetrodotoxin (2 microm) or ca2+ withdrawal did not block the ability of ethanol to increase nucleus accumbens dopamine. |
2004-08-24 |
2023-08-12 |
rat |
| Qing-Shan Ya. Involvement of non-exocytotic mechanisms in ethanol-induced in vivo dopamine release: comparisons with cocaine. European journal of pharmacology. vol 477. issue 1. 2004-08-24. PMID:14512096. |
the blockade of dopamine autoreceptors by local infusion of sulpiride did not significantly alter the effect of ethanol on nucleus accumbens dopamine either. |
2004-08-24 |
2023-08-12 |
rat |
| Qing-Shan Ya. Involvement of non-exocytotic mechanisms in ethanol-induced in vivo dopamine release: comparisons with cocaine. European journal of pharmacology. vol 477. issue 1. 2004-08-24. PMID:14512096. |
as opposed to ethanol, however, cocaine-induced increases in nucleus accumbens dopamine were sensitive to tetrodotoxin or ca2+ omission. |
2004-08-24 |
2023-08-12 |
rat |
| Qing-Shan Ya. Involvement of non-exocytotic mechanisms in ethanol-induced in vivo dopamine release: comparisons with cocaine. European journal of pharmacology. vol 477. issue 1. 2004-08-24. PMID:14512096. |
these differences in responses to tetrodotoxin, ca2+ withdrawal and inhibition of dopamine autoreceptors suggest that a non-exocytotic mechanism may be involved in dopamine release in the nucleus accumbens evoked by focally applied ethanol. |
2004-08-24 |
2023-08-12 |
rat |
| Lawrence D Middaugh, Karen K Szumlinski, Yancy Van Patten, Angela-Leigh Bandy Marlowe, Peter W Kaliva. Chronic ethanol consumption by C57BL/6 mice promotes tolerance to its interoceptive cues and increases extracellular dopamine, an effect blocked by naltrexone. Alcoholism, clinical and experimental research. vol 27. issue 12. 2004-08-13. PMID:14691376. |
chronic ethanol consumption by c57bl/6 mice promotes tolerance to its interoceptive cues and increases extracellular dopamine, an effect blocked by naltrexone. |
2004-08-13 |
2023-08-12 |
mouse |
| Lawrence D Middaugh, Karen K Szumlinski, Yancy Van Patten, Angela-Leigh Bandy Marlowe, Peter W Kaliva. Chronic ethanol consumption by C57BL/6 mice promotes tolerance to its interoceptive cues and increases extracellular dopamine, an effect blocked by naltrexone. Alcoholism, clinical and experimental research. vol 27. issue 12. 2004-08-13. PMID:14691376. |
in this study, we determined the amounts of ethanol voluntarily consumed by b6 mice and associated blood ethanol levels (bel), the effects of this consumption on extracellular dopamine (da) and how this was altered by naltrexone, as well as on its interoceptive discriminative cues. |
2004-08-13 |
2023-08-12 |
mouse |
| Maria Castorina, Rossana Marino, Michele Grassi, Filippo Drag. Very low doses of ethanol induce behavioral changes involving dopamine D2 receptors. European journal of pharmacology. vol 477. issue 2. 2004-08-11. PMID:14519416. |
very low doses of ethanol induce behavioral changes involving dopamine d2 receptors. |
2004-08-11 |
2023-08-12 |
rat |
| Maria Castorina, Rossana Marino, Michele Grassi, Filippo Drag. Very low doses of ethanol induce behavioral changes involving dopamine D2 receptors. European journal of pharmacology. vol 477. issue 2. 2004-08-11. PMID:14519416. |
the pretreatment with the dopamine d2 receptor agonist, (+/-)-2-(n-phenethyl-n-propylamino)-5-hydroxytetralin (ppht, 0.1 mg/kg), enhanced the effects of ethanol very low doses in delaying the initiation and reducing the duration of narcosis induced by an acute i.p. |
2004-08-11 |
2023-08-12 |
rat |
| Maria Castorina, Rossana Marino, Michele Grassi, Filippo Drag. Very low doses of ethanol induce behavioral changes involving dopamine D2 receptors. European journal of pharmacology. vol 477. issue 2. 2004-08-11. PMID:14519416. |
thus, repeated administration of ethanol very low doses may have affected the sensitivity of presynaptic dopamine d2 receptors. |
2004-08-11 |
2023-08-12 |
rat |
| Maria Castorina, Rossana Marino, Michele Grassi, Filippo Drag. Very low doses of ethanol induce behavioral changes involving dopamine D2 receptors. European journal of pharmacology. vol 477. issue 2. 2004-08-11. PMID:14519416. |
the influence on dopamine release through an action on presynaptic receptors may be involved in these effects of small doses of ethanol. |
2004-08-11 |
2023-08-12 |
rat |
| Susan E Senogle. D2s dopamine receptor mediates phospholipase D and antiproliferation. Molecular and cellular endocrinology. vol 209. issue 1-2. 2004-07-30. PMID:14604817. |
addition of ethanol, at 0.5% reduced the ability of dopamine agonists to inhibit growth in d2s-hek 293 cells, suggesting that pld is involved in the antiproliferative effects of d2s signaling. |
2004-07-30 |
2023-08-12 |
Not clear |
| Herman H Samson, Ann M Chappel. Effects of raclopride in the core of the nucleus accumbens on ethanol seeking and consumption: the use of extinction trials to measure seeking. Alcoholism, clinical and experimental research. vol 28. issue 4. 2004-07-23. PMID:15100604. |
a previous study using a sipper procedure of ethanol self-administration found that blockade of the d2 dopamine (da) receptors in the nucleus accumbens resulted in a reduction in ethanol-seeking behavior with only slight effects on ethanol drinking. |
2004-07-23 |
2023-08-12 |
Not clear |
| Douglas P Dohrman And, Cindy K Reite. Chronic ethanol reduces nicotine-induced dopamine release in PC12 cells. Alcoholism, clinical and experimental research. vol 27. issue 11. 2004-06-29. PMID:14634503. |
chronic ethanol reduces nicotine-induced dopamine release in pc12 cells. |
2004-06-29 |
2023-08-12 |
Not clear |
| Manoranjan S D'Souza, Aiko Ikegami, Christopher M Olsen, Christine L Duvauchell. Chronic D1 agonist and ethanol coadministration facilitate ethanol-mediated behaviors. Pharmacology, biochemistry, and behavior. vol 76. issue 2. 2004-06-18. PMID:14592686. |
separate lines of evidence suggest that neuroadaptations associated with ethanol (etoh) reinforcement can be initiated by chronic etoh preexposure and a signaling pathway activated by dopamine (da) d1 receptor stimulation. |
2004-06-18 |
2023-08-12 |
Not clear |
| William M Doyon, Jennifer L York, Laurea M Diaz, Herman H Samson, Cristine L Czachowski, Rueben A Gonzale. Dopamine activity in the nucleus accumbens during consummatory phases of oral ethanol self-administration. Alcoholism, clinical and experimental research. vol 27. issue 10. 2004-06-15. PMID:14574227. |
dopamine activity in the nucleus accumbens during consummatory phases of oral ethanol self-administration. |
2004-06-15 |
2023-08-12 |
Not clear |