All Relations between mog and oligodendrocyte

Publication Sentence Publish Date Extraction Date Species
Vicky W W Tsai, Mohammad G Mohammad, Ornella Tolhurst, Samuel N Breit, Paul E Sawchenko, David A Brow. CCAAT/enhancer binding protein-δ expression by dendritic cells regulates CNS autoimmune inflammatory disease. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 31. issue 48. 2012-02-09. PMID:22131422. mice deficient in c/ebpδ expression exhibited less severe clinical disease than wild-type littermates in response to induction of experimental autoimmune encephalomyelitis (eae) by vaccination with a myelin oligodendrocyte glycoprotein (mog) fragment. 2012-02-09 2023-08-12 mouse
Xiaoying Zhuang, Xiaoyu Xiang, William Grizzle, Dongmei Sun, Shuangqin Zhang, Robert C Axtell, Songwen Ju, Jiangyao Mu, Lifeng Zhang, Lawrence Steinman, Donald Miller, Huang-Ge Zhan. Treatment of brain inflammatory diseases by delivering exosome encapsulated anti-inflammatory drugs from the nasal region to the brain. Molecular therapy : the journal of the American Society of Gene Therapy. vol 19. issue 10. 2012-02-03. PMID:21915101. the results generated from three inflammation-mediated disease models, i.e., a lipopolysaccharide (lps)-induced brain inflammation model, experimental autoimmune encephalitis and a gl26 brain tumor model, showed that mice treated intranasally with exo-cur or exo-jsi124 are protected from lps-induced brain inflammation, the progression of myelin oligodendrocyte glycoprotein (mog) peptide induced experimental autoimmune encephalomyelitis (eae), and had significantly delayed brain tumor growth in the gl26 tumor model. 2012-02-03 2023-08-12 mouse
Patrice H Lalive, Nicolas Molnarfi, Mahdia Benkhoucha, Martin S Weber, Marie-Laure Santiago-Rabe. Antibody response in MOG(35-55) induced EAE. Journal of neuroimmunology. vol 240-241. 2012-01-30. PMID:21993076. we show that mice immunized with encephalitogenic myelin oligodendrocyte glycoprotein (mog(35-55)) peptide develop significant serum levels of anti-mog antibodies in parallel with disease progression. 2012-01-30 2023-08-12 mouse
Kerstin Berer, Marsilius Mues, Michail Koutrolos, Zakeya Al Rasbi, Marina Boziki, Caroline Johner, Hartmut Wekerle, Gurumoorthy Krishnamoorth. Commensal microbiota and myelin autoantigen cooperate to trigger autoimmune demyelination. Nature. vol 479. issue 7374. 2012-01-26. PMID:22031325. we show further that recruitment and activation of autoantibody-producing b cells from the endogenous immune repertoire depends on availability of the target autoantigen, myelin oligodendrocyte glycoprotein (mog), and commensal microbiota. 2012-01-26 2023-08-12 mouse
Walter Maetzler, Daniela Berg, Matthis Synofzik, Kathrin Brockmann, Jana Godau, Arthur Melms, Thomas Gasser, Stephanie Hörnig, Markus Langkam. Autoantibodies against amyloid and glial-derived antigens are increased in serum and cerebrospinal fluid of Lewy body-associated dementias. Journal of Alzheimer's disease : JAD. vol 26. issue 1. 2012-01-10. PMID:21593566. autoantibody levels against α-synuclein, amyloid-β₄₂ (aβ₄₂), myelin oligodendrocyte glycoprotein (mog), myelin basic protein (mbp), and s100b were determined. 2012-01-10 2023-08-12 human
Ani Grigorian, Lindsey Araujo, Nandita N Naidu, Dylan J Place, Biswa Choudhury, Michael Demetrio. N-acetylglucosamine inhibits T-helper 1 (Th1)/T-helper 17 (Th17) cell responses and treats experimental autoimmune encephalomyelitis. The Journal of biological chemistry. vol 286. issue 46. 2012-01-05. PMID:21965673. here, we report that oral glcnac inhibits t-helper 1 (th1) and t-helper 17 (th17) responses and attenuates the clinical severity of myelin oligodendrocyte glycoprotein (mog)-induced eae when administered after disease onset. 2012-01-05 2023-08-12 mouse
Hyun Hor, Luca Bartesaghi, Zoltán Kutalik, José L Vicário, Clara de Andrés, Corinne Pfister, Gert J Lammers, Nicolas Guex, Roman Chrast, Mehdi Tafti, Rosa Peraita-Adrado. A missense mutation in myelin oligodendrocyte glycoprotein as a cause of familial narcolepsy with cataplexy. American journal of human genetics. vol 89. issue 3. 2011-11-22. PMID:21907016. transient expression of mutant myelin oligodendrocyte glycoprotein (mog) in mouse oligodendrocytes showed abnormal subcellular localization, suggesting an altered function of the mutant mog. 2011-11-22 2023-08-12 mouse
Haolong Cong, Yue Jiang, Po Tie. Identification of the myelin oligodendrocyte glycoprotein as a cellular receptor for rubella virus. Journal of virology. vol 85. issue 21. 2011-11-21. PMID:21880773. in this study, we found that the myelin oligodendrocyte glycoprotein (mog) specifically bound to the e1 envelope glycoprotein of rv, and an antibody against mog could block rv infection. 2011-11-21 2023-08-12 Not clear
Rohit D Divekar, Cara L Haymaker, Jason A Cascio, Betul F Guloglu, Jason S Ellis, Danielle M Tartar, Christine M Hoeman, Craig L Franklin, Bernd H Zinselmeyer, Jennifer N Lynch, Mark J Miller, Habib Zaghouan. T cell dynamics during induction of tolerance and suppression of experimental allergic encephalomyelitis. Journal of immunology (Baltimore, Md. : 1950). vol 187. issue 8. 2011-11-15. PMID:21911603. fcγr-deficient (fcγr(-/-)) mice were unable to resist or alleviate experimental allergic encephalomyelitis when treated with ig-myelin oligodendrocyte glycoprotein (mog) tolerogen, an ig carrying the mog35-55 peptide. 2011-11-15 2023-08-12 mouse
Xiang T Chen, Siow T Chan, Hamid Hosseini, Daniel Layton, Richard Boyd, Frank Alderuccio, Ban-Hock Toh, James Cha. Transplantation of retrovirally transduced bone marrow prevents autoimmune disease in aged mice by peripheral tolerance mechanisms. Autoimmunity. vol 44. issue 5. 2011-10-28. PMID:21244332. transplantation of bone marrow (bm) engineered to express self-antigen has been shown to protect 100% of young mice from myelin oligodendrocyte glycoprotein (mog)-induced experimental autoimmune encephalomyelitis (eae), with thymic clonal deletion as a tolerance mechanism. 2011-10-28 2023-08-12 mouse
A K Pröbstel, K Dornmair, R Bittner, P Sperl, D Jenne, S Magalhaes, A Villalobos, C Breithaupt, R Weissert, U Jacob, M Krumbholz, T Kuempfel, A Blaschek, W Stark, J Gärtner, D Pohl, K Rostasy, F Weber, I Forne, M Khademi, T Olsson, F Brilot, E Tantsis, R C Dale, H Wekerle, R Hohlfeld, B Banwell, A Bar-Or, E Meinl, T Derfus. Antibodies to MOG are transient in childhood acute disseminated encephalomyelitis. Neurology. vol 77. issue 6. 2011-09-30. PMID:21795651. to study the longitudinal dynamics of anti-myelin oligodendrocyte glycoprotein (mog) autoantibodies in childhood demyelinating diseases. 2011-09-30 2023-08-12 Not clear
Khadir Raddassi, Sally C Kent, Junbao Yang, Kasia Bourcier, Elizabeth M Bradshaw, Vicki Seyfert-Margolis, Gerald T Nepom, William W Kwok, David A Hafle. Increased frequencies of myelin oligodendrocyte glycoprotein/MHC class II-binding CD4 cells in patients with multiple sclerosis. Journal of immunology (Baltimore, Md. : 1950). vol 187. issue 2. 2011-09-23. PMID:21653833. in this study, we investigated whether myelin oligodendrocyte glycoprotein (mog)-specific t cells could be detected and their frequency was measured using drb1*0401/mog(97-109(107e-s)) tetramers in ms subjects and healthy controls expressing hla class ii drb1*0401. 2011-09-23 2023-08-12 human
Socorro Miranda-Hernandez, Nicole Gerlach, Julie M Fletcher, Erik Biros, Matthias Mack, Heinrich Körner, Alan G Baxte. Role for MyD88, TLR2 and TLR9 but not TLR1, TLR4 or TLR6 in experimental autoimmune encephalomyelitis. Journal of immunology (Baltimore, Md. : 1950). vol 187. issue 2. 2011-09-23. PMID:21685327. in this study, we examined the effects of targeted deletions of tlr1, tlr2, tlr4, tlr6, tlr9, and myd88 on the induction of myelin oligodendrocyte glycoprotein 35-55 (mog(35-55)) peptide/cfa/pertussis toxin-induced autoimmune encephalomyelitis. 2011-09-23 2023-08-12 mouse
Carlo Guardiani, Simone Marsili, Stefania Marchetti, Cecilia Gambi, Piero Procacci, Roberto Liv. Conformational structure of the MOG-derived peptide 101-108 in solution. Biopolymers. vol 96. issue 3. 2011-09-20. PMID:20549678. one of the most important targets in the autoimmune attack in experimental autoimmune encephalomyielitis is the myelin oligodendrocyte glycoprotein (mog). 2011-09-20 2023-08-12 Not clear
Alexander Annenkov, Anne Rigby, Sandra Amor, Dun Zhou, Nasim Yousaf, Bernhard Hemmer, Yuti Chernajovsk. A chimeric receptor of the insulin-like growth factor receptor type 1 (IGFR1) and a single chain antibody specific to myelin oligodendrocyte glycoprotein activates the IGF1R signalling cascade in CG4 oligodendrocyte progenitors. Biochimica et biophysica acta. vol 1813. issue 8. 2011-08-30. PMID:21600935. in order to generate neural stem cells with increased ability to survive after transplantation in brain parenchyma we developed a chimeric receptor (chr) that binds to myelin oligodendrocyte glycoprotein (mog) via its ectodomain and activates the insulin-like growth factor receptor type 1 ‎‎(igf1r) signalling cascade. 2011-08-30 2023-08-12 Not clear
Mascha S Recks, Klaus Addicks, Stefanie Kuerte. Spinal cord histopathology of MOG peptide 35-55-induced experimental autoimmune encephalomyelitis is time- and score-dependent. Neuroscience letters. vol 494. issue 3. 2011-08-17. PMID:21406210. in the present study, we demonstrate that the histopathologic features of myelin oligodendrocyte glycoprotein (mog) peptide 35-55-induced experimental autoimmune encephalomyelitis (eae) in c57bl/6 mice closely mirror the hallmarks of ms pathology. 2011-08-17 2023-08-12 mouse
Paula Dore-Duffy, Marie Wencel, Vladimir Katyshev, Kristen Clear. Chronic mild hypoxia ameliorates chronic inflammatory activity in myelin oligodendrocyte glycoprotein (MOG) peptide induced experimental autoimmune encephalomyelitis (EAE). Advances in experimental medicine and biology. vol 701. 2011-08-16. PMID:21445784. chronic mild hypoxia ameliorates chronic inflammatory activity in myelin oligodendrocyte glycoprotein (mog) peptide induced experimental autoimmune encephalomyelitis (eae). 2011-08-16 2023-08-12 mouse
Paula Dore-Duffy, Marie Wencel, Vladimir Katyshev, Kristen Clear. Chronic mild hypoxia ameliorates chronic inflammatory activity in myelin oligodendrocyte glycoprotein (MOG) peptide induced experimental autoimmune encephalomyelitis (EAE). Advances in experimental medicine and biology. vol 701. 2011-08-16. PMID:21445784. although aspects of adaptive angiogenesis are induced in ms and experimental autoimmune encephalomyelitis (eae), vascular remodeling is ineffective and the balance between metabolic need and oxygen (o(2)) and glucose availability is disrupted.we hypothesized that restoration of metabolic homeostasis in the cns would ameliorate tissue damage and promote repair in myelin oligodendrocyte glycoprotein(mog)-induced eae in mice. 2011-08-16 2023-08-12 mouse
Thomas Tourdias, Bassem Hiba, Gerard Raffard, Marc Biran, Tomokazu Nishiguchi, Justine Aussudre, Jean-Michel Franconi, Bruno Brochet, Klaus G Petry, Vincent Dousse. Adapted focal experimental autoimmune encephalomyelitis to allow MRI exploration of multiple sclerosis features. Experimental neurology. vol 230. issue 2. 2011-08-15. PMID:21575634. we adapted a two-hit model based on pre-sensitization of the lewis rat with myelin oligodendrocyte protein (mog) followed by stereotaxic injection of pro-inflammatory cytokines (tnfα+ifnγ) within the internal capsule. 2011-08-15 2023-08-12 rat
Kerstin Göbel, Susann Pankratz, Tilman Schneider-Hohendorf, Stefan Bittner, Michael K Schuhmann, Harald F Langer, Guido Stoll, Heinz Wiendl, Christoph Kleinschnitz, Sven G Meut. Blockade of the kinin receptor B1 protects from autoimmune CNS disease by reducing leukocyte trafficking. Journal of autoimmunity. vol 36. issue 2. 2011-07-22. PMID:21216565. we studied the expression of b1r and b2r and the effect of their inhibition on the disease course, bbb integrity and t cell migration following myelin oligodendrocyte glycoprotein (mog(35-55))-induced eae. 2011-07-22 2023-08-12 mouse