| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| N Arai, M I Papp, P L Lanto. New observation on ubiquitinated neurons in the cerebral cortex of multiple system atrophy (MSA). Neuroscience letters. vol 182. issue 2. 1995-05-17. PMID:7715809. |
to investigate neuronal damage in the cerebral cortex in patients with multiple system atrophy (msa), immunohistochemical stainings were carried out on the prefrontal cortex, the hippocampus, the precentral gyrus, the supplementary motor cortex and the occipital cortex in 6 cases of msa and 6 controls, using antibodies against ubiquitin, tau protein and neurofilaments (bf10, rt97, 147). |
1995-05-17 |
2023-08-12 |
Not clear |
| J P Brion, J N Octave, A M Couc. Distribution of the phosphorylated microtubule-associated protein tau in developing cortical neurons. Neuroscience. vol 63. issue 3. 1995-04-26. PMID:7898684. |
we have investigated the developmental distribution of the phosphorylated fetal-type tau in the developing rat cortex and in cultures of embryonic cortical neurons, using antibodies which react with tau in a phosphorylation-dependent manner. |
1995-04-26 |
2023-08-12 |
rat |
| J P Brion, J N Octave, A M Couc. Distribution of the phosphorylated microtubule-associated protein tau in developing cortical neurons. Neuroscience. vol 63. issue 3. 1995-04-26. PMID:7898684. |
the phosphorylated fetal-type tau was present in the developing cortex at 20 days but not at 18 days of embryonic life and was not detected before four to five days in neuronal culture. |
1995-04-26 |
2023-08-12 |
rat |
| J P Brion, J N Octave, A M Couc. Distribution of the phosphorylated microtubule-associated protein tau in developing cortical neurons. Neuroscience. vol 63. issue 3. 1995-04-26. PMID:7898684. |
the timing of appearance of phosphorylated tau in the cortex, by comparison with the expression of other developmental markers, indicates that phosphorylated tau is present at a high level only during the period of intense neuritic outgrowth and that it disappears during the period of neurite stabilization and synaptogenesis, concomitantly to the expression of adult tau isoforms. |
1995-04-26 |
2023-08-12 |
rat |
| H Mori, M Nishimura, Y Namba, M Od. Corticobasal degeneration: a disease with widespread appearance of abnormal tau and neurofibrillary tangles, and its relation to progressive supranuclear palsy. Acta neuropathologica. vol 88. issue 2. 1995-01-04. PMID:7985491. |
immunohistochemistry for tau showed tau-positive neurons in the cerebral cortex, brain stem, subcortical nuclei and spinal cord, and tau-positive glial cells were seen in the cerebral cortex, white matter and subcortical nuclei, and thread-like structures were seen in the cerebral cortex and white matter. |
1995-01-04 |
2023-08-12 |
Not clear |
| P Vermersch, Y Robitaille, L Bernier, A Wattez, D Gauvreau, A Delacourt. Biochemical mapping of neurofibrillary degeneration in a case of progressive supranuclear palsy: evidence for general cortical involvement. Acta neuropathologica. vol 87. issue 6. 1994-10-17. PMID:8091949. |
we found that: (i) the abnormal tau proteins can be detected in all cortical areas, sometimes in larger amounts than in some subcortical areas; (ii) these abnormal tau proteins consist of a doublet called tau 64 and 69, except for in the entorhinal cortex where we detected, as for alzheimer brains, the triplet of tau proteins called tau 55, 64 and 69; (iii) the amounts of abnormal tau proteins were higher in some neocortical regions, especially in the frontal lobe, than in the hippocampal formation. |
1994-10-17 |
2023-08-12 |
Not clear |
| H Braak, E Braak, M Strothjohan. Abnormally phosphorylated tau protein related to the formation of neurofibrillary tangles and neuropil threads in the cerebral cortex of sheep and goat. Neuroscience letters. vol 171. issue 1-2. 1994-10-10. PMID:7521944. |
abnormally phosphorylated tau protein related to the formation of neurofibrillary tangles and neuropil threads in the cerebral cortex of sheep and goat. |
1994-10-10 |
2023-08-12 |
human |
| P Sindou, M Lesort, P Couratier, C Yardin, F Esclaire, J Hugo. Glutamate increases tau phosphorylation in primary neuronal cultures from fetal rat cerebral cortex. Brain research. vol 646. issue 1. 1994-09-15. PMID:7914466. |
glutamate increases tau phosphorylation in primary neuronal cultures from fetal rat cerebral cortex. |
1994-09-15 |
2023-08-12 |
human |
| T Fukuda, J Tanaka, K Watabe, R T Numoto, M Minamitan. Immunohistochemistry of neuronal inclusions in the cerebral cortex and brain-stem in Lewy body disease. Acta pathologica japonica. vol 43. issue 10. 1994-02-24. PMID:8291442. |
the cerebral cortex was immuno-reactive with antibodies against tyrosine hydroxylase (th) and tau protein, and differed from bs. |
1994-02-24 |
2023-08-12 |
Not clear |
| T Priestley, J A Kem. Agonist response kinetics of N-methyl-D-aspartate receptors in neurons cultured from rat cerebral cortex and cerebellum: evidence for receptor heterogeneity. Molecular pharmacology. vol 44. issue 6. 1994-01-26. PMID:7903418. |
the difference between the two cell types in the tau off for glycine was more substantial, i.e., 558 +/- 53 (n = 15) and 2214 +/- 125 msec (n = 19, p < 0.0001) for cerebellum and cortex, respectively. |
1994-01-26 |
2023-08-12 |
rat |
| K Nihei, A C McKee, N W Kowal. Patterns of neuronal degeneration in the motor cortex of amyotrophic lateral sclerosis patients. Acta neuropathologica. vol 86. issue 1. 1993-10-08. PMID:8396837. |
we examined patterns of neuronal degeneration in the motor cortex of amyotrophic lateral sclerosis (als) patients using traditional cell stains and several histochemical markers including neurofilament, parvalbumin, nadph-diaphorase, ubiquitin, alz-50 and tau. |
1993-10-08 |
2023-08-12 |
Not clear |
| K Oyanagi, F Ikut. [The aging of central nervous system in methylmercury intoxication in Niigata, Japan--quantitative investigation of neurofibrillary tangles and senile plaques]. No to shinkei = Brain and nerve. vol 45. issue 3. 1993-08-06. PMID:8323818. |
in order to elucidate the progression of aging process in the brains of methylmercury intoxication in humans, neurofibrillary tangles (nft) and senile plaques (sp) were examined quantitatively in the temporal lobe and calcarine cortex of 16 patients with methylmercury intoxication and 12 control subjects by using tau and beta-peptide immunostained preparations. |
1993-08-06 |
2023-08-12 |
human |
| P Vermersch, A Delacourte, F Javoy-Agid, J J Hauw, Y Agi. Dementia in Parkinson's disease: biochemical evidence for cortical involvement using the immunodetection of abnormal Tau proteins. Annals of neurology. vol 33. issue 5. 1993-06-21. PMID:8498822. |
the frequency and intensity of immunodetection of the abnormal tau triplet were higher in the demented parkinsonian subgroups than in the nondemented parkinsonian subgroup in the prefrontal area, temporal cortex, and entorhinal cortex but not in either the occipital or the cingular cortex. |
1993-06-21 |
2023-08-12 |
human |
| P Vermersch, A Delacourte, F Javoy-Agid, J J Hauw, Y Agi. Dementia in Parkinson's disease: biochemical evidence for cortical involvement using the immunodetection of abnormal Tau proteins. Annals of neurology. vol 33. issue 5. 1993-06-21. PMID:8498822. |
a quantification of abnormal tau triplet by densitometry showed that unlike the results obtained in alzheimer patients, the intensity of lesions in the cerebral cortex of the most demented parkinsonian patients was more severe in the prefrontal area versus the temporal area. |
1993-06-21 |
2023-08-12 |
human |
| M P Mattson, M A Lovell, W D Ehmann, W R Markesber. Comparison of the effects of elevated intracellular aluminum and calcium levels on neuronal survival and tau immunoreactivity. Brain research. vol 602. issue 1. 1993-04-12. PMID:8448655. |
we examined the effects of experimentally elevated intraneuronal levels of aluminum ([al]i) and/or calcium ([ca2+]i) on neuronal degeneration and antigenic alterations in the microtubule-associated protein tau in cell cultures of rat hippocampus and human cerebral cortex. |
1993-04-12 |
2023-08-12 |
human |
| P Vermersch, B Frigard, J P David, C Fallet-Bianco, A Delacourt. Presence of abnormally phosphorylated Tau proteins in the entorhinal cortex of aged non-demented subjects. Neuroscience letters. vol 144. issue 1-2. 1992-12-18. PMID:1436694. |
presence of abnormally phosphorylated tau proteins in the entorhinal cortex of aged non-demented subjects. |
1992-12-18 |
2023-08-11 |
human |
| P Vermersch, B Frigard, J P David, C Fallet-Bianco, A Delacourt. Presence of abnormally phosphorylated Tau proteins in the entorhinal cortex of aged non-demented subjects. Neuroscience letters. vol 144. issue 1-2. 1992-12-18. PMID:1436694. |
the immunodetection of tau 55, 64 and 69 was positive in all cortical areas from alzheimer patients, in the entorhinal cortex from each control aged more than 65 but not in cortical samples from younger controls. |
1992-12-18 |
2023-08-11 |
human |
| K Arima, S Murayama, S Oyanagi, T Akashi, T Inos. Presenile dementia with progressive supranuclear palsy tangles and Pick bodies: an unusual degenerative disorder involving the cerebral cortex, cerebral nuclei, and brain stem nuclei. Acta neuropathologica. vol 84. issue 2. 1992-10-15. PMID:1381857. |
immunocytochemical study using anti-phosphorylated tau and anti-ubiquitin antibodies in conjunction with ultrastructural observations revealed two types of inclusions: neurofibrillary tangles (nfts) of progressive supranuclear palsy (psp) in the edinger-westphal nucleus, locus coeruleus, cerebellar dentate nucleus, inferior olivary nucleus, and posterior horn of the spinal cord; and pick bodies (pbs) in the atrophied cerebral cortex and red nucleus. |
1992-10-15 |
2023-08-11 |
Not clear |
| F F Cruz-Sanchez, M L Rossi, A Cardozo, P Deacon, E Tolos. Clinical and pathological study of two patients with progressive supranuclear palsy and Alzheimer's changes. Antigenic determinants that distinguish cortical and subcortical neurofibrillary tangles. Neuroscience letters. vol 136. issue 1. 1992-08-24. PMID:1321966. |
a topographic and immunohistological study (with neurofilament subunit and tau and ubiquitin antibodies) of the distribution of neurofibrillary tangles (nfts) was performed in order to compare the characteristics of nfts from cortex and brainstem. |
1992-08-24 |
2023-08-11 |
Not clear |
| R W Shin, T Iwaki, T Kitamoto, Y Sato, J Tateish. Massive accumulation of modified tau and severe depletion of normal tau characterize the cerebral cortex and white matter of Alzheimer's disease. Demonstration using the hydrated autoclaving method. The American journal of pathology. vol 140. issue 4. 1992-05-11. PMID:1373272. |
massive accumulation of modified tau and severe depletion of normal tau characterize the cerebral cortex and white matter of alzheimer's disease. |
1992-05-11 |
2023-08-11 |
human |