All Relations between reward and dopamine

Publication Sentence Publish Date Extraction Date Species
Travis E Baker, Tim Stockwell, Gordon Barnes, Roderick Haesevoets, Clay B Holroy. Reward Sensitivity of ACC as an Intermediate Phenotype between DRD4-521T and Substance Misuse. Journal of cognitive neuroscience. vol 28. issue 3. 2016-12-13. PMID:26601911. we used structural equation modeling to examine whether two related electrophysiological phenomena associated with the control and reinforcement learning functions of acc-theta power and the reward positivity-mediated the relationship between the degree of substance misuse and genetic polymorphisms that regulate dopamine processing in frontal cortex. 2016-12-13 2023-08-13 Not clear
Hugo Juárez Olguín, David Calderón Guzmán, Ernestina Hernández García, Gerardo Barragán Mejí. The Role of Dopamine and Its Dysfunction as a Consequence of Oxidative Stress. Oxidative medicine and cellular longevity. vol 2016. 2016-12-13. PMID:26770661. the level of dopamine transmission increases in response to any type of reward and by a large number of strongly additive drugs. 2016-12-13 2023-08-13 Not clear
Brian A Anderson, Hiroto Kuwabara, Dean F Wong, Emily G Gean, Arman Rahmim, James R Brašić, Noble George, Boris Frolov, Susan M Courtney, Steven Yanti. The Role of Dopamine in Value-Based Attentional Orienting. Current biology : CB. vol 26. issue 4. 2016-12-13. PMID:26877079. dopamine signaling within the ventral striatum plays an important role in reward learning, representing the expected reward initiated by a cue. 2016-12-13 2023-08-13 Not clear
Brian A Anderson, Hiroto Kuwabara, Dean F Wong, Emily G Gean, Arman Rahmim, James R Brašić, Noble George, Boris Frolov, Susan M Courtney, Steven Yanti. The Role of Dopamine in Value-Based Attentional Orienting. Current biology : CB. vol 26. issue 4. 2016-12-13. PMID:26877079. how dopamine and the striatum may be involved in maintaining behaviors that have been shaped by reward learning, even after reward expectancies have changed, is less well understood. 2016-12-13 2023-08-13 Not clear
Brian A Anderson, Hiroto Kuwabara, Dean F Wong, Emily G Gean, Arman Rahmim, James R Brašić, Noble George, Boris Frolov, Susan M Courtney, Steven Yanti. The Role of Dopamine in Value-Based Attentional Orienting. Current biology : CB. vol 26. issue 4. 2016-12-13. PMID:26877079. we show that, in the explicit absence of reward, the magnitude of attentional capture by previously reward-associated but currently task-irrelevant distractors is correlated across individuals with changes in available d2/d3 dopamine receptors (presumably due to intrasynaptic dopamine) linked to distractor processing within the right caudate and posterior putamen. 2016-12-13 2023-08-13 Not clear
Brian A Anderson, Hiroto Kuwabara, Dean F Wong, Emily G Gean, Arman Rahmim, James R Brašić, Noble George, Boris Frolov, Susan M Courtney, Steven Yanti. The Role of Dopamine in Value-Based Attentional Orienting. Current biology : CB. vol 26. issue 4. 2016-12-13. PMID:26877079. our findings provide direct evidence linking dopamine signaling within the striatum to the involuntary orienting of attention, and specifically to the attention-grabbing quality of learned reward cues. 2016-12-13 2023-08-13 Not clear
W Goutier, J P Lowry, A C McCreary, J J O'Conno. Frequency-Dependent Modulation of Dopamine Release by Nicotine and Dopamine D1 Receptor Ligands: An In Vitro Fast Cyclic Voltammetry Study in Rat Striatum. Neurochemical research. vol 41. issue 5. 2016-12-13. PMID:26975318. nicotine is a highly addictive drug and exerts this effect partially through the modulation of dopamine release and increasing extracellular dopamine in regions such as the brain reward systems. 2016-12-13 2023-08-13 rat
J D Slade, B E Stavele. Manipulation of components that control feeding behavior in Drosophila melanogaster increases sensitivity to amino acid starvation. Genetics and molecular research : GMR. vol 15. issue 1. 2016-11-11. PMID:26909968. this action may manipulate the post-feeding reward circuit in that lowered dopamine levels depress feeding, and excess dopamine levels encourage feeding. 2016-11-11 2023-08-13 drosophila_melanogaster
Ewa A Miendlarzewska, Daphne Bavelier, Sophie Schwart. Influence of reward motivation on human declarative memory. Neuroscience and biobehavioral reviews. vol 61. 2016-11-09. PMID:26657967. existing evidence suggests that the neurotransmitter dopamine boosts the formation of declarative memory for rewarded information and may also control the generalization of reward values. 2016-11-09 2023-08-13 human
Elena H Chartoff, Shayla R Ebner, Angela Sparrow, David Potter, Phillip M Baker, Michael E Ragozzino, Mitchell F Roitma. Relative Timing Between Kappa Opioid Receptor Activation and Cocaine Determines the Impact on Reward and Dopamine Release. Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology. vol 41. issue 4. 2016-11-08. PMID:26239494. relative timing between kappa opioid receptor activation and cocaine determines the impact on reward and dopamine release. 2016-11-08 2023-08-13 rat
Elena H Chartoff, Shayla R Ebner, Angela Sparrow, David Potter, Phillip M Baker, Michael E Ragozzino, Mitchell F Roitma. Relative Timing Between Kappa Opioid Receptor Activation and Cocaine Determines the Impact on Reward and Dopamine Release. Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology. vol 41. issue 4. 2016-11-08. PMID:26239494. we hypothesize that the initial aversive effects of kor activation increase, whereas the delayed rewarding effects decrease, the net effects of cocaine on reward and dopamine release. 2016-11-08 2023-08-13 rat
Elena H Chartoff, Shayla R Ebner, Angela Sparrow, David Potter, Phillip M Baker, Michael E Ragozzino, Mitchell F Roitma. Relative Timing Between Kappa Opioid Receptor Activation and Cocaine Determines the Impact on Reward and Dopamine Release. Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology. vol 41. issue 4. 2016-11-08. PMID:26239494. using intracranial self-stimulation and fast scan cyclic voltammetry, we found that cocaine-induced increases in brain stimulation reward and evoked dopamine release in the nac core were potentiated when cocaine was administered within 1 h of salva, but attenuated when administered 24 h after salva. 2016-11-08 2023-08-13 rat
Zhi-Bing You, Bin Wang, Qing-Rong Liu, Yan Wu, Laszlo Otvos, Roy A Wis. Reciprocal Inhibitory Interactions Between the Reward-Related Effects of Leptin and Cocaine. Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology. vol 41. issue 4. 2016-11-08. PMID:26243270. in addition to neuronal inputs, forebrain dopamine circuits are modulated by hormonal influences; one of these is leptin, an adipose-derived hormone that attenuates the rewarding effects of food- and hunger-associated brain stimulation reward. 2016-11-08 2023-08-13 rat
A Benyamina, M Reynau. [Management of alcohol use disorders in ambulatory care: Which follow-up and for how long?]. L'Encephale. vol 42. issue 1. 2016-10-31. PMID:26796554. on the contrary, alcohol dependence has been defined as a primary chronic disease of the brain reward, motivation, memory and related circuitry, involving the signalling pathway of neurotransmitters such as dopamine, opioid peptides, and gamma-aminobutyric acid. 2016-10-31 2023-08-13 Not clear
Kimberly Renk, Neil W Boris, Ellen Kolomeyer, Amanda Lowell, Jayme Puff, Annelise Cunningham, Maria Khan, Meagan McSwigga. The state of evidence-based parenting interventions for parents who are substance-involved. Pediatric research. vol 79. issue 1-2. 2016-10-24. PMID:26484624. these parenting interventions should be considered within the context of the neurobiology of substance abuse, which emphasizes the role of dopamine in the reward systems that promote substance use. 2016-10-24 2023-08-13 Not clear
Y Li, S Ge, N Li, L Chen, S Zhang, J Wang, H Wu, X Wang, X Wan. NMDA and dopamine D1 receptors within NAc-shell regulate IEG proteins expression in reward circuit during cocaine memory reconsolidation. Neuroscience. vol 315. 2016-10-19. PMID:26674058. nmda and dopamine d1 receptors within nac-shell regulate ieg proteins expression in reward circuit during cocaine memory reconsolidation. 2016-10-19 2023-08-13 Not clear
Y Li, S Ge, N Li, L Chen, S Zhang, J Wang, H Wu, X Wang, X Wan. NMDA and dopamine D1 receptors within NAc-shell regulate IEG proteins expression in reward circuit during cocaine memory reconsolidation. Neuroscience. vol 315. 2016-10-19. PMID:26674058. in summary, n-methyl-d-aspartate (nmda) and dopamine d1 receptors, but not dopamine d2 or β adrenergic receptors, within the nac-shell, may regulate zif 268 and fos b expression in most brain nuclei of the reward circuit after cocaine memory reactivation. 2016-10-19 2023-08-13 Not clear
b' Mateusz Dudek, Usama Abo-Ramadan, Derik Hermann, Matthew Brown, Santiago Canals, Wolfgang H Sommer, Petri Hyyti\\xc3\\xa. Brain activation induced by voluntary alcohol and saccharin drinking in rats assessed with manganese-enhanced magnetic resonance imaging. Addiction biology. vol 20. issue 6. 2016-10-17. PMID:25146961.' these data show that voluntary alcohol recruits an extensive network that includes the ascending dopamine systems and their afferent connections, and that this network is largely shared with saccharin reward. 2016-10-17 2023-08-13 rat
Jeffery C Sobieraj, Airee Kim, McKenzie J Fannon, Chitra D Mandya. Chronic wheel running-induced reduction of extinction and reinstatement of methamphetamine seeking in methamphetamine dependent rats is associated with reduced number of periaqueductal gray dopamine neurons. Brain structure & function. vol 221. issue 1. 2016-10-14. PMID:25273280. given that the pag dopamine neurons project onto the structures of the extended amygdala, the present findings also suggest that wheel running may be preventing certain allostatic changes in the brain reward and stress systems contributing to the negative reinforcement and perpetuation of the addiction cycle. 2016-10-14 2023-08-13 rat
Laura G Rosen, Jordan Zunder, Justine Renard, Jennifer Fu, Walter Rushlow, Steven R Laviolett. Opiate Exposure State Controls a D2-CaMKIIα-Dependent Memory Switch in the Amygdala-Prefrontal Cortical Circuit. Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology. vol 41. issue 3. 2016-10-06. PMID:26174594. in the opiate-naïve state, reward memory formation in the bla involves a functional link between dopamine (da) d1 receptor (d1r) and extracellular signal-related kinase 1/2 (erk1/2) signaling substrates, but switches to a da d2 (d2r)/ca(2+)/calmodulin-dependent protein kinase iiα (camkiiα)-dependent memory substrate following chronic opiate exposure and spontaneous withdrawal. 2016-10-06 2023-08-13 rat