| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| J D Sinden, D M Atren. Dopaminergic and noradrenergic inhibition of hypothalamic self-stimulation: differentiation of reward and performance effects. European journal of pharmacology. vol 86. issue 2. 1983-04-15. PMID:7160436. |
dopaminergic and noradrenergic inhibition of hypothalamic self-stimulation: differentiation of reward and performance effects. |
1983-04-15 |
2023-08-12 |
Not clear |
| R A Wise, M A Bozart. Action of drugs of abuse on brain reward systems: an update with specific attention to opiates. Pharmacology, biochemistry, and behavior. vol 17. issue 2. 1982-12-16. PMID:6127721. |
in addressing the role that the substrate of brain stimulation reward might play in drug abuse, wise [47] reviewed evidence relating brain stimulation and psychomotor stimulant reward to dopaminergic but not noradrenergic elements identified with brain reward circuitry. |
1982-12-16 |
2023-08-12 |
Not clear |
| R A Wise, M A Bozart. Action of drugs of abuse on brain reward systems: an update with specific attention to opiates. Pharmacology, biochemistry, and behavior. vol 17. issue 2. 1982-12-16. PMID:6127721. |
he then speculated that one possible mechanism of opiate, ethanol, barbiturate or benzodiazepine reward might involve a specified disinhibition of the dopaminergic element. |
1982-12-16 |
2023-08-12 |
Not clear |
| R A Wise, M A Bozart. Action of drugs of abuse on brain reward systems: an update with specific attention to opiates. Pharmacology, biochemistry, and behavior. vol 17. issue 2. 1982-12-16. PMID:6127721. |
he suggested that these drugs might have inhibitory actions on locus coeruleus, which in turn might send an inhibitory projection to the dopaminergic link in reward circuitry. |
1982-12-16 |
2023-08-12 |
Not clear |
| R A Wise, M A Bozart. Action of drugs of abuse on brain reward systems: an update with specific attention to opiates. Pharmacology, biochemistry, and behavior. vol 17. issue 2. 1982-12-16. PMID:6127721. |
rewarding opiate injections appear to activate the same or a similar dopaminergic link in brain reward circuitry as that thought to be activated through its afferent inputs in the case of brain stimulation reward and activated at its synaptic terminals in the case of psychomotor stimulant reward. |
1982-12-16 |
2023-08-12 |
Not clear |
| R A Wise, M A Bozart. Action of drugs of abuse on brain reward systems: an update with specific attention to opiates. Pharmacology, biochemistry, and behavior. vol 17. issue 2. 1982-12-16. PMID:6127721. |
whether other drugs of abuse activate links in brain reward circuitry which function in parallel or in series with the dopaminergic link identified with opiates and stimulants remains an open question. |
1982-12-16 |
2023-08-12 |
Not clear |
| S Xenakis, A Sclafan. The dopaminergic mediation of a sweet reward in normal and VMH hyperphagic rats. Pharmacology, biochemistry, and behavior. vol 16. issue 2. 1982-06-24. PMID:6951206. |
the dopaminergic mediation of a sweet reward in normal and vmh hyperphagic rats. |
1982-06-24 |
2023-08-12 |
rat |
| M A Bozarth, R A Wis. Heroin reward is dependent on a dopaminergic substrate. Life sciences. vol 29. issue 18. 1982-02-12. PMID:7311722. |
heroin reward is dependent on a dopaminergic substrate. |
1982-02-12 |
2023-08-12 |
Not clear |
| S D Glick, L M Weaver, R C Meibac. Amphetamine enhancement of reward asymmetry. Psychopharmacology. vol 73. issue 4. 1981-09-15. PMID:6789352. |
it is proposed that schizophrenia results from a lateralized overactivity of dopaminergic neuronal systems mediating reward and that amphetamine mimics schizophrenic symptomatology by enhancing lateralization of the same systems. |
1981-09-15 |
2023-08-12 |
rat |
| D R Royall, W R Klem. Dopaminergic mediation of reward: evidence gained using a natural reinforcer in a behavioral contrast paradigm. Neuroscience letters. vol 21. issue 2. 1981-06-13. PMID:7219871. |
dopaminergic mediation of reward: evidence gained using a natural reinforcer in a behavioral contrast paradigm. |
1981-06-13 |
2023-08-12 |
rat |
| D R Royall, W R Klem. Dopaminergic mediation of reward: evidence gained using a natural reinforcer in a behavioral contrast paradigm. Neuroscience letters. vol 21. issue 2. 1981-06-13. PMID:7219871. |
previous studies suggest a dopaminergic basis for the apparent reward properties of self-stimulation of certain brain areas with electrical current. |
1981-06-13 |
2023-08-12 |
rat |
| A Routtenber. Participation of brain stimulation reward substrates in memory: anatomical and biochemical evidence. Federation proceedings. vol 38. issue 11. 1979-12-18. PMID:385350. |
in view of the biological specificity of brain reinforcement pathways, i suggest the hypothesis that activity in the mesocortical dopaminergic brain stimulation reward pathways participates in the memory consolidation process. |
1979-12-18 |
2023-08-11 |
Not clear |
| G J Mogenson, M Takigawa, A Robertson, M W. Self-stimulation of the nucleus accumbens and ventral tegmental area of Tsai attenuated by microinjections of spiroperidol into the nucleus accumbens. Brain research. vol 171. issue 2. 1979-10-17. PMID:572734. |
these observations provide additional evidence implicating dopaminergic neurons in brain-stimulation reward and suggest that dopaminergic neurons contribute to self-stimulation of the nucleus accumbens but not the prefrontal cortex. |
1979-10-17 |
2023-08-11 |
Not clear |
| G Fouriezos, P Hansson, R A Wis. Neuroleptic-induced attenuation of brain stimulation reward in rats. Journal of comparative and physiological psychology. vol 92. issue 4. 1978-12-02. PMID:29060. |
these data support the view that central dopaminergic systems are important components of the neural mechanisms mediating reward. |
1978-12-02 |
2023-08-11 |
rat |
| N Francis, E Marley, J D Stephenso. Effects of spiperone on self-stimulation and other activities of the Mongolian gerbil. British journal of pharmacology. vol 63. issue 1. 1978-07-15. PMID:565659. |
5 the spiperone-induced attentuation of self-stimulation was not necessarily a result of its action on dopaminergic reward pathways since the effects could equally well be explained by a failure to initiate responding. |
1978-07-15 |
2023-08-11 |
Not clear |
| H C Fibiger, D A Carter, A G Phillip. Decreased intracranial self-stimulation after neuroleptics or 6-hydroxydopamine: evidence for mediation by motor deficits rather than by reduced reward. Psychopharmacology. vol 47. issue 1. 1976-10-20. PMID:959465. |
while these experiments do not exclude the possibility that dopaminergic (da) systems participate in some central reinforcement mechanisms, they suggest that neuroleptics and 6-ohda decrease responding for food or ics primarily by impairing the function of da systems critically involved in the initiation or maintenance of operant behaviour rather than by interfering with reward. |
1976-10-20 |
2023-08-11 |
rat |
| B L Baxter, M I Gluckman, R A Scern. Apomorphine self-injection is not affected by alpha-methylparatyrosine treatment: support for dopaminergic reward. Pharmacology, biochemistry, and behavior. vol 4. issue 5. 1976-10-02. PMID:951437. |
apomorphine self-injection is not affected by alpha-methylparatyrosine treatment: support for dopaminergic reward. |
1976-10-02 |
2023-08-11 |
Not clear |
| S L Brown, R Fia. Intracranial alpha-methyl-P-tyrosine and response for electrical brain stimulation. Pharmacology, biochemistry, and behavior. vol 3. issue 4. 1976-01-16. PMID:1187727. |
this result lends support to data reported in the literature on the use of ampt administered intraperitoneally or orally, and suggests a noradrenergic or dopaminergic system of reward in the lateral hypothalamus. |
1976-01-16 |
2023-08-11 |
rat |
| A G Phillips, S M Brooke, H C Fibige. Effects of amphetamine isomers and neuroleptics on self-stimulation from the nucleus accumbens and dorsal noradrenergic bundle. Brain research. vol 85. issue 1. 1975-04-23. PMID:1109690. |
it is suggested that neuroleptic drugs may produce a general disruption of operant behavior and that the decrease in ics produced by these agents does not therefore necessarily implicate dopaminergic mechanisms in the neurochemistry of reward. |
1975-04-23 |
2023-08-11 |
rat |