| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| S Inoue, S Satoh, M Saito, M Naitoh, H Suzuki, M Egaw. Effects of selective vagotomy on circadian rhythms of plasma glucose, insulin and food intake in control and ventromedial hypothalamic (VMH) lesioned rats. Obesity research. vol 3 Suppl 5. 1996-07-31. PMID:8653558. |
in vmh lesioned rats, celiac vagotomy destroyed circadian rhythm of food intake due to the reduction of plasma insulin by removal of efferent function without affecting the loss of circadian rhythms of plasma glucose and insulin. |
1996-07-31 |
2023-08-12 |
rat |
| M Orosco, C Rouch, S Nicolaïdi. Rostromedial hypothalamic monoamine changes in response to intravenous infusions of insulin and glucose in freely feeding obese Zucker rats: a microdialysis study. Appetite. vol 26. issue 1. 1996-07-30. PMID:8660029. |
rostromedial hypothalamic monoamine changes in response to intravenous infusions of insulin and glucose in freely feeding obese zucker rats: a microdialysis study. |
1996-07-30 |
2023-08-12 |
rat |
| N I Williams, M J Lancas, J L Camero. Stimulation of luteinizing hormone secretion by food intake: evidence against a role for insulin. Endocrinology. vol 137. issue 6. 1996-07-18. PMID:8641210. |
stimulation of luteinizing hormone secretion by food intake: evidence against a role for insulin. |
1996-07-18 |
2023-08-12 |
monkey |
| S G Miller, P De Vos, M Guerre-Millo, K Wong, T Hermann, B Staels, M R Briggs, J Auwer. The adipocyte specific transcription factor C/EBPalpha modulates human ob gene expression. Proceedings of the National Academy of Sciences of the United States of America. vol 93. issue 11. 1996-07-17. PMID:8643605. |
ob gene expression is increased in obese rodents and regulated by feeding, insulin, and glucocorticoids, which supports the concept that ob gene expression is under hormonal control, which is expected for a key factor controlling body weight homeostasis and energy balance. |
1996-07-17 |
2023-08-12 |
human |
| T Kazumi, T Hirano, H Odaka, T Ebara, N Amano, T Hozumi, Y Ishida, G Yoshin. VLDL triglyceride kinetics in Wistar fatty rats, an animal model of NIDDM: effects of dietary fructose alone or in combination with pioglitazone. Diabetes. vol 45. issue 6. 1996-07-09. PMID:8635657. |
although feeding fructose into fatty rats did not change plasma glucose and insulin levels, it produced a twofold increase in tg levels (8.74 +/- 1.15 mmol/l). |
1996-07-09 |
2023-08-12 |
rat |
| C E Wiseman, J A Higgins, G S Denyer, J C Mille. Amylopectin starch induces nonreversible insulin resistance in rats. The Journal of nutrition. vol 126. issue 2. 1996-07-01. PMID:8632213. |
starches that are high in amylopectin are digested and absorbed more quickly than starches with a high amylose content and produce insulin resistance in rats during long-term feeding. |
1996-07-01 |
2023-08-12 |
rat |
| C E Wiseman, J A Higgins, G S Denyer, J C Mille. Amylopectin starch induces nonreversible insulin resistance in rats. The Journal of nutrition. vol 126. issue 2. 1996-07-01. PMID:8632213. |
the aim of this study was to determine whether amylopectin-induced insulin resistance could be prevented or reversed by a period of high amylose feeding. |
1996-07-01 |
2023-08-12 |
rat |
| C E Wiseman, J A Higgins, G S Denyer, J C Mille. Amylopectin starch induces nonreversible insulin resistance in rats. The Journal of nutrition. vol 126. issue 2. 1996-07-01. PMID:8632213. |
all rats were fed two 10-g meals per day (300 kj/d), and insulin sensitivity was assessed by intravenous glucose tolerance test (ivgtt) after 8 or 16 wk of feeding. |
1996-07-01 |
2023-08-12 |
rat |
| C E Wiseman, J A Higgins, G S Denyer, J C Mille. Amylopectin starch induces nonreversible insulin resistance in rats. The Journal of nutrition. vol 126. issue 2. 1996-07-01. PMID:8632213. |
insulin responses, however, were 50% higher (p < 0.01) after 16 wk of high amylopectin feeding [area under the plasma insulin curve (auc) = 18.1 +/- 1.4 nmol.l-1 x 15 min] compared with high amylose feeding (auc = 13.0 +/- 1.2 nmol.l-1 x 15 min). |
1996-07-01 |
2023-08-12 |
rat |
| C E Wiseman, J A Higgins, G S Denyer, J C Mille. Amylopectin starch induces nonreversible insulin resistance in rats. The Journal of nutrition. vol 126. issue 2. 1996-07-01. PMID:8632213. |
the two groups which received both diets developed a similar degree of insulin resistance, equivalent to that after 16 wk of high amylopectin feeding. |
1996-07-01 |
2023-08-12 |
rat |
| C E Wiseman, J A Higgins, G S Denyer, J C Mille. Amylopectin starch induces nonreversible insulin resistance in rats. The Journal of nutrition. vol 126. issue 2. 1996-07-01. PMID:8632213. |
the findings suggest that amylopectin-induced insulin resistance cannot be reversed or prevented by either a subsequent or previous period of amylose feeding. |
1996-07-01 |
2023-08-12 |
rat |
| L J Slieker, K W Sloop, P L Surface, A Kriauciunas, F LaQuier, J Manetta, J Bue-Valleskey, T W Stephen. Regulation of expression of ob mRNA and protein by glucocorticoids and cAMP. The Journal of biological chemistry. vol 271. issue 10. 1996-06-20. PMID:8621378. |
it has been demonstrated that exogenous human ob protein (leptin) treatment reduces food intake and weight gain, as well as insulin, glucose, and corticosterone levels in ob/ob mice. |
1996-06-20 |
2023-08-12 |
mouse |
| J K Kim, J K Wi, J H You. Metabolic impairment precedes insulin resistance in skeletal muscle during high-fat feeding in rats. Diabetes. vol 45. issue 5. 1996-06-14. PMID:8621018. |
metabolic impairment precedes insulin resistance in skeletal muscle during high-fat feeding in rats. |
1996-06-14 |
2023-08-12 |
rat |
| J K Kim, J K Wi, J H You. Metabolic impairment precedes insulin resistance in skeletal muscle during high-fat feeding in rats. Diabetes. vol 45. issue 5. 1996-06-14. PMID:8621018. |
to examine whether impairment of intracellular glucose metabolism precedes insulin resistance, we determined the time courses of changes in insulin-stimulated glucose uptake, glycolysis, and glycogen synthesis during high-fat feeding in rats. |
1996-06-14 |
2023-08-12 |
rat |
| S C Woods, M Chavez, C R Park, C Riedy, K Kaiyala, R D Richardson, D P Figlewicz, M W Schwartz, D Porte, R J Seele. The evaluation of insulin as a metabolic signal influencing behavior via the brain. Neuroscience and biobehavioral reviews. vol 20. issue 1. 1996-06-14. PMID:8622820. |
finally, experimentally-induced changes of insulin in the brain cause predictable changes of food intake and body weight. |
1996-06-14 |
2023-08-12 |
Not clear |
| S C Woods, M Chavez, C R Park, C Riedy, K Kaiyala, R D Richardson, D P Figlewicz, M W Schwartz, D Porte, R J Seele. The evaluation of insulin as a metabolic signal influencing behavior via the brain. Neuroscience and biobehavioral reviews. vol 20. issue 1. 1996-06-14. PMID:8622820. |
given these observations, the question is then asked: since endogenous insulin, acting within the brain, appears to decrease food intake, can a decrease of food intake caused by exogenous insulin administered into the same area of the brain be ascribed to the same, naturally-occurring response system, or should it be attributed to malaise or a non-specific depression of behavior? |
1996-06-14 |
2023-08-12 |
Not clear |
| S C Woods, M Chavez, C R Park, C Riedy, K Kaiyala, R D Richardson, D P Figlewicz, M W Schwartz, D Porte, R J Seele. The evaluation of insulin as a metabolic signal influencing behavior via the brain. Neuroscience and biobehavioral reviews. vol 20. issue 1. 1996-06-14. PMID:8622820. |
arguments are presented supporting the former position that exogenous insulin, when administered in small quantities directly into the brain, taps into the natural caloric/metabolic system and hence influences food intake and body weight. |
1996-06-14 |
2023-08-12 |
Not clear |
| I Bergerot, N Fabien, A Mayer, C Thivole. Active suppression of diabetes after oral administration of insulin is determined by antigen dosage. Annals of the New York Academy of Sciences. vol 778. 1996-05-24. PMID:8610991. |
we have previously demonstrated that feeding six-week-old female mice with 20 units of human insulin every 2 - 3 days for 15 or 30 days induced an active mechanism of suppression through the generation of regulatory t cells that reduced the number of successful diabetic transfers in irradiated nod recipients. |
1996-05-24 |
2023-08-12 |
mouse |
| I Bergerot, N Fabien, A Mayer, C Thivole. Active suppression of diabetes after oral administration of insulin is determined by antigen dosage. Annals of the New York Academy of Sciences. vol 778. 1996-05-24. PMID:8610991. |
the effects of the dose of insulin feeding were therefore compared during cotransfer experiments of 5 x 10(6) t cells from diabetic mice and 5 x 10(6) t cells from the spleen of mice receiving 10 units, 20 units, or 40 units of insulin or saline every 2 - 3 days for 15 days. |
1996-05-24 |
2023-08-12 |
mouse |
| J A Higgins, J C Brand Miller, G S Denye. Development of insulin resistance in the rat is dependent on the rate of glucose absorption from the diet. The Journal of nutrition. vol 126. issue 3. 1996-04-25. PMID:8598543. |
amylopectin-fed animals became progressively insulin resistant from 12 to 26 wk of feeding. |
1996-04-25 |
2023-08-12 |
rat |