| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| D Darmau. Role of nutrients in the regulation of in vivo protein metabolism in humans. Acta paediatrica (Oslo, Norway : 1992). Supplement. vol 88. issue 433. 2000-01-27. PMID:10626554. |
whereas insulin may primarily suppress rates of proteolysis, amino acids are responsible for the stimulation of protein synthesis that follows food intake. |
2000-01-27 |
2023-08-12 |
Not clear |
| C D McMahon, L T Chapin, K J Lookingland, R P Radcliff, H A Tucke. Feeding-induced increases in insulin do not suppress secretion of growth hormone. Domestic animal endocrinology. vol 17. issue 4. 2000-01-27. PMID:10628433. |
we hypothesized that increased secretion of insulin after feeding inhibits release of gh from the anterior pituitary gland. |
2000-01-27 |
2023-08-12 |
Not clear |
| C D McMahon, L T Chapin, K J Lookingland, R P Radcliff, H A Tucke. Feeding-induced increases in insulin do not suppress secretion of growth hormone. Domestic animal endocrinology. vol 17. issue 4. 2000-01-27. PMID:10628433. |
our objectives were to determine whether: 1) alloxan prevents concentrations of insulin from increasing after feeding steers; 2) concentrations of gh remain high after feeding alloxan-treated steers; and 3) gh-releasing hormone (ghrh) stimulates greater release of gh in alloxan-treated, than in control, steers after feeding. |
2000-01-27 |
2023-08-12 |
Not clear |
| C D McMahon, L T Chapin, K J Lookingland, R P Radcliff, H A Tucke. Feeding-induced increases in insulin do not suppress secretion of growth hormone. Domestic animal endocrinology. vol 17. issue 4. 2000-01-27. PMID:10628433. |
concentrations of insulin were not different (p = 0.61) between control and alloxan-treated steers before feeding (87.5 +/- 33.6 pmol/l). |
2000-01-27 |
2023-08-12 |
Not clear |
| C D McMahon, L T Chapin, K J Lookingland, R P Radcliff, H A Tucke. Feeding-induced increases in insulin do not suppress secretion of growth hormone. Domestic animal endocrinology. vol 17. issue 4. 2000-01-27. PMID:10628433. |
however, alloxan prevented insulin from increasing (p < 0.001) after feeding (131.8 pmol/1) compared with control steers (442.0 pmol/l) (pooled sem = 47.5). |
2000-01-27 |
2023-08-12 |
Not clear |
| C D McMahon, L T Chapin, K J Lookingland, R P Radcliff, H A Tucke. Feeding-induced increases in insulin do not suppress secretion of growth hormone. Domestic animal endocrinology. vol 17. issue 4. 2000-01-27. PMID:10628433. |
we conclude that increased concentrations of insulin after feeding do not mediate feeding-induced suppression of gh secretion in steers. |
2000-01-27 |
2023-08-12 |
Not clear |
| C V Anuradha, S D Balakrishna. Taurine attenuates hypertension and improves insulin sensitivity in the fructose-fed rat, an animal model of insulin resistance. Canadian journal of physiology and pharmacology. vol 77. issue 10. 2000-01-11. PMID:10588478. |
fructose feeding induces moderate increases in blood pressure levels in normal rats, which is associated with hyperinsulinemia, insulin resistance, and impaired glucose tolerance. |
2000-01-11 |
2023-08-12 |
rat |
| M C Cam, B Rodrigues, J H McNeil. Distinct glucose lowering and beta cell protective effects of vanadium and food restriction in streptozotocin-diabetes. European journal of endocrinology. vol 141. issue 5. 1999-12-28. PMID:10576774. |
as food restriction has been reported to enhance insulin sensitivity and reduce insulin demand, we assessed the contribution of a reduced food intake to the glucose lowering and beta-cell protective effects of vanadium. |
1999-12-28 |
2023-08-12 |
rat |
| G E Demas, T J Bartnes. Effects of food deprivation and metabolic fuel utilization on food hoarding by jirds (Meriones shawi). Physiology & behavior. vol 67. issue 2. 1999-12-17. PMID:10477056. |
in the present study, the effects of food deprivation as well as alterations in metabolic fuel utilization (i.e., 2-deoxy-d-glucose and isophane insulin) on food hoarding and food intake were tested in female jirds using a simulated burrow system. |
1999-12-17 |
2023-08-12 |
Not clear |
| N Gletsu, W Dixon, M T Clandini. Insulin receptor at the mouse hepatocyte nucleus after a glucose meal induces dephosphorylation of a 30-kDa transcription factor and a concomitant increase in malic enzyme gene expression. The Journal of nutrition. vol 129. issue 12. 1999-12-17. PMID:10573543. |
insulin stimulation was achieved in vivo by oral glucose feeding of mice deprived of food for 24 h. hepatocytes were fractionated after the glucose meal and nuclei were purified. |
1999-12-17 |
2023-08-12 |
mouse |
| C R Pedersen, I Hagemann, T Bock, K Buschar. Intermittent feeding and fasting reduces diabetes incidence in BB rats. Autoimmunity. vol 30. issue 4. 1999-12-01. PMID:10524500. |
food intake may be one of several factors which influence the risk of development of insulin dependent diabetes mellitus, but the influence of the pattern of food supply has not been studied previously. |
1999-12-01 |
2023-08-12 |
rat |
| C Schubring, F Prohaska, A Prohaska, P Englaro, W Blum, T Siebler, J Kratzsch, W Kies. Leptin concentrations in maternal serum and amniotic fluid during the second trimenon: differential relation to fetal gender and maternal morphometry. European journal of obstetrics, gynecology, and reproductive biology. vol 86. issue 2. 1999-11-23. PMID:10509783. |
transient increase in obese gene expression after food intake or insulin administration. |
1999-11-23 |
2023-08-12 |
mouse |
| L C Pickavance, P S Widdowson, R G Vernon, G William. Neuropeptide Y receptor alterations in the hypothalamus of lactating rats. Peptides. vol 20. issue 9. 1999-11-22. PMID:10499422. |
however, reducing food intake by 35% had a more profound effect on npy receptor density in lactating than in control rats, producing down-regulation of non-y1 receptors in the ventromedial, dorsomedial, and perifornical lateral areas (all p < 0.05; n = 7/group) and reduction of plasma insulin and leptin levels (both p < 0.01). |
1999-11-22 |
2023-08-12 |
rat |
| K J Kaiyala, R L Prigeon, S E Kahn, S C Woods, D Porte, M W Schwart. Reduced beta-cell function contributes to impaired glucose tolerance in dogs made obese by high-fat feeding. The American journal of physiology. vol 277. issue 4. 1999-11-22. PMID:10516125. |
because high-fat feeding reduces both insulin sensitivity and glucose tolerance, we hypothesized that it also reduces beta-cell compensation. |
1999-11-22 |
2023-08-12 |
Not clear |
| K J Kaiyala, R L Prigeon, S E Kahn, S C Woods, D Porte, M W Schwart. Reduced beta-cell function contributes to impaired glucose tolerance in dogs made obese by high-fat feeding. The American journal of physiology. vol 277. issue 4. 1999-11-22. PMID:10516125. |
to test this hypothesis, we used intravenous glucose tolerance testing with minimal model analysis to measure glucose tolerance (k(g)), insulin sensitivity (s(i)), and the acute insulin response to glucose (air(g)) in nine dogs fed a chow diet and again after 7 wk of high-fat feeding. |
1999-11-22 |
2023-08-12 |
Not clear |
| K J Kaiyala, R L Prigeon, S E Kahn, S C Woods, D Porte, M W Schwart. Reduced beta-cell function contributes to impaired glucose tolerance in dogs made obese by high-fat feeding. The American journal of physiology. vol 277. issue 4. 1999-11-22. PMID:10516125. |
high-fat feeding also elicited a 44% lower 24-h insulin level (p = 0.004) in association with an 8% reduction of glucose (p = 0.0003). |
1999-11-22 |
2023-08-12 |
Not clear |
| K J Kaiyala, R L Prigeon, S E Kahn, S C Woods, D Porte, M W Schwart. Reduced beta-cell function contributes to impaired glucose tolerance in dogs made obese by high-fat feeding. The American journal of physiology. vol 277. issue 4. 1999-11-22. PMID:10516125. |
we conclude that high-fat feeding causes insulin resistance that is not compensated for by increased insulin secretion and that this contributes to the development of glucose intolerance. |
1999-11-22 |
2023-08-12 |
Not clear |
| T H Elsasser, J L Sartin, A Martínez, S Kahl, L Montuenga, R Pío, R Fayer, M J Miller, F Cuttitt. Underlying disease stress augments plasma and tissue adrenomedullin (AM) responses to endotoxin: colocalized increases in AM and inducible nitric oxide synthase within pancreatic islets. Endocrinology. vol 140. issue 11. 1999-11-22. PMID:10537172. |
these acute complications rapidly progress into a more chronic state characterized by diminished insulin response to feeding stimulus and colocalized increases in pancreatic islet am and inos. |
1999-11-22 |
2023-08-12 |
Not clear |
| C Ploix, I Bergerot, A Durand, C Czerkinsky, J Holmgren, C Thivole. Oral administration of cholera toxin B-insulin conjugates protects NOD mice from autoimmune diabetes by inducing CD4+ regulatory T-cells. Diabetes. vol 48. issue 11. 1999-11-19. PMID:10535448. |
to optimize the induction of tolerance, we have shown that feeding insulin conjugated to cholera toxin b-subunit (ctb), a potent mucosal adjuvant, reduced by 5,000 the amounts of antigen necessary for delaying diabetes onset in nod mice. |
1999-11-19 |
2023-08-12 |
mouse |
| J W Blum, H Hammo. Endocrine and metabolic aspects in milk-fed calves. Domestic animal endocrinology. vol 17. issue 2-3. 1999-11-17. PMID:10527125. |
metabolic and endocrine changes, such as insulin resistance and disturbed glucose metabolism, can be observed in part in association with high feeding intensity in veal calves. |
1999-11-17 |
2023-08-12 |
Not clear |