All Relations between apoptosis and spinal cord

Publication Sentence Publish Date Extraction Date Species
Kiran Kumar Soni, Jinsu Hwang, Mahesh Ramalingam, Choonghyo Kim, Byeong C Kim, Han-Seong Jeong, Sujeong Jan. Endoplasmic Reticulum Stress Causing Apoptosis in a Mouse Model of an Ischemic Spinal Cord Injury. International journal of molecular sciences. vol 24. issue 2. 2023-01-21. PMID:36674822. in conclusion, an isci increases the er stress proteins, resulting in apoptosis in neuronal cells in the spinal cord. 2023-01-21 2023-08-14 mouse
Yu Tang, Zonghan Xu, Jincheng Tang, Yichang Xu, Ziang Li, Wenbo Wang, Liang Wu, Kun Xi, Yong Gu, Liang Che. Architecture-engineered electrospinning cascade regulates spinal microenvironment to promote nerve regeneration. Advanced healthcare materials. 2023-01-18. PMID:36652529. the inflammatory cascade after spinal cord injury (sci) causes necrotizing apoptosis of local stem cells, which limits nerve regeneration. 2023-01-18 2023-08-14 rat
Yanming Liu, Feiyue Zhang, Qi Sun, Lichao Lian. Adalimumab combined with erythropoietin improves recovery from spinal cord injury by suppressing microglial M1 polarization-mediated neural inflammation and apoptosis. Inflammopharmacology. 2023-01-15. PMID:36642757. adalimumab combined with erythropoietin improves recovery from spinal cord injury by suppressing microglial m1 polarization-mediated neural inflammation and apoptosis. 2023-01-15 2023-08-14 Not clear
Ying Chen, Yan Dong, Zai-Li Zhang, Jie Han, Feng-Shou Chen, Xiang-Yi Tong, Hong M. Fra-1 induces apoptosis and neuroinflammation by targeting S100A8 to modulate TLR4 pathways in spinal cord ischemia/reperfusion injury. Brain pathology (Zurich, Switzerland). vol 33. issue 1. 2023-01-12. PMID:36634215. fra-1 induces apoptosis and neuroinflammation by targeting s100a8 to modulate tlr4 pathways in spinal cord ischemia/reperfusion injury. 2023-01-12 2023-08-14 rat
Ying Chen, Yan Dong, Zai-Li Zhang, Jie Han, Feng-Shou Chen, Xiang-Yi Tong, Hong M. Fra-1 induces apoptosis and neuroinflammation by targeting S100A8 to modulate TLR4 pathways in spinal cord ischemia/reperfusion injury. Brain pathology (Zurich, Switzerland). vol 33. issue 1. 2023-01-12. PMID:36634215. spinal cord ischemia/reperfusion injury (scii) is a severe complication driven by apoptosis and neuroinflammation. 2023-01-12 2023-08-14 rat
Shining Xiao, Yu Zhang, Zihao Liu, Anan Li, Weilai Tong, Xu Xiong, Jiangbo Nie, Nanshan Zhong, Guoqing Zhu, Jiaming Liu, Zhili Li. Alpinetin inhibits neuroinflammation and neuronal apoptosis via targeting the JAK2/STAT3 signaling pathway in spinal cord injury. CNS neuroscience & therapeutics. 2023-01-11. PMID:36627822. alpinetin inhibits neuroinflammation and neuronal apoptosis via targeting the jak2/stat3 signaling pathway in spinal cord injury. 2023-01-11 2023-08-14 Not clear
Majid Keyhanifard, Helia Helali, Marjan Gholami, Mitra Akbari, Melody Omraninava, Hossein Mohammad. Quercetin in combination with hyperbaric oxygen therapy synergistically attenuates damage progression in traumatic spinal cord injury in a rat model. Journal of chemical neuroanatomy. 2023-01-10. PMID:36627061. oxidative stress, inflammation and cell apoptosis are the most important destructive factors in the spread of damage following trauma to the spinal cord. 2023-01-10 2023-08-14 rat
Fuchuang Qin, Guorong He, Yu Sun, Guangning Chen, Qijian Yu, Xilie M. Overexpression of long non-coding RNA LINC00158 inhibits neuronal apoptosis by promoting autophagy in spinal cord injury. The Chinese journal of physiology. vol 65. issue 6. 2023-01-02. PMID:36588354. overexpression of long non-coding rna linc00158 inhibits neuronal apoptosis by promoting autophagy in spinal cord injury. 2023-01-02 2023-08-14 rat
Fuchuang Qin, Guorong He, Yu Sun, Guangning Chen, Qijian Yu, Xilie M. Overexpression of long non-coding RNA LINC00158 inhibits neuronal apoptosis by promoting autophagy in spinal cord injury. The Chinese journal of physiology. vol 65. issue 6. 2023-01-02. PMID:36588354. after ogd treatment, the expression of linc00158 decreased significantly and the apoptosis of spinal cord neurons increased. 2023-01-02 2023-08-14 rat
Fuchuang Qin, Guorong He, Yu Sun, Guangning Chen, Qijian Yu, Xilie M. Overexpression of long non-coding RNA LINC00158 inhibits neuronal apoptosis by promoting autophagy in spinal cord injury. The Chinese journal of physiology. vol 65. issue 6. 2023-01-02. PMID:36588354. 3-methyladenine and chloroquine (autophagy inhibitor) reversed the inhibitory effect of linc00158 overexpression on apoptosis of primary spinal cord neurons. 2023-01-02 2023-08-14 rat
Chuan Qin, Yi Liu, Pei-Pei Xu, Xin Zhang, Zuliyaer Talifu, Jia-Yi Liu, Ying-Li Jing, Fan Bai, Li-Xi Zhao, Yan Yu, Feng Gao, Jian-Jun L. Inhibition by rno-circRNA-013017 of the apoptosis of motor neurons in anterior horn and descending axonal degeneration in rats after traumatic spinal cord injury. Frontiers in neuroscience. vol 16. 2023-01-02. PMID:36590290. inhibition by rno-circrna-013017 of the apoptosis of motor neurons in anterior horn and descending axonal degeneration in rats after traumatic spinal cord injury. 2023-01-02 2023-08-14 rat
Fang Yang, Wen-Zhi Chen, Shi-Shi Jiang, Xiao-Hua Wang, Ren-Shi X. A candidate protective factor in amyotrophic lateral sclerosis: heterogenous nuclear ribonucleoprotein G. Neural regeneration research. vol 18. issue 7. 2022-12-26. PMID:36571358. in this study, we studied the potential mechanism of heterogenous nuclear ribonucleoprotein g in neuronal death in the spinal cord of tg and wild-type mice and examined the mechanism by which heterogenous nuclear ribonucleoprotein g induces apoptosis. 2022-12-26 2023-08-14 mouse
Fang Yang, Wen-Zhi Chen, Shi-Shi Jiang, Xiao-Hua Wang, Ren-Shi X. A candidate protective factor in amyotrophic lateral sclerosis: heterogenous nuclear ribonucleoprotein G. Neural regeneration research. vol 18. issue 7. 2022-12-26. PMID:36571358. we analyzed heterogenous nuclear ribonucleoprotein g distribution in spinal cord in the amyotrophic lateral sclerosis model at various time points and the expressions of apoptosis and proliferation-related proteins. 2022-12-26 2023-08-14 mouse
Xuegang He, Xudong Guo, Bo Deng, Jihe Kang, Wenzhao Liu, Guangzhi Zhang, Yonggang Wang, Yong Yang, Xuewen Kan. HSPA1A ameliorated spinal cord injury in rats by inhibiting apoptosis to exert neuroprotective effects. Experimental neurology. 2022-12-20. PMID:36538982. hspa1a ameliorated spinal cord injury in rats by inhibiting apoptosis to exert neuroprotective effects. 2022-12-20 2023-08-14 rat
Xuegang He, Xudong Guo, Bo Deng, Jihe Kang, Wenzhao Liu, Guangzhi Zhang, Yonggang Wang, Yong Yang, Xuewen Kan. HSPA1A ameliorated spinal cord injury in rats by inhibiting apoptosis to exert neuroprotective effects. Experimental neurology. 2022-12-20. PMID:36538982. traumatic spinal cord injury (tsci) is a serious nervous system insult, and apoptosis in secondary injury is an important barrier to recovery from tsci. 2022-12-20 2023-08-14 rat
Xuegang He, Xudong Guo, Bo Deng, Jihe Kang, Wenzhao Liu, Guangzhi Zhang, Yonggang Wang, Yong Yang, Xuewen Kan. HSPA1A ameliorated spinal cord injury in rats by inhibiting apoptosis to exert neuroprotective effects. Experimental neurology. 2022-12-20. PMID:36538982. however, whether hspa1a can inhibit apoptosis after spinal cord injury, and the potential mechanism of this inhibition, remain unclear. 2022-12-20 2023-08-14 rat
Ye Bai, Ning Guo, Zhanwu Xu, Yuxi Chen, Wenjin Zhang, Qinghe Chen, Zhenggang B. S100A1 expression is increased in spinal cord injury and promotes inflammation, oxidative stress and apoptosis of PC12 cells induced by LPS via ERK signaling. Molecular medicine reports. vol 27. issue 2. 2022-12-16. PMID:36524376. s100a1 expression is increased in spinal cord injury and promotes inflammation, oxidative stress and apoptosis of pc12 cells induced by lps via erk signaling. 2022-12-16 2023-08-14 rat
Riyun Yang, Jingying Pan, Yankai Wang, Panhui Xia, Mingliang Tai, Zhihao Jiang, Gang Che. Application and prospects of somatic cell reprogramming technology for spinal cord injury treatment. Frontiers in cellular neuroscience. vol 16. 2022-12-05. PMID:36467604. after sci, many neurons in the injured area die due to necrosis or apoptosis, and astrocytes, oligodendrocytes, microglia and other non-neuronal cells become dysfunctional, hindering the repair of the injured spinal cord. 2022-12-05 2023-08-14 Not clear
Mingdong Li, Hasiqiqige, Yanqiang Huan, Xiaolei Wang, Mingkai Tao, Tianqi Jiang, Hongbin Xie, Wu Jisiguleng, Wei Xing, Zhibo Zhu, Aitao Wang, Yongxiong H. Calycosin ameliorates spinal cord injury by targeting Hsp90 to inhibit oxidative stress and apoptosis of nerve cells. Journal of chemical neuroanatomy. 2022-11-19. PMID:36402284. calycosin ameliorates spinal cord injury by targeting hsp90 to inhibit oxidative stress and apoptosis of nerve cells. 2022-11-19 2023-08-14 Not clear
Taotao Lin, Yujie Zhao, Shengyu Guo, Zhengru Wu, Wenwen Li, Rongcan Wu, Zhenyu Wang, Wenge Li. Apelin-13 Protects Neurons by Attenuating Early-Stage Postspinal Cord Injury Apoptosis In Vitro. Brain sciences. vol 12. issue 11. 2022-11-11. PMID:36358441. this research identified and verified the differential expression of apelin in rat spinal cord injured tissues and normal spinal cord tissues by transcriptome sequencing in vivo and proved that apelin-13 protects neurons by strengthening autophagy and attenuating early-stage postspinal cord injury apoptosis in vitro. 2022-11-11 2023-08-14 rat