| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Liang Shi, Liang Duan, Dapeng Duan, Honghai Xu, Xiaoming Li, Wei Zha. Long non-coding RNA DANCR increases spinal cord neuron apoptosis and inflammation of spinal cord injury by mediating the microRNA-146a-5p/MAPK6 axis. European spine journal : official publication of the European Spine Society, the European Spinal Deformity Society, and the European Section of the Cervical Spine Research Society. 2024-03-29. PMID:38551688. |
long non-coding rna dancr increases spinal cord neuron apoptosis and inflammation of spinal cord injury by mediating the microrna-146a-5p/mapk6 axis. |
2024-03-29 |
2024-03-31 |
Not clear |
| Jie Zeng, Wei-Wei Gao, Hao Yang, Ya-Nang Wang, Yang Mei, Ting-Ting Liu, Min Wang, Li Tang, Dong-Chuan Ma, Wei L. Sodium tanshinone IIA sulfonate suppresses microglia polarization and neuroinflammation possibly via regulating miR-125b-5p/STAT3 axis to ameliorate neuropathic pain. European journal of pharmacology. 2024-03-29. PMID:38552937. |
after treatment with sts, the mechanical thresholds, expression of mir-125b-5p, and microglial m2 marker such as arg-1 in the spinal cord horn increased significantly, whereas multiple pro-inflammatory cytokines and apoptosis were significantly reduced. |
2024-03-29 |
2024-04-01 |
rat |
| Yiru Wang, Zhi Chen, Qingquan Liu, Yongman L. LncTCONS_00058568 is involved in the pathophysiologic processes mediated by P2X7R in the lower thoracic spinal cord after acute kidney injury. FASEB journal : official publication of the Federation of American Societies for Experimental Biology. vol 38. issue 6. 2024-03-18. PMID:38498358. |
in the present study, we found that the upregulated expression of lnctcons_00058568 in lower thoracic spinal cord significantly ameliorated aki-induced renal tissue injury, kidney morphology, inflammation and apoptosis, and suppressed renal sympathetic nerve activity. |
2024-03-18 |
2024-03-21 |
rat |
| Jinpeng Gao, ShuRui Chen, Sen Lin, Xifan Me. Prussian blue analogues improves the microenvironment after spinal cord injury by regulating Zn. International immunopharmacology. vol 131. 2024-03-17. PMID:38493692. |
mitochondrial injury, neuronal apoptosis and phenotypic transformation of macrophages are the main mechanisms of spinal cord injury. |
2024-03-17 |
2024-03-20 |
Not clear |
| Jinpeng Gao, ShuRui Chen, Sen Lin, Xifan Me. Prussian blue analogues improves the microenvironment after spinal cord injury by regulating Zn. International immunopharmacology. vol 131. 2024-03-17. PMID:38493692. |
in vivo experiments, it shows that prussian blue zirconium can improve inflammation, apoptosis and oxidative stress of spinal cord tissue, promote regenerative therapy after spinal cord injury, and improve motor function. |
2024-03-17 |
2024-03-20 |
Not clear |
| Rui-Guang Zhang, Bo-Wen Zheng, Jing Zhang, Ming-Yu Hao, Yu-Hang Diao, Xiao- Jun Hu, Xuan-Hui Liu, Tao Zhu, Zi-Long Zhao, Hong-Tao Ron. Spinal lymphatic dysfunction aggravates the recovery process after spinal cord injury. Neuroscience. 2024-03-09. PMID:38460904. |
the spinal cord edema was evaluated by dry and wet weight.terminal deoxynucleotidyl transferase dutp nick-end labeling (tunel) assay was used to evaluate apoptosis after spinal cord injury. |
2024-03-09 |
2024-03-12 |
Not clear |
| Weijun Huang, Depeng Wu, Chaoyang Cai, Hui Yao, Zhenming Tian, Yang Yang, Mao Pang, Limin Rong, Bin Li. Inhibition of MST1 ameliorates neuronal apoptosis via GSK3β/β-TrCP/NRF2 pathway in spinal cord injury accompanied by diabetes. Redox biology. vol 71. 2024-03-02. PMID:38430683. |
inhibition of mst1 ameliorates neuronal apoptosis via gsk3β/β-trcp/nrf2 pathway in spinal cord injury accompanied by diabetes. |
2024-03-02 |
2024-03-06 |
Not clear |
| Shuangmei Liu, Huali Liu, Chunyan Gong, Guiliang Li, Qiaofen Li, Zhipeng Pan, Xiaona He, Zhilv Jiang, Heng Li, Chunjun Zhan. MiR-10b-5p regulates neuronal autophagy and apoptosis induced by spinal cord injury through UBR7. Neuroscience. 2024-02-21. PMID:38382692. |
mir-10b-5p regulates neuronal autophagy and apoptosis induced by spinal cord injury through ubr7. |
2024-02-21 |
2024-02-24 |
rat |
| Shuangmei Liu, Huali Liu, Chunyan Gong, Guiliang Li, Qiaofen Li, Zhipeng Pan, Xiaona He, Zhilv Jiang, Heng Li, Chunjun Zhan. MiR-10b-5p regulates neuronal autophagy and apoptosis induced by spinal cord injury through UBR7. Neuroscience. 2024-02-21. PMID:38382692. |
this study aimed to explore the effects of mir-10b-5p on autophagy and apoptosis in neuronal cells after spinal cord injury (sci) and the molecular mechanism. |
2024-02-21 |
2024-02-24 |
rat |
| Shuangmei Liu, Huali Liu, Chunyan Gong, Guiliang Li, Qiaofen Li, Zhipeng Pan, Xiaona He, Zhilv Jiang, Heng Li, Chunjun Zhan. MiR-10b-5p regulates neuronal autophagy and apoptosis induced by spinal cord injury through UBR7. Neuroscience. 2024-02-21. PMID:38382692. |
treatment with the mir-10b-5p mimic effectively improved motor function, inhibited neuronal cell apoptosis, and promoted spinal cord tissue repair in sci rats. |
2024-02-21 |
2024-02-24 |
rat |
| Wei Mao, Feng Jiang, Chunping Zhu, Jun Liu, Zhao Lu, Yinwei Qian, Jinchun Xia. Effect of CTLA-4 Inhibition on Inflammation and Apoptosis After Spinal Cord Injury. Neurochemical research. 2024-02-17. PMID:38366208. |
effect of ctla-4 inhibition on inflammation and apoptosis after spinal cord injury. |
2024-02-17 |
2024-02-19 |
rat |
| Wei Mao, Feng Jiang, Chunping Zhu, Jun Liu, Zhao Lu, Yinwei Qian, Jinchun Xia. Effect of CTLA-4 Inhibition on Inflammation and Apoptosis After Spinal Cord Injury. Neurochemical research. 2024-02-17. PMID:38366208. |
spinal cord injury (sci) encompasses various pathological processes, notably neuroinflammation and apoptosis, both of which play significant roles. |
2024-02-17 |
2024-02-19 |
rat |
| Cai Wang, Chengcheng Wang, Weizhao Lu, Yanjiao Wang, Qianwen Yue, Dongyuan Xin, Baoliang Sun, Jingguo Wu, Jingyi Sun, Ying Wan. Novel SERS Signal Amplification Strategy for Ultrasensitive and Specific Detection of Spinal Cord Injury-Related miRNA. ACS sensors. 2024-02-12. PMID:38346401. |
spinal cord injury (sci) is a serious disease of the central nervous system, accompanied by inflammation, cell apoptosis, neuronal necrosis, axonal rupture, demyelination, and other pathological processes, resulting in impaired sensory and motor functions of patients. |
2024-02-12 |
2024-02-15 |
Not clear |
| Longyu Zhang, Jiao Xu, Shi Yin, Qiang Wang, Zhiwei Jia, Tianlin We. Albiflorin Attenuates Neuroinflammation and Improves Functional Recovery After Spinal Cord Injury Through Regulating LSD1-Mediated Microglial Activation and Ferroptosis. Inflammation. 2024-02-10. PMID:38340239. |
meanwhile, af attenuated neuron apoptosis and microglia activation, reduced the production of pro-inflammatory cytokines and iron accumulation, and inhibited spinal cord ferroptosis following sci in rats. |
2024-02-10 |
2024-02-14 |
rat |
| Qinyuan Yang, Linxuan Tian, Weiwei Wang, Xiong Chen, Junyan Ta. Post-fertilization 2-ethylhexyl-4-methoxycinnamate (EHMC) exposure affects axonal growth, muscle fiber length, and motor behavior in zebrafish embryos. Ecotoxicology and environmental safety. vol 272. 2024-02-02. PMID:38306815. |
moreover, ehmc exposure induced excessive cell apoptosis in the head and spinal cord regions, increased the production of reactive oxygen species (ros) and malondialdehyde (mda), and reduced the level of glutathione (gsh). |
2024-02-02 |
2024-02-05 |
zebrafish |
| W-L Gong, Y-N Zhang, J Zhang, X Jiang, W-J Xia. Biological differences and molecular mechanism of different peripheral nerves after injury. European review for medical and pharmacological sciences. vol 28. issue 2. 2024-02-02. PMID:38305620. |
the aim of the study was to analyze the apoptosis of neurons and the differences in expression of bcl-2 and bax protein in the neurons in the corresponding spinal cord segment after the repair of the tibial nerve (tn) and common peroneal nerve (cpn) in rats. |
2024-02-02 |
2024-02-04 |
rat |
| Siqiao Wang, Liming Chen. The role of apoptosis in spinal cord injury: a bibliometric analysis from 1994 to 2023. Frontiers in cellular neuroscience. vol 17. 2024-01-31. PMID:38293650. |
the role of apoptosis in spinal cord injury: a bibliometric analysis from 1994 to 2023. |
2024-01-31 |
2024-02-02 |
Not clear |
| Siqiao Wang, Liming Chen. The role of apoptosis in spinal cord injury: a bibliometric analysis from 1994 to 2023. Frontiers in cellular neuroscience. vol 17. 2024-01-31. PMID:38293650. |
apoptosis after spinal cord injury (sci) plays a pivotal role in the secondary injury mechanisms, which cause the ultimate neurologic insults. |
2024-01-31 |
2024-02-02 |
Not clear |
| Dasheng Tian, Xingyu You, Jianan Ye, Gan Chen, Hang Yu, Jianwei Lv, Fangli Shan, Chao Liang, Yihui Bi, Juehua Jing, Meige Zhen. hBcl2 overexpression in BMSCs enhances resistance to myelin debris-induced apoptosis and facilitates neuroprotection after spinal cord injury in rats. Scientific reports. vol 14. issue 1. 2024-01-21. PMID:38246980. |
hbcl2 overexpression in bmscs enhances resistance to myelin debris-induced apoptosis and facilitates neuroprotection after spinal cord injury in rats. |
2024-01-21 |
2024-01-24 |
human |
| Xigong Li, Jing Fu, Ming Guan, Haifei Shi, Wenming Pan, Xianfeng Lo. Biochanin A attenuates spinal cord injury in rats during early stages by inhibiting oxidative stress and inflammasome activation. Neural regeneration research. vol 19. issue 9. 2024-01-16. PMID:38227535. |
the results showed that biochanin a effectively alleviated spinal cord neuronal injury and spinal cord tissue injury, reduced inflammation and oxidative stress in spinal cord neurons, and reduced apoptosis and pyroptosis. |
2024-01-16 |
2024-01-18 |
rat |