| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| R A Baiocchi, V P Khatri, M J Lindemann, M E Ross, G Papoff, A J Caprio, T V Caprio, R Fenstermaker, G Ruberti, Z P Bernstein, M A Caligiur. Phenotypic and functional analysis of Fas (CD95) expression in primary central nervous system lymphoma of patients with acquired immunodeficiency syndrome. Blood. vol 90. issue 5. 1997-09-30. PMID:9292506. |
no evidence for intrinsic resistance to fas-mediated apoptosis (ie, secreted or truncated forms of fas) could be shown. |
1997-09-30 |
2023-08-12 |
mouse |
| R A Baiocchi, V P Khatri, M J Lindemann, M E Ross, G Papoff, A J Caprio, T V Caprio, R Fenstermaker, G Ruberti, Z P Bernstein, M A Caligiur. Phenotypic and functional analysis of Fas (CD95) expression in primary central nervous system lymphoma of patients with acquired immunodeficiency syndrome. Blood. vol 90. issue 5. 1997-09-30. PMID:9292506. |
radiation-induced apoptosis of neoplastic ebv+ b cells was enhanced by activation of fas, and prolonged exposure to interleukin-2 increased both fas expression and fas-induced apoptosis. |
1997-09-30 |
2023-08-12 |
mouse |
| K J Sung, E M Paik, K A Jang, H S Suh, J H Cho. Apoptosis is induced by anti-Fas antibody alone in cultured human keratinocytes. The Journal of dermatology. vol 24. issue 7. 1997-09-29. PMID:9267102. |
fas is a well-known cell surface receptor whose main function is the induction of apoptosis in many cell types including human keratinocytes. |
1997-09-29 |
2023-08-12 |
human |
| K J Sung, E M Paik, K A Jang, H S Suh, J H Cho. Apoptosis is induced by anti-Fas antibody alone in cultured human keratinocytes. The Journal of dermatology. vol 24. issue 7. 1997-09-29. PMID:9267102. |
because ifn gamma is synthesized by activated t cells, but not by keratinocytes, these results suggest that fas may only be effective in apoptosis occurring in t-cell mediated inflammatory skin diseases. |
1997-09-29 |
2023-08-12 |
human |
| K J Sung, E M Paik, K A Jang, H S Suh, J H Cho. Apoptosis is induced by anti-Fas antibody alone in cultured human keratinocytes. The Journal of dermatology. vol 24. issue 7. 1997-09-29. PMID:9267102. |
we hypothesized that fas alone might mediate apoptosis in normal human keratinocytes without any other help and thus play a role in normal epidermal homeostasis. |
1997-09-29 |
2023-08-12 |
human |
| A Imai, S Horibe, A Takagi, T Ohno, T Tamay. Frequent expression of Fas in gonadotropin-releasing hormone receptor-bearing tumors. European journal of obstetrics, gynecology, and reproductive biology. vol 74. issue 1. 1997-09-24. PMID:9243207. |
fas, a cell surface receptor, mediates cell death by means of apoptosis in a variety of cell types. |
1997-09-24 |
2023-08-12 |
Not clear |
| J Estaquier, J C Ameise. A role for T-helper type-1 and type-2 cytokines in the regulation of human monocyte apoptosis. Blood. vol 90. issue 4. 1997-09-24. PMID:9269781. |
th1 cytokines reduced monocyte apoptosis induced by several stimuli: il-2 reduced apoptosis induced by either il-10 or high concentrations of ifn-gamma, il-12 reduced apoptosis induced by either the ligation of the fas (cd95) molecule or gamma-irradiation, and ifn-gamma (at low doses that did not trigger apoptosis) reduced apoptosis induced by gamma-irradiation. |
1997-09-24 |
2023-08-12 |
human |
| J K Wang, B Zhu, S T Ju, J Tschopp, A Marshak-Rothstei. CD4+ T cells reactivated with superantigen are both more sensitive to FasL-mediated killing and express a higher level of FasL. Cellular immunology. vol 179. issue 2. 1997-09-19. PMID:9268499. |
to determine whether the dissimilarity between the 1 degrees and 2 degrees cultures could be attributed to differences in fasl cytotoxic activity or in the sensitivity of the fas apoptosis signaling pathway, we compared these parameters during the 1 degrees and 2 degrees responses of lpr and gld cd4+ t cells (which do not undergo aicd due to a lack of fas and an inactive fasl, respectively) so that each parameter could be evaluated independently. |
1997-09-19 |
2023-08-12 |
Not clear |
| L E French, A Wilson, M Hahne, I Viard, J Tschopp, H R MacDonal. Fas ligand expression is restricted to nonlymphoid thymic components in situ. Journal of immunology (Baltimore, Md. : 1950). vol 159. issue 5. 1997-09-19. PMID:9278307. |
the cell surface receptor fas (apo-1/cd95) and its ligand (fasl) are mediators of apoptosis that have been shown to be implicated in activation-induced death of mature t cells and in killing mediated by cytolytic t cells. |
1997-09-19 |
2023-08-12 |
Not clear |
| L E French, A Wilson, M Hahne, I Viard, J Tschopp, H R MacDonal. Fas ligand expression is restricted to nonlymphoid thymic components in situ. Journal of immunology (Baltimore, Md. : 1950). vol 159. issue 5. 1997-09-19. PMID:9278307. |
the role of the fas pathway in apoptosis associated with thymic selection events is, however, controversial. |
1997-09-19 |
2023-08-12 |
Not clear |
| L E French, A Wilson, M Hahne, I Viard, J Tschopp, H R MacDonal. Fas ligand expression is restricted to nonlymphoid thymic components in situ. Journal of immunology (Baltimore, Md. : 1950). vol 159. issue 5. 1997-09-19. PMID:9278307. |
the coexistence of fasl-expressing stromal cells and fas-expressing thymocytes may have important implications for the role of the fas pathway in apoptosis associated with thymic selection events. |
1997-09-19 |
2023-08-12 |
Not clear |
| D Balomenos, R Rumold, A N Theofilopoulo. The proliferative in vivo activities of lpr double-negative T cells and the primary role of p59fyn in their activation and expansion. Journal of immunology (Baltimore, Md. : 1950). vol 159. issue 5. 1997-09-19. PMID:9278315. |
we conclude that 1) dn lpr t cells, rather than being end stage, retain the capacity to be activated and repeatedly divide before reaching an anergic or replicative senescence stage; 2) the cd3 zeta-chain-associated fyn kinase is important to dn t cell signal transduction; 3) dn cells, as functional intermediates of the cd8+ subset, are highly dependent on fas participation for apoptosis; and 4) dn cells contribute to the early development of the serologic and histologic features of the mrl-lpr lupus disease. |
1997-09-19 |
2023-08-12 |
mouse |
| M Okamoto, M Makino, I Kitajima, I Maruyama, M Bab. HIV-1-infected myelomonocytic cells are resistant to Fas-mediated apoptosis: effect of tumor necrosis factor-alpha on their Fas expression and apoptosis. Medical microbiology and immunology. vol 186. issue 1. 1997-09-18. PMID:9255761. |
hiv-1-infected myelomonocytic cells are resistant to fas-mediated apoptosis: effect of tumor necrosis factor-alpha on their fas expression and apoptosis. |
1997-09-18 |
2023-08-12 |
Not clear |
| M Okamoto, M Makino, I Kitajima, I Maruyama, M Bab. HIV-1-infected myelomonocytic cells are resistant to Fas-mediated apoptosis: effect of tumor necrosis factor-alpha on their Fas expression and apoptosis. Medical microbiology and immunology. vol 186. issue 1. 1997-09-18. PMID:9255761. |
to get insight into the involvement of tumor necrosis factor-alpha (tnf-alpha) and fas (cd95) ligand in apoptosis (programmed cell death) of monocyte/macrophages in hiv-1-infected individuals, various t cell and myelomonocytic cell lines, including the hiv-1-infected clones om-10.1 and u1 cells, were cultured in the presence of either tnf-alpha alone, anti-fas agonist monoclonal antibody (fas-mab) alone, or their combinations. |
1997-09-18 |
2023-08-12 |
Not clear |
| M Okamoto, M Makino, I Kitajima, I Maruyama, M Bab. HIV-1-infected myelomonocytic cells are resistant to Fas-mediated apoptosis: effect of tumor necrosis factor-alpha on their Fas expression and apoptosis. Medical microbiology and immunology. vol 186. issue 1. 1997-09-18. PMID:9255761. |
however, they would become sensitive to the apoptosis if the expression of fas could be up-regulated by tnf-alpha or other factors. |
1997-09-18 |
2023-08-12 |
Not clear |
| C M Eischen, P J Leibso. The Fas pathway in apoptosis. Advances in pharmacology (San Diego, Calif.). vol 41. 1997-09-17. PMID:9204143. |
the fas pathway in apoptosis. |
1997-09-17 |
2023-08-12 |
Not clear |
| S Hirose, K Yan, M Abe, Y Jiang, Y Hamano, H Tsurui, T Shira. Precursor B cells for autoantibody production in genomically Fas-intact autoimmune disease are not subject to Fas-mediated immune elimination. Proceedings of the National Academy of Sciences of the United States of America. vol 94. issue 17. 1997-09-17. PMID:9256475. |
while splenic b cells from 2-month-old mice before overt sle expressed fas poorly, in vitro stimulation with an agonistic anti-cd40 mab up-regulated their fas expression, thus revealing the existence of two populations: one was fashigh and highly susceptible to anti-fas mab-induced apoptosis, and the other was faslow and apoptosis-resistant. |
1997-09-17 |
2023-08-12 |
mouse |
| C M Eischen, P J Leibso. Role for NK-cell-associated Fas ligand in cell-mediated cytotoxicity and apoptosis. Research in immunology. vol 148. issue 3. 1997-09-16. PMID:9255868. |
role for nk-cell-associated fas ligand in cell-mediated cytotoxicity and apoptosis. |
1997-09-16 |
2023-08-12 |
Not clear |
| S M Srinivasula, M Ahmad, S Ottilie, F Bullrich, S Banks, Y Wang, T Fernandes-Alnemri, C M Croce, G Litwack, K J Tomaselli, R C Armstrong, E S Alnemr. FLAME-1, a novel FADD-like anti-apoptotic molecule that regulates Fas/TNFR1-induced apoptosis. The Journal of biological chemistry. vol 272. issue 30. 1997-09-09. PMID:9228018. |
interestingly, flame-1 is recruited to the fas receptor complex and can abrogate fas/tnfr-induced apoptosis upon expression in fasl/tumor necrosis factor-sensitive mcf-7 cells, possibly by acting as a dominant-negative inhibitor. |
1997-09-09 |
2023-08-12 |
human |
| G Tai, J Eun-Young, H Yuji, K Masahiko, H Toshio, K Kenji, F Kenshi, M Mitsufum. Different effects of cyclic AMP and butyrate on eosinophilic differentiation, apoptosis and bcl-2 expression of a human eosinophilic leukemia cell line, EoL-1. Hematological oncology. vol 14. issue 4. 1997-09-09. PMID:9267464. |
eol-1 cells constitutively expressed fas and anti-fas antibody induced eol-1 cell death, but the fas/fas ligand system was not involved in dbcamp-induced eol-1 cell apoptosis. |
1997-09-09 |
2023-08-12 |
human |